ライフサイエンスコーパス: mucous

1 acuoles in the macrophages of the intestinal mucous.

2 has been measured in hypoxic cystic fibrosis mucous.

3 morphogenesis, and a lack of differentiated mucous acinar cells in submandibular and sublingual glan

4 f IRF6 in the developing ductal, serous, and mucous acinar cells of salivary glands.

5 n of prolonged diarrhea containing blood and mucous after recovery from an acute phase of myositis pr

6 cies underwent mass mortality, formed large, mucous aggregates, and sank out en masse (carbon sinkers

7 a serum-free medium that induces endocrine, mucous and absorptive lineage commitment by HRA-19 cells

8 ay 15.5-18.5), epithelial differentiation to mucous and chief cell lineages was rudimentary, with no

9 ed differentiation, with inhibition inducing mucous and endocrine cell differentiation while activati

10 ls and gland thread cells, which produce the mucous and fibrous components of the slime, respectively

11 copy to obtain force maps of human preocular mucous and purified ocular mucins by probing and locatin

12 es, mammary ducts, and glands such as sweat, mucous and sebaceous glands, are initiated in developmen

13 anism against pathogens that complements the mucous and secretory IgA Ab-mediated system in the prote

14 an normal (p = 0.031), but the proportion of mucous and serous cells in CF glands was normal.

15 variant clones induced pathology akin to the mucous and squamous metaplasia, neutrophilic inflammatio

16 echanisms involved in the deployment of both mucous and thread cells during the transition from thick

17 ent stem cells that give rise to absorptive, mucous, and endocrine lineages.

18 genesis and how Giardia's disruptions of the mucous barrier facilitate bacterial translocation that m

19 e interplay between diet, microbiome and the mucous barrier in these diseases has gained significant

20 The mucous barrier of our digestive tract is the first line

21 Diet modulates the mucous barrier via alterations in gut microbiota, result

22 print associated with the composition of the mucous barrier, which is further influenced by host-micr

23 ents and micronutrients), gut microbiota and mucous barriers (gastrointestinal and respiratory tract)

24 responses are often manifested as increased mucous cell density (mucous cell metaplasia) associated

25 utosomal recessive mutation, sld, attenuates mucous cell expression in murine sublingual glands with

26 Mucous cell hyperplasia and airway smooth muscle (ASM) h

27 educes allergen- and cigarette smoke-induced mucous cell hyperplasia in mice and in differentiated pr

28 n the lower airways of patients with asthma, mucous cell hyperplasia is shown to stem from a novel mu

29 l resolution process of inflammation-induced mucous cell hyperplasia may lead to sustained mucous hyp

30 , and during prolonged exposure to allergen, mucous cell hyperplasia remained elevated in STAT1(-/-)

31 d significantly less pulmonary inflammation, mucous cell hyperplasia, and eosinophilia compared with

32 osure of mice to allergen, IFN-gamma reduces mucous cell hyperplasia, but the signaling responsible f

33 Features of allergic airway disease such as mucous cell hyperplasia, infiltration of airway eosinoph

34 AECs and the resolution of allergen-induced mucous cell hyperplasia.

35 xyuridine, and for expression of the surface mucous cell marker Ulex europaeus.

36 to regulate epithelial responses, including mucous cell metaplasia (MCM), but their roles in mucoinf

37 endai virus triggered a switch to persistent mucous cell metaplasia and airway hyperreactivity after

38 ff and immunohistochemical staining revealed mucous cell metaplasia and muc5AC expression in RV1B- bu

39 or CLCA family members in the development of mucous cell metaplasia and possibly airway hyperreactivi

40 Indeed, mucous cell metaplasia is considered the critical preneo

41 uction on day 4 postinfection and subsequent mucous cell metaplasia on day 21 postinfection.

42 ute inflammatory response and the subsequent mucous cell metaplasia that accompanies the asthma pheno

43 Cell proliferation and mucous cell metaplasia were detected in newborn, but not

44 manifested as increased mucous cell density (mucous cell metaplasia) associated with mucus obstructio

45 nificant increase in pulmonary inflammation, mucous cell metaplasia, airway hyperresponsiveness, and

46 3, dramatically blocked the allergen-induced mucous cell metaplasia, airway mucus formation, and the

47 tokines, C-C chemokines, IgE production, and mucous cell metaplasia, as compared with the adoptive tr

48 indicates that at least one of these traits, mucous cell metaplasia, depends on members of the calciu

49 onse that recruits CD4(+) T cells and drives mucous cell metaplasia, thus linking antiviral responses

50 Bcl-2 sustains mucous cell metaplasia, whereas p53(-/-) mice display ch

51 w enhanced expression of SPDEF and Bcl-2 and mucous cell metaplasia.

