ライフサイエンスコーパス: multiplicity of infection

1 -type (wt) HSV-1 was highly dependent on the multiplicity of infection.

2 ble to infection-induced cell death at a low multiplicity of infection.

3 , the magnitude of which is dependent on the multiplicity of infection.

4 between 6 and 24 h postinfection with a low multiplicity of infection.

5 nnings of virus transmission, especially the multiplicity of infection.

6 fection in the absence of ICP0 at the chosen multiplicity of infection.

7 copy is dependent on both the cell type and multiplicity of infection.

8 nomes was unimpaired at either a low or high multiplicity of infection.

9 us monocytes infected with adenovirus at low multiplicity of infection.

10 ransmission, resulting in an increase in the multiplicity of infection.

11 gering of the lytic cycle, especially at low multiplicity of infection.

12 ven after 3 h of infection at a dose of 1000 multiplicity of infection.

13 id inclusion of citrate and the use of a low multiplicity of infection.

14 total protein of the cells, depending on the multiplicity of infection.

15 nclusions per cell varying directly with the multiplicity of infection.

16 t 10 to 15 h postinfection, depending on the multiplicity of infection.

17 F and exhibits a replication defect at a low multiplicity of infection.

18 the findings of a virus yield assay at a low multiplicity of infection.

19 were added to RPE and THP-1 cells with a 5:1 multiplicity of infection.

20 sages in cell culture of the virus at a high multiplicity of infection.

21 with CJ2-gD2 and wild-type HSV-2 at the same multiplicity of infection.

22 tent of the relief being highly dependent on multiplicity of infection.

23 rug-resistant bacterial colonies at very low multiplicity of infections.

24 in controlling viral gene expression at low multiplicities of infection.

25 was both long lasting and detectable at low multiplicities of infection.

26 s well as the wild type at both low and high multiplicities of infection.

27 us or Salmonella serovar Dublin at different multiplicities of infection.

28 ith enhanced responses at low, but not high, multiplicities of infection.

29 h and protein synthesis, particularly at low multiplicities of infection.

30 decrease in SIV replication, even after high multiplicities of infection.

31 in cells lacking DNA-PKcs, especially at low multiplicities of infection.

32 nitiation of DNA synthesis especially at low multiplicities of infection.

33 d in PML(-/-) infected at low, but not high, multiplicities of infection.

34 roduct required for viral replication at low multiplicities of infection.

35 uired for efficient viral replication at low multiplicities of infection.

36 rtant for efficient viral replication at low multiplicities of infection.

37 1-47 and gKDelta31-117 mutant virions at low multiplicities of infection.

38 cted with Mtb strains H(37)Ra and H(37)Rv at multiplicities of infection 0.1 and 1.

39 ng strain), or RSV (A/2001/2-20 strain) with multiplicity of infection 0.01 to 1.

40 h greater progeny production of Sy2 at a low multiplicity of infection (0.01) in tissue culture, but

41 th cytomegalovirus (CMV) strain AD169 at low multiplicity of infection (0.03) and harvesting the cell

42 ar stomatitis virus (VSV) replication at low multiplicity of infection (0.1 pfu/cell).

43 culum was increased at the time of exposure (multiplicity of infection = 0.05), the inhibitory effect

44 number detected in Huh-7.5/JFH-1 coculture (multiplicity of infection: 0.01) at an effector:target r

45 3-transduced lung cancer cell lines at a low multiplicity of infection (1 MOI).

46 s (> 52 h postinjection), high vector doses (multiplicity of infection, 1.5) led to a decrease of FIA

47 ro with live M. tuberculosis H37Ra or H37Rv (multiplicity of infection, 1:1) or cultured with lipopol

48 (10 ng/muL), and heat-killed whole bacteria (multiplicity of infection, 1:100) for 16 hours with or w

49 Increasing the titer of adenoviral vectors (multiplicity of infection 10-1000) led to a dose-depende

50 kin-4, interleukin-10), although a very high multiplicity of infection (10,000 infectious units/islet

51 versity (pi = 2.4 x 10(-4)), moderately high multiplicity of infection (2.7), and low linkage disequi

52 Infection of hematopoietic cells at modest multiplicities of infection (20-40) required special con

53 ges before or after Schu 4 or LVS challenge (multiplicity of infection, 20:1) had significantly reduc

54 Incubation of Ad.PKG-infected RPaSMC (multiplicity of infection = 200) with 8-Br-cGMP decrease

55 Human MSC viability was 96.7% (multiplicity of infection, 250).

56 ide range simply by changing the baculovirus multiplicity of infection; 3) HBV replication is readily

57 mpared with those infected with Ad-CMV/hNIS (multiplicity of infection = 30).

