ライフサイエンスコーパス: motor cortex

1 uit (i.e., primary sensory cortex to primary motor cortex).

2 stigated such mechanisms in human pharyngeal motor cortex.

3 13% increase in (18)F-DPA714 binding in the motor cortex.

4 particular phase of the beta activity in the motor cortex.

5 otor function after injury in monkey primary motor cortex.

6 l fibers originating from the contralesional motor cortex.

7 nscranial magnetic stimulation over the left motor cortex.

8 nts were uniquely represented in the primary motor cortex.

9 s have potential computational advantages in motor cortex.

10 ment of layer-specific activity in the human motor cortex.

11 leech central nervous system to the primate motor cortex.

12 and myelin sheath remodeling in the forelimb motor cortex.

13 y single elements are represented in primary motor cortex.

14 ecially during task engagement, in secondary motor cortex.

15 and changes in functional connectivity with motor cortex.

16 ion of preparatory activity in anterolateral motor cortex.

17 endrites of layer 5 pyramidal neurons in the motor cortex.

18 hown to reflect motor control in the primary motor cortex.

19 ise of gamma rhythm over right somatosensory-motor cortex.

20 the putamen and the hand and arm regions of motor cortex.

21 EEG was recorded over leg and hand area of motor cortex.

22 and physiological inhibition in the primary motor cortex.

23 ween head and forelimb areas of peri-infarct motor cortex.

24 fter focal cortical stroke in mouse forelimb motor cortex.

25 ocalized to ALM or widely distributed within motor cortex.

26 ynx and the jaw muscles in the human primary motor cortex.

27 ressing GGGGCC repeats and in C9-ALS patient motor cortex.

28 ) over the caudal forelimb area (CFA) of the motor cortex.

29 peration in mouse visual, somatosensory, and motor cortex.

30 be situated near the jaw region in the human motor cortex.

31 ral and ventromedial prefrontal cortices and motor cortex.

32 spatially clustered dendritic spines in the motor cortex.

33 ationships and its projection to the primary motor cortex.

34 consolidated and stratified in the premotor-motor cortex.

35 t via descending tracts originating from the motor cortex.

36 plicated in setting the initial state of the motor cortex(5,6), but they may also have a pattern-gene

37 of layer 5 pyramidal neurons in the primary motor cortex, a cortical region important for the acquis

38 were weakly represented in the anterolateral motor cortex, a region necessary for generating choices.

39 These results suggest that motor cortex acquires skillful control by leveraging bot

40 ibration cue and delayed frontal and lateral motor cortex activation.

41 A recent study in which primary motor cortex activity was imaged with sub-laminar resolu

42 ncertainty to compute choice is reflected in motor cortex activity.

43 how that disrupting left PPC but not primary motor cortex after learning has been allowed to occur bl

44 s and PV responses in mouse anterior lateral motor cortex (ALM) and barrel cortex (S1) upon PV photos

45 preparatory activity in the anterior lateral motor cortex (ALM) and cerebellum is related to the pred

46 emory, neurons in the mouse anterior lateral motor cortex (ALM) show persistent activity that instruc

47 mary motor cortex (wMC) and anterior lateral motor cortex (ALM).

48 ased KL expression via DNAm age residuals in motor cortex among older white non-Hispanics decedents (

49 h PTSD to predict advanced epigenetic age in motor cortex among the subset of relatively older (>=45

50 theta band power over the left somatosensory-motor cortex and a significant rise of gamma rhythm over

51 , alongside supraspinal regions, such as the motor cortex and brainstem.

52 tial tremor patients is associated with both motor cortex and cerebellar connectivity.

53 terconnected structures, such as the primary motor cortex and cerebellum, as well as the contribution

54 In motor cortex and dorsal premotor cortex, we find that th

55 , we recorded neural activity in the primary motor cortex and dorsolateral striatum during reach-to-g

56 Furthermore, inactivation of the primary motor cortex and dorsolateral striatum had distinct effe

57 ve network degeneration originating from the motor cortex and expanding in a spatiotemporal manner.

58 d women in the premotor cortex/supplementary motor cortex and left medial superior frontal gyrus.

59 ced directional metaplasticity in pharyngeal motor cortex and new insights into its clinical applicat

60 umbar locomotor network independently of the motor cortex and other supraspinal locomotor centers.