52 tokines, C-C chemokines, IgE production, and mucous cell metaplasia.

53 nd that FceRIa(-/-) mice no longer developed mucous cell metaplasia.

54 events, whereas blockade of CCL28 inhibited mucous cell metaplasia.

55 display enhanced airway hyperreactivity and mucous cell metaplasia.

56 sensitization, TH2 cytokine production, and mucous cell metaplasia.

57 ng phagocyte recruitment, IgA secretion, and mucous cell metaplasia.

58 13, and alpha7-nAChRs are critical in airway mucous cell metaplasia/hyperplasia and mucus production

59 th loss of acid-secreting parietal cells and mucous cell metaplasias.

60 ealed edema within the submucosa and altered mucous cell morphology.

61 ated with Notch agonists displayed increased mucous cell numbers and decreased ciliated cell numbers.

62 ia, in which there are dramatic increases in mucous cell numbers.

63 tch misexpression resulted in an increase in mucous cells and a decrease in ciliated cells in the air

64 ecretory products from two cell types, gland mucous cells and gland thread cells, which produce the m

65 demonstrate MUC19 glycoproteins in glandular mucous cells and saliva.

66 nd-organ alterations (particularly in airway mucous cells and smooth muscle) as critical steps leadin

67 nd-organ alterations (particularly in airway mucous cells and smooth muscle) as critical steps leadin

68 Accordingly, more mucous cells are present in primary human airway culture

69 Normally, airway mucous cells are rare.

70 combination of no ciliated cells and excess mucous cells can account for the chronic rhinitis and in

71 ume, but serous cell transdifferentiation to mucous cells does not occur, nor are gland acini inflame

72 restricted localization to subpopulations of mucous cells in sld mice during postnatal gland developm

73 R, results in selective expansion of surface mucous cells in the body and fundus of the stomach.

74 Mucous cells of MECs were positive for CK7, CEA, as well

75 In addition, ER Bax was detected in mucous cells of STAT1(+/+) but not STAT1(-/-) airways of

76 Primary canine parietal or mucous cells were treated with IL-1beta.

77 ng LGR5-expressing cells, surface and antral mucous cells, and a diversity of gastric endocrine cells

78 were differentially expressed, in serous and mucous cells, respectively.

79 ehog (Ihh) and hyperproliferation of surface mucous cells.

80 p53(-/-) mice display chronically increased mucous cells.

81 ent accumulation near the luminal borders of mucous cells.

82 on, displayed little or no redistribution in mucous cells.

83 t suppress Shh expression in primary gastric mucous cells.

84 ression patterns distinguished tumor goblet (mucous) cells from nontumorigenic airway and intestinal

85 ll hyperplasia is shown to stem from a novel mucous ciliated cell state, as well as goblet cell hyper

86 usea, vomiting, discomfort, and even gastric mucous damage.

87 n the disc consisted of cellular changes and mucous degeneration.

88 he cysteine protease inhibitor E64 prevented mucous degradation, mucin depletion, and the increase in

89 008 to April 2010, episodes of hematochezia, mucous diarrhea and epigastric pain intermittently manif

90 early biological effects of RA during normal mucous differentiation in NHTBE cells.

91 taining Ets-like factor (SPDEF), a driver of mucous differentiation.

92 igns of NLDO (epiphora, increased tear lake, mucous discharge) in both eyes on masked examination at

93 symptoms (photophobia, tearing, itching, and mucous discharge), and quality of life (QoL) assessed by

94 d to assess the effect of decreased PTCH1 on mucous expression and airway epithelial phenotypes.

95 Ptch1(+/-) mice had reduced mucous expression compared to wildtype mice following mu

96 PTCH1 knockdown attenuated wound closure and mucous expression in airway epithelial cell lines.

97 CH1 siRNA knockdown on epithelial repair and mucous expression were evaluated using human epithelial

98 in COPD airway epithelium and may upregulate mucous expression.

99 l surface that helps them evade predation by mucous filter feeders.

100 only partially exposed at the surface of the mucous gel.

101 to an exogenous probe in isolated mucins and mucous gels.