58 Macrophages were exposed to HRV16 (multiplicity of infection 5), polyinosinic:polycytidylic

59 ithin 48 h of exposure to an exceedingly low multiplicity of infection (5 x 10(-4)), 50% of ES cell-d

60 1 infectivity reduced 4 logs and high-input (multiplicity of infection = 5.0) replication completely

61 In contrast, AdHIF-1alpha-no-TAD (multiplicity of infection 50) ablated the anoxic resista

62 ction with an ICP0 mutant was greater at low multiplicity of infection, a condition in which ICP0 mut

63 ble but growth impaired at both high and low multiplicities of infection and exhibits a kinetic defec

64 y of gene transfer increased with increasing multiplicities of infection and incubation time, with 45

65 e-dependent relationships between the vector multiplicities of infection and the efficiencies of lacZ

66 is necessary for productive infection at low multiplicities of infection and therefore likely to be i

67 A mutant had normal growth properties at low multiplicities of infection and was more effective than

68 ental outcome is determined by the choice of multiplicity of infection and cell type and by whether c

69 -to-cell contact and increased together with multiplicity of infection and HCV protein levels.

70 required a threshold level of SipA linked to multiplicity of infection and may be a limiting factor c

71 When inoculated at the same multiplicity of infection and observed 24-48 h after inf

72 d by using the 6-h standard, because the low multiplicity of infection and short culture time did not

73 man (Ad.hHGF) adenoviral HGF (Ad.HGF) at low multiplicity of infection and studied in vitro.

74 luorescent and viable SA increased at higher multiplicity of infection and when SA presented to PMN h

75 the effect of different aspects: perfusion, multiplicity of infection, and temporal patterns of infe

76 The models identify multiplicity of infection as a key factor.

77 thelial cells were infected with HPV31b at a multiplicity of infection as low as 1 to 10 viral genome

78 derived DC are permissive to Ad infection at multiplicity of infection between 100 and 500 and occurs

79 cells infected with Ad-hACE2-eGFP (10 to 100 multiplicities of infection), but not Ad-eGFP (100 multi

80 KK1 is important for HCMV infection at a low multiplicity of infection, but is dispensable for AMPK a

81 caused apoptosis of MPhis at a wide range of multiplicity of infection, but smaller inocula resulted

82 on severely attenuated viral growth at a low multiplicity of infection by modestly reducing viral DNA

83 ll parasitemic individuals were assessed for multiplicity of infections by nPCR and gametocyte carria

84 Furthermore, we observe that high multiplicity of infection can induce the conversion of L

85 In contrast, high multiplicity of infection conditions caused a host cell

86 lls (MECs) were infected with HHV-8 at a low multiplicity of infection, considerable latent replicati

87 n was pathogen specific, heat sensitive, and multiplicity of infection dependent and required chlamyd

88 of HSV-1(vCPc0) beta or gamma genes was also multiplicity of infection dependent.

89 restriction was cell type dependent but not multiplicity of infection dependent.

90 oxia/SD-induced apoptosis at 24 h in an moi (multiplicity of infection)-dependent manner.

91 al gene expression in rabbit skin cells in a multiplicity of infection-dependent fashion.

92 or viral gene expression in a cell type- and multiplicity of infection-dependent fashion.

93 replication in cells silenced for BAG3 in a multiplicity of infection-dependent manner.

94 uclear translocation in a time-dependent and multiplicity-of-infection-dependent manner as determined

95 At low multiplicities of infection, dl1520 had an apparent p53-

96 At higher multiplicities of infection, dl1520 viral replication wa

97 o with varicella-zoster virus (VZV) at a low multiplicity of infection does not result in a cytopathi

98 Finally, we show that at a low multiplicity of infection, either UL97 or UL21a can part

99 henicol treatment of macrophages infected at multiplicities of infection exceeding 10,000 prevented c

100 licities of infection), but not Ad-eGFP (100 multiplicities of infection), exhibit dose-dependent ACE

101 Retesting at a defined multiplicity of infection followed by in vitro growth an

102 on the C. pneumoniae strain examined at low multiplicities of infection following 24 h of incubation

103 led or UV-inactivated C. pneumoniae at a low multiplicity of infection for 24 to 72 h stimulated both

104 is within 48 h after exposure to low titers (multiplicity of infection > 0.07) of an African lineage

105 city observed with smooth strains at extreme multiplicities of infection (>1,000).

106 After enrollment, mixed infections (multiplicity of infection, >1) were only present in the

107 These results suggest that, at low multiplicities of infection, ICP0 blocks CoREST-mediated

108 ood allelic discrimination and detected high multiplicities of infection in 63 P. vivax infections in

109 (Ad-ZTA), whose level of expression at a low multiplicity of infection in normal human diploid fibrob

110 ss between HIV strains in the cohort and the multiplicity of infection in seroconverters.

