ライフサイエンスコーパス: mononuclear cell

1 t forming cells per 1 x 106 peripheral blood mononuclear cells).

2 ancerous cells (MCF-10A and peripheral blood mononuclear cells).

3 ot seen in the fibers but in adjacent F4/80+ mononuclear cells.

4 l immune profiling of human peripheral blood mononuclear cells.

5 ptomic data from stimulated peripheral blood mononuclear cells.

6 FN-alpha in healthy control peripheral blood mononuclear cells.

7 antified TET2 expression in peripheral blood mononuclear cells.

8 reactivation in plasma and peripheral blood mononuclear cells.

9 uated cytokine responses by peripheral blood mononuclear cells.

10 capacity of primary patient AML bone marrow mononuclear cells.

11 but variably in plasma and peripheral blood mononuclear cells.

12 ies in three cell lines and peripheral blood mononuclear cells.

13 1 T cells and donor-derived peripheral blood mononuclear cells.

14 secreting cells per million peripheral blood mononuclear cells.

15 hepatic stellate cells and peripheral blood mononuclear cells.

16 ng with HIV and produced in peripheral blood mononuclear cells.

17 surements on whole blood or peripheral blood mononuclear cells.

18 the Seahorse XFe96 in fresh peripheral blood mononuclear cells.

19 is induces a specific transcriptome in human mononuclear cells, a stronger cytokine response compared

20 and patient-derived primary peripheral blood mononuclear cells, A-196 increased FXN expression by up

21 pha plasma levels and lower peripheral blood mononuclear cell activation versus both groups.

22 and PPARD were decreased in peripheral blood mononuclear cells after replacing SFAs with PUFAs.

23 cytotoxicity profile toward peripheral blood mononuclear cells and bone marrow-derived stromal cells.

24 pes of samples: cell lines, peripheral blood mononuclear cells and brain tissue, generating 36 librar

25 ro cytotoxicity assays with peripheral blood mononuclear cells and CEA-expressing MKN-45 gastric or F

26 profile from primary human peripheral blood mononuclear cells and demonstrated efficacy in generatin

27 performed mass cytometry of peripheral blood mononuclear cells and discovered an immune signature of

28 d by measuring HIV-1 DNA in peripheral blood mononuclear cells and inducible cell-free HIV-1 RNA in C

29 Methods: Peripheral blood mononuclear cells and nasal scrapings were obtained from

30 her-newborn dyads, maternal peripheral blood mononuclear cells and neonatal umbilical cord blood mono

31 We analyzed peripheral blood mononuclear cells and plasma collected following human H

32 vesicles derived from both peripheral blood mononuclear cells and plasma from HIV+ patient blood sam

33 ranscriptional profiling of peripheral blood mononuclear cells and promoter-based bioinformatic analy

34 ustion immunophenotyping on peripheral blood mononuclear cells and quantified interferon-stimulated g

35 Using flow cytometry on peripheral blood mononuclear cells and serum immunoassays, we analyzed th

36 Cytokine production by peripheral blood mononuclear cells and the frequency of cells expressing

37 NA levels in ventricles and peripheral blood mononuclear cells and their changes in patients with acu

38 and IL8 gene expression in peripheral blood mononuclear cells and tumors of patients treated with at

39 h levels of IL-8 in plasma, peripheral blood mononuclear cells and tumors were associated with decrea

40 methylation in normal human peripheral blood mononuclear cells and utilize a broad proteomic approach

41 m could induce apoptosis in peripheral blood mononuclear cells, and the underlying molecular mechanis

42 egrated HIV-1 DNA per 10(6) peripheral blood mononuclear cells, and were in generally good health.

43 Infiltrating host mononuclear cells are a likely source of TNF as these ch

44 evels of SQSTM1 and INSR in peripheral blood mononuclear cells are related to the severity of sepsis

45 Third, peripheral blood mononuclear cells are used to explore heterogeneous immu

46 ed inflammatory response in peripheral blood mononuclear cells, as well as a compensatory mechanism t

47 in IFN-gamma production by peripheral blood mononuclear cells at day 42 in the adjuvanted vaccine gr

48 We collected peripheral blood mononuclear cells at influenza diagnosis and four weeks

49 Peripheral blood mononuclear cells before and after cardiopulmonary bypas

50 Analysis of peripheral blood mononuclear cells before and after desensitization in on

51 the patients received up to 1.3 x 1010 donor mononuclear cells before transplantation.