61 m128C cells in the superficial layers of the motor cortex and performed in vivo calcium imaging in mi

62 -high field (1)H-MRS scans of the upper limb motor cortex and pons, ALS Functional Rating Scale-Revis

63 the presence of NK cells in post-mortem ALS motor cortex and spinal cord tissues, and the expression

64 , we demonstrate NK cell accumulation in the motor cortex and spinal cord, with an early CCL2-depende

65 xpressing an ALS-causing TDP-43 mutation, in motor cortex and spinal motor neurons from patients with

66 ggest that the disease might initiate in the motor cortex and spread to its targets along the cortico

67 systematically varied the size of strokes in motor cortex and surrounding regions to assess effects o

68 layers II/III and V striatal afferents from motor cortex and that cortical BDNF is essential for mot

69 al magnetic stimulation (TMS) of the primary motor cortex and the measurement of motor evoked potenti

70 00 Gy to 5-7.5 mm fields in the left primary motor cortex and the right subcortical white matter, and

71 ted predominately in the white matter of the motor cortex and the spinal cord.

72 Neuronal activity mapping revealed bilateral motor cortex and unilateral, ischemic somatosensory cort

73 psilateral activity suppresses contralateral motor cortex and, accordingly, that inhibiting ipsilater

74 Most work to date has focused on primary motor cortex, and although there is ample evidence of le

75 of cortical projections to contralateral pre-motor cortex, and upregulation of CREB and DLK signaling

76 and vocal motor thalamus, auditory and vocal motor cortex, and VTA.

77 in the anterior cingulate cortex and primary motor cortex; and (iii) abnormal CSF phosphorylated tau

78 The cerebellum and the motor cortex appear to be critical for our ability to le

79 s suggest that neural population dynamics in motor cortex are invariant to the number of recorded neu

80 excitable phase of beta oscillations in the motor cortex are known to lead to muscle responses of gr

81 ranial magnetic stimulation over the primary motor cortex arrive at corticospinal-motor neuronal syna

82 has been made using rodents to establish the motor cortex as an adaptive structure that supports moto

83 sitive cells were observed in postmortem ALS motor cortex as compared with controls, and these cells

84 recisely during the most active phase of the motor cortex as patients attempted to perform a movement

85 us, entrains the excitability of the primary motor cortex, as reflected by the phase-amplitude coupli

86 y 30 min over the hand representation of the motor cortex at an interstimulus interval mimicking the

87 a unique activation was found in the primary motor cortex (BA4) at MNI coordinates -38 -26 56.

88 ater brain activity than controls in primary motor cortex (BA4), supramarginal gyrus (BA40), inferior

89 Changes in neural activity occur in the motor cortex before movement, but the nature and purpose

90 studies found that preconditioning the hand motor cortex before rTMS could enhance stimulation outco

91 duration of beta bursts in the contralateral motor cortex before the go-cue, but not with averaged be

92 ospinal cells from primary and supplementary motor cortex bilaterally.

93 the gray/white matter spinal cord and brain motor cortex/brainstem, substantially restored capillary

94 ex itself or reflect abnormal modulations of motor cortex by other schizophrenia-related brain areas.

95 on the brain surface, known as the secondary motor cortex, can also reverse movement disorders in mic

96 cilitation, but not inhibition, in the human motor cortex, challenging the generalizability of the pu

97 The human motor cortex comprises a microcircuit of five interconne

98 l current stimulation of the rat (all males) motor cortex consisting of a continuous subthreshold sin

99 corticospinal volley from stimulation of the motor cortex consists of two parts: one that originates

100 ility, a mechanism through which the primary motor cortex controls motor output, is unknown.

101 The motor cortex controls skilled arm movement by sending te

102 ynaptic inputs to OPCs residing in secondary motor cortex, corpus callosum, and primary somatosensory

103 ium-activated potassium channel Kcnn2 in the motor cortex correlated with motor learning deficits in

104 d show increased (18)F-DPA714 binding in the motor cortex correlating with Iba1 expression (glial cel

105 have shown that preconditioning of the hand motor cortex could increase cortical capacity for neurop

106 lly, spine maturation and persistence in the motor cortex depend on synaptic mobilization of the glut

107 e evidence of learning-related plasticity in motor cortex, direct evidence for its involvement in mem

108 es a decrease in hyperdirect inputs from the motor cortex directly to the STN and that rescuing this

109 quantify the concentration of GABA in human motor cortex during a period of motor learning, as well

110 5-25 Hz) scalp EEG signals recorded over the motor cortex during a pre-movement preparatory phase wer

111 e we examined BSC based on Hyperalignment of motor cortex during a task of motor imagery of three nat

112 larity analysis in primary somatosensory and motor cortex during missing and intact hand movements.

