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1 way to induce rapid goblet cell emptying and mucus secretion.
2 es were assessed for airway inflammation and mucus secretion.
3  recently been reported to promote excessive mucus secretion.
4 tegrity, regeneration, pathogen-sensing, and mucus secretion.
5 nflammation, microvascular permeability, and mucus secretion.
6 f autophagosomes were required for efficient mucus secretion.
7 leukin-13 (IL-13) is a mediator of pulmonary mucus secretion.
8 d several mechanisms that inhibit neurogenic mucus secretion.
9 sal glands do not appear to be involved with mucus secretion.
10 ad to particles that rapidly penetrate human mucus secretions.
11 a disease characterized by hyperviscoelastic mucus secretions.
12 les are similar to those in many other human mucus secretions.
13                                    Increased mucus secretion accompanies ciliary motion defects, but
14 eosinophilia, type 2 cytokine production and mucus secretion after allergen inhalation.
15 ole in murine asthma, mediating both AHR and mucus secretion after HDM exposure.
16 h2 cytokine production, airway inflammation, mucus secretion, airway hyperresponsiveness, and serum o
17 triggered defensive behavior such as copious mucus secretion and a range of other anomalous behaviors
18 tic tissues, including the absence of acidic mucus secretion and aberrant adherens junctions in the e
19  bronchoconstriction, airway plasma leakage, mucus secretion and cough.
20 matory, and infectious insults induce airway mucus secretion and goblet cell metaplasia to preserve a
21 d signal to intestinal goblet cells to drive mucus secretion and gut protection.
22 ermeability and immune activation; excessive mucus secretion and impaired mucociliary clearance that
23 tered secretory state can lead to changes in mucus secretion and luminal pH.
24 utonomic and trigeminal innervation controls mucus secretion and may release neurotransmitters into n
25  healthy, intact epithelium results in rapid mucus secretion and movement of Ly6C(+)7/4(+) monocytes
26 negative regulatory role in allergen-induced mucus secretion and MUC5AC expression by regulating STAT
27 downstream effects, including reduced airway mucus secretion and protection from endothelial barrier
28 l asthma, such as smooth muscle contraction, mucus secretion and recruitment of inflammatory cells, a
29 ammatory responses, and direct activation of mucus secretion and smooth muscle cell constriction.
30 creased IgE production, eosinophil activity, mucus secretion and smooth muscle reactivity, effected t
31 erentiated epithelium with functional cilia, mucus secretion and subepithelial fibroblasts within typ
32                                  We measured mucus secretion and the expression of Rho-kinase in the
33 am immune responses, including eosinophilia, mucus secretion and type 2 immunity.
34 in the protective epithelial barrier through mucus secretion and yet sample lumenal substances for im
35  vicious feedback cycle of hyperconcentrated mucus secretions and persistent inflammation in the CF a
36 loped epithelial cell hyperplasia, increased mucus secretion, and airway hyperreactivity.
37 here it regulates eosinophilic inflammation, mucus secretion, and airway hyperresponsiveness.
38 ncluding gastric acid/bicarbonate secretion, mucus secretion, and cell migration.
39 d mediators involved in bronchoconstriction, mucus secretion, and cell trafficking in asthmatic patie
40 ce showed increased Th2 cytokine production, mucus secretion, and lung infiltration of eosinophils an
41 issue eosinophilia, goblet cell hyperplasia, mucus secretion, and peribronchial edema and also inhibi
42 vented eosinophilia, airway hyperreactivity, mucus secretion, and Th2 cyto-kine production.
43 y shown to regulate airway cell cytokine and mucus secretion, and transepithelial Cl(-) current.
44 of the nanoscale barrier properties of human mucus secretions, and to achieve more uniform and longer
45 d IL-5, IL-13, and TNF-alpha levels; reduced mucus secretion; and improved airway function.
46 red areas rather than unexplored areas using mucus secretion as a buffer.
47 lation of human epithelial cells resulted in mucus secretion as measured by MUC5AC mRNA and protein.
48                           Here, we show that mucus secretion by goblet cells is altered in the colon
49 clude reflex stimulation of submucosal gland mucus secretion by sensory neurons that release substanc
50 the NLRP6 inflammasome, enhancing protective mucus secretion by sentinel goblet cells.
51 dition, DF of BR increased significantly the mucus secretion compared to control group.
52 nfected IL-27rKO mice showed exacerbation of mucus secretion compared with wild type, as well as enha
53 crease microvascular permeability, stimulate mucus secretion, decrease mucociliary clearance, and app
54 rrhea, coughing, bronchoconstriction, airway mucus secretion, dysphagia, altered gastrointestinal mot
55 se, metabolic imbalances, hypoxia/hyperoxia, mucus secretion, dysregulated autophagy, and alters pulm
56                                 Constitutive mucus secretion ejected beads from crypt lumens in 8-10
57 mediate host-pathogen interactions including mucus secretion, flow and air-liquid interface (ALI), wh
58 n as the most likely mechanism for defective mucus secretion from CF glands.
59 ncrease the rate of cholinergically mediated mucus secretion from CF glands.