102 A and protein are expressed predominantly in mucous gland cells of the antrum and in mucous neck cell

103 s required for terminal maturation of antral mucous gland cells to protect animals from gastric infla

104 Spdef impaired terminal maturation of antral mucous gland cells, as reflected in reduced expression o

105 earch groups conclude that changes in airway mucous gland function in CFTR-deficient animals of these

106 have approximately the same number of airway mucous glands as humans.

107 TTX was detected in mucous glands in both populations, contradicting the gen

108 and exocrine secretion of mucins by salivary mucous glands is regulated predominantly by parasympathe

109 LDA) was isolated from extracts derived from mucous glands that coat the dart before it is stabbed th

110 es abnormally viscous secretions from airway mucous glands that may be a major factor in CF pathology

111 Mouse airways have few mucous glands, and the mouse model of CF exhibits no sig

112 Cases of cutaneous and mucous hyperpigmentations during interferon alfa treatme

113 triction leading to airflow obstruction, and mucous hypersecretion are clinical hallmarks of asthma.

114 Goblet cell hyperplasia and mucous hypersecretion contribute to the pathogenesis of

115 ucous cell hyperplasia may lead to sustained mucous hypersecretion in chronic diseases.

116 Mucous hypersecretion is an important feature of obstruc

117 n lymphocytes, eosinophils, IL-13, IL-5, and mucous hypersecretion to wild-type levels, whereas eotax

118 ralizing antibody attenuated ILC2 expansion, mucous hypersecretion, and airways responsiveness.

119 ulmonary abnormalities of COPD/CF, including mucous hypersecretion, inflammatory and emphysematous ph

120 ner et al. show, in a mouse model of chronic mucous hypersecretion, that ciliated epithelial cell apo

121 the PRD of p53 as a determinant for chronic mucous hypersecretion.

122 mice, as were airway hyperresponsiveness and mucous hypersecretion.

123 l in airway diseases associated with chronic mucous hypersecretion.Bcl-2 interacting killer (Bik) dec

124 GFR ligands and IL-13 are known to stimulate mucous induction, but the detailed mechanisms of epithel

125 pression compared to wildtype mice following mucous induction.

126 ce infected with G. duodenalis had a thinner mucous layer and demonstrated differential Muc2 and Muc5

127 vD2 likely assists in the maintenance of the mucous layer of the tear film to sustain ocular surface

128 The intestinal mucous layer provides a critical host defense against pa

129 abitat for the commensal flora and the inner mucous layer that protects the epithelial cells by being

130 networks that in colon build the loose outer mucous layer that provides the habitat for the commensal

131 coadhesion of microparticles was tested on a mucous layer under shear stress, mimicking the human bli

132 ajor protein present in the small intestinal mucous layer, and that removal of this barrier in vitro

133 ls, between G. duodenalis and the intestinal mucous layer.

134 ella propel bacteria through urine and along mucous layers during ascension to the upper urinary trac

135 rmatologist when there were cutaneous and/or mucous lesions.

136 We show that endocrine and mucous lineage commitment is blocked in the presence of

137 9 cells significantly enhanced endocrine and mucous lineage commitment, while cells expressing the no

138 d negligible ability for either endocrine or mucous lineage commitment.

139 gnosis, most patients had other cutaneous or mucous localizations of cGVHD.

140 on, especially in case of other cutaneous or mucous localizations of cGVHD.

141 cal findings such as petechiae, bruising and mucous membrane bleeding, examination of peripheral bloo

142 ensitivity and UV radiation-induced skin and mucous membrane cancers.

143 entification of Candida species from skin or mucous membrane cultures, and/or by culture or staining

144 actions characterized by widespread skin and mucous membrane detachments, including bronchial mucosa,

145 ugs have been reported to induce skin and/or mucous membrane discoloration.

146 efinitive management (n = 342) that included mucous membrane grafting (MMG), prosthetic replacement o

147 servatively, but 2 eyes required free labial-mucous membrane grafting for persistent corneal exposure

148 ally alleviated with autologous serum drops, mucous membrane grafting to replace scarred tarsal conju

149 Free labial-mucous membrane grafting was performed in all cases of p

150 t secretion of components of the peritrophic/mucous membrane in the larval digestive tract.

151 Formaldehyde, a known human carcinogen and mucous membrane irritant, is emitted from a variety of b

152 rom adhesion of Trichomonas vaginalis to the mucous membrane of the urethra or vagina.