113 scranial magnetic brain stimulation over the motor cortex during motor imagination leads to enhanced

114 d sequential activation in the mouse primary motor cortex during motor skill learning.

115 es have hypothesized that neural activity in motor cortex during preparation acts as an 'initial cond

116 hared markers between RA and human laryngeal motor cortex (e.g. SLIT1, RTN4R, LINGO1, PLXNC1).

117 to the optimal phase of the beta EEG in the motor cortex enjoy transmission amplitude gain, but may

118 that both layer II/III and V neurons in the motor cortex express BDNF as a potential regulator of pl

119 was most optimal for suppressing pharyngeal motor cortex (F(1,13) = 13.547; P = 0.003).

120 ediation analysis showed that the prefrontal-motor cortex FC significantly mediated the corona radiat

121 In schizophrenia, left prefrontal cortex-motor cortex FC was inversely associated with SICI but p

122 Higher resting-state left prefrontal-motor cortex FC, accompanied by a higher fractional anis

123 we probed sensorimotor interactions in face motor cortex (fM1) using short-afferent inhibition (SAI)

124 In contrast, cTBS to motor cortex following learning had little effect on ret

125 elated words (e.g., "write"), but in ventral motor cortex for face-related words ("talk").

126 ulation therapies in addition to the primary motor cortex for patients who do not respond adequately

127 centrally and can be alleviated by altering motor cortex function.

128 anscranial magnetic stimulation (TMS) of the motor cortex generated plastic changes in motor output.

129 We used electrode arrays to record from the motor cortex 'hand knob' in two people with tetraplegia,

130 Increased beta bursts detected in the motor cortex have also been associated with longer react

131 halography (EEG) recordings over the primary motor cortex have been associated with the preparation a

132 unctional metaplasticity in human pharyngeal motor cortex have been identified.

133 ch with monkeys has shown how neurons in the motor cortex have firing rates tuned to movement directi

134 show that inactivation of the mouse primary motor cortex impairs both the acquisition and extinction

135 hat optogenetic activation of secondary (M2) motor cortex improves motor functions in dopamine-deplet

136 sults suggest opposing roles for sensory and motor cortex in behavioral choice via distinct influence

137 ter unilateral photothrombotic stroke of the motor cortex in mice using anterograde tracing.

138 Conclusion NAA/Cr levels decreased in the motor cortex in patients with CSM 6 months after success

139 pecific manipulation of sensory signaling in motor cortex in rats by locally concentrating and releas

140 Inhibitory deficits in motor cortex in schizophrenia have been well demonstrate

141 , suggesting that the inhibitory deficits in motor cortex in schizophrenia may in part be mediated by

142 in the wild, is well represented within the motor cortex in the form of muscle synergies between dif

143 l and functional organisation of the primary motor cortex in the human brain.

144 Early evolution of the motor cortex included development of connections to brai

145 profiles from mouse astrocytic membralin KO motor cortex indicated significant perturbation in KEGG

146 by previous noninvasive TMS studies of human motor cortex indicating a reduction of corticospinal exc

147 Yet, when and how motor cortex influences muscle activity during movement

148 chical control of vocal production, with the motor cortex influencing the pacing of singing behavior

149 onal spaces of the motor imagery task in the motor cortex into a common model representational space.

