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1 riod onset are poorly tuned and are rendered monocular.
2 ts of short-term exposure (2 or 48 hours) to monocular +10 and -10 diopter (D) lenses, on RPE gene ex
7 o 6 months after surgery) with the following monocular and binocular assessments: high- and low-contr
8 ntile nystagmus syndrome and myopia improved monocular and binocular BCVA and contrast sensitivity.
10 with and without optical correction, and in monocular and binocular conditions; one condition was me
11 alens AO and ReSTOR +3.0 demonstrated better monocular and binocular contrast sensitivity without gla
12 modal versus unimodal responses of the adult monocular and binocular cortices also mirror regional sp
13 Comparison of the adaptation of the medial monocular and binocular cortices to long-term ME or dark
14 ria and supernormal VFV underwent MRI during monocular and binocular fixation of a centered, near tar
15 ight level on normal, age-related changes in monocular and binocular functional contrast sensitivity.
20 TCOME MEASURE(S): Three-months-postoperative monocular and binocular UCVA and DCVA in 4 m, 80 cm, and
21 1 and 3 months included manifest refraction; monocular and binocular uncorrected (UCVA) and distance-
22 stalens AO demonstrated significantly better monocular and binocular uncorrected and distance-correct
23 The ReSTOR+3.0 lens had significantly better monocular and binocular uncorrected and distance-correct
25 patients underwent: monocular defocus curve; monocular and binocular uncorrected visual acuity in pho
26 ed at the Wills Eye Institute using standard monocular and binocular VF testing, as well as an object
32 sed eye response that normally occurs in the monocular as well as binocular zone is delayed, but only
33 riority of TFNT00 to SN60AT in mean photopic monocular BCDVA (95% upper confidence limit of the diffe
36 of the BEFIE test was assessed by comparing monocular BEFIE test results with those of standard conv
38 ry effectiveness outcomes were mean photopic monocular best-corrected distance visual acuity (BCDVA;
43 respectively, even in neurons classified as monocular by conventional ocular dominance (OD) measurem
45 nths after the surgery, 95% of eyes achieved monocular CDVA equal or better than 0.3 logMAR, mean pos
47 cro F, binocular UDVA, 0.01 logMAR +/- 0.05; monocular CDVA, 0.03 logMAR +/- 0.06; binocular UNVA, 0.
49 ands that control both eyes or from separate monocular commands that control the eyes independently.S
51 table for laser in situ keratomileusis, with monocular corrected distance visual acuity of 20/32 or b
54 s followed by cross-modal adaptations in the monocular cortex, in which whiskers become a dominant no
56 .SIGNIFICANCE STATEMENT Motion parallax is a monocular cue to depth that commonly arises during obser
63 R] margin), and superiority in mean photopic monocular DCNVA (difference of 0.42 logMAR; P < 0.001) a
65 s established that amblyopia is not simply a monocular deficit, and therefore the most promising new
66 One month after surgery patients underwent: monocular defocus curve; monocular and binocular uncorre
70 loping primary visual cortex is initiated by monocular deprivation (MD) and consolidated during subse
71 adult mice that visuomotor experience during monocular deprivation (MD) augmented enhancement of visu
75 to recover cortical responsiveness following monocular deprivation (MD) during the critical period, a
79 on dendritic spine turnover before and after monocular deprivation (MD) in adult V1 with chronic in v
80 f-century of research on the consequences of monocular deprivation (MD) in animals has revealed a gre
83 s in OD index following a short-term (2-3 d) monocular deprivation (MD) of the contralateral eye with
84 ging to characterize the effects of juvenile monocular deprivation (MD) on the responses of neurons i
87 , we visualized V1 activity before and after monocular deprivation (MD) using intrinsic signal optica
90 city in primary visual cortex in response to monocular deprivation (MD), the maturation of inhibition
91 ts on unit activity during the first 48 h of monocular deprivation (MD), we show that PNN removal res
97 s receptor alters the microglial response to monocular deprivation and abrogates ocular dominance pla
98 al period peak, postnatal day 28 (P28) after monocular deprivation and dark rearing, and in the adult
99 lar dominance (OD) plasticity resulting from monocular deprivation and stimulus-selective response po
100 lly induced activity-dependent plasticity by monocular deprivation caused rapid changes in single uni
101 sensitivity called the critical period (CP), monocular deprivation causes a shift in the response of
