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1 on was detected in one individual with acute monocytic leukemia.
2 retroviral insertional mutagenesis in murine monocytic leukemias.
3 between proliferation and differentiation in monocytic leukemias.
4          Cluster F (n = 33) was dominated by monocytic leukemias (97% of cases), also showing increas
5 ding WM164 melanoma, WM35 melanoma, U937 pro-monocytic leukemia, and HT1080 fibrosarcoma cells, XAF1
6 of IL-1B and TNF-a mRNA expressed in a human monocytic leukemia cell line (THP-1) is visualized and c
7 in the presence of OPN increased human acute monocytic leukemia cell line (THP-1) monocyte binding, w
8   LXA4 also induced a rise in [Ca2+]i in the monocytic leukemia cell line (time to reach maximum = 15
9 E. sennetsu, but not HGE agent, in the acute monocytic leukemia cell line THP-1 almost completely inh
10 nduce proinflammatory cytokines in the human monocytic leukemia cell line THP-1 and bone marrow-deriv
11 es are detectable in human monocytes and the monocytic leukemia cell line THP-1.
12 phospholipid surfaces and THP-1 (human acute monocytic leukemia cell line) monocytes.
13 ing in control of differentiation in a human monocytic leukemia cell line, THP-1 cells were transient
14 n of the E. chaffeensis inclusion in a human monocytic leukemia cell line, THP-1 cells, implying that
15 man peripheral blood monocytes and the acute monocytic leukemia cell line, THP-1.
16 oids was further evaluated using THP-1 human monocytic leukemia cell line.
17  the binding of phospholipid vesicles to the monocytic leukemia cell lines THP-1 and J774A.1 with a f
18 uman diploid fibroblasts (WI-38, IMR-90) and monocytic leukemia cells (THP-1), and tissues of mice in
19 ion to paclitaxel-induced apoptosis in human monocytic leukemia cells (U937).
20 ctin ligands in leukocytes and myelocytic or monocytic leukemia cells are carried by transmembrane gl
21 lso induced in primary macrophages and THP-1 monocytic leukemia cells by the phorbol ester 12-O-tetra
22 ochondrial dysfunction and apoptosis in U937 monocytic leukemia cells exposed to the proteasome inhib
23                                We found that monocytic leukemia cells express elevated levels of CEBP
24 1WAF1/CIP1 on the apoptotic response of U937 monocytic leukemia cells to 1-beta-D-arabinofuranosylcyt
25                             Exposure of U937 monocytic leukemia cells to a marginally toxic concentra
26                             Exposure of U937 monocytic leukemia cells to minimally toxic concentratio
27     Simultaneous exposure (30 hours) of U937 monocytic leukemia cells to minimally toxic concentratio
28   Here, we show that acute exposure of THP-1 monocytic leukemia cells to the phorbol ester 12-O-tetra
29 e demonstrated that treatment of THP-1 human monocytic leukemia cells with Z-LLL-CHO, a reversible pr
30 cium ([Ca2+]i) in PBM and THP-1 cells (acute monocytic leukemia cells) as well as on the functional r
31 atic ductal epithelial-like cells, and THP-1 monocytic leukemia cells).
32                        This suggests that in monocytic leukemia cells, an antagonism exists between t
33                             In monocytes and monocytic leukemia cells, integrin-mediated adhesion res
34                                     In THP-1 monocytic leukemia cells, phorbol ester-induced stabiliz
35  compound inhibited sLe(X) formation in U937 monocytic leukemia cells, suggesting that it had inhibit
36 titutively higher H(2)O(2) content than U937 monocytic leukemia cells.
37 RhD systems, including cancer of the tongue, monocytic leukemia, cervical cancer, osteoarthrosis, ast
38 lin, like venetoclax, is ineffective against monocytic leukemia (French-American-British [FAB] subtyp
39 trated in two variants derived from the U937 monocytic leukemia in the absence of exogenous inhibitor
40                                        Acute monocytic leukemia is a distinct subtype of acute myeloi
41                          In patient 3, acute monocytic leukemia-M3 with t(15;17) arose 18 months afte
42                          Human fetal RPE and monocytic leukemia macrophage (THP-1) cell lines were cu
43 e superior in patients with severe anemia or monocytic leukemias or when requiring rapid treatment.
44 l lines, and on neoplastic cell lines: acute monocytic leukemia THP-1 and lung adenocarcinoma A549.
45 oliferator-activated receptor-alpha in human monocytic leukemia THP-1 cells and human aortic endothel
46 detected in E. chaffeensis cultured in human monocytic leukemia THP-1 cells.
47  Ca2+-independent activity of CaM-KII in the monocytic leukemia, U937.
48  the t(8;16)(p11;p13) translocation in acute monocytic leukemia with erythrophagocytosis that fuses M
49                                          The monocytic leukemia zinc finger (MOZ) gene encodes a larg
50                                    The human monocytic leukemia zinc finger (MOZ) protein is an essen
51                                              Monocytic leukemia zinc finger (MOZ)/KAT6A is a MOZ, Ybf
52                 Here, we use deletion of the monocytic leukemia zinc finger gene (Moz/Kat6a/Myst3) to
53 FK activity was linked to recruitment of the monocytic leukemia zinc finger protein (MOZ) histone ace
54             Using stage-specific deletion of monocytic leukemia zinc finger protein (MOZ), a histone
55 V-1 Tat interactive protein Tip60, the human monocytic leukemia zinc finger protein MOZ and the yeast
56 ransferase complexes, where the gene of MOZ (monocytic leukemia zinc finger protein) was first identi
57 in reaction (RT-PCR) that the genes for MOZ, monocytic leukemia zinc finger protein, and TIF2, transc
58  to 72.1 kb within the third intron of MORF (monocytic leukemia zinc finger protein-related factor or
59 man oncogenic histone acetyltransferase MOZ (monocytic leukemia zinc finger) is required for specifyi
60                                   MORF [MOZ (monocytic leukemia zinc-finger protein)-related factor]