ライフサイエンスコーパス: motor seizure

1 re, locomotion, and susceptibility to limbic motor seizures.

2 physiological, and behavioral adaptations to motor seizures.

3 which protects against focally evoked limbic motor seizures.

4 neuronal circuit active during frontal lobe motor seizures.

5 one of the SwHi rats, had spontaneous limbic motor seizures 5 weeks following pilocarpine-induced sta

6 ypical group (4 cases presenting with simple motor seizures and a tendency for more frequent generali

7 contralateral hemisphere following the first motor seizure, and the pattern of its expression reflect

8 nly a few seizures, 1 had more than 30 focal motor seizures, and 1 had 4 witnessed generalized tonic-

9 e neurological disorder resulting in ataxia, motor seizures, and behavioral absence seizures resembli

10 cognised because they usually present as non-motor seizures, and can overlap with other symptoms of t

11 amocortical oscillations amplify focal onset motor seizures, and corpus callosum spreads them bilater

12 tage change in the mean monthly frequency of motor seizures at 12 weeks.

13 thogenic CDKL5 variant and at least 16 major motor seizures (defined as bilateral tonic, generalised

14 (200 pmol) into STN protected against limbic motor seizures evoked either by intravenous bicuculline

15 substantia nigra, protected rats from limbic motor seizures evoked focally from area tempestas, an ep

16 ttent EEGs with behavioural observations for motor seizures failed to demonstrate spontaneous seizure

17 rats exhibited a decreased latency to limbic motor seizures following acute pilocarpine administratio

18 was percentage change in median 28-day major motor seizure frequency from the baseline period to the

19 s a median percentage change in 28-day major motor seizure frequency of -30.7% (IQR -49.5 to -1.9) in

20 Limbic motor seizures in animals, analogous to complex partial

21 imol (a GABAA agonist) and susceptibility to motor seizures in rats.

22 directly related to the suppression of tonic motor seizures in the electroshock model of epilepsy.

23 nctional role of MD in focally evoked limbic motor seizures in the rat.

24 was significantly longer in the generalized motor seizures of the SUDEP group (p < 0.001).

25 us, and substantia nigra during frontal lobe motor seizures on the cellular level, revealing a comple

26 ater, after displaying spontaneous recurrent motor seizures, their dentate gyri were examined.

27 d ECS treatment caused progressively shorter motor seizures (tolerance) in both rats and wild-type mi

28 The median monthly frequency of motor seizures was 30.0 (IQR 11.0-96.0) at baseline and

29 The median reduction in monthly motor seizures was 36.5% (IQR 0-64.7).

30 ons of the percentage change in frequency of motor seizures were done with a Mann-Whitney U test.

31 ilepticus (n = 25), including 15 with subtle motor seizures, were more likely to die than those with

32 e day prior to his admission, he had a focal motor seizure with rotation of the head and eyes to the