60 nor amiloride increased forskolin-stimulated mucus secretion from porcine submucosal glands (75 gland
61 ing regulation of airway smooth-muscle tone, mucus secretion from submucosal glands and surface epith
62                                              Mucus secretions from X. laevis previously exposed to B.
63 eatment of disorders of epithelial fluid and mucus secretion, hypertension, asthma, and possibly canc
64 at nanoparticles can rapidly penetrate human mucus secretions if they are densely coated with low MW
65 s the airways, resulting in inflammation and mucus secretion in both mice and humans.
66              Although clarithromycin reduced mucus secretion in both rhinitis patients and normal sub
67 olinergic pathways still elicit strong gland mucus secretion in CF subjects, it is unclear whether VI
68                 Loss of "housekeeping" gland mucus secretion in CF, in combination with demonstrated
69 rinic receptor subtypes mediating neurogenic mucus secretion in ferret trachea were characterized in
70 w levels of VIP and ACh produced significant mucus secretion in human glands via strong synergistic i
71                           Notably, FR blocks mucus secretion in human lung slices from asthmatic pati
72 rine lactone (C4-HSL), on cell viability and mucus secretion in LS174T cells.
73 bition of TMEM16A-CaCC significantly impairs mucus secretion in primary human airway surface epitheli
74 mucin gene expression, mucus composition, or mucus secretion in response to intestinal microbes or ho
75                 show that autophagy controls mucus secretion in the colons of mice.
76 T-I CD8 T cells, attenuated eosinophilia and mucus secretion in the lungs of sensitized mice in an an
77                                Thus, altered mucus secretion in TMF(-/-) mouse colons is accompanied
78 inistration to the lung resulted in enhanced mucus secretion, inflammatory cell recruitment, and cyto
79 ajor contributor to increased vagal tone and mucus secretion, inhaled long-acting muscarinic antagoni
80 tive autophagy in goblet cells and abrogated mucus secretion into the large intestinal lumen.
81                                    Increased mucus secretion is an important clinical symptom and con
82                                              Mucus secretion is an important protective mechanism for
83  The dominant neural control of human airway mucus secretion is cholinergic.
84 tic nanoparticles must rapidly penetrate the mucus secretions lining the surfaces of the respiratory,
85 une regulatory pathway governing goblet cell mucus secretion, linking nonhematopoietic inflammasome s
86 cant depletion of goblet cell metaplasia and mucus secretion markers after HDM exposure.
87 formation, some studies imply that in-sucker mucus secretion may be another critical factor in helpin
88  resume after mucus removal, suggesting that mucus secretion may mediate MCC deterioration.
89  vasculature, inflammatory cell recruitment, mucus secretion, mucociliary clearance and airway surfac
90 TMEM16B regulate diverse processes including mucus secretion, neuronal excitability, smooth muscle co
91 ts that may be functionally analogous to the mucus secretions of higher eukaryotes.
92 ese two hypotheses, we measured single gland mucus secretion optically and applied ENaC inhibitors to
93 hohexital nor propofol significantly affects mucus secretion or clearance in healthy dogs.
94 rocess begins when the bacteria aggregate in mucus secretions outside the light organ.
95 d dependent on progeny viruses shedding into mucus secretions overlaying the apical surface of WD-HAE
96 2 expression or increases in goblet cells or mucus secretion pathways.
97 d tracheas, using optical methods to monitor mucus secretion rates from multiple glands in parallel.
98 ild infection induces a strong activation of mucus secretion-related genes in young immunocompetent r
99 s and cohesive forces predicted to limit SMG mucus secretion/release.
100 ory pathways involved in goblet cell-induced mucus secretion remain largely unknown.
101 hma is characterized by airway inflammation, mucus secretion, remodeling and hyperresponsiveness (AHR
102 , spatially complex chemical attributes, and mucus secretion response to stimulation, with the potent
103                    It appears that increased mucus secretion results from increased mucin gene expres
104 ng of the airways with significantly reduced mucus secretion, subepithelial fibrosis, smooth muscle t
105       Cholinergic stimulation, which elicits mucus secretion, substantially reduced microdisk movemen
106 al range, indicating a rapid recovery in the mucus secretion system.
107 unction, leukocyte migration activation, and mucus secretion that are affected by both SCIT and SLIT,
108 ormally active and stimulate low-level gland mucus secretion that is a component of innate mucosal de
109 istics of human and porcine small intestinal mucus secretions to sub-micron sized particles have been
110  therefore investigated the role of HCO3- in mucus secretion using mouse small intestine segments ex
111                    PGE(2) is known to induce mucus secretion, vasodilation, and edema, and acts as an
112 hea, cholinergic nerve stimulation increases mucus secretion via muscarinic M3 receptors on the submu
113 ction of biological suckers' soft bodies and mucus secretion, we propose a multiscale suction mechani
114  increases in airway hyperresponsiveness and mucus secretion were similar to those observed in wild-t
115 rovide secondary protection by orchestrating mucus secretion when microbes breach the mucus barrier.
116 tor for snails' sliding suction behaviour is mucus secretion, which reduces friction and enhances suc

 
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