ライフサイエンスコーパス: mononuclear

1 - c:3",2"- h:2''',3'''- j]phenazine], with a mononuclear analogue with a modified intercalating ligan

2 stant k(s) (~5 orders of magnitude) over the mononuclear analogue.

3 l sorting analyses suggest that most BAs are mononuclear and diploid.

4 bligate intracellular bacterium that infects mononuclear and endothelial cells to cause the emerging

5 and induce cytotoxicity by peripheral blood mononuclear and engineered NK cells.

6 ator cell line, and primary peripheral blood mononuclear and T cells, along with bioactivity, flow cy

7 S. japonicus hyphae also remain mononuclear and undergo complete cell divisions, which a

8 ch is counter to expectations based on known mononuclear BDFE(OH)s which increase with the oxidation

9 ysis was also proposed involving monodentate-mononuclear/bidentate-binuclear As-Fe complex formation

10 transition theory to predict how peripheral mononuclear blood cells in mice transition from a health

11 mprovement, after human umbilical cord blood mononuclear cell (hUCBC) infusion.

12 We performed peripheral blood mononuclear cell (PBMC) analysis on a subgroup of 26 IgG

13 extracts were determined in peripheral blood mononuclear cell (PBMC) cultures.

14 -ABL inhibitor dasatinib in peripheral blood mononuclear cell (PBMC) lysates from individual donors.

15 ersistence, we analyzed 181 peripheral blood mononuclear cell (PBMC) samples from 64 PLWH starting AR

16 ed consistent modulation in peripheral blood mononuclear cell (PBMC) samples from HIV-positive (HIV(+

17 Thus, peripheral blood mononuclear cell (PBMC)-derived induced pluripotent stem

18 pha plasma levels and lower peripheral blood mononuclear cell activation versus both groups.

19 Prior work has shown peripheral blood mononuclear cell conditioned media causes sublethal dege

20 The effect of peripheral blood mononuclear cell conditioned medium from vaccinated catt

21 ogy) (n = 99), and analyzed peripheral blood mononuclear cell gene expression (n = 95) by quantitativ

22 collagen-1a, alpha-smooth muscle actin, and mononuclear cell infiltration (CD11b(+) Ly-6c(hi) monocy

23 markers and frequencies of peripheral blood mononuclear cell phenotypes (range 0.19 to 3.54 fold), a

24 weaning gene expression and peripheral blood mononuclear cell profiling may be useful as predictors o

25 es demonstrated that infant peripheral blood mononuclear cell responses were dominated by monocyte-as

26 e ever smokers, had shorter peripheral blood mononuclear cell telomeres, and were more likely to carr

27 geneous tissue comprised of muscle fiber and mononuclear cell types that, in addition to movement, in

28 We identified 11 human skeletal muscle mononuclear cell types, including two fibro-adipogenic p

29 nanomolar potency in cells (peripheral blood mononuclear cell, IC(50,free) = 6.2 nM) and good in vivo

30 K(+) channel expression in peripheral blood mononuclear cell, which strongly correlated to that in v

31 and revealed significant changes in specific mononuclear cell-type proportions related to age, sex, a

32 y increased the proliferation of bone marrow mononuclear cells (BMNC) and, at the same time, inhibite

33 Bone marrow mononuclear cells (BMNC) are a source of naive macrophag

34 Bone marrow mononuclear cells (BMNCs) are a rich source of macrophag

35 zed RRGS animals with human peripheral blood mononuclear cells (hPBMCs) to set up a human acute GVHD

36 tudied with activated human peripheral blood mononuclear cells (hPBMCs).

37 in vitro from treated human peripheral blood mononuclear cells (hPBMCs).

