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1 o now of the epidemiology of hand, foot, and mouth disease.
2 ction are usually limited, such as hand-foot-mouth disease.
3 ions in the event of an outbreak of foot-and-mouth disease.
4 pment as effective vaccines against foot-and-mouth disease.
5 h CNS involvement and 29 with skin, eye, and mouth disease.
6 clinical data presented with hand, foot, and mouth disease.
7 spreading the much more contagious foot and mouth disease.
8 ere respiratory complications, and hand-foot-mouth disease.
9 apan that are known to cause hand, foot, and mouth disease.
10 ant animal pathogen responsible for foot-and-mouth disease.
11 4% (95% CI: 16.63 to 16.66) against foot and mouth disease, 33.80% (33.43 to 34.38) against lumpy ski
12 1 (EV71) is a common cause of hand, foot and mouth disease-a disease endemic especially in the Asia-P
13 es to cause large outbreaks of hand foot and mouth disease across Asia, associated with neurological
14 DSLINE database was searched using the term "mouth diseases." Additional references were identified f
15 ential route of vaccination against foot-and-mouth disease and may be useful for eliciting protection
17 ng pathogens associated with hand, foot, and mouth disease and pediatric respiratory disease worldwid
19 is the major cause of severe hand, foot, and mouth disease and viral encephalitis in children across
20 sease), two datasets of serotype A (Foot-and-Mouth disease) and two datasets of influenza when the sc
21 as frequently associated with hand, foot and mouth disease, and EV-D68 with respiratory infections.
22 enteroviruses responsible for hand, foot and mouth disease, and plays a key role in cell entry(2).
23 7,200,092 probable cases of hand, foot, and mouth disease (annual incidence, 1.2 per 1000 person-yea
24 cts children, manifesting as hand, foot, and mouth disease, aseptic meningitis, poliomyelitis-like ac
25 the largest receptor-group of hand-foot-and-mouth disease causing viruses, which includes CV-A10.
26 ve samples) from suspected cases of foot-and-mouth disease collected from 65 countries between 1965 a
28 present an analysis of the current foot-and-mouth disease epidemic in Great Britain over the first 2
33 tainty as management proceeds, with foot-and-mouth disease (FMD) culling and measles vaccination as c
35 e the occurrence of transmission of foot-and-mouth disease (FMD) during the incubation phase amongst
39 fox-hunting during the outbreak of foot-and-mouth disease (FMD) in 2001 to examine this issue and fo
40 e show that, during the outbreak of foot and mouth disease (FMD) in 2001, there was a significant red
41 essing IFNs can effectively control foot-and-mouth disease (FMD) in cattle and swine during experimen
43 In 1997, a devastating outbreak of foot-and-mouth disease (FMD) in Taiwan was caused by a serotype O
56 table vaccine candidates.IMPORTANCE Foot-and-mouth disease (FMD) is the most devastating disease affe
58 of infection with, or exposure to, foot-and-mouth disease (FMD) over the same time period using reco
64 ase virus (FMDV) is responsible for foot-and-mouth disease (FMD), an important disease of farmed anim
65 spongiform encephalopathy (BSE) and foot-and-mouth disease (FMD), and the advent of new technologies,
67 ith FMDV developed typical signs of foot-and-mouth disease (FMD), including fever, vesicular lesions,
68 ase of notifiable diseases, such as foot-and-mouth disease (FMD), these analyses provide important in
69 ase virus (FMDV) is the pathogen of foot-and-mouth disease (FMD), which is a highly contagious diseas
72 e major etiological agents of hand, foot and mouth disease (HFMD) and are often associated with neuro
73 of a 31-year-old patient with Hand, Foot and Mouth Disease (HFMD) and concurrent acute monocular macu
74 an emerging pathogen causing hand, foot, and mouth disease (HFMD) and fatal neurological diseases in
75 (EV-A71) is a major cause of hand, foot, and mouth disease (HFMD) and is particularly prevalent in pa
76 mily and are major causes of hand, foot, and mouth disease (HFMD) and pediatric respiratory disease w
77 rus 71 (EV71) and associated hand, foot, and mouth disease (HFMD) are recognized as emerging public h
78 Epidemiology and etiology of hand, foot, and mouth disease (HFMD) based on large sample size or evalu
79 million/11.3 million) of all hand, foot, and mouth disease (HFMD) cases reported to WHO during 2010-2
82 rovirus A71 (EV-A71)-related hand, foot, and mouth disease (HFMD) imposes a substantial clinical burd
83 is a major pathogen for the hand, foot, and mouth disease (HFMD) in children and can also lead to AF
84 To monitor search trends on Hand, Foot and Mouth Disease (HFMD) in Guangdong Province, China, we te
91 ework to simulate and optimize hand foot and mouth disease (HFMD) surveillance in a high-burden regio
92 ary causes of the epidemics of hand-foot-and-mouth disease (HFMD) that affect more than a million chi
94 aviridae), a common cause of hand, foot, and mouth disease (HFMD), may also cause severe neurological
95 rus that causes outbreaks of hand, foot, and mouth disease (HFMD), primarily in the Asia-Pacific area
96 1) causes large outbreaks of hand, foot, and mouth disease (HFMD), with severe neurological complicat
101 y emerged as a major cause of hand, foot and mouth disease in children worldwide but no vaccine is av
103 cterised the epidemiology of hand, foot, and mouth disease in China on the basis of enhanced surveill
106 k is used to analyse an outbreak of foot-and-mouth disease in the UK, enhancing current understanding
108 ms (in 1197 [17%] patients), hand, foot, and mouth disease (in 528 [7% patients), and myocarditis (in
109 rus infection and in the control of foot-and-mouth disease infection highlight the problems caused by
110 and animal (bovine brucellosis and foot-and-mouth disease) infections clearly differentiating infect
111 924, 1.939]; p < 0.001), and hand, foot, and mouth disease (IRR: 2.501, 95% CI [2.491, 2.510]; p < 0.