111 eporter gene was dose dependent and at a low multiplicity of infection increased progressively over t

112 ious studies, using the stated cell type and multiplicities of infection, indicated that the original

113 At a low multiplicity of infection, induction of autophagy by RNa

114 ses to better immune control if a sufficient multiplicity of infection is attained in vivo, but this

115 The multiplicity of infection is determined by the rate of n

116 At low multiplicities of infection, it was difficult to detect

117 ild-type infections generally occur at a low multiplicity of infection, it is unlikely that these del

118 hages with Mycobacterium tuberculosis at low multiplicities of infection leads 48-72 h after the infe

119 However, infection of these cells at a low multiplicity of infection leads to no discernible cytopa

120 though transduction of fibroblasts at higher multiplicities of infection led to a marked reduction of

121 At low multiplicity of infection (m.o.i.), delivery of influenz

122 the M. tuberculosis strain, time course, and multiplicity of infection, may have predominant features

123 zygosity, 0.843 [range 0.570-0.989] with low multiplicity of infection (mean, 1.05) reflecting a low

124 nuated strains of pathogenic mycobacteria at multiplicities of infection (MOI) < or = 10 triggers TNF

125 Ad5 at high (1.0), low (0.1), or zero (0.0) multiplicities of infection (MOI) and harvested at diffe

126 ells infected with poliovirus at low or high multiplicities of infection (MOI) and measured viral gen

127 of these genes was significantly greater at multiplicities of infection (MOI) of 10 PFU/cell or grea

128 with HRV or UV-irradiated HRV at increasing multiplicities of infection (MOI) without or with IFN-be

129 pulations generated in environments with low multiplicities of infection (MOI), a phenomenon that may

130 ls of viral components, particularly for low multiplicities of infection (MOI), where few virus parti

131 a growth defect on swine macrophages at low multiplicities of infection (MOI), yielding 0.1 to 1.0%

132 -1 is critical for IE gene expression at low multiplicities of infection (moi).

133 t transcription and viral replication at low multiplicities of infection (MOI).

134 ed kinetically over a wide range of starting multiplicities of infection (MOI; from 0.02 to 20,000 ba

135 ) using a single addition of vector at a low multiplicity of infection (MOI = 5).

136 After a low multiplicity of infection (MOI) (0.01 PFU/cell), recombi

137 and complex: 67% polyclonal infections, mean multiplicity of infection (MOI) 2.2, and mean within-hos

138 pheral blood neutrophils incubated with RSV (multiplicity of infection (MOI) = 10) induced IL-8, macr

139 ations, along with estimates of the cellular multiplicity of infection (MOI) and MOI model selection,

140 e distal enhancer replicate normally at high multiplicity of infection (MOI) but replicate poorly at

141 Multiplicity of infection (MOI) by Amp-Seq was 2.32 vers

142 has not been previously shown how the viral multiplicity of infection (MOI) can affect IFN induction

143 Multiplicity of infection (MOI) can be an indicator of i

144 The data indicate that a low multiplicity of infection (MOI) challenge (MOI = 0.1) re

145 e of F-pili from host cells, and that higher multiplicity of infection (MOI) correlates with detachme

146 t diversity has been defined in terms of the multiplicity of infection (MOI) derived by PCR-based gen

147 rotavirus (RRV) is serially passaged at high multiplicity of infection (MOI) in cells permitting high

148 ls of MIE promoter activity at a high or low multiplicity of infection (MOI) in human foreskin fibrob

149 However, infection at a low multiplicity of infection (MOI) in the presence of acycl

150 wide range of virus inoculum, ranging from a multiplicity of infection (moi) of 0.0005 to 0.05, with

151 on the dose of vector, ranging from 4% at a multiplicity of infection (MOI) of 0.1 to 99% at an MOI

152 l progeny production as much as 50-fold at a multiplicity of infection (MOI) of 0.5 and 80-fold at an

153 At a multiplicity of infection (MOI) of 1 at 24 h postinfecti

154 At a multiplicity of infection (MOI) of 1, viruses containing

155 The highest IL-8 secretion was at the multiplicity of infection (MOI) of 1,000:1 in bacterial

156 h more permissive to infection with SV5 at a multiplicity of infection (MOI) of 10 PFU/cell compared

157 ned from healthy individuals and primed at a multiplicity of infection (MOI) of 10(2) with different

158 evident only at ~72 hpi at the corresponding multiplicity of infection (MOI) of 10.

159 ematopoietic precursor cells infected with a multiplicity of infection (MOI) of 100 of AdGFP show tha

160 Macrophages infected with a multiplicity of infection (MOI) of 25:1 developed chroma

161 49, and RAW 264.7 cells when inoculated at a multiplicity of infection (MOI) of 3 PFU/cell.

162 ted with herpes simplex virus 1 (HSV-1) at a multiplicity of infection (MOI) of 3 with the majority o

163 that most N18 cells were infected by TE at a multiplicity of infection (MOI) of 50 to 500 and by 633

164 s were infected with the rAAV-CIP vectors at multiplicity of infection (MOI) of 5000, in the absence

165 After infection with 100 multiplicity of infection (MOI) of AdCat, cellular catal

166 st in the gastric mucosa, and because a high multiplicity of infection (MOI) of H. pylori is needed t

167 ntaneous gene targeting are dependent on the multiplicity of infection (MOI) of rAAV.

168 n RAW264.7 and THP-1 macrophages infected at multiplicity of infection (MOI) of ~1.0.

169 rus infection in vitro and the effect of the multiplicity of infection (MOI) on costimulatory ligand

170 that viruses can be transmitted at a higher multiