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1 sured by real-time PCR from blood and tissue mononuclear leukocytes.
2 elatively sparing of normal peripheral blood mononuclear leukocytes.
3  TLR agonists, in the presence or absence of mononuclear leukocytes.
4 tein-coupled receptor kinase 2 expression in mononuclear leukocytes.
5  CCR3, and produced a normal calcium flux in mononuclear leukocytes.
6 ells (SMC), extracellular matrix and admixed mononuclear leukocytes.
7  chemotactic activity for various subsets of mononuclear leukocytes.
8 CR4 expression was localized to infiltrating mononuclear leukocytes.
9 s investigated, except polymorphonuclear and mononuclear leukocytes.
10 ression in rodent cardiomyocytes and patient mononuclear leukocytes.
11  disease is the perivascular accumulation of mononuclear leukocytes.
12 ed on the majority of human peripheral blood mononuclear leukocytes.
13 sulted in the presence of erythorbic acid in mononuclear leukocytes.
14 WMSKVKRFM (DcPLA2) was cloned from PMNLs and mononuclear leukocytes.
15 ly recognized kidney tissue compartments and mononuclear leukocytes.
16 ear stress per se is sufficient to stimulate mononuclear leukocyte adhesion and, presumptively, migra
17                      These data suggest that mononuclear leukocyte adhesion-dependent pathologies con
18                                              Mononuclear leukocytes also bound to poly(I.C)-treated M
19 nt protein-1 (MCP-1) is a potent agonist for mononuclear leukocytes and has been implicated in the pa
20 se include an increase in intestinal mucosal mononuclear leukocytes and hyperplasia of the muscularis
21           DPP4 was also found in a subset of mononuclear leukocytes and in serous cells of submucosal
22 injection stimulated OPGL expression in both mononuclear leukocytes and osteoblastic cells.
23 proteoglycans are both secreted by activated mononuclear leukocytes and released as a consequence of
24 therosclerosis is interaction of circulating mononuclear leukocytes and the endothelium.
25  and JFL relative to normal peripheral blood mononuclear leukocytes and the other cell lines.
26                             Peripheral blood mononuclear leukocytes and U937 monocytic cells adhered
27 cle, colon, testes, bone marrow cells, blood mononuclear leukocytes, and blood erythrocytes of mrp(-/
28                           The recruitment of mononuclear leukocytes, and the migration, growth and ac
29                 This study demonstrates that mononuclear leukocytes are markedly more sensitive than
30                                        These mononuclear leukocytes are mostly monocyte-derived, but
31 potent chemotactic and activating factor for mononuclear leukocytes, are mediated by specific binding
32 receptor ligands inhibited TNF-alpha-induced mononuclear leukocyte arrest by 60-65%.
33 e activated than those incubated with normal mononuclear leukocytes, as judged by the increased endot
34                             Proliferation of mononuclear leukocytes (assessed by expression of Ki-67,
35                                        Naive mononuclear leukocytes bind specifically to inflammation
36 ll surface hyaluronan (HA), and nonactivated mononuclear leukocytes bind to virus-induced HA structur
37 ere measured in 24-h culture supernatants of mononuclear leukocytes by immunoassays.
38 or T(H)1-type T cells (CXCL9 and CXCL10) and mononuclear leukocytes (CCL2, CCL3) among others.
39                  We have reported that human mononuclear leukocytes contain large amounts of angioten
40                         This accumulation of mononuclear leukocytes could be reversed by the administ
41 transgenic mice show cardiac infiltration by mononuclear leukocytes, culminating in dilated cardiomyo
42                       The addition of Ags to mononuclear leukocyte cultures typically elicits modest
43 din E2, IL-10, and IL-4 production in atopic mononuclear leukocyte cultures.
44 ternal-fetal interface, density of placental mononuclear leukocytes decreased with stress, yet matern
45 lear changes over time in the proportions of mononuclear leukocytes, especially in B-cells and T lymp
46  livers of IL-12-treated mice were primarily mononuclear leukocytes expressing LFA-1, VLA-4, MAC-1, a
47 ood lipids and HMG CoA reductase activity in mononuclear leukocytes fell at week 8 during both diets,
48 the total infectivity was recovered from the mononuclear leukocyte fraction.
49 phox mRNA was less than 10% of normal in the mononuclear leukocytes from 3 of the 4 patients analyzed
50                                     Although mononuclear leukocytes from DKO mice did not mount a pro
51  respectively, and the cytosolic fraction of mononuclear leukocytes from NAT1*4/*4 and NAT1*10/*10 ho
52 termine the mRNA transcriptome of cord blood mononuclear leukocytes from term neonates to identify ke
53                                              Mononuclear leukocytes frozen at the baseline investigat
54 genotyping was performed on peripheral blood mononuclear leukocyte genomic DNA.