153 fibrosis in the immunobullous disease ocular mucous membrane pemphigoid (MMP) causes blindness; the p

154 Mucous membrane pemphigoid (MMP) describes an autoimmune

155 dity of the First International Consensus on Mucous Membrane Pemphigoid (MMP) guidance, which recomme

156 Mucous membrane pemphigoid (MMP) is a heterogeneous grou

157 A rare variant of mucous membrane pemphigoid (MMP) is characterized by cir

158 ancements in the diagnosis and management of mucous membrane pemphigoid (MMP) published in the Englis

159 A case of anti-laminin-332 mucous membrane pemphigoid (MMP) that developed shortly

160 ial pemphigoid (OCP), also labeled as ocular mucous membrane pemphigoid (MMP).

161 ) in patients with clinically typical ocular mucous membrane pemphigoid (MMP).

162 Ocular mucous membrane pemphigoid (OMMP) is an autoimmune disea

163 he immunosuppressive drugs used for treating mucous membrane pemphigoid and certain definitive catego

164 teers and 40 patients with other variants of mucous membrane pemphigoid and mAb GoH3 and BQ16 to inte

165 in we present a case of treatment-refractory mucous membrane pemphigoid and propose a mechanism to ex

166 Successful entropion repair in patients with mucous membrane pemphigoid can be achieved if control of

167 phigoid and certain definitive categories of mucous membrane pemphigoid continue to evolve.

168 Mucous membrane pemphigoid continues to be a potentially

169 ohnson syndrome in 41.7% (20 of 48 eyes) and mucous membrane pemphigoid in 41.7% (20 of 48 eyes).

170 Mucous membrane pemphigoid is an autoantibody-mediated d

171 Ocular mucous membrane pemphigoid is an immunobullous disease i

172 ocular disease among patients with oral only mucous membrane pemphigoid is estimated at 15-20% at 5 y

173 About 25% of mucous membrane pemphigoid patients generate autoantibod

174 aim of this article is to review articles on mucous membrane pemphigoid published in the English lite

175 However, not all cases of mucous membrane pemphigoid respond to rituximab.

176 Treatment for mucous membrane pemphigoid typically involves immunosupp

177 Moreover, patients with ocular mucous membrane pemphigoid were characterized by margina

178 specific enough to confirm the diagnosis of mucous membrane pemphigoid with consistency.

179 agnoses (excluding Stevens-Johnson syndrome, mucous membrane pemphigoid, and congenital disorders) wh

180 indings and consisted of pemphigus vulgaris, mucous membrane pemphigoid, lichen planus, linear immuno

181 patients with lid malposition resulting from mucous membrane pemphigoid, particularly cicatricial ent

182 ts with an altered phenotype exist in ocular mucous membrane pemphigoid, we compared the functional c

183 good efficacy in the treatment of refractory mucous membrane pemphigoid.

184 t immunofluorescent testing in patients with mucous membrane pemphigoid.

185 e two more common are oral lichen planus and mucous membrane pemphigoid.

186 e anti-mLAMalpha3 IgG-induced mouse model of mucous membrane pemphigoid.

187 olysis bullosa acquisita or anti-laminin-332 mucous membrane pemphigoid.

188 pemphigoid, epidermolysis bullosa acquisita, mucous membrane pemphigoid/cicatricial pemphigoid, or an

189 The conjunctiva is a mucous membrane that covers the sclera and lines the ins

190 Hyperpigmentation of the oral mucous membrane, acquired longitudinal melononychia, and

191 ften affects upper and lower gingiva, buccal mucous membrane, the retromolar triangle and the palate.

192 n of leptospirosis occurs through contact of mucous membranes and abraded skin with freshwater contam

193 Further research into contact allergies of mucous membranes and identification of its allergens is

194 ase that features blistering of the skin and mucous membranes caused by autoantibodies directed again

195 an autoimmune blistering disease of skin and mucous membranes caused by autoantibodies to the desmogl

196 y, by tense blisters and erosions on skin or mucous membranes close to the skin surface.

197 all other shallow periodontal sites, and at mucous membranes followed by removal of calculus using c

198 In pemphigus, keratinocytes in epidermis and mucous membranes lose cell-cell adhesion, and in pemphig

199 ruses (HPVs) infect the epidermis as well as mucous membranes of humans.

200 red patchy depigmentation of skin, hair, and mucous membranes results from loss of melanocytes from i

201 d, natural exposure of the oral and/or nasal mucous membranes seems certain.