52 y increased the proliferation of bone marrow mononuclear cells (BMNC) and, at the same time, inhibite

53 Bone marrow mononuclear cells (BMNC) are a source of naive macrophag

54 Bone marrow mononuclear cells (BMNCs) are a rich source of macrophag

55 ivity and high specificity in the imaging of mononuclear cells both in vitro and in vivo.

56 s markedly induced in AH livers, not only in mononuclear cells but also notably in hepatocytes.

57 Tomato mice, we found CX(3)CR1 expression in mononuclear cells, but not in multinucleated TRAP(+) OC.

58 mIDH1 clearance in bone marrow mononuclear cells by BEAMing (beads, emulsion, amplifica

59 EBV) DNA were quantified in peripheral blood mononuclear cells by droplet digital PCR (ddPCR).

60 ysis of CD3/CD28-stimulated peripheral blood mononuclear cells by RNA sequencing confirmed the stimul

61 s measured noninvasively in peripheral blood mononuclear cells can serve as novel biomarkers of sepsi

62 ia exposure during infancy, peripheral blood mononuclear cells collected from participants at age 2.5

63 osphorylation events within peripheral blood mononuclear cells collected prior to vaccination identif

64 the single-cell level from peripheral blood mononuclear cells collected ~2 weeks or 6-7 months postp

65 ed to scRNA-seq data from pancreatic tissue, mononuclear cells, colorectal tumor biopsies, and circul

66 Prior work has shown peripheral blood mononuclear cell conditioned media causes sublethal dege

67 The effect of peripheral blood mononuclear cell conditioned medium from vaccinated catt

68 We used a human peripheral blood mononuclear cell culture system and a nontransgenic mous

69 th in THP-1 cells and human peripheral blood mononuclear cell-derived macrophages, and attenuated M.

70 t T cells and primary human peripheral blood mononuclear cells, despite exhibiting severe defects in

71 soluble mucosal immune factors and cervical mononuclear cells during hormone titer-defined phases of

72 d therapy group had pre-ART peripheral blood mononuclear cells evaluated for drug resistance to non-n

73 ter cardiopulmonary bypass, peripheral blood mononuclear cells exhibited significantly reduced levels

74 omic DNA were isolated from peripheral blood mononuclear cells for transcriptional and DNA methylatio

75 lls were sorted from frozen peripheral blood mononuclear cells from 11 RV-infected hypogammaglobuline

76 and without asthma.Methods: Peripheral blood mononuclear cells from 17 children with asthma and 19 co

77 Here, we characterize peripheral blood mononuclear cells from 172 healthy adults 22-93 years of

78 Peripheral blood mononuclear cells from 23 participants collected longitu

79 ncing, we profiled ~276,000 peripheral blood mononuclear cells from 33 children with SLE with differe

80 is method to lymph node and peripheral blood mononuclear cells from 5 ART-treated donors to determine

81 Profiling of ~82,000 single peripheral blood mononuclear cells from adults with SLE confirmed the exp

82 Clinical leukapheresis can concentrate mononuclear cells from almost the entire blood volume, b

83 Stimulating peripheral blood mononuclear cells from COVID-19 convalescent patients wi

84 We apply Harmony to peripheral blood mononuclear cells from datasets with large experimental

85 By performing scRNA-seq on peripheral blood mononuclear cells from four untreated individuals before

86 was induced by transferring peripheral blood mononuclear cells from giant cell arteritis patients int

87 unomodulatory effects using peripheral blood mononuclear cells from healthy donors exposed to IFN-alp

88 T cells in peripheral blood mononuclear cells from healthy donors were stimulated wi

89 Metformin added to peripheral blood mononuclear cells from healthy volunteers enhanced in vi

90 man muscle fibers and single cell RNA-seq of mononuclear cells from human vastus lateralis, mouse qua

91 rasplenically injected with peripheral blood mononuclear cells from long-term ART-suppressed HIV-infe

92 T cells and lamina propria mononuclear cells from mice were analyzed by flow cytome

93 We evaluated peripheral blood mononuclear cells from MOG-AAD patients by flow cytometr

94 g MCL/MINCLE stimulation in peripheral blood mononuclear cells from MS patients.

