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1 ugh impaired cholinergic pathway, leading to motor disorder.
2 ation of conversion disorder or dissociative motor disorder.
3 d different conclusions on the nature of the motor disorder.
4  understanding of the pathophysiology of the motor disorder.
5 r HIV-associated dementia or minor cognitive-motor disorder.
6 rectly implicating auditory function in this motor disorder.
7 atment for selected patients with functional motor disorder.
8 accompanied by a pyramidal or extrapyramidal motor disorder.
9 t of therapeutics for spinal cord injury and motor disorders.
10 estinal inflammation, take place long before motor disorders.
11  neural activity associated with symptoms of motor disorders.
12 g can lead to failure in behavioral tasks or motor disorders.
13 K(+) (BK) channels also result in tremor and motor disorders.
14 apeutic strategies for CBG-related vocal and motor disorders.
15 und to aid and assist recovery in functional motor disorders.
16  and aid in development of drugs for gastric motor disorders.
17 ommonly used to relief spasticity related to motor disorders.
18 s in the treatment of esophageal sensory and motor disorders.
19 ther studying, monitoring, and even treating motor disorders.
20 ory dysfunction as a component of esophageal motor disorders.
21 edical, and surgical therapies for childhood motor disorders.
22  regions during normal motor function and in motor disorders.
23 lasting muscle relaxation effects on spastic motor disorders.
24 pports treatment decisions for patients with motor disorders.
25 riggers ROS and PD pathologies, resulting in motor disorders.
26 s disease, and is often also associated with motor disorders(1).
27 incidence of speech/language, scholastic, or motor disorders (829, 187, and 285 instances, respective
28 n on voluntary motor output in subjects with motor disorders affected by spasticity.
29  Parkinson's disease (PD) is the most common motor disorder affecting the elderly.
30 rget for enhancing voluntary motor output in motor disorders affecting the corticospinal tract.
31                        Although defined as a motor disorder, ALS can arise concurrently with frontote
32 ine interventions for people with functional motor disorder and develop evidence-based methods to gui
33 ithout fibromyalgia, 35 with both functional motor disorder and fibromyalgia, 35 with fibromyalgia on
34 h functional disorders, including functional motor disorder and fibromyalgia, suggesting a shared def
35 "fibromyalgianess" in people with functional motor disorder and fibromyalgia.
36 e electron transport chain (ETC) induces the motor disorder and PD pathologies in neuronal Thy1-C/EBP
37 that suffer cerebellar insults often develop motor disorders and cognitive difficulty.
38 port widens the phenotype of dopa-responsive motor disorders and the range of young children with pri
39  insight into the pathogenesis of esophageal motor disorders and treatment of gastroesophageal reflux
40 chological impairment, 5 had minor cognitive/motor disorder, and 1 was demented.
41 ppear in the absence of any other sensory or motor disorder, and is sufficient to cause a literacy im
42 remains the most investigated and understood motor disorder, and the year's research focused heavily
43 vents that serve as signatures of esophageal motor disorders, and esophageal manometry retains its po
44  high-resolution manometry to rule out major motor disorders, and pH monitoring off PPI therapy (or p
45 ircadian behavior, neuronal differentiation, motor disorders, anxiety, and neurodegenerative diseases
46 sting studies of the prognosis of functional motor disorder are nearly all retrospective, small and u
47 Reliable data on the prognosis of functional motor disorder are scarce, as existing studies of the pr
48 Importance: Speech/language, scholastic, and motor disorders are common in children.
49       While half of the mutations that cause motor disorders are positioned to affect enzyme activity
50  mice facilitates PD pathologies and elicits motor disorders associated with augmentation of d-secret
51  mice facilitates PD pathologies and elicits motor disorders associated with augmentation of delta-se
52 an their littermates and showed very obvious motor disorders, associated with a dramatic loss of larg
53 the vague and variable presentations of this motor disorder become appreciated.
54 ot only in patients with levodopa-responsive motor disorders, but also in patients with developmental
55            Rodent models of DYT1 dystonia, a motor disorder caused by a single gene mutation, demonst
56 on developed from 5 mo of age, a progressive motor disorder characterized at onset by impaired rota-r
57 key risk factor for dystonia, a debilitating motor disorder characterized by cocontractions of muscle
58 mutations in humans lead to hyperekplexia, a motor disorder characterized by startle responses, the z
59                     Although predominantly a motor disorder, cognitive impairment and dementia are im
60        Although PD is primarily considered a motor disorder, cognitive impairment is often present at
61 st physiotherapy intervention for functional motor disorder compared with treatment as usual.
62 s particularly relevant to participants with motor disorders coupled with an increased incidence of s
63     More recent studies suggest instead that motor disorders derive from abnormal firing patterns, an
64  clinically definite diagnosis of functional motor disorder, diagnosed by a neurologist, were include
65                          The childhood-onset motor disorder DYT6 dystonia is caused by loss-of-functi
66 otions in the legs of mice with neurological motor disorders, enabling the animals to kick a ball, wa
67 ular interactors that probably bear on human motor disorders, especially those affecting speech.
