戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ence of excess ATP, suggesting that they are monomeric G proteins.
2 n-enriched member of the Ras family of small monomeric G proteins.
3  is a direct link between heterotrimeric and monomeric G proteins.
4 closely related members of the Rho family of monomeric G-proteins.
5 with the GDP-bound form of the Rho family of monomeric G proteins, and thus may serve as a nodal poin
6 ne is stimulated by protein kinase C and the monomeric G proteins Arf, RhoA, Cdc42, and Rac1, resulti
7 ide exchange factor for the Rap subfamily of monomeric G proteins, as an important regulator of insul
8 important for the steady-state enrichment of monomeric G proteins at the plasma membrane, but it is n
9 infestans piypt1 gene, a gene that encodes a monomeric G-protein believed to be involved in regulatio
10 lar dynamics simulations to show that Ras, a monomeric G protein, can generate mechanical force upon
11                                          The monomeric G protein Cdc42 is believed to play an importa
12 B (TcdB), glucosyltransferases that modulate monomeric G-protein function and alter cytoskeletal func
13 ve mutations for investigating Ras and other monomeric G proteins inspired us to create a functionall
14                                   This small monomeric G protein is both necessary and sufficient for
15 xis pathway consisting of heterotrimeric and monomeric G proteins is sufficient for chemotaxis.
16 is study, we test the hypothesis that Rad, a monomeric G protein, is a novel regulatory mechanism in
17 at the calcium-channel inhibitor Rad(8,9), a monomeric G protein, is enriched in the Ca(V)1.2 microen
18                                       Rad, a monomeric G protein, is upregulated in uterine arteries
19 es expression of the mRNA encoding the small monomeric G protein Ki-RasA.
20            To determine whether any of these monomeric G-proteins mediate the response to BK, we have
21 r-like proteins (Arls) are a family of small monomeric G proteins of unknown function.
22 kinases were reported to interact with small monomeric G-proteins of the RHO of plant (ROP; also call
23 strate that activated Rap1A, but not related monomeric G proteins, promotes ribosomal protein S6 phos
24 alization of a marker for the Golgi Ras-like monomeric G-protein RAB2:GFP expressed in transgenic tob
25 ly stimulates GTP hydrolytic activity of the monomeric G protein Rap1 and thus is believed to functio
26                                          The monomeric G protein Rap1 has been implicated in cancer t
27 -like domain with structural homology to the monomeric G protein Ras and a helical domain comprised o
28          Among the substrates of Icmt is the monomeric G protein Ras, a critical component of many si
29 alpha2betagamma, and an activation step of a monomeric G-protein Ras.
30                                          The monomeric G protein, Ras, transduces multiple signaling
31 perfamily of Ras GTPase signalling proteins (monomeric G proteins) require post-translational carboxy
32  guanine nucleotide exchange factors for the monomeric G protein Rho (RhoGEFs) are well characterized
33 ionally uncoupled from signaling through the monomeric G protein Rho, a protein known to propagate se
34 eceptor kinase pathways or inhibition of the monomeric G proteins Rho, Rac and CDC42.
35  it has been shown that barley RACB, a small monomeric G-protein (ROP, Rho of plants), is required fo
36 ioning to directly link Galpha activation to monomeric G-protein signaling.
37  like the GAPs that regulate the activity of monomeric G proteins such as Ras.
38                            RalA and RalB are monomeric G proteins that are 83% identical in amino aci
39 els, a paradigm of ion channel regulation by monomeric G-proteins with significant physiological rami