95 Peripheral blood mononuclear cells from naive and matched acutely (n = 4)

96 pG methylation of the INS gene in cord blood mononuclear cells from offspring with a susceptible INS

97 A sequencing (scRNA-seq) of peripheral blood mononuclear cells from opioid-dependent individuals and

98 Peripheral blood mononuclear cells from patients with active CD differed

99 se of active UC in that the peripheral blood mononuclear cells from patients with CD had increased IL

100 re significantly reduced in peripheral blood mononuclear cells from patients with COPD compared with

101 iogenesis) noninvasively in peripheral blood mononuclear cells from patients with sepsis and correlat

102 of primary peripheral blood and bone marrow mononuclear cells from pediatric B-ALL patients, culture

103 f IL-35 subsets Ebi3 and p35 was elevated in mononuclear cells from peripheral blood, spleen, bone ma

104 Peripheral blood mononuclear cells from premalignant cases (1 year predia

105 in primary peripheral blood and bone marrow mononuclear cells from ten pediatric B-ALL patients.

106 In primary peripheral blood mononuclear cells from the patients, downstream type I i

107 itial enrichment by routine leukapheresis of mononuclear cells from very large blood volumes, followe

108 ogy) (n = 99), and analyzed peripheral blood mononuclear cell gene expression (n = 95) by quantitativ

109 zed RRGS animals with human peripheral blood mononuclear cells (hPBMCs) to set up a human acute GVHD

110 tudied with activated human peripheral blood mononuclear cells (hPBMCs).

111 in vitro from treated human peripheral blood mononuclear cells (hPBMCs).

112 mprovement, after human umbilical cord blood mononuclear cell (hUCBC) infusion.

113 nanomolar potency in cells (peripheral blood mononuclear cell, IC(50,free) = 6.2 nM) and good in vivo

114 cells, the Jurkat TAg T-cell line, and donor mononuclear cells in a SERINC5-dependent manner.

115 ics of the transcriptome of peripheral blood mononuclear cells in a two-dimensional state-space repre

116 somal activity in heart and peripheral blood mononuclear cells in all protocols except protocols 5A a

117 Most of the mononuclear cells in Coats' disease samples were CD163(+

118 ecreased PD-1 expression in peripheral blood mononuclear cells in monoculture by 41% and 55%, and co-

119 lowing engraftment of human peripheral blood mononuclear cells in NSG mice deficient in MHC class I a

120 on was acutely decreased in peripheral blood mononuclear cells in pediatric sepsis despite an increas

121 naive precursors from human peripheral blood mononuclear cells in the presence of various adjuvants,

122 mic profiling of plasma and peripheral blood mononuclear cells including metabolome, lipidome, immuno

123 collagen-1a, alpha-smooth muscle actin, and mononuclear cell infiltration (CD11b(+) Ly-6c(hi) monocy

124 ng cells [SFCs] per million peripheral blood mononuclear cells [IQR 841-2428], n=24; 56-69 years: 797

125 t-forming cells per million peripheral blood mononuclear cells, IQR 493-1802; n=43).

126 n T cell stimulation experiments using blood mononuclear cells isolated from both healthy donors and

127 f TNF-alpha and IL-6 in the peripheral blood mononuclear cells isolated from cutaneous lupus erythema

128 a patients, as well as from peripheral blood mononuclear cells isolated from leukoreduced donor whole

129 ntibody in a yeast display screen built from mononuclear cells isolated from the immunized camel and

130 n M158-66-specific CD8+ T cells in tonsillar mononuclear cells (MNC) of HLA-matched individuals.