202 rvids commonly sustain minor lesions on oral mucous membranes that could have an impact on susceptibi

203 dermoscopic examination of skin, nails, and mucous membranes was performed, and skin biopsies were p

204 Most importantly, mucous membranes were touched, on average, 34.3 (SE = 2.

205 ired (e.g., orally or via lesions to skin or mucous membranes).

206 toxicities were as follows: esophagus, 4.7%; mucous membranes, 3.1%; and xerostomia, 3.1%.

207 neoplasia in sun-exposed areas of the skin, mucous membranes, and ocular surfaces.

208 utoimmune blistering disease of the skin and mucous membranes, characterized by autoantibodies agains

209 ), autoantibodies induce blisters on skin or mucous membranes, or both.

210 Nitrates are rapidly absorbed from mucous membranes, the gastrointestinal tract, and the sk

211 on can improve the integrity of the skin and mucous membranes, today's accomplishments are only the f

212 Damage is exclusive to the skin and mucous membranes, with no known direct compromise of the

213 affecting the epithelial layers of skin and mucous membranes, without extracutaneous manifestations,

214 potentially fatal blistering of the skin and mucous membranes.

215 ing in blisters and erosions of the skin and mucous membranes.

216 ketosugars that are ubiquitous on mammalian mucous membranes.

217 pigmented spots of skin, overlying hair, and mucous membranes.

218 noglobulin G (IgG) and IgA antibodies across mucous membranes.

219 s) is a common colonizer of healthy skin and mucous membranes.

220 ermatologic disorders involving the skin and mucous membranes.

221 onditions that initially affect the skin and mucous membranes.

222 s characterized by the fragility of skin and mucous membranes.

223 s characterized by the fragility of skin and mucous membranes.

224 eding via particle capture occurs on a fine, mucous mesh (fiber diameter d approximately 0.1 microm)

225 o infection in lung diseases associated with mucous metaplasia and a mechanism by which patients with

226 of 6-d-old mice, but not mature mice, causes mucous metaplasia and airway hyperresponsiveness that ar

227 infection of 6-day-old immature mice causes mucous metaplasia and airway hyperresponsiveness which i

228 ven type 2 response, which causes persistent mucous metaplasia and airways hyperresponsiveness.

229 ablished role as an inflammatory mediator of mucous metaplasia and functions through Stat6-mediated g

230 d 11 mpi had less severe gastritis, atrophy, mucous metaplasia and hyperplasia (P < 0.01) and, additi

231 -IL-13 neutralizing Ab attenuated RV-induced mucous metaplasia and methacholine responses, and IL-4R

232 K19-kras transgenic mice uniquely displayed mucous metaplasia as early as 3 months and progressed to

233 hage IL-1beta limits type 2 inflammation and mucous metaplasia following RV infection by suppressing

234 The generation of mucous metaplasia in immature RV-infected mice involves

235 at Notch ligands, however, are able to cause mucous metaplasia in Stat6-null cultured trachea, thus i

236 During mucous metaplasia induced under inflammatory conditions,

237 nd a mechanism by which patients with florid mucous metaplasia may tolerate microbial burdens that ar

238 infection of 6-day-old BALB/c mice induces a mucous metaplasia phenotype that is dependent on type 2

239 Mucous metaplasia was accompanied by induction of gob-5,

240 pposite effect, decreasing IL-25, IL-33, and mucous metaplasia while increasing IL-17 expression.

241 opment of three cardinal features of asthma: mucous metaplasia, AHR, and the presence of inflammatory

242 rse agonist treatment, namely, reductions in mucous metaplasia, airway hyperresponsiveness (AHR), and

243 re sufficient for eosinophilic inflammation, mucous metaplasia, and airways hyperresponsiveness.

244 on leads to persistent airways inflammation, mucous metaplasia, and hyperresponsiveness, which are me

245 ed decreases in HDM-induced inflammation and mucous metaplasia, as well as reduced IL-4, IL-25, CD68,

246 Whether these twinned phenomena (mucous metaplasia, compromised host defenses) are causal

247 RV-induced mucous metaplasia, ILC2 expansion, airway hyperresponsiv

248 ses of the airway are often characterized by mucous metaplasia, in which there are dramatic increases

249 VAMP8 knock-out (KO) mice with IL-13-induced mucous metaplasia, mucin content in the bronchoalveolar

250 on of bronchoalveolar lavage eosinophils and mucous metaplasia.

251 nd IL-4R null mice failed to show RV-induced mucous metaplasia.

252 ably, Notch antagonists blocked IL13-induced mucous metaplasia.

