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1 d chance of achieving clinical pregnancy and multiple pregnancy.
2 apies to reduce the risk of preterm birth in multiple pregnancy.
3 ore than 1 embryo with its inherent risks of multiple pregnancy.
4 d, with 51 children (12%) known to be from a multiple pregnancy.
5  associated with the incidence of high-order multiple pregnancy.
6 oking, not White ethnicity, nulliparity, and multiple pregnancy.
7 rategy for increasing baby survival rates of multiple pregnancies.
8 d preterm delivery of artificially conceived multiple pregnancies.
9 ght less than 1000 g and 317 (20%) were from multiple pregnancies.
10 mor virus (MMTV)-infected females even after multiple pregnancies.
11  "best" embryos for transfer and to minimize multiple pregnancies.
12 ult in greater weight retention in mice with multiple pregnancies.
13 egenerated a differentiated gland even after multiple pregnancies.
14 rates and reducing the risks associated with multiple pregnancies.
15 es of spontaneous abortion, termination, and multiple pregnancies.
16  benefit of revaccination over the course of multiple pregnancies.
17 ne stimulation with pituitary isografts; (3) multiple pregnancies; (4) DMBA alone; and (5) DMBA+pitui
18                                        Thus, multiple pregnancies and allostimuli appear to have prov
19 sk of non-Hodgkin's lymphoma associated with multiple pregnancies and an increased risk of non-Hodgki
20  brain volume in singletons and offspring of multiple pregnancies and, in singletons, with cognitive
21  included singletons (ie, not twins or other multiple pregnancies) and children for whom the mother w
22 regnancy or gestational diabetes, age <18 y, multiple pregnancy, and fetal anomaly.
23 ily history of pre-eclampsia, nulliparity or multiple pregnancies; and previous pre-eclampsia or intr
24                                              Multiple pregnancy (aOR = 5.75; 95% CI 1.54-21.45) and i
25 rk inconsistency per outcome was deemed low (Multiple pregnancy chi(2): 0.11, OHSS chi(2): 0.26), mod
26 igation of the relation of the occurrence of multiple pregnancy complications to CVD death over 5 dec
27 uded potential confounders and accounted for multiple pregnancies contributed by each man.
28 n the adult build alveolar structures during multiple pregnancies, demonstrating the existence of a W
29 years at cardiomyopathy diagnosis; 2.6% with multiple pregnancies) developed cardiomyopathy.
30 es of ongoing pregnancy, clinical pregnancy, multiple pregnancy, ectopic pregnancy, or miscarriage.
31 inal progesterone was associated with higher multiple pregnancy events, [OR 7.09 (95% CrI 2.49, 31.)]
32                                              Multiple pregnancies, fetal abnormalities or births befo
33 ipients with female donors who had undergone multiple pregnancies had a higher rate of chronic GVHD t
34 omary may reduce the incidence of high-order multiple pregnancy in infertile women, though only to a
35                                Sesquizygotic multiple pregnancy is an exceptional intermediate betwee
36 of guidance on care specifically for twin or multiple pregnancies: None of the countries provided cle
37 eatment within 5 years of RNA positivity and multiple pregnancies occurred before treatment, resultin
38 aternal age, body mass index, ethnicity, and multiple pregnancy (odds ratio, 0.99; 95% CI, 0.93-1.03;
39                              Children from a multiple pregnancy or with chromosomal disorders or unce
40 . falciparum parasitemia during pregnancy on multiple pregnancy outcomes.
41 omes and biochemical pregnancy, miscarriage, multiple pregnancy, ovarian hyperstimulation syndrome (O
42 cantly with an increasing risk of high-order multiple pregnancy (P<0.001), as did younger age (P=0.00
43 ultivariable regression analysis showed that multiple pregnancies, parity >= 1, maternal BMI, and dem
44 -effects Cox regression models adjusting for multiple pregnancies per individual.
45        Log-linear regression, accounting for multiple pregnancies per woman, generated adjusted preva
46 easures analysis was used to account for the multiple pregnancies per woman.
47 riance estimates to account for inclusion of multiple pregnancy periods per unique pregnancy.
48 nital mutilation, sepsis, no antenatal care, multiple pregnancy, placenta praevia, assisted reproduct
49                                   Women with multiple pregnancy, preterm birth prior to 37 weeks, con
50  1.16, 95% CI = 1.00-1.36, I(2) = 48.3%) and multiple pregnancy rates (OR = 1.50, 95% CI = 1.11-2.01,
51 ant results of clinical pregnancy as well as multiple pregnancy rates were observed among women who r
52                                      She had multiple pregnancy-related complications, including toxe
53 oving clinical decision-making, and reducing multiple pregnancy risks.
54 hile incidences of history of preterm birth, multiple pregnancies, serious or severe psychological di
55                                              Multiple pregnancies should be monitored closely, and th
56 that long-term use of oral contraceptives or multiple pregnancies significantly increases the risk fo
57 rn weighing less than 1500 g--and those from multiple pregnancies than in infants of normal birthweig
58                                        After multiple pregnancies the p100 transgenics exhibited a du
59             Whether the number of high-order multiple pregnancies (those with three or more fetuses)
60     Adverse effects of gonadotropins include multiple pregnancy (up to 36% of cycles, depending on sp
61               Graft survival in mothers with multiple pregnancies was poorer than those with a single
62               Among pregnancies, the rate of multiple pregnancy was 6.0% in the clomiphene group, 0%
63                       The risk of high-order multiple pregnancy was significantly increased in women
64 nd for multiparous women, scarred uterus and multiple pregnancies were risk factors.