107 responsiveness to open-eye stimulation after monocular deprivation during the critical period is a ho
110 y.SIGNIFICANCE STATEMENT We demonstrate that monocular deprivation during the developmental critical
112 ng in vivo optical imaging, we observed that monocular deprivation in adult EE mice (i) caused a very
113 and to restore cortical plasticity following monocular deprivation in vivo Together, our results show
115 itivity of ocular dominance to regulation by monocular deprivation is the canonical model of plastici
116 tiation of open eye responses resulting from monocular deprivation relies on a homeostatic response t
117 binocular-like excitatory firing rates after monocular deprivation results from a rapid, although tra
119 ar matching process is completely blocked by monocular deprivation spanning the entire critical perio
121 Loss of visual acuity in response to brief monocular deprivation was concomitantly delayed and resc
122 but did occur in wild-type littermates when monocular deprivation was imposed during the critical pe
123 ivity in V1 can be unmasked following 4 d of monocular deprivation when the mice older than 2 months
124 NT Microglia in the visual cortex respond to monocular deprivation with increased lysosome content, b
125 equence of occluding vision through one eye (monocular deprivation) is a rapid loss of excitatory syn
126 l in adult Long Evans rats following chronic monocular deprivation, a manipulation that reduces the s
129 cells phenocopies the changes observed after monocular deprivation, suggesting that glutamate may con
130 male and female mice before and after a 7 d monocular deprivation, which allowed us to examine both
131 emoval of tPA in Lynx1 KO mice can block the monocular deprivation-dependent reduction of dendritic s
132 markably, sustained MET signaling eliminates monocular deprivation-induced ocular dominance plasticit
144 eal distinct duration encoding mechanisms at monocular, depth-selective and depth-invariant stages of
145 R] 0.69, 95% confidence interval 0.52-0.91), monocular deviation (OR 0.64), complex surgery (OR 1.63)
150 diplopia), 1 (4%) optical/refractive error (monocular diplopia), 2 (8%) mixed retinal misregistratio
152 visual acuity (UDVA), -0.01 logMAR +/- 0.06; monocular distance corrected visual acuity (CDVA), 0.02
153 med comprehensive eye examinations including monocular distance visual acuity (VA), cover testing, an
154 condary effectiveness outcomes included mean monocular distance-corrected intermediate visual acuity
157 urations associated with states of exclusive monocular dominance and states of mixed perception durin
158 Neural concomitants of this improvement in monocular dominance are reflected in measurements of bra
159 Withdrawing attention causes the alternating monocular dominance that characterizes rivalry to cease,
162 t when intereye competition is eliminated by monocular enucleation, blocking cholinergic stage II ret
164 s lower than for hyperopic subjects for both Monocular Estimation Method (1.03 +/- 0.51 D vs 2.03 +/-
166 y and facilitation of OD plasticity by prior monocular experience were both present in GluA1(-/-) mic
168 reater than this limit cause the two unfused monocular features to appear flattened into the fixation
175 these novel observations with insights from monocular, frontoparallel motion studies concurrently in
176 S definitions, DME and CSME prevalences from monocular fundus photographs (28.5% and 21.0%, respectiv
177 ross-sectional study of DME grading based on monocular fundus photographs and OCT images obtained fro
179 many eyes diagnosed as having DME or CSME on monocular fundus photographs have no DME based on OCT CS
181 nically significant macular edema (CSME), on monocular fundus photographs used definitions from the M
182 12.9%-24.2%) were diagnosed as having DME on monocular fundus photographs using MESA and NHANES defin
183 ot diagnosed as having either DME or CSME on monocular fundus photographs using MESA and NHANES defin
184 d CSME (48.5%) based on MESA definitions for monocular fundus photographs were greater than the DME p
185 Diagnosing diabetic macular edema (DME) from monocular fundus photography vs optical coherence tomogr
186 ies and telemedicine screening typically use monocular fundus photography, while treatment of DME use
192 The purpose of this study was to compare the monocular Humphrey Visual Field (HVF) with the binocular
196 -Fos, and zinc finger protein, Zif268, after monocular inactivation (MI) to identify ODCs in V1 of Ne
198 associated with the individual stimuli from monocular inputs (self-terms) and responses due to inter
199 two things can happen: sufficiently similar monocular inputs are combined into a fused representatio
