ライフサイエンスコーパス: mouse cytomegalovirus

1 ccinia virus, but dispensable in the case of mouse cytomegalovirus.

2 Ds prevented necroptosis upon infection with mouse cytomegalovirus.

3 UTX deficiency showed increased lethality to mouse cytomegalovirus.

4 ntiation, we have analyzed an infection with mouse cytomegalovirus, a persistent-latent virus that el

5 both profound susceptibility to infection by mouse cytomegalovirus and approximately 20,000-fold sens

6 f LPS, AIM2 activators Francisella novicida, mouse cytomegalovirus and DNA, and the infectious agents

7 o Francisella tularensis, vaccinia virus and mouse cytomegalovirus and had a partial role in the sens

8 o caused hypersusceptibility to infection by mouse cytomegalovirus and other microbes.

9 similar to that of the miRNAs described for mouse cytomegalovirus, but they do not share any substan

10 pDC activation states in animals infected by mouse cytomegalovirus by combining Ifnb1 reporter mice w

11 es simplex virus 1 (HSV-1), HSV-2, HCMV, and mouse cytomegalovirus, by 30- to 700-fold, depending on

12 vely replicate in human cells and found that mouse cytomegalovirus can produce infectious particles a

13 This finding demonstrates that mouse cytomegalovirus can undergo all processes of its l

14 We reexamined the dogma that mouse cytomegalovirus cannot productively replicate in h

15 During mouse cytomegalovirus (CMV) infection, a population of L

16 importance for detecting cells infected with mouse cytomegalovirus (CMV) via recognition of the viral

17 ions, we challenged old mice carrying latent mouse cytomegalovirus (CMV), herpes simplex virus 1 (HSV

18 We observed that the mouse cytomegalovirus encoded protein m18 is necessary a

19 The mouse cytomegalovirus encodes an MHC-like protein, m157,

20 effects of LPS, and are hypersusceptible to mouse cytomegalovirus, failing to produce type I interfe

21 s as a transgene vector, manipulation of the mouse cytomegalovirus genome to allow limited spread to

22 Tlr9(CpG1) allele are highly susceptible to mouse cytomegalovirus infection and show impaired infect

23 ither pathway offers full protection against mouse cytomegalovirus infection in the absence of the ot

24 ain mediators of NK cell-mediated control of mouse cytomegalovirus infection in vivo.

25 s replication, were dependent on AIM2 during mouse cytomegalovirus infection in vivo.

26 hermore, Ly49H(+) NK cells that responded to mouse cytomegalovirus infection primarily developed from

27 Mouse cytomegalovirus infection triggers NK receptor-ind

28 fect NK cell function in vivo in response to mouse cytomegalovirus infection.

29 ls are defective in protecting the host from mouse cytomegalovirus infection.

30 lso observed after Listeria monocytogenes or mouse cytomegalovirus infection.

31 or the expansion of Ly49H(+) NK cells during mouse cytomegalovirus infection.

32 ls to coordinate the NK cell response during mouse cytomegalovirus infection.

33 tion and calcium signaling in the context of mouse cytomegalovirus infection.

34 ctor NK cell (NK(eff)) populations following mouse cytomegalovirus infection.

35 The mouse cytomegalovirus M33 protein is highly homologous t

36 The mouse cytomegalovirus major immediate-early (IE) transcr

37 By screening a panel of deletion mutants of mouse cytomegalovirus (MCMV) a mutant was identified tha

38 infection by the double-stranded DNA viruses mouse cytomegalovirus (MCMV) and human adenovirus.

39 nce of the major immediate-early gene ie3 of mouse cytomegalovirus (MCMV) and that of the correspondi

40 Mouse cytomegalovirus (MCMV) and Toll-like receptor-medi

41 ing Ly49H, an activating NK receptor for the mouse cytomegalovirus (MCMV) antigen m157, show enhanced

42 ies specificity and similar tropisms suggest mouse cytomegalovirus (mCMV) as a potential vector for t

43 Using infection of newborn mice with mouse cytomegalovirus (MCMV) as a reliable model that re

44 daptive and innate immune cells critical for mouse cytomegalovirus (MCMV) clearance.

45 d cytolytic activity, and mice infected with mouse cytomegalovirus (MCMV) displayed elevated titers i

46 Mouse cytomegalovirus (MCMV) encodes an MHC-like protein

47 Mouse cytomegalovirus (MCMV) encodes two potential seven

48 ne, we selectively deleted 32 genes from the mouse cytomegalovirus (MCMV) genome.

49 The mouse cytomegalovirus (MCMV) immediate early 3 protein (

50 Transcription of mouse cytomegalovirus (MCMV) immediate early ie1 and ie3

51 We investigated the mouse cytomegalovirus (MCMV) immediate-early 1 protein (

52 49D receptor impacts the NK cell response to mouse cytomegalovirus (MCMV) infection by comparing the

53 is up-regulated in activated NK cells during mouse cytomegalovirus (MCMV) infection in response to si

54 Mouse cytomegalovirus (MCMV) infection promotes a rapid

55 Early during mouse cytomegalovirus (MCMV) infection, NK cells up-regu

56 During mouse cytomegalovirus (MCMV) infection, the first wave o

57 investigated how NOD NK cells respond after mouse cytomegalovirus (MCMV) infection, using NOD mice c

58 -18 stimulation and by mouse NK cells during mouse cytomegalovirus (MCMV) infection.

59 in expansion and memory cell formation after mouse cytomegalovirus (MCMV) infection.

60 oradrenaline, in modulating host response to mouse cytomegalovirus (MCMV) infection.

61 ygotes displayed increased susceptibility to mouse cytomegalovirus (MCMV) infection.

62 sion to become long-lived memory cells after mouse cytomegalovirus (MCMV) infection; therefore, we ex

63 ller (NK) cell receptor Ly49H recognizes the mouse cytomegalovirus (MCMV) m157 glycoprotein expressed

64 ng NK cells in Nfil3(-/-) mice infected with mouse cytomegalovirus (MCMV) or recombinant viruses expr

65 ereas engagement of the AIM2 inflammasome by mouse cytomegalovirus (MCMV) or transfected double-stran

66 Mouse cytomegalovirus (MCMV) susceptibility often result

67 scovered the first two epitopes derived from mouse cytomegalovirus (MCMV) that are recognized by CD4

68 Mouse cytomegalovirus (MCMV), a beta-herpesvirus that es

69 focuses on genes required for resistance to mouse cytomegalovirus (MCMV), as identified through unbi

70 rs, including the Ly49H molecule recognizing mouse cytomegalovirus (MCMV), can stimulate NK cell expa

71 In infections with mouse cytomegalovirus (MCMV), MCMV-specific NK cells und

72 Ly49H binds with high affinity to a mouse cytomegalovirus (MCMV)-encoded glycoprotein, m157,

73 t have arisen from these recent studies: (i) mouse cytomegalovirus (MCMV)-induced memory; (ii) cytoki

74 Currently, no mouse cytomegalovirus (MCMV)-specific CD4 T cell respons

75 interleukin-12 (IL-12) is indispensible for mouse cytomegalovirus (MCMV)-specific NK cell expansion

76 V-7, than to another murine betaherpesvirus, mouse cytomegalovirus (MCMV).

77 isted after the resolution of infection with mouse cytomegalovirus (MCMV).

78 While the possibility that mouse cytomegalovirus might replicate in human cells rai