55 a albicans by Toll-like receptors 2 and 4 on mononuclear leukocytes has recently been discovered to d
56                       Previous studies using mononuclear leukocytes have shown that the lipopolysacch
57 on of polymorphonuclear leukocytes (PMN) and mononuclear leukocytes, hemorrhage, increased concentrat
58 AL2 cleaves HA into fragments that stimulate mononuclear leukocytes in the immediate microenvironment
59 as the major quantitative adduct detected in mononuclear leukocytes in vivo and tumour cell lines in
60                               In circulating mononuclear leukocytes, in stark contrast, no inflammato
61     The expression of BST-2 was increased on mononuclear leukocytes, including CD4-positive T lymphoc
62 al pneumonia associated with a predominantly mononuclear leukocyte infiltrate.
63 he development of an atherosclerotic plaque, mononuclear leukocytes infiltrate the artery wall throug
64 ized by neuronal degeneration, necrosis, and mononuclear leukocyte infiltrates, was observed in the d
65  vs. abnormal kidney tissue compartments and mononuclear leukocyte infiltrates.
66         In vivo, DDR1 mRNA was detectable in mononuclear leukocytes infiltrating human renal tumor ti
67           Synovial tissue analysis confirmed mononuclear leukocyte infiltration in response to MSU cr
68 cular degeneration (AMD) is characterized by mononuclear leukocyte infiltration of the outer blood-re
69 eutropenia resulted in significantly reduced mononuclear leukocyte infiltration possibly due to reduc
70 sions specific for interstitium, tubules and mononuclear leukocyte infiltration.
71 lectin, and VCAM-1, particularly in areas of mononuclear leukocyte infiltration.
72 istic pathological changes include increased mononuclear leukocyte influx into the intestinal mucosa
73            Isolated autologous 99mTc-labeled mononuclear leukocytes injected into the left atrium loc
74 ver, they exhibited defective recruitment of mononuclear leukocytes into thioglycollate-induced perit
75 at transfusions of purified peripheral blood mononuclear leukocytes irradiated with 1,200 mJ/cm2 UVB
76                          The accumulation of mononuclear leukocytes is an early and persistent findin
77 cular mechanisms that mediate recruitment of mononuclear leukocytes (lymphocytes and monocytes) and d
78    Less is known about placental patterns of mononuclear leukocyte (MNL) density and PTD.
79      It has been shown that peripheral-blood mononuclear leukocytes (MNL) are responsible for transfu
80 died the transendothelial migration of blood mononuclear leukocytes (MNL) from 76 HIV+ patients and 4
81 ge of tolerant mixed chimeras with 5 million mononuclear leukocytes (MNL) from naive syngeneic mice w
82 raviolet B (UVB)-irradiated peripheral blood mononuclear leukocytes (MNL) have been shown to induce h
83 ll cytotoxic activities in the population of mononuclear leukocytes (MNL) in the liver, but not in MN
84 o produce proinflammatory cytokines, promote mononuclear leukocytes (MNL) transendothelial migration,
85      Endogenous polymorphonuclear (PMNs) and mononuclear leukocytes (MNLs) attached and rolled in HEV
86 ring polymorphonuclear leukocytes (PMNs) and mononuclear leukocytes (MNLs) from diluted human blood (
87   To further identify which subset of spleen mononuclear leukocytes (MNLs) in the tolerant CBA mice i
88 ers ex vivo and transmit infectious virus to mononuclear leukocytes (MNLs) lodged beneath this endoth
89 drocytes were cultured with autologous blood mononuclear leukocytes (MNLs).
90                   By contrast, the patients' mononuclear leukocytes, particularly monocytes, were hyp
91 tential of adenovirus vectors for studies of mononuclear leukocyte recruitment in vitro, we studied t
92 f retinoid X receptor (RXR)alpha on arterial mononuclear leukocyte recruitment is poorly understood,
93 a4 integrin signaling can selectively impair mononuclear leukocyte recruitment to sites of inflammati
94 elial cells and induces a temporal switch to mononuclear leukocyte recruitment.
95 ortant than the epithelium in recruitment of mononuclear leukocytes responsible for cell-mediated imm
96 -1) is a surface molecule expressed on human mononuclear leukocytes that functions as an inhibitory r
97 al intima as a result of the infiltration of mononuclear leukocytes, the proliferation of vascular sm
98 pound to mice led to impaired recruitment of mononuclear leukocytes to a site of inflammation in vivo
99 ar factor-kappa B activation was measured in mononuclear leukocytes using electrophoretic mobility sh
100 anism and recruitment of bone marrow-derived mononuclear leukocytes were evident in uterine tissue 1
101 othelial cells (MVEC) with sickle and normal mononuclear leukocytes were incubated, and endothelial a
102      Endothelial cells incubated with sickle mononuclear leukocytes were more activated than those in

 
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