116 irus (FMDV), the causative agent of foot-and-mouth disease, is an Aphthovirus within the Picornavirid
117 irus (FMDV), the causative agent of foot-and-mouth disease, is an Apthovirus within the Picornavirida
118 irus), jump dispersal on a network (foot-and-mouth disease), or a combination of these (Sudden oak de
119 del run for the 2001 United Kingdom foot and mouth disease outbreak and compare the efficacy of diffe
123 tio-temporal model of the spread of foot-and-mouth disease, parameterized to match the 2001 UK outbre
125 contagious livestock viral disease, foot-and-mouth disease poses a great threat to the beef-cattle in
126 respiratory diseases, such as hand, foot and mouth disease, rebounded substantially towards the end o
127 aboratory data from cases of hand, foot, and mouth disease reported to the Chinese Center for Disease
130 problem using the specific case of foot-and-mouth disease spreading between farms using the formulat
131 he severe, atypical cases of hand, foot, and mouth disease that have been reported worldwide since 20
132 71 is a picornavirus causing hand, foot, and mouth disease that may progress to fatal encephalitis in
133 e performed experimental studies of foot-and-mouth disease transmission in cattle and estimated this
134 results on three datasets of SAT2 (Foot-and-Mouth disease), two datasets of serotype A (Foot-and-Mou
139 retion are induced by expression of foot-and-mouth disease virus (FMDV) 3C(pro) and that this require
142 a from transmission experiments for foot-and-mouth disease virus (FMDV) and African swine fever virus
143 sted in this study: one recognizing foot-and-mouth disease virus (FMDV) and another recognizing the 1
144 encephalomyocarditis virus (EMCV), foot-and-mouth disease virus (FMDV) and other picornaviruses comp
146 t to which the genetic diversity of foot-and-mouth disease virus (FMDV) arising over the course of in
148 t the leader proteinase (L(pro)) of foot-and-mouth disease virus (FMDV) blocks cap-dependent mRNA tra
151 tabilizing SAT2 vaccines.IMPORTANCE Foot-and-mouth disease virus (FMDV) causes a highly contagious ac
156 rains; however, the pathogenesis of foot-and-mouth disease virus (FMDV) coinfections is largely unkno
159 ifferential laboratory detection of foot-and-mouth disease virus (FMDV) from viruses that cause clini
166 ith clearance versus persistence of foot-and-mouth disease virus (FMDV) in micro-dissected compartmen
170 Here, we show that the picornavirus foot-and-mouth disease virus (FMDV) induces the formation of auto
171 lymphocyte subsets in recovery from foot-and-mouth disease virus (FMDV) infection in calves was inves
172 The pathogenesis of persistent foot-and-mouth disease virus (FMDV) infection was investigated in
174 investigation, the manner in which foot-and-mouth disease virus (FMDV) interacts with the innate and
175 t the leader proteinase (L(pro)) of foot-and-mouth disease virus (FMDV) interferes with the innate im
176 slation initiation dependent on the foot-and-mouth disease virus (FMDV) internal ribosome entry site
177 ors were constructed containing the foot-and-mouth disease virus (FMDV) internal ribosome entry site
179 ction in African buffalo.IMPORTANCE Foot-and-mouth disease virus (FMDV) is a highly contagious virus
181 esponse.IMPORTANCE The picornavirus foot-and-mouth disease virus (FMDV) is a notorious animal pathoge
183 ersity in endemic regions.IMPORTANCEFoot-and-mouth disease virus (FMDV) is a pathogen of domestic liv
186 he final steps in the maturation of foot-and-mouth disease virus (FMDV) is cleavage of the VP0 protei
187 reviously shown that replication of foot-and-mouth disease virus (FMDV) is highly sensitive to alpha/
189 The leader proteinase (L(pro)) of foot-and-mouth disease virus (FMDV) is involved in antagonizing t
196 animals with chemically inactivated foot-and-mouth disease virus (FMDV) is widely practiced to contro
198 an aromatic hydrophobic residue in foot-and-mouth disease virus (FMDV) leader proteinase (Lpro) (W10
202 feron [IFN-alpha/beta]) can inhibit foot-and-mouth disease virus (FMDV) replication in cell culture,
203 Ns have proven effective to inhibit foot-and-mouth disease virus (FMDV) replication in swine, a simil
204 rative analysis, of 103 isolates of foot-and-mouth disease virus (FMDV) representing all seven seroty
208 Adsorption and plaque formation of foot-and-mouth disease virus (FMDV) serotype A12 are inhibited by
209 human rhinovirus type 2 (HRV2) and foot-and-mouth disease virus (FMDV) to control the translation of
210 ly demonstrated that the ability of foot-and-mouth disease virus (FMDV) to form plaques in cell cultu