131 fter ex vivo stimulation of peripheral blood mononuclear cells (MNCs) and bone marrow MNCs was higher

132 Autologous bone marrow mononuclear cells modulate joint homeostasis in an equin

133 in reduced DAP1 protein in peripheral blood mononuclear cells, monocytes, and lymphoblastoid cell li

134 Likewise, their peripheral blood mononuclear cells mounted a negligible cellular immune r

135 In PPD-stimulated peripheral blood mononuclear cells, MPA and dexamethasone, but not NET-A,

136 Mobilized peripheral blood mononuclear cells (mPBMCs) from B6 mice were transplante

137 mucosa samples (n = 87) and peripheral blood mononuclear cells (n = 85) from patients with active or

138 We investigated peripheral blood mononuclear cells (n=24), plasma (n=27), endomyocardial

139 HIV-1 DNA was quantified in peripheral blood mononuclear cells obtained from acutely (Fiebig I-III) a

140 Frozen peripheral blood mononuclear cells of 30 human immunodeficiency virus (HI

141 increased IDO1 activity in peripheral blood mononuclear cells of a significant proportion of MS pati

142 6 peptide-specific CD8+ T cells in tonsillar mononuclear cells of HLA-matched individuals.

143 nd content were measured in peripheral blood mononuclear cells on days 1-2, 3-5, and 8-14 after sepsi

144 ither coinjected with human peripheral blood mononuclear cells or engrafted with human hematopoietic

145 Human peripheral blood mononuclear cells, or immature dendritic cells derived f

146 We performed peripheral blood mononuclear cell (PBMC) analysis on a subgroup of 26 IgG

147 extracts were determined in peripheral blood mononuclear cell (PBMC) cultures.

148 -ABL inhibitor dasatinib in peripheral blood mononuclear cell (PBMC) lysates from individual donors.

149 ersistence, we analyzed 181 peripheral blood mononuclear cell (PBMC) samples from 64 PLWH starting AR

150 ed consistent modulation in peripheral blood mononuclear cell (PBMC) samples from HIV-positive (HIV(+

151 hnology Information for 120 peripheral blood mononuclear cell (PBMC) samples of patients with idiopat

152 included 57 BM samples, 29 peripheral blood mononuclear cell (PBMC) samples, and 32 plasma samples.

153 Here, we modify peripheral blood mononuclear cell (PBMC)-derived CD8 T cells to express t

154 Thus, peripheral blood mononuclear cell (PBMC)-derived induced pluripotent stem

155 ral DNA from peripheral blood and lymph node mononuclear cells (PBMC and LNMC) during ART suppression

156 isoforms of GR and MKP-1 in peripheral blood mononuclear cells (PBMC) after in vitro stimulation with

157 In both peripheral blood mononuclear cells (PBMC) and macrophages, omega-3 increa

158 quantitation of RRV DNA in peripheral blood mononuclear cells (PBMC) by real-time PCR revealed level

159 and functional responses of peripheral blood mononuclear cells (PBMC) collected from male rhesus maca

160 ome-wide gene expression in peripheral blood mononuclear cells (PBMC) derived from these IPF study su

161 ctive disease biomarkers in peripheral blood mononuclear cells (PBMC) from 25 premanifest subjects, 5

162 lity on 75 split samples of peripheral blood mononuclear cells (PBMC) from 5 antiretroviral therapy (

163 ome-wide gene expression in peripheral blood mononuclear cells (PBMC) from 77 Holocaust survivor offs

164 expression of both genes in peripheral blood mononuclear cells (PBMC) from ASD children, their non-AS

165 rasplenically injected with peripheral blood mononuclear cells (PBMC) from long-term ART-suppressed H

166 Peripheral blood mononuclear cells (PBMC) from perinatally infected child

167 as replication competent in peripheral blood mononuclear cells (PBMC) of all RMs tested.

168 tagonism of activated human peripheral blood mononuclear cells (PBMC) proliferation, significantly ou

169 rmed microarray analyses of peripheral blood mononuclear cells (PBMC) RNA from subjects enrolled in t

170 ed on pre- and on-treatment peripheral blood mononuclear cells (PBMC) samples.

171 While telomere length in peripheral blood mononuclear cells (PBMC) significantly decreased with ag

172 lel samples from plasma and peripheral blood mononuclear cells (PBMC) were also genotyped.