201 reduction in PV-cell-evoked responses after monocular lid suture is restricted to the critical perio
205 cted VA than the ReSTOR +3.0 and better mean monocular low-contrast DCVA than the Tecnis Multifocal l
206 nd Mouth Disease (HFMD) and concurrent acute monocular maculopathy, and to describe multimodal imagin
207 with retinal outputs maintained as separate monocular maps en route through the lateral geniculate n
209 blood glucose (BG) concentration, HbA1c, and monocular mfERG were performed on 115 adolescent patient
210 oveal optokinetic contribution suggests that monocular nasotemporal optokinetic asymmetry is partly a
211 to play an important role in maintaining the monocular nasotemporal optokinetic asymmetry seen in pat
212 The initial screening included testing of monocular near and distance visual acuity, stereoacuity,
213 are established by conversion of well-tuned monocular neurons as they gain matched input from the ot
214 ow that, despite responding to only one eye, monocular neurons in all layers, including the input lay
215 an previously appreciated, as even so-called monocular neurons in V1's input layers encode what is sh
217 main input layers of V1 contain most of the monocular neurons while binocular neurons dominate the l
218 rons respond to stimulation of only one eye (monocular neurons), while most neurons respond to stimul
221 ) and near (0.3 m) fixation after >1 hour of monocular occlusion at preoperative and postoperative ex
223 nkeys reared under conditions of alternating monocular occlusion during their first few months of lif
224 the esodeviated eye can supplement temporal monocular optokinetic responses in the fixating eye unde
228 of mouse simple cells is nearly identical to monocular orientation selectivity in both anaesthetized
229 of the retinal nerve fiber layer (RNFL), and monocular pattern reversal visually evoked potentials (p
232 visual system's anatomical progression from monocular, pre-cortical neurons to their binocular, cort
233 l for in-phase and antiphase conditions, and monocular presentation, but increased a little at interm
237 isible to both eyes do indeed form part of a monocular representation of the contralateral visual fie
238 eous yet separate presence of two segregated monocular representations, rather than a joint represent
240 depth and thus require the co-ordination of monocular saccade amplitudes and binocular vergence eye
241 inocular visual field was estimated from the monocular SAP tests, and rates of change in mean sensiti
244 ion occurs after spatiotemporal filtering of monocular signals, which leads to restrictions on dispar
245 the DSpecs was comparable to the integrated monocular standard automated perimetry based on point-by
246 rivation, GABA concentration measured during monocular stimulation correlated with the deprived eye d
247 ratio of excitation to inhibition evoked by monocular stimulation decreased mainly for nonpreferred
248 ms can be recruited in vivo by pairing brief monocular stimulation with pharmacological or chemogenet
250 (1-4) numbers of dots, is facilitated in the monocular, subcortical portions of the visual system.
252 f 127 subjects, 11 (8.7%) could not complete monocular TAC testing in either eye; 39 (30.7%) could no
253 In interocular suppression, a suprathreshold monocular target can be rendered invisible by a salient
255 nstrate the benefit of binocular relative to monocular text presentation for both parafoveal and fove
257 ring response in the fellow eye when using a monocular trial eliminates the need for additional offic
263 OLs displayed in tabular format include mean monocular uncorrected distance, intermediate, and near v
265 omprehensive ophthalmic evaluation including monocular VA testing, cover testing, cycloplegic autoref
274 onviewing (mean, 39.7% +/- 6.2%) eyes during monocular viewing conditions, even in cases with large a
275 We tested smooth pursuit adaptation during monocular viewing in strabismic monkeys with exotropia.
282 nt comprehensive eye examinations, including monocular visual acuity testing, stereoacuity testing, a
288 ic and postsynaptic sites in mice undergoing monocular visual deprivation (MD) were compared to those
290 short period of time: contrary to intuition, monocular visual deprivation actually improves the depri
294 ssesses two distinct visual fields-a focused monocular visual field suitable for detecting features e
297 sion-weighted imaging (DWI) in patients with monocular visual loss of presumed ischemic origin (MVL).
298 near, intermediate and distance compared to monocular visual outcome at the same distances in patien
300 s binocular zone is delayed, but only in the monocular zone in GluA1(-/-) mice and only in a backgrou