253 identifying a novel pathway that stimulates mucous metaplasia.

254 intranasal IL-33 and TSLP was sufficient for mucous metaplasia.

255 are required for maximum ILC2 expansion and mucous metaplasia.

256 airway epithelial cell cultures with induced mucous metaplasia/mucin hypersecretion.

257 XBP1 is also required for loss of mucous NC markers as ZCs form.

258 Compared with controls, numbers of total mucous neck and zymogen cells were significantly decreas

259 ronic gastritis followed by oxyntic atrophy, mucous neck cell hyperplasia, spasmolytic polypeptide-ex

260 cells (ZCs) as they differentiate from their mucous neck cell progenitors.

261 The transition of gastric epithelial mucous neck cells (NCs) to digestive enzyme-secreting zy

262 We investigated transcriptional programs in mucous neck cells and chief cells as they progress to me

263 Although mucous neck cells and chief cells from healthy stomachs

264 ectory analysis confirmed the convergence of mucous neck cells and chief cells into a pre-metaplastic

265 ons of chronic inflammation, chief cells and mucous neck cells are plastic and converge into a pre-me

266 Our findings indicated that mucous neck cells do not contribute substantially to gen

267 s study demonstrates that Agr2 expression by mucous neck cells in the stomach promotes the differenti

268 tiation of the zymogen cell lineage from the mucous neck cells in the stomachs of HKCre/Shh(KO) mice.

269 When Agr2 expression is absent, gastric mucous neck cells increased in number as does the number

270 y in mucous gland cells of the antrum and in mucous neck cells of the glandular corpus.

271 d base are believed to arise from descending mucous neck cells, which arise from stem cells.

272 und weak expression of FoxM1b protein in the mucous neck region of gastric mucosa, whereas we observe

273 and within 20 days, giving rise to parietal, mucous neck, and chief cells, but not to enterochromaffi

274 ISMCs express robust numbers of surface pit, mucous neck, chief, endocrine and parietal cells.

275 cells express robust numbers of surface pit, mucous neck, chief, endocrine and parietal cells.

276 ineages of the corpus including surface pit, mucous neck, zymogenic, and parietal cells expressed Shh

277 can evade filtration by slipping through the mucous nets of both pelagic and benthic tunicates.

278 uman tracheobronchial epithelial (NHTBE) and mucous NHTBE cells.

279 us metaplastic NHTBE cells but not in normal mucous NHTBE or normal bronchial epithelium.

280 h inflammation remains intact, heterogeneous mucous occlusion decreases by 74%.

281 moglobin degradation products 3. melanin, 4. mucous- or protein-containing lesions, 5. highly cellula

282 rt foreign particulate trapped in a layer of mucous out of the upper airway.

283 Existing dogma defines mucous phenotypes in asthma and chronic obstructive pulm

284 n cystic fibrosis) peribronchial thickening, mucous plugging and many other disorders that occur in t

285 l slices for the presence of bronchiectasis, mucous plugging, or other airway abnormalities (inspirat

286 ent revealed that ivacaftor decreased airway mucous plugging.

287 RSV rA2-line19F infection induces pulmonary mucous production and increased breathing effort in BALB

288 s without reducing goblet cell metaplasia or mucous production and may explain the lower risk of alle

289 of tissue loss, sclerite enlargement, excess mucous production, bleached commensal ophiuroids, and co

290 consider the therapeutic effects of blocking mucous production.

291 n, but the detailed mechanisms of epithelial mucous regulation have not been well defined.

292 tected in a total of 52 of 126 (41.3%) nasal mucous samples, showing the nasal cavity is a reservoir

293 Fasudil also reduced mucous secretion and MUC5AC expression in OVA-challenged

294 kotrienes stimulate conjunctival goblet cell mucous secretion with LTD(4) using the CysLT(1) receptor

295 ERK1/2 to stimulate conjunctival goblet cell mucous secretion, and that activation of all four histam

296 contributor to the viscoelastic property of mucous secretion, which is the major barrier to trapping

297 formation to pulmonary diseases, thrombosis, mucous secretions in the airways, and cytokine productio

298 ses by permitting continuous accumulation of mucous secretions on the epithelial surface, establishin

299 Deletion of IL-4Ralpha abolished transient mucous secretory cell (MuSC) abundance and eosinophilia

300 t least monthly leakage of solid, liquid, or mucous stool), and pelvic organ prolapse (seeing/feeling

301 trend towards reduction of symptom score and mucous weight.