173 of IFN-gamma and IP-10 when peripheral blood mononuclear cells (PBMC) were co-cultured with INH, with

174 nd TFV-DP concentrations in peripheral blood mononuclear cells (PBMC) were found to be at least 10-fo

175 Cryopreserved peripheral blood mononuclear cells (PBMC) were obtained from infants from

176 -pregnancy-induced genes in peripheral blood mononuclear cells (PBMC); and (2) characterize the tempo

177 fic CAR-T cells using human peripheral blood mononuclear cells (PBMCs) and a CAR construct that enabl

178 tein was shown to adhere to peripheral blood mononuclear cells (PBMCs) and DF-1 cells and cause apopt

179 st time, ME/CFS miRNomes in peripheral blood mononuclear cells (PBMCs) and extracellular vesicles (EV

180 in peripheral blood mononuclear cells (PBMCs) and in cell pellets from CSF.

181 of immune response genes in peripheral blood mononuclear cells (PBMCs) and lesion biopsy specimens ob

182 ytokine production in human peripheral blood mononuclear cells (PBMCs) and rodent PaSCs, indicating t

183 and TFV-diphosphate (dp) in peripheral blood mononuclear cells (PBMCs) and seminal mononuclear cells

184 fied in allergen-stimulated peripheral blood mononuclear cells (PBMCs) and skin punch biopsies of IBH

185 PUUC were additive in human peripheral blood mononuclear cells (PBMCs) and synergistic in human FLT3-

186 sing cells, including human peripheral blood mononuclear cells (PBMCs) and the SupT1 cell line.

187 ses detected in circulating peripheral blood mononuclear cells (PBMCs) are referred to as "defective"

188 ated epigenetic patterns in peripheral blood mononuclear cells (PBMCs) as models to predict future ra

189 cRNA reduction are found in peripheral blood mononuclear cells (PBMCs) derived from patients with aut

190 as developed by challenging peripheral blood mononuclear cells (PBMCs) derived from patients with con

191 ed HRV-specific peptides on peripheral blood mononuclear cells (PBMCs) from 14 HLA I typed subjects.

192 l glucocorticoid bursts and peripheral blood mononuclear cells (PBMCs) from 17 adult asthmatics after

193 Peripheral blood mononuclear cells (PBMCs) from 25 patients with chronic

194 tocol using neutrophils and peripheral blood mononuclear cells (PBMCs) from CFS/ME patients (10) and

195 -1 was further evaluated in peripheral blood mononuclear cells (PBMCs) from FRDA patients and from no

196 regulation/inflammation, in peripheral blood mononuclear cells (PBMCs) from healthy donors and patien

197 n of innate immune cells in peripheral blood mononuclear cells (PBMCs) from healthy donors incubated

198 cation of kBET to data from peripheral blood mononuclear cells (PBMCs) from healthy donors to disting

199 CyTOF was performed on peripheral blood mononuclear cells (PBMCs) from PBC patients (n = 33) and

200 pare cytokine profiles in stimulated primary mononuclear cells (PBMCs) from RVVC and healthy individu

201 cing (scRNA-seq) to profile peripheral blood mononuclear cells (PBMCs) from seven patients hospitaliz

202 ies were sequenced from 2 million peripheral mononuclear cells (PBMCs) in 14 HAM/TSP patients, 34 MS

203 n, and serologic testing of peripheral blood mononuclear cells (PBMCs) in a subset of study participa

204 an exploratory analysis of peripheral blood mononuclear cells (PBMCs) isolated from healthy controls

205 evels of DNA methylation in peripheral blood mononuclear cells (PBMCs) longitudinally collected from

206 In the peripheral blood mononuclear cells (PBMCs) of COVID-19 patients, we obser

207 tokine staining (ICS) using peripheral blood mononuclear cells (PBMCs) of individuals naturally expos

208 inflammasome activation in peripheral blood mononuclear cells (PBMCs) of patients with BD and health

209 viability of healthy donor peripheral blood mononuclear cells (PBMCs) or NK cells, even at high (25

210 incubated with third-party peripheral blood mononuclear cells (PBMCs) pretreated with anti-HLA antib

211 transcriptomic profiles in peripheral blood mononuclear cells (PBMCs) pretreated with Whole Smoke-Co

212 hment of B cells from whole peripheral blood mononuclear cells (PBMCs) results in the most reliable B