38 fter ex vivo stimulation of peripheral blood mononuclear cells (MNCs) and bone marrow MNCs was higher

39 mucosa samples (n = 87) and peripheral blood mononuclear cells (n = 85) from patients with active or

40 We investigated peripheral blood mononuclear cells (n=24), plasma (n=27), endomyocardial

41 ral DNA from peripheral blood and lymph node mononuclear cells (PBMC and LNMC) during ART suppression

42 In both peripheral blood mononuclear cells (PBMC) and macrophages, omega-3 increa

43 quantitation of RRV DNA in peripheral blood mononuclear cells (PBMC) by real-time PCR revealed level

44 ome-wide gene expression in peripheral blood mononuclear cells (PBMC) derived from these IPF study su

45 lity on 75 split samples of peripheral blood mononuclear cells (PBMC) from 5 antiretroviral therapy (

46 ome-wide gene expression in peripheral blood mononuclear cells (PBMC) from 77 Holocaust survivor offs

47 Peripheral blood mononuclear cells (PBMC) from perinatally infected child

48 rmed microarray analyses of peripheral blood mononuclear cells (PBMC) RNA from subjects enrolled in t

49 ed on pre- and on-treatment peripheral blood mononuclear cells (PBMC) samples.

50 lel samples from plasma and peripheral blood mononuclear cells (PBMC) were also genotyped.

51 nd TFV-DP concentrations in peripheral blood mononuclear cells (PBMC) were found to be at least 10-fo

52 Cryopreserved peripheral blood mononuclear cells (PBMC) were obtained from infants from

53 -pregnancy-induced genes in peripheral blood mononuclear cells (PBMC); and (2) characterize the tempo

54 fic CAR-T cells using human peripheral blood mononuclear cells (PBMCs) and a CAR construct that enabl

55 tein was shown to adhere to peripheral blood mononuclear cells (PBMCs) and DF-1 cells and cause apopt

56 st time, ME/CFS miRNomes in peripheral blood mononuclear cells (PBMCs) and extracellular vesicles (EV

57 in peripheral blood mononuclear cells (PBMCs) and in cell pellets from CSF.

58 of immune response genes in peripheral blood mononuclear cells (PBMCs) and lesion biopsy specimens ob

59 fied in allergen-stimulated peripheral blood mononuclear cells (PBMCs) and skin punch biopsies of IBH

60 PUUC were additive in human peripheral blood mononuclear cells (PBMCs) and synergistic in human FLT3-

61 sing cells, including human peripheral blood mononuclear cells (PBMCs) and the SupT1 cell line.

62 ses detected in circulating peripheral blood mononuclear cells (PBMCs) are referred to as "defective"

63 cRNA reduction are found in peripheral blood mononuclear cells (PBMCs) derived from patients with aut

64 as developed by challenging peripheral blood mononuclear cells (PBMCs) derived from patients with con

65 ed HRV-specific peptides on peripheral blood mononuclear cells (PBMCs) from 14 HLA I typed subjects.

66 -1 was further evaluated in peripheral blood mononuclear cells (PBMCs) from FRDA patients and from no

67 regulation/inflammation, in peripheral blood mononuclear cells (PBMCs) from healthy donors and patien

68 cation of kBET to data from peripheral blood mononuclear cells (PBMCs) from healthy donors to disting

69 CyTOF was performed on peripheral blood mononuclear cells (PBMCs) from PBC patients (n = 33) and

70 pare cytokine profiles in stimulated primary mononuclear cells (PBMCs) from RVVC and healthy individu

71 cing (scRNA-seq) to profile peripheral blood mononuclear cells (PBMCs) from seven patients hospitaliz

72 ies were sequenced from 2 million peripheral mononuclear cells (PBMCs) in 14 HAM/TSP patients, 34 MS

73 n, and serologic testing of peripheral blood mononuclear cells (PBMCs) in a subset of study participa

74 an exploratory analysis of peripheral blood mononuclear cells (PBMCs) isolated from healthy controls

75 evels of DNA methylation in peripheral blood mononuclear cells (PBMCs) longitudinally collected from

76 In the peripheral blood mononuclear cells (PBMCs) of COVID-19 patients, we obser

77 tokine staining (ICS) using peripheral blood mononuclear cells (PBMCs) of individuals naturally expos

78 incubated with third-party peripheral blood mononuclear cells (PBMCs) pretreated with anti-HLA antib

79 hment of B cells from whole peripheral blood mononuclear cells (PBMCs) results in the most reliable B

80 ulates the bioenergetics of peripheral blood mononuclear cells (PBMCs) under basal conditions or in s

81 nockout (GTKO), and TKO pig peripheral blood mononuclear cells (PBMCs) using sera from humans, severa

82 epidemiologic associations, peripheral blood mononuclear cells (PBMCs) were collected from 21 childre

83 irculating, activated human peripheral blood mononuclear cells (PBMCs) were suppressed by MSC bioreac

84 d IL-18 upon stimulation of peripheral blood mononuclear cells (PBMCs) with antigen.

