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1 arly half of the infected spiders exhibiting multiple infection.
2 h facilitates the treatment of patients with multiple infections.
3 ci that influence humoral immune response to multiple infections.
4  with patients often displaying a history of multiple infections.
5 ndosymbiotic bacteria and frequently exhibit multiple infections.
6  other and therefore represented evidence of multiple infections.
7 tion depending on the presence or absence of multiple infections.
8 fect cross-immunity has on the prevalence of multiple infections.
9 d, senescent states ("Hayflick limit") after multiple infections.
10 re enhanced among older women and women with multiple infections.
11  T-cell immunodeficiencies, who are prone to multiple infections.
12 tion and thus appeared to result solely from multiple infections.
13 d beneficial fitness effects associated with multiple infections.
14 ls the multiplicity of subtypes results from multiple infections.
15 selection of more-resistant C. albicans over multiple infections.
16 ity of an infection varies by pathogen or by multiple infections.
17 % [n=97]; isolated thoracotomy, 0.02% [n=5]; multiple infections, 0.6% [n=130]).
18    We apply this method to study the role of multiple infection and intracellular interactions among
19 timing is discussed from the perspectives of multiple infection and life-history trait evolution.
20 t mechanisms underlie the reliance of IAV on multiple infection and underscore the importance of viru
21  widespread use as vaccine platforms against multiple infections and cancers, and multiple serotypes
22 ow these relationships change from single to multiple infections and impact helminth dynamics remains
23  withstand parasitic infections, the role of multiple infections and the trade-off in multiple defenc
24 ploying organoids demonstrates the hazard of multiple infections and the unique cellular microenviron
25 ided substantially better protection against multiple infections and tumors.
26       Analyses of viral load need to address multiple infections and type attribution to evaluate whe
27 ger women with low-grade histology/cytology, multiple infections, and the highest viral load.
28 isolates from three continents, we find that multiple infections are common, especially in regions wi
29                                     However, multiple infections are common, making attribution of ca
30 , the mechanisms that lead to such extensive multiple infections are not understood.
31 gs) play an important role in the CNS during multiple infections, as well as autoimmune inflammation,
32 in a single-round infectivity assay and in a multiple-infection assay in H9 and CEMx174 cells.
33 at MHC heterozygote superiority emerges over multiple infections because MHC-mediated resistance is g
34                                              Multiple infections before age 40 conveyed the greatest
35    Here, we show that the reliance of IAV on multiple infection can form an important species barrier
36                                              Multiple infection control improvements led to the reduc
37 s, a similar distribution was observed after multiple infection cycles, and among recombinant sequenc
38 yping directly from tissue sections resolved multiple infections detected in exfoliated cells to a si
39                            The proportion of multiple infections detected increased with the PGMY09/1
40  and the ARLG Master Protocol for Evaluating Multiple Infection Diagnostics (MASTERMIND) initiative f
41 RLG developed Master Protocol for Evaluating Multiple Infection Diagnostics (MASTERMIND), an innovati
42 d MASTERMIND (Master Protocol for Evaluating Multiple Infection Diagnostics) for advancement of infec
43                            We also find that multiple infections do not influence viral dynamics when
44                                           If multiple infections do occur, then only a single haploty
45 hat by expediting the deterioration of BAMs, multiple infection episodes might be associated with acc
46  a mode of HIV transmission that can lead to multiple infection events per target cell.
47 vs. susceptibility to infection, we included multiple infection experiments including one with consta
48 s between the single-infection group and the multiple-infection group regarding the number of DNA sub
49                       Also, patients who had multiple infections had lower CD4 counts than those with
50 urst across all phagocytes, who are prone to multiple infections, including both Bacillus Calmette-Gu
51 merase has been implicated in the success of multiple infection-induced pathologies, including the su
52                                This need for multiple infection is greatly reduced in the natural hos
53                                              Multiple infection is predicted to increase the number o
54             PQS is required for virulence in multiple infection models and has been found in the lung
55                                              Multiple infections occurred significantly more frequent
56 f viruses, which increased the likelihood of multiple infection of a host cell and ultimately enabled
57 if one virus is transferred per synapse, and multiple infection of cells increases susceptibility.
58 virus population to anti-viral drugs through multiple infection of cells, contributing to low-level v
59 irus to overcome the antiviral state through multiple infection of cells.
60 ute HIV-1 infection, and further include the multiple infection of cells.
61 assortment of influenza through simultaneous multiple infection of individual hosts and the generatio
62 rger numbers of transferred viruses, because multiple infection of the same cell wastes viruses that
63                                              Multiple infections of Agaricus may represent a diverse,
64 o study the immune response that occurs with multiple infections of C. trachomatis in the female uppe
65                        The high incidence of multiple infections of cells by HIV sets the stage for r
66                 The patient had a history of multiple infections of central vascular catheters with o
67                                              Multiple infections of different bacteria in a single ho
68 itional survival costs to aphids of carrying multiple infections of symbiont species or strains, and
69 ct minor populations of mutant strain within multiple infection, offering high sensitivity and specif
70 d provide key insights into the influence of multiple infections on HIV dynamics.
71 ients were significantly more likely to have multiple infections over time than HIV- patients, highli
72 ought to investigate the effect of single or multiple infections (pathogen burden) on atopy and wheez
73  and experimentally validate the model using multiple infections per cell by cell-free HIV in the pre
74                   We propose a model whereby multiple infections per cell lead to reduced sensitivity
75  was to identify the strains responsible for multiple infections, presumably through recent transmiss
76  common HPV types but were probably due to a multiple infection rather than to a cross-reaction.
77           Yet how HIV dynamics proceeds with multiple infections remains poorly understood.
78 the results of our study show that even when multiple infections seem to have no effect on a host, th
79 tation accounts for the moderate reliance on multiple infection seen in avian cells but not the added
80               Escalation was associated with multiple infection sites (2.54, 1.34-4.83) and a positiv
81 ng hormone as a signal to differentiate into multiple infection structure and thus time the infection
82  may explain why extant models, which ignore multiple infections, successfully describe viral dynamic
83 at in vitro phenotypes should be examined in multiple infection systems to fully understand their rol
84 time PCR format that has been used to detect multiple infection targets.
85 esponse is cumulative and aberrant as, after multiple infections, the epithelium was markedly thicken
86  cause limited immunosuppression and require multiple infections to bypass CRISPR defenses, a single
87                                       Hence, multiple infection via synapses does not simply lower tr
88                               At enrollment, multiple infection was detected in only 3 of 58 (5.2%) p
89 Sim, allowing us to test control measures in multiple infection waves caused by the spread of a new v
90                         In a murine model of multiple infections, we show that exposure of mice to re
91 f wheeze and atopic outcomes with single and multiple infections were analyzed by means of logistic r
92  [15%]), and cytomegalovirus (34 [12%]), and multiple infections were common.
93                                              Multiple infections were found to be more common than ex
94                                        While multiple infections were more common in young women and
95                                              Multiple infections were observed in 76%, 17%, and 3% of
96                                              Multiple infections were required to elicit T-helper 17
97                 Multiple genotypes (implying multiple infections) were detected in tissues from 8 of
98 nate between trypanosome species involved in multiple infections with a high sensitivity.
99                                Children with multiple infections with Cryptosporidium were more likel
100                                              Multiple infections with different strains of Mycobacter
101                                              Multiple infections with HPV genotypes of different risk
102                                              Multiple infections with nearly identical rhinoviruses c
103                                              Multiple infections with the attenuated plasmid-deficien

 
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