213 igated responses of IgE and peripheral blood mononuclear cells (PBMCs) to flour extracts used in the

214 ulates the bioenergetics of peripheral blood mononuclear cells (PBMCs) under basal conditions or in s

215 nockout (GTKO), and TKO pig peripheral blood mononuclear cells (PBMCs) using sera from humans, severa

216 epidemiologic associations, peripheral blood mononuclear cells (PBMCs) were collected from 21 childre

217 hosphate (TFV-DP) levels in peripheral blood mononuclear cells (PBMCs) were similar among infected an

218 irculating, activated human peripheral blood mononuclear cells (PBMCs) were suppressed by MSC bioreac

219 d IL-18 upon stimulation of peripheral blood mononuclear cells (PBMCs) with antigen.

220 to antigenic stimulation of peripheral blood mononuclear cells (PBMCs) with novel Kgp synthetic pepti

221 Coculture with human peripheral blood mononuclear cells (PBMCs) with poly I:C-activated hPDL c

222 V-immune sera were added to peripheral blood mononuclear cells (PBMCs), and ADE and NK cell activatio

223 inflammatory activation in peripheral blood mononuclear cells (PBMCs), and the potential antiinflamm

224 diabetes was separated into peripheral blood mononuclear cells (PBMCs), plasma, and serum.

225 alidated preclinical model, peripheral blood mononuclear cells (PBMCs), that is highly predictive of

226 to CD4(+) T cells and human peripheral blood mononuclear cells (PBMCs), the propagation of the two vi

227 tochondrial respiration) of peripheral blood mononuclear cells (PBMCs), which was normalized in metfo

228 pithelial cells (NECs), and peripheral blood mononuclear cells (PBMCs).

229 (IgG) and 16.7/106 (IgA) in peripheral blood mononuclear cells (PBMCs).

230 Cs compared to normal human peripheral blood mononuclear cells (PBMCs).

231 zed by phosphoproteomics in peripheral blood mononuclear cells (PBMCs).

232 mouse splenocytes and human peripheral blood mononuclear cells (PBMCs).

233 expressed in both hepatocytes and peripheral mononuclear cells (PBMCs).

234 ular markers in circulating peripheral blood mononuclear cells (PBMCs).

235 sed on the surface of human peripheral blood mononuclear cells (PBMCs).

236 neuroblastoma (SH-SY5Y) and peripheral blood mononuclear cells (PBMCs).

237 muM) was observed in human peripheral blood mononuclear cells (PBMCs).

238 drained the muscle, and the peripheral blood mononuclear cells (PBMCs)isolated from the circulating b

239 We profiled total peripheral blood mononuclear cells (PBMCs, 106,545 cells) and dendritic c

240 onses (median 521 SFU/10(6) peripheral blood mononuclear cells [PBMCs] in the tetravalent group and m

241 teins in both BM and blood (peripheral blood mononuclear cells [PBMCs]).

242 markers and frequencies of peripheral blood mononuclear cell phenotypes (range 0.19 to 3.54 fold), a

243 found to be induced in the Peripheral Blood mononuclear cells (PMBCs) of asthmatic pre-school childr

244 Transcriptomic analysis of blood mononuclear cells post-Sm treatment shows IFN-I pathway

245 We collected peripheral blood mononuclear cells pre- and postimmunization from 60 pati

246 prior to DENV infection of peripheral blood mononuclear cells prevents activation of human vascular

247 weaning gene expression and peripheral blood mononuclear cell profiling may be useful as predictors o

248 re transfused with 1 x 10(7) of human spleen mononuclear cells reconstituted human CD45(+) cells that

249 es demonstrated that infant peripheral blood mononuclear cell responses were dominated by monocyte-as

250 Immunophenotyping of peripheral blood mononuclear cells revealed an increase in activated CD8+

251 ractions and unfractionated peripheral blood mononuclear cell samples longitudinally collected from s

252 Peripheral mononuclear cells, serum creatinine levels, and renal bi

253 2 diabetes patient-derived peripheral blood mononuclear cells show reduced expression of DBC1 and EL

254 uring co-culture with human peripheral blood mononuclear cells, showing that TZM-gfp can support outg

