1 d in the prevention of cognitive decline and multi-infarct cerebrovascular disease.

2  Sourander and Walinder described hereditary multi-infarct dementia (MID) in a Swedish family.

3 for Diagnosis of Dementia of Alzheimer Type, Multi-infarct Dementia and Dementia of other Etiology ac

4 ive astrocytes, and damaged blood vessels in multi-infarct dementia when compared to AD.

5 decline in cognitive function by influencing multi-infarct dementia, but data are sparse.

6 including the hereditary vascular dementias, multi-infarct dementia, post-stroke dementia, subcortica

7 sease were generally higher among women; for multi-infarct dementia, rates for men and women were sim

8 agnosed with Alzheimer's disease and 24 with multi-infarct dementia.

9 F had a higher rate of non-lacunar infarcts, multi-infarct patterns and more severe periventricular w

10  the temporal cortex of patients with either multi-infarct vascular dementia or SND, compared to age-

11 ty to develop novel therapies for stroke and multi-infarct vascular dementia patients.

12 including large artery territory infarction (multi-infarct vascular dementia) and sub-cortical ischae

13 nd temporal cortices of patients with either multi-infarct vascular dementia, sub-cortical ischaemic

14 nt distinction between the neurochemistry of multi-infarct vascular dementia/SND and sub-cortical isc