68                     Patients with functional motor disorder (FMD) including weakness and paralysis ar
69 nderlying various acquired and developmental motor disorders have been described, as well as the effe
70        This work reviews supranuclear ocular motor disorders, highlighting new data published during
71  about pathophysiology of spastic esophageal motor disorders, however, with its recent update (Chicag
72 r conversion disorder (DSM-IV), dissociative motor disorder (ICD-10) and 'psychogenic' paralysis.
73  known as congenital nystagmus, is an ocular motor disorder in humans characterized by spontaneous ey
74 entre international prospective study of the motor disorder in the largest cohort of patients studied
75 cracker" esophagus in two, and a nonspecific motor disorder in two patients.
76 dging the presence of additional sensory and motor disorders in certain individuals.
77 aneously could also be developed to classify motor disorders in humans.
78 ciated with speech/language, scholastic, and motor disorders in offspring up to early adolescence.
79       Children with autism exhibit a host of motor disorders including poor coordination, poor tool u
80 deficiency virus type 1-associated cognitive-motor disorder, including the AIDS dementia complex, is
81           Mouse GAT1 (mGAT1) KO mice exhibit motor disorders, including gait abnormality, constant 25
82                                     "A major motor disorder is unlikely in the setting of a 'normal'
83 ent of achalasia, the best-understood distal motor disorder, is resolving as the benefits and disadva
84  met diagnostic criteria for minor cognitive/motor disorder (MCMD) or HIV-associated dementia (HAD).
85 ile Parkinson's disease (PD) is considered a motor disorder, motor signs of PD can be exacerbated by
86                  In patients with functional motor disorder, no association was found between prepuls
87 he pathogenesis and management of esophageal motor disorders occurred over the past year.
88 nfidence interval = 1.9-16.8; P = 0.002) and motor disorder (odds ratio = 3.3; confidence interval =
89 re provided, along with a summary of how the motor disorder of LND relates to current understanding o
90                                          The motor disorders of CP are frequently accompanied by impa
91  muscle in health, as well as in sensory and motor disorders of the esophagus.
92 ophagus in normal subjects and patients with motor disorders of the esophagus.
93                             The concept that motor disorders of the gallbladder, cystic duct and sphi
94 ical relevance, because it helps explain why motor disorders of the LES and fundus frequently occur t
95 al projections as well as changes induced by motor disorders or injuries are not known in nonhuman pr
96 s a target antigen in a human paraneoplastic motor disorder [paraneoplastic opsoclonus-myoclonus atax
97 iduals present with a rare negative episodic motor disorder, paroxysmal nonkinesigenic dyskinesia (PN
98 tapping) Nineteen subjects without diagnosed motor disorders participated in the study.
99 hythms, have been studied in a wide range of motor disorders, providing a foundation for developing r
100 ) has expanded as an effective treatment for motor disorders, providing a valuable opportunity for in
101                Accordingly, individuals with motor disorders receiving mild intermittent hypoxia as a
102 t the mechanisms underlying dopamine-related motor disorders remain under debate.
103  (2.38) for speech/language, scholastic, and motor disorders, respectively.
104 ic and cortical fiber loss and a progressive motor disorder responsive to l-DOPA.
105 nication disorders, intellectual disability, motor disorders, specific learning disorders, and tic di
106              When focusing on the functional motor disorder subtype (FND-motor, n=46), a classifier r
107 development of therapeutics for upper airway motor disorders such as obstructive sleep apnoea.
108 sent targets in nondopaminergic treatment of motor disorders such as Parkinson's disease (PD).
109  of particular interest as it is elevated in motor disorders such as Parkinson's disease and modulate
110 vation and movement, and is dysfunctional in motor disorders such as Parkinson's disease.
111 ested this theory in children with cognitive-motor disorders, such as developmental coordination diso
112           The pathogenesis of complex ocular motor disorders, such as paradoxical pupillary constrict
113 ssociated dementia and related cognitive and motor disorders that affect 20 to 30% of treatment-naive
114 , epilepsy, and pyramidal and extrapyramidal motor disorders) that are primarily attributed to defici
115                                   Functional motor disorder-the motor variant of functional neurologi
116                        In all three types of motor disorders, there was a uniform and strong decrease
117                           For scholastic and motor disorders, there were no differences between offsp
118 TBE) is commonly used to evaluate esophageal motor disorders, though its accuracy is limited.
119 nd is involved in the development of enteric motor disorders through an increase in tachykininergic a
120 llectual disabilities, and communication and motor disorders usually emerge early in life and are ass
121 Previous studies have suggested this gastric motor disorder was a consequence of an enteric neuropath
122                                An esophageal motor disorder was diagnosed only by 24-hour ambulatory
123 ve approach to alleviate intractable colonic motor disorders, whose effectiveness can be evaluated th
124 s-of-function mutations in Na(v)1.6 underlie motor disorders, with homozygous-null mutations causing
125 ients clinically categorized with functional motor disorder without fibromyalgia, 35 with both functi

 
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