85 to antigenic stimulation of peripheral blood mononuclear cells (PBMCs) with novel Kgp synthetic pepti

86 Coculture with human peripheral blood mononuclear cells (PBMCs) with poly I:C-activated hPDL c

87 inflammatory activation in peripheral blood mononuclear cells (PBMCs), and the potential antiinflamm

88 diabetes was separated into peripheral blood mononuclear cells (PBMCs), plasma, and serum.

89 alidated preclinical model, peripheral blood mononuclear cells (PBMCs), that is highly predictive of

90 tochondrial respiration) of peripheral blood mononuclear cells (PBMCs), which was normalized in metfo

91 muM) was observed in human peripheral blood mononuclear cells (PBMCs).

92 pithelial cells (NECs), and peripheral blood mononuclear cells (PBMCs).

93 (IgG) and 16.7/106 (IgA) in peripheral blood mononuclear cells (PBMCs).

94 Cs compared to normal human peripheral blood mononuclear cells (PBMCs).

95 zed by phosphoproteomics in peripheral blood mononuclear cells (PBMCs).

96 sed on the surface of human peripheral blood mononuclear cells (PBMCs).

97 neuroblastoma (SH-SY5Y) and peripheral blood mononuclear cells (PBMCs).

98 drained the muscle, and the peripheral blood mononuclear cells (PBMCs)isolated from the circulating b

99 We profiled total peripheral blood mononuclear cells (PBMCs, 106,545 cells) and dendritic c

100 found to be induced in the Peripheral Blood mononuclear cells (PMBCs) of asthmatic pre-school childr

101 ng cells [SFCs] per million peripheral blood mononuclear cells [IQR 841-2428], n=24; 56-69 years: 797

102 onses (median 521 SFU/10(6) peripheral blood mononuclear cells [PBMCs] in the tetravalent group and m

103 teins in both BM and blood (peripheral blood mononuclear cells [PBMCs]).

104 and PPARD were decreased in peripheral blood mononuclear cells after replacing SFAs with PUFAs.

105 pes of samples: cell lines, peripheral blood mononuclear cells and brain tissue, generating 36 librar

106 ro cytotoxicity assays with peripheral blood mononuclear cells and CEA-expressing MKN-45 gastric or F

107 profile from primary human peripheral blood mononuclear cells and demonstrated efficacy in generatin

108 performed mass cytometry of peripheral blood mononuclear cells and discovered an immune signature of

109 d by measuring HIV-1 DNA in peripheral blood mononuclear cells and inducible cell-free HIV-1 RNA in C

110 Methods: Peripheral blood mononuclear cells and nasal scrapings were obtained from

111 her-newborn dyads, maternal peripheral blood mononuclear cells and neonatal umbilical cord blood mono

112 vesicles derived from both peripheral blood mononuclear cells and plasma from HIV+ patient blood sam

113 ranscriptional profiling of peripheral blood mononuclear cells and promoter-based bioinformatic analy

114 ustion immunophenotyping on peripheral blood mononuclear cells and quantified interferon-stimulated g

115 Using flow cytometry on peripheral blood mononuclear cells and serum immunoassays, we analyzed th

116 NA levels in ventricles and peripheral blood mononuclear cells and their changes in patients with acu

117 and IL8 gene expression in peripheral blood mononuclear cells and tumors of patients treated with at

118 h levels of IL-8 in plasma, peripheral blood mononuclear cells and tumors were associated with decrea

119 methylation in normal human peripheral blood mononuclear cells and utilize a broad proteomic approach

120 Infiltrating host mononuclear cells are a likely source of TNF as these ch

121 evels of SQSTM1 and INSR in peripheral blood mononuclear cells are related to the severity of sepsis

122 Third, peripheral blood mononuclear cells are used to explore heterogeneous immu

123 in IFN-gamma production by peripheral blood mononuclear cells at day 42 in the adjuvanted vaccine gr

124 We collected peripheral blood mononuclear cells at influenza diagnosis and four weeks

125 Peripheral blood mononuclear cells before and after cardiopulmonary bypas

126 Analysis of peripheral blood mononuclear cells before and after desensitization in on

127 the patients received up to 1.3 x 1010 donor mononuclear cells before transplantation.