150 omotor-learning tasks induced a reduction in motor cortex intracortical inhibition but did not modula

151 orticospinal excitability but did not change motor cortex intracortical inhibition or sensorimotor in

152 erhemispheric functional connectivity in the motor cortex is also reduced, consistent with disrupted

153 e data invite the hypothesis that the speech motor cortex is best modelled as a neural oscillator, a

154 ease, the LTP-like plasticity of the primary motor cortex is impaired, and gamma oscillations are alt

155 Whether and how the primary motor cortex is involved in modulating freezing response

156 n learning, somatosensory cortex rather than motor cortex is involved in the consolidation of motor m

157 The primary motor cortex is known to participate in the planning, co

158 the ventral premotor cortex, and the primary motor cortex is necessary for transforming visual inform

159 al exercise in the adult, BDNF expression in motor cortex is reinduced, especially in layer II/III pr

160 these deficits originate from dysfunction of motor cortex itself or reflect abnormal modulations of m

161 rtical circuits which may refer to different motor cortex layers in humans, by exploiting small time

162 ost-stroke neuronal knockdown of CCR5 in pre-motor cortex leads to early recovery of motor control.

163 very of function after injury of the primary motor cortex, likely through enhancing plasticity in per

164 Primary motor cortex (M1) almost exclusively controls the contra

165 In the present study, we investigated motor cortex (M1) and a small portion of premotor and pa

166 excitability were measured in vitro in mouse motor cortex (M1) and in human postoperative neocortex,

167 wer components thought to arise from primary motor cortex (M1) and other supraspinal areas.

168 or areas such as contralateral S1BC, primary motor cortex (M1) and secondary somatosensory cortex (S2

169 to stimulation of different parts of primary motor cortex (M1) and supplementary motor area (SMA) bil

170 magnetic stimulation (TMS) over the primary motor cortex (M1) appear to activate distinct interneuro

171 nnections between the cerebellum and primary motor cortex (M1) are essential for performing daily lif

172 nscranial magnetic stimulation (TMS) of hand motor cortex (M1) as a model, but in this model it is di

173 examined neural activity in SMA and primary motor cortex (M1) as monkeys cycled various distances th

174 nial magnetic stimulation applied to primary motor cortex (M1) at different coil orientations.

175 ng current stimulation (tACS) to the primary motor cortex (M1) at different frequencies during an ind

176 Motor cortex (M1) has lateralized outputs, yet neurons c

177 The forelimb representation in motor cortex (M1) is an important model system in contem

178 nts in M1.SIGNIFICANCE STATEMENT The primary motor cortex (M1) is important for producing individuate

179 To examine whether primary motor cortex (M1) is involved in maintenance of sequenti

180 rameters influence the effects of TUS on the motor cortex (M1) of 16 healthy subjects by probing cort

181 How is the primary motor cortex (M1) organized to control fine finger movem

182 ed that antidromic activation of the primary motor cortex (M1) plays a significant role in mediating

183 ATEMENT Previous work has shown that primary motor cortex (M1) represents information related to the

184 how that during the belief of agency primary motor cortex (M1) shows stronger functional connectivity

185 igh density microelectrode arrays in primary motor cortex (M1) to record neuronal population response

186 ranial magnetic stimulation over the primary motor cortex (M1) we examined motor evoked potentials (M

187 tion (TMS) over the hand area of the primary motor cortex (M1) when humans tracked with the eyes a vi

188 d in neuronal population activity in primary motor cortex (M1) when monkeys make reaching movements.

189 or hotspot (thought to represent the primary motor cortex (M1)) to modulate motor network excitabilit

190 interactions between premotor ventral (PMv)-motor cortex (M1), anterior inferior parietal lobule (aI

191 st measured after stimulation of the primary motor cortex (M1), corticospinal tract (CST), and reticu

192 magined movements directly influence primary motor cortex (M1), how this might occur remains unknown

193 ent in the pars opercularis IFG/PMv, primary motor cortex (M1), IPL/supramarginal gyrus and middle oc

194 After subtotal infarcts of primary motor cortex (M1), motor rehabilitative training (RT) pr

195 luding anterior parietal areas, from primary motor cortex (M1), premotor cortex (PM), the supplementa

196 In human motor cortex (M1), recruitment of indirect waves (I-wave

197 In the primary motor cortex (M1), residual subperceptual hand touch sig

198 heir neuronal and network realization in the motor cortex (M1), which directs skilled movements, is l

199 motor network and, specifically, in primary motor cortex (M1), which results from overall disinhibit

200 ndary somatosensory cortex (S2), and primary motor cortex (M1), with similar distributions in the ips

201 hanges in local circuitry within the primary motor cortex (M1).

