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1 on was detected in one individual with acute monocytic leukemia.
2 retroviral insertional mutagenesis in murine monocytic leukemias.
3 between proliferation and differentiation in monocytic leukemias.
5 ding WM164 melanoma, WM35 melanoma, U937 pro-monocytic leukemia, and HT1080 fibrosarcoma cells, XAF1
6 of IL-1B and TNF-a mRNA expressed in a human monocytic leukemia cell line (THP-1) is visualized and c
7 in the presence of OPN increased human acute monocytic leukemia cell line (THP-1) monocyte binding, w
8 LXA4 also induced a rise in [Ca2+]i in the monocytic leukemia cell line (time to reach maximum = 15
9 E. sennetsu, but not HGE agent, in the acute monocytic leukemia cell line THP-1 almost completely inh
10 nduce proinflammatory cytokines in the human monocytic leukemia cell line THP-1 and bone marrow-deriv
13 ing in control of differentiation in a human monocytic leukemia cell line, THP-1 cells were transient
14 n of the E. chaffeensis inclusion in a human monocytic leukemia cell line, THP-1 cells, implying that
17 the binding of phospholipid vesicles to the monocytic leukemia cell lines THP-1 and J774A.1 with a f
18 uman diploid fibroblasts (WI-38, IMR-90) and monocytic leukemia cells (THP-1), and tissues of mice in
20 ctin ligands in leukocytes and myelocytic or monocytic leukemia cells are carried by transmembrane gl
21 lso induced in primary macrophages and THP-1 monocytic leukemia cells by the phorbol ester 12-O-tetra
22 ochondrial dysfunction and apoptosis in U937 monocytic leukemia cells exposed to the proteasome inhib
24 1WAF1/CIP1 on the apoptotic response of U937 monocytic leukemia cells to 1-beta-D-arabinofuranosylcyt
27 Simultaneous exposure (30 hours) of U937 monocytic leukemia cells to minimally toxic concentratio
28 Here, we show that acute exposure of THP-1 monocytic leukemia cells to the phorbol ester 12-O-tetra
29 e demonstrated that treatment of THP-1 human monocytic leukemia cells with Z-LLL-CHO, a reversible pr
30 cium ([Ca2+]i) in PBM and THP-1 cells (acute monocytic leukemia cells) as well as on the functional r
35 compound inhibited sLe(X) formation in U937 monocytic leukemia cells, suggesting that it had inhibit
37 RhD systems, including cancer of the tongue, monocytic leukemia, cervical cancer, osteoarthrosis, ast
38 lin, like venetoclax, is ineffective against monocytic leukemia (French-American-British [FAB] subtyp
39 trated in two variants derived from the U937 monocytic leukemia in the absence of exogenous inhibitor
43 e superior in patients with severe anemia or monocytic leukemias or when requiring rapid treatment.
44 l lines, and on neoplastic cell lines: acute monocytic leukemia THP-1 and lung adenocarcinoma A549.
45 oliferator-activated receptor-alpha in human monocytic leukemia THP-1 cells and human aortic endothel
48 the t(8;16)(p11;p13) translocation in acute monocytic leukemia with erythrophagocytosis that fuses M
53 FK activity was linked to recruitment of the monocytic leukemia zinc finger protein (MOZ) histone ace
55 V-1 Tat interactive protein Tip60, the human monocytic leukemia zinc finger protein MOZ and the yeast
56 ransferase complexes, where the gene of MOZ (monocytic leukemia zinc finger protein) was first identi
57 in reaction (RT-PCR) that the genes for MOZ, monocytic leukemia zinc finger protein, and TIF2, transc
58 to 72.1 kb within the third intron of MORF (monocytic leukemia zinc finger protein-related factor or
59 man oncogenic histone acetyltransferase MOZ (monocytic leukemia zinc finger) is required for specifyi