128 s markedly induced in AH livers, not only in mononuclear cells but also notably in hepatocytes.

129 mIDH1 clearance in bone marrow mononuclear cells by BEAMing (beads, emulsion, amplifica

130 EBV) DNA were quantified in peripheral blood mononuclear cells by droplet digital PCR (ddPCR).

131 s measured noninvasively in peripheral blood mononuclear cells can serve as novel biomarkers of sepsi

132 osphorylation events within peripheral blood mononuclear cells collected prior to vaccination identif

133 the single-cell level from peripheral blood mononuclear cells collected ~2 weeks or 6-7 months postp

134 d therapy group had pre-ART peripheral blood mononuclear cells evaluated for drug resistance to non-n

135 omic DNA were isolated from peripheral blood mononuclear cells for transcriptional and DNA methylatio

136 lls were sorted from frozen peripheral blood mononuclear cells from 11 RV-infected hypogammaglobuline

137 and without asthma.Methods: Peripheral blood mononuclear cells from 17 children with asthma and 19 co

138 Here, we characterize peripheral blood mononuclear cells from 172 healthy adults 22-93 years of

139 Peripheral blood mononuclear cells from 23 participants collected longitu

140 ncing, we profiled ~276,000 peripheral blood mononuclear cells from 33 children with SLE with differe

141 is method to lymph node and peripheral blood mononuclear cells from 5 ART-treated donors to determine

142 Profiling of ~82,000 single peripheral blood mononuclear cells from adults with SLE confirmed the exp

143 Clinical leukapheresis can concentrate mononuclear cells from almost the entire blood volume, b

144 Stimulating peripheral blood mononuclear cells from COVID-19 convalescent patients wi

145 By performing scRNA-seq on peripheral blood mononuclear cells from four untreated individuals before

146 unomodulatory effects using peripheral blood mononuclear cells from healthy donors exposed to IFN-alp

147 man muscle fibers and single cell RNA-seq of mononuclear cells from human vastus lateralis, mouse qua

148 T cells and lamina propria mononuclear cells from mice were analyzed by flow cytome

149 We evaluated peripheral blood mononuclear cells from MOG-AAD patients by flow cytometr

150 g MCL/MINCLE stimulation in peripheral blood mononuclear cells from MS patients.

151 Peripheral blood mononuclear cells from naive and matched acutely (n = 4)

152 pG methylation of the INS gene in cord blood mononuclear cells from offspring with a susceptible INS

153 A sequencing (scRNA-seq) of peripheral blood mononuclear cells from opioid-dependent individuals and

154 Peripheral blood mononuclear cells from patients with active CD differed

155 se of active UC in that the peripheral blood mononuclear cells from patients with CD had increased IL

156 re significantly reduced in peripheral blood mononuclear cells from patients with COPD compared with

157 iogenesis) noninvasively in peripheral blood mononuclear cells from patients with sepsis and correlat

158 of primary peripheral blood and bone marrow mononuclear cells from pediatric B-ALL patients, culture

159 f IL-35 subsets Ebi3 and p35 was elevated in mononuclear cells from peripheral blood, spleen, bone ma

160 Peripheral blood mononuclear cells from premalignant cases (1 year predia

161 in primary peripheral blood and bone marrow mononuclear cells from ten pediatric B-ALL patients.

162 In primary peripheral blood mononuclear cells from the patients, downstream type I i

163 itial enrichment by routine leukapheresis of mononuclear cells from very large blood volumes, followe

164 cells, the Jurkat TAg T-cell line, and donor mononuclear cells in a SERINC5-dependent manner.