255 RNA-sequencing analysis of peripheral blood mononuclear cells, single-cell bioinformatics analysis a

256 blood mononuclear cells (PBMCs) and seminal mononuclear cells (SMCs) at the end of the dosing interv

257 Human peripheral blood mononuclear cells stimulated with M. tuberculosis displa

258 to those produced by human peripheral blood mononuclear cells stimulated with PG(Bb) In addition, sy

259 ne expression profiles from peripheral blood mononuclear cells taken at several time points from chil

260 e ever smokers, had shorter peripheral blood mononuclear cell telomeres, and were more likely to carr

261 In human peripheral blood mononuclear cells, these molecules suppressed Candida al

262 show that exposure of human peripheral blood mononuclear cells to bacterial lipopolysaccharide (LPS)

263 ranscriptional profiling of peripheral blood mononuclear cells to determine immunological signatures

264 pase-8 sensitized patients' peripheral blood mononuclear cells to RIPK1 activation, apoptosis and nec

265 d proliferative response of peripheral blood mononuclear cells to WHV peptides.

266 and revealed significant changes in specific mononuclear cell-type proportions related to age, sex, a

267 geneous tissue comprised of muscle fiber and mononuclear cell types that, in addition to movement, in

268 We identified 11 human skeletal muscle mononuclear cell types, including two fibro-adipogenic p

269 l and B-cell responses from peripheral blood mononuclear cells using flow cytometry and enzyme-linked

270 from polyclonally activated peripheral blood mononuclear cells using SAB assays and compared HLA anti

271 e-genome gene expression of peripheral blood mononuclear cells was performed before and after the int

272 Cathepsin Z mRNA in human peripheral blood mononuclear cells was significantly differentially-expre

273 e (CpG) sites measured from peripheral blood mononuclear cells, we identified 15 sites that were sign

274 Transcriptomic profiles of peripheral blood mononuclear cells were analyzed in n=6 patients with HF

275 content within circulating peripheral blood mononuclear cells were associated with organ dysfunction

276 lear cells and neonatal umbilical cord blood mononuclear cells were collected and cryopreserved short

277 Plasma samples and peripheral blood mononuclear cells were collected at 3 timepoints (pretra

278 Peripheral blood mononuclear cells were collected from 20 transplant reci

279 Peripheral blood mononuclear cells were collected from 30 transplant reci

280 Peripheral blood mononuclear cells were collected from donor-recipient pa

281 Peripheral blood mononuclear cells were cultured with atabecestat and its

282 Patient peripheral blood mononuclear cells were incubated in different arginine c

283 Peripheral blood mononuclear cells were infected with recombinant vesicul

284 heral blood was drawn from all children, and mononuclear cells were polyclonally activated.

285 Peripheral blood mononuclear cells were quantified for T and B cell subse

286 Peripheral blood mononuclear cells were sampled from acute myeloid leukem

287 levels of all four genes in peripheral blood mononuclear cells were significantly reduced in septic p

288 Peripheral blood mononuclear cells were stained and analyzed for frequenc

289 Peripheral blood mononuclear cells were stimulated with Staphylococcus au

290 itochondrial alterations in peripheral blood mononuclear cells were unrelated to organ dysfunction, p

291 duced cytokine responses in peripheral blood mononuclear cells were used to assess trained immunity.R

292 ic cells derived from human peripheral blood mononuclear cells, were cultured in medium containing TI

293 K(+) channel expression in peripheral blood mononuclear cell, which strongly correlated to that in v

294 ysing more than 1.5 billion peripheral blood mononuclear cells, which raises the possibility that a s

295 human livers, LCN2 expressed exclusively in mononuclear cells, while its expression was markedly ind

296 After stimulating peripheral blood mononuclear cells with anti-CD3/CD28 beads for 7 days, w

297 es following stimulation of peripheral blood mononuclear cells with complete peptide pools representi

298 e studies of whole blood or peripheral blood mononuclear cells with concordant direction of effect (h

299 coculturing HLA-II-matched peripheral blood mononuclear cells with HL cell lines and showed IS forma

300 n human Th17 induction from peripheral blood mononuclear cells, with ramifications for immunogenetic