211 Adaptation of field isolates of foot-and-mouth disease virus (FMDV) to grow in cells in culture c
213 netic and epidemiological data in a Foot and Mouth Disease Virus (FMDV) veterinary outbreak in Englan
215 velopment of a serological test for foot-and-mouth disease virus (FMDV) which is quick and easy to us
218 The viruses in question spanned foot and mouth disease virus (FMDV), African swine fever virus (A
219 rin receptors on cultured cells for foot-and-mouth disease virus (FMDV), and high-efficiency utilizat
221 for several diverse species such as foot-and-mouth disease virus (FMDV), hemagglutinin (HA) and neura
228 that the key replication enzyme of foot-and-mouth disease virus (FMDV), the RNA-dependent RNA polyme
229 esentatives of several serotypes of foot-and-mouth disease virus (FMDV), we discovered a putative cre
230 We have previously reported that Foot-and-mouth disease virus (FMDV), which is virulent for cattle
231 were used to map antigenic sites on foot-and-mouth disease virus (FMDV), which resulted in the identi
232 documented as an essential ITAF for foot-and-mouth disease virus (FMDV), with no apparent role in cel
246 y the method to two UK epidemics of Foot-and-Mouth Disease Virus (FMDV): the 2007 outbreak, and a sub
248 ike other viruses, the picornavirus foot-and-mouth disease virus (FMDV; genus Aphthovirus), one of th
249 (n=3 each, 18.8%); Brucella spp and foot and mouth disease virus (n=2 each, 12.5%); and variola virus
250 ntibiotic resistance gene, with the foot and mouth disease virus 2A self-cleaving sequence placed bet
251 binding and explains the ability of foot-and-mouth disease virus 3C(pro) to cleave sequences containi
252 The X-ray crystal structure of the foot-and-mouth disease virus 3C(pro), mutated to replace the cata
254 he gene encoding the 2A protease of foot-and-mouth disease virus and then inserted in frame between t
258 nction derived from domain 3 of the foot-and-mouth disease virus internal ribosome entry site (IRES);
259 x 10(7) c.f.u./ml, indicating that foot-and-mouth disease virus IRES provides high-titer bicistronic
261 with a multiple cloning site 3' to foot-and-mouth disease virus IRES, was used to construct vectors
262 1) British field strain serotype of foot-and-mouth disease virus is a high-affinity ligand for alpha
265 se of core catalytic domains of the foot-and-mouth disease virus leader protease and coronavirus PLPs
266 hese methods to analyze data from a foot-and-mouth disease virus outbreak in the United Kingdom in 20
268 predict the antigenic similarity in foot-and-mouth disease virus strains and in influenza strains, wh
269 s a 20-mer peptide derived from the foot-and-mouth disease virus that exhibits nanomolar and selectiv
270 e genome-linked protein, VPg wheras foot-and-mouth disease virus uniquely encodes three copies of VPg
271 te responses against infection with foot-and-mouth disease virus was analyzed on consecutive 5 d foll
272 (e.g., poliovirus, rhinovirus, and foot-and-mouth disease virus), the capsid precursor protein VP0 i
274 ed a salient genome segmentation of foot-and-mouth disease virus, an important animal pathogen whose
275 has been documented for poliovirus, foot-and-mouth disease virus, and coxsackievirus B3 and can lead
277 us family, including poliovirus and foot-and-mouth disease virus, are widespread pathogens of humans
278 QVLAQKVART (A20FMDV2), derived from foot-and-mouth disease virus, as a potent inhibitor of alphavbeta
279 ovirus, coxsackievirus, poliovirus, foot-and-mouth disease virus, enterovirus D-68, and a wide range
281 nent G-H loop of the VP1 protein of foot-and-mouth disease virus, raised substantial levels of antipe
282 ture of the corresponding domain of foot-and-mouth disease virus, revealing an analogous domain organ
283 larger picornavirus IRESs (those of foot-and-mouth disease virus, rhinovirus, encephalomyocarditis vi
284 s, including all seven serotypes of foot-and-mouth disease virus, two serotypes of vesicular stomatit
292 ogens: classical swine fever virus; foot-and-mouth disease virus; vesicular stomatitis virus, New Jer
294 affinity ligands of alpha v beta6 (foot-and-mouth-disease virus, latency associated peptide), have a
295 s-neutralization between serotype O foot-and-mouth disease viruses (FMDVs) is critical for guiding va
296 investigated how highly contagious foot-and-mouth disease viruses persist in the African buffalo, wh
299 ols during a nationwide epidemic of foot and mouth disease, which substantially delayed removal of TB
300 ent large-scale outbreaks of hand, foot, and mouth disease worldwide and represent a major etiologica