165 ics of the transcriptome of peripheral blood mononuclear cells in a two-dimensional state-space repre

166 Most of the mononuclear cells in Coats' disease samples were CD163(+

167 ecreased PD-1 expression in peripheral blood mononuclear cells in monoculture by 41% and 55%, and co-

168 naive precursors from human peripheral blood mononuclear cells in the presence of various adjuvants,

169 mic profiling of plasma and peripheral blood mononuclear cells including metabolome, lipidome, immuno

170 n T cell stimulation experiments using blood mononuclear cells isolated from both healthy donors and

171 f TNF-alpha and IL-6 in the peripheral blood mononuclear cells isolated from cutaneous lupus erythema

172 ntibody in a yeast display screen built from mononuclear cells isolated from the immunized camel and

173 Likewise, their peripheral blood mononuclear cells mounted a negligible cellular immune r

174 HIV-1 DNA was quantified in peripheral blood mononuclear cells obtained from acutely (Fiebig I-III) a

175 increased IDO1 activity in peripheral blood mononuclear cells of a significant proportion of MS pati

176 6 peptide-specific CD8+ T cells in tonsillar mononuclear cells of HLA-matched individuals.

177 nd content were measured in peripheral blood mononuclear cells on days 1-2, 3-5, and 8-14 after sepsi

178 We collected peripheral blood mononuclear cells pre- and postimmunization from 60 pati

179 prior to DENV infection of peripheral blood mononuclear cells prevents activation of human vascular

180 re transfused with 1 x 10(7) of human spleen mononuclear cells reconstituted human CD45(+) cells that

181 Immunophenotyping of peripheral blood mononuclear cells revealed an increase in activated CD8+

182 2 diabetes patient-derived peripheral blood mononuclear cells show reduced expression of DBC1 and EL

183 to those produced by human peripheral blood mononuclear cells stimulated with PG(Bb) In addition, sy

184 ranscriptional profiling of peripheral blood mononuclear cells to determine immunological signatures

185 pase-8 sensitized patients' peripheral blood mononuclear cells to RIPK1 activation, apoptosis and nec

186 d proliferative response of peripheral blood mononuclear cells to WHV peptides.

187 l and B-cell responses from peripheral blood mononuclear cells using flow cytometry and enzyme-linked

188 from polyclonally activated peripheral blood mononuclear cells using SAB assays and compared HLA anti

189 Cathepsin Z mRNA in human peripheral blood mononuclear cells was significantly differentially-expre

190 Transcriptomic profiles of peripheral blood mononuclear cells were analyzed in n=6 patients with HF

191 lear cells and neonatal umbilical cord blood mononuclear cells were collected and cryopreserved short

192 Plasma samples and peripheral blood mononuclear cells were collected at 3 timepoints (pretra

193 Peripheral blood mononuclear cells were collected from 30 transplant reci

194 Peripheral blood mononuclear cells were collected from donor-recipient pa

195 Peripheral blood mononuclear cells were cultured with atabecestat and its

196 Peripheral blood mononuclear cells were infected with recombinant vesicul

197 heral blood was drawn from all children, and mononuclear cells were polyclonally activated.

198 Peripheral blood mononuclear cells were quantified for T and B cell subse

199 Peripheral blood mononuclear cells were sampled from acute myeloid leukem

200 levels of all four genes in peripheral blood mononuclear cells were significantly reduced in septic p

201 Peripheral blood mononuclear cells were stained and analyzed for frequenc

202 Peripheral blood mononuclear cells were stimulated with Staphylococcus au

203 duced cytokine responses in peripheral blood mononuclear cells were used to assess trained immunity.R

204 e studies of whole blood or peripheral blood mononuclear cells with concordant direction of effect (h

205 coculturing HLA-II-matched peripheral blood mononuclear cells with HL cell lines and showed IS forma

206 t forming cells per 1 x 106 peripheral blood mononuclear cells).

207 is induces a specific transcriptome in human mononuclear cells, a stronger cytokine response compared

208 and patient-derived primary peripheral blood mononuclear cells, A-196 increased FXN expression by up

209 ed inflammatory response in peripheral blood mononuclear cells, as well as a compensatory mechanism t

210 Tomato mice, we found CX(3)CR1 expression in mononuclear cells, but not in multinucleated TRAP(+) OC.

211 t-forming cells per million peripheral blood mononuclear cells, IQR 493-1802; n=43).

212 in reduced DAP1 protein in peripheral blood mononuclear cells, monocytes, and lymphoblastoid cell li

213 Human peripheral blood mononuclear cells, or immature dendritic cells derived f

214 Peripheral mononuclear cells, serum creatinine levels, and renal bi

215 uring co-culture with human peripheral blood mononuclear cells, showing that TZM-gfp can support outg

216 RNA-sequencing analysis of peripheral blood mononuclear cells, single-cell bioinformatics analysis a

217 In human peripheral blood mononuclear cells, these molecules suppressed Candida al

218 e (CpG) sites measured from peripheral blood mononuclear cells, we identified 15 sites that were sign

219 ic cells derived from human peripheral blood mononuclear cells, were cultured in medium containing TI

220 ysing more than 1.5 billion peripheral blood mononuclear cells, which raises the possibility that a s

221 human livers, LCN2 expressed exclusively in mononuclear cells, while its expression was markedly ind

222 the Seahorse XFe96 in fresh peripheral blood mononuclear cells.

223 ot seen in the fibers but in adjacent F4/80+ mononuclear cells.

224 l immune profiling of human peripheral blood mononuclear cells.

225 ptomic data from stimulated peripheral blood mononuclear cells.

226 FN-alpha in healthy control peripheral blood mononuclear cells.

227 antified TET2 expression in peripheral blood mononuclear cells.

228 reactivation in plasma and peripheral blood mononuclear cells.

229 1 T cells and donor-derived peripheral blood mononuclear cells.

230 secreting cells per million peripheral blood mononuclear cells.

231 ies in three cell lines and peripheral blood mononuclear cells.

232 hepatic stellate cells and peripheral blood mononuclear cells.

233 ng with HIV and produced in peripheral blood mononuclear cells.

234 surements on whole blood or peripheral blood mononuclear cells.

235 Irs4 (L683F), but show that neither affects mononuclear CM level individually.

236 ffer substantially (6.6% vs. 14.3%) in adult mononuclear CM level.

237 A substantially higher percentage of mononuclear CMs in BALB/cByJ are tetraploid (66.7% vs. 3

238 In this work, we obtained an unprecedented mononuclear Co(IV)-dinitrate complex (2) upon one-electr

239 TSCs grew as mononuclear colonies, whereas upon induction of syncytio

240 ectronic pyroelectricity in the crystal of a mononuclear complex, [Co(phendiox)(rac-cth)](ClO(4)).0.5

241 BDFE(OH)s were measured and analyzed for the mononuclear complexes [LCu(OH(2))](1+) and [LCu(OH(2))](

242 These results indicate the presence of a mononuclear copper center in both PmoB and PmoC.

243 /C-Cl bond cleavage in biology mediated by a mononuclear copper center.

244 copper binding protein CopC share a similar mononuclear copper site.

245 displays enhanced reactivity compared to its mononuclear counterpart due to bimetallic cooperativity

246 crystal X-ray crystallography shows that the mononuclear [CuOH](1+) core is stabilized via intramolec

247 itially bind to different Mg centers and the mononuclear dimethyl magnesium.

248 in BALB/cJ), such that the overall level of mononuclear diploid CMs between the two strains is simil

249 L-1beta) is upregulated in lesional skin and mononuclear donor cells exposed to recombinant mutant pr

250 Unique examples of dianionic mononuclear eta(4) -P(4) complexes are presented that ca

251 mplex 2 undergoes NO dissociation to yield a mononuclear Fe(III) complex, [Fe((TMS)PS2)((TMS)PS2CH(3)

252 avage of the cysteine C3-S bond, producing a mononuclear [Fe(I)(CO)(2)(CN)S] species that serves as t

253 of the Rieske-type [2Fe-2S] cluster and the mononuclear [Fe] center of CntA were identified.

254 maturation, Rieske [2Fe-2S] followed by the mononuclear [Fe] center.

255 bited inflammation, and dramatically reduced mononuclear immune cells.

256 y cytokines and MMPs, together with apparent mononuclear infiltrate and increased collagenolysis conf

257 rane thickness (by 10% to 25%) and increased mononuclear infiltrate inside the membrane.

258 keletal muscle phenotypes including elevated mononuclear invasion, central nucleation, fibrosis and d

259 The active site consists of a mononuclear iron coordinated by two Tyr side-chain pheno

260 tional domains (the Rieske and the catalytic mononuclear iron domains) are located >40 angstrom apart

261 orted the first example of N(2) binding at a mononuclear iron site supported by only S and C donors.

262 ized by neuronal degeneration, necrosis, and mononuclear leukocyte infiltrates, was observed in the d

263 ternal-fetal interface, density of placental mononuclear leukocytes decreased with stress, yet matern

264 yeast two-hybrid screen (human leukocyte and mononuclear library) and confirmed by coimmunoprecipitat

265 lar, Fe(3+)-hydroxamate-based polyhedra with mononuclear metal nodes.

266 mechanism of NO reduction to form N(2)O at a mononuclear metal site that provides insight into the re

267 ese products are challenging to access under mononuclear metal-catalyzed hydrostannylation conditions

268 t symmetry-breaking phase transitions in the mononuclear Mn(3+) compound [Mn(3,5-diBr-sal(2) (323))]B

269 Mononuclear molybdoenzymes of the dimethyl sulfoxide red

270 yeloid-derived suppressor cells (PMN-MDSCs), mononuclear myeloid-derived suppressor cells (M-MDSCs),

271 Our survey includes complexes in which mononuclear N(2) complexes are used as building blocks t

272 Herein, we report a mononuclear non-heme iron(II)-cyclam complex, 1-trans, t

273 A mononuclear nonheme cobalt(III) iodosylbenzene complex,

274 s a rare example of dioxygen activation at a mononuclear nonheme iron(II) complex that produces both

275 The mononuclear phagocyte system (MPS) is a family of cells

276 ifferentiation, and survival of cells of the mononuclear phagocyte system (MPS; progenitors, monocyte

277 Monocytes are key effectors of the mononuclear phagocyte system, playing critical roles in

278 human liver contains specialized subsets of mononuclear phagocytes (MNPs) and T cells, but whether t

279 ractions between effector CD4(+) T cells and mononuclear phagocytes (MPs).

280 2B) prevents the degeneration of spinal cord mononuclear phagocytes and modulates motility of murine

281 cell transcriptional profiling of intestinal mononuclear phagocytes and multidimensional flow cytomet

282 We found that recruited Dock8(-/-)CX3CR1(+) mononuclear phagocytes are exquisitely sensitive to migr

283 Human mononuclear phagocytes comprise phenotypically and funct

284 In single-cell analysis of mononuclear phagocytes from CLM tissues, S-TAM and L-TAM

285 milar to wild type, although accumulation of mononuclear phagocytes increased in postischemic Ackr2(-

286 he development, homeostasis, and function of mononuclear phagocytes is the colony stimulating factor-

287 characterization of human and murine myeloid mononuclear phagocytes revealed the presence of a conser

288 urfaces of humans and mice host a network of mononuclear phagocytes that differ in their ontogeny, su

289 Exposure of mononuclear phagocytes to beta-glucan, a naturally occur

290 Recruited lung mononuclear phagocytes were abundant in CF and were sepa

291 Non-microglial mononuclear phagocytes, such as CNS-associated macrophag

292 damage, including the activation of resident mononuclear phagocytes, tissue infiltration by non-resid

293 re they activate the inflammasome in colonic mononuclear phagocytes, triggering inflammation.

294 health and disease with a specific focus on mononuclear phagocytes.

295 e editing and the effect of this mutation on mononuclear phagocytic cells was examined.

296 Recent exciting developments in the area of mononuclear photoactive complexes with Earth-abundant me

297 The reaction rates for the mononuclear rearrangement of the Z-phenylhydrazones of 3

298 report the activation of acetylene gas at a mononuclear tris(phosphino)silyl-iron center, (SiP(3))Fe

299 post-reaction solids showed the presence of mononuclear U(VI) species rather than a solid U(VI) phas

300 Weak adsorption of mononuclear uranyl(VI) complexes is found on stoichiomet