202 In our case, the target was the primary motor cortex (M1).

203 e dominant oscillatory activity in the human motor cortex (M1).

204 No changes were observed in the primary motor cortex (M1).

205 ntal (PFC), somatosensory (SS2), and primary motor cortex (M1).

206 ns of large groups of neurons in the primary motor cortex (M1).

207 ic plasticity and E/I balance in the primary motor cortex (M1).

208 stimulation (iTBS) is reduced in the primary motor cortex (M1).

209 the organization of intrinsic connections in motor cortex (M1).

210 The secondary motor cortex (M2) is a prime candidate for this role giv

211 vo tractography, in addition to the cerebral motor cortex, major portions of CPC streamlines leave th

212 n over the leg representation of the primary motor cortex, maximal voluntary contractions (MVCs), and

213 n over the arm representation of the primary motor cortex, maximal voluntary contractions, the StartR

214 This manifold-based view of motor cortex may lead to a better understanding of how t

215 connections between separate neural sites in motor cortex (MC).

216 How do neurons in orofacial motor cortex (MCtx) orchestrate behaviors?

217 hine produced increased ventral striatum and motor cortex metabolism in females, and increased ventra

218 to integrate physiological accounts of this motor cortex microcircuit with the pathophysiology of ne

219 guided by functional mapping of the primary motor cortex, naive participants were awakened from REM

220 BF increases in the dorsomedial thalamus and motor cortex near the vertex ECT electrode, as well as d

221 deficits that there are specific changes in motor cortex neuron activity associated with the graspin

222 During spindles, primary motor cortex neurons fired at a preferred phase, with ne

223 We thus recorded activity from the primary motor cortex of 4 male sleeping rats to investigate how

224 ngs of parkinsonian movement deficits in the motor cortex of chronic unilateral 6-hydroxydopamine les

225 neuron-to-population coupling for neurons in motor cortex of freely moving rats.

226 TMS was delivered to the motor cortex of healthy human subjects, and baseline MEP

227 We delivered single-pulse TMS to the motor cortex of healthy human volunteers (10 females and

228 m signals in pyramidal cell dendrites in the motor cortex of mice performing a tactile decision task.

229 rgeted or traditional photothrombosis to the motor cortex of mice, post-ischemic cerebral blood flow

230 a period of synapse turnover in peri-infarct motor cortex of mice.

231 We recorded data using Neuropixels probes in motor cortex of nonhuman primates and reanalyzed data fr

232 However, in motor cortex of the awake macaque monkey, we observe ver

233 udy is the first detailed exploration of the motor cortex of these primates using long-train intracor

234 potential (LFP) activity within the primary motor cortex of three cats during a prism visuomotor ada

235 recordings of neural activity in the primary motor cortex of two female nonhuman primates during free

236 rs, the effects of disruption of left speech motor cortex on responses to tone changes were inconclus

237 This a priori suggests that motor cortex operates in a noncritical regime, which in

238 ea and functional anticorrelation to primary motor cortex (p < 0.001).

239 y associated with advanced epigenetic age in motor cortex (p = 0.039, n = 114).

240 An understanding of motor cortex plasticity has been advanced greatly using

241 y GABA are thought to be crucial in inducing motor cortex plasticity.

242 riatum (PPC-STR) and the posterior secondary motor cortex (PPC-pM2).

243 the somatosensory cortex, as well as in the motor cortex, preferentially track time-varying postures

244 h originated overwhelmingly from the primary motor cortex, primary somatosensory cortex, and secondar

245 ry activity similar to that in anterolateral motor cortex prior to reward acquisition.

246 re than 1,300 neurons in adult mouse primary motor cortex, providing a morpho-electric annotation of

247 ion normalizes pathological hyperactivity of motor cortex pyramidal cells, while concurrently activat

248 rominent efferent pathways from HVC to vocal-motor cortex (RA, robust nucleus of the arcopallium) and

249 adigms are known to induce plasticity in the motor cortex, reflected by changes in the motor evoked p

250 primary somatosensory cortex, and secondary motor cortex, regions that are linked to skeletomotor co

251 gest that the functional organization of the motor cortex represents not just muscles of the body, bu

252 tivities of layer 5 pyramidal neurons in the motor cortex respectively.

253 immediately after lesions in the adult mouse motor cortex restored damaged cortical pathways.

254 Local field potentials in monkey motor cortex revealed characteristic signatures of a Kal

255 song learning in the avian analog of primary motor cortex (robust nucleus of the arcopallium, RA) in

256 main sources of feedback to rat S1: primary motor cortex, secondary somatosensory cortex, and second

257 changes in the number of interneurons while motor cortex, showed no obvious loss in any of the subse

258 In rat motor cortex, SMI32-postive pyramidal neurons expressing

259 We recorded myogenic MEPs after transcranial motor cortex stimulation in 6 lambs aged 1-2 days.

260 ower, attested in the regions of a cortical (motor cortex, supplementary motor area, premotor cortex)

261 ges, disruption of left but not right speech motor cortex suppressed responses in both language group

262 guage groups: disruption of the right speech motor cortex suppressed responses to tone changes in non

263 akers, whereas disruption of the left speech motor cortex suppressed responses to tone changes in ton

264 eural- and circuit-level mechanisms by which motor cortex, thalamus, and striatum support motor learn

265 ct more than 1,000 projection neurons in the motor cortex, thalamus, subiculum, and hypothalamus.

266 d patterns, such as 7 Hz oscillations in rat motor cortex that are not time locked to measured behavi

267 diated by synaptic plasticity in a region of motor cortex that, before lesions, is not essential for

268 In motor cortex, this phenomenon was indicative of disconti

269 Motor cortex tNAA:mIns and ALSFRS-R at baseline were sig

270 e contributions of the right and left speech motor cortex to auditory speech processing are determine

271 etic and electrical stimulation over the leg motor cortex to elicit motor evoked potentials (MEPs) in

272 to align neural responses across subject in motor cortex to enable BSC of motor imagery.

273 rain slice and in vivo experiments on monkey motor cortex to investigate the output timing from layer

274 ied transcranial magnetic stimulation to the motor cortex, to assess movement preparation and executi

275 Motor cortex total N-acetylaspartate to myo-inositol rat

276 changes in recurrent connections in the rat motor cortex upon training on a novel motor task.

277 precision after photothrombotic infarcts in motor cortex using longitudinal 2-photon and multiexposu

278 l metaplastic properties in human pharyngeal motor cortex using preconditioned rTMS.

279 mporarily disrupted the right or left speech motor cortex using transcranial magnetic stimulation (TM

280 gle pulse TMS was administered over the left motor cortex, using anatomical scans of each subject to

281 expression in sections from macaque and rat motor cortex, using two different antibodies (NeuroMab,

282 sensory cortex, vS1, together with vibrissal motor cortex, vM1 (a frontal cortex target of vS1), whil

283 Decreased beta-band power over contralateral motor cortex was associated with a focal shift from rela

284 connectivity between the dorsal striatum and motor cortex was associated with more severe negative PL

285 etention at all; retention following cTBS to motor cortex was not different than following sham TMS s

286 ate component (BP2, generated in the primary motor cortex) was found to be linked to motor execution

287 Within primary motor cortex, we evoked finger movements involving flexi

288 ranial recordings from the basal ganglia and motor cortex, we found that response-conflict increases

289 thin long-term neural recordings from monkey motor cortex, we found two distinct patterns of sequenti

290 biotin-dextran-amine injections in the left motor cortex were equally labelled across groups and red

291 Stimulation sites for SICI at motor cortex were used as the seeds to obtain whole-brai

292 ceived 10 min of 1 Hz rTMS to the pharyngeal motor cortex which elicited the largest PMEPs to suppres

293 inantly from area 2), posterior parietal and motor cortex, which could provide the substrate for repr

294 e copy related to orientation from secondary motor cortex, which is involved in controlling HOMs.

295 rst report of sequential sleep replay in the motor cortex, which may play an important role in consol

296 ent site of seizure origin, to the bilateral motor cortex, which mediates convulsive seizures, have n

297 dapting internal model of visuomotor gain in motor cortex while two macaques performed a reaching tas

298 ndicated that short-latency pathways linking motor cortex with spinal motor neurons are selectively a

299 at the noxious stimulation activates the pre-motor cortex with the highest activation shown in the ce

300 including both the whisker region of primary motor cortex (wMC) and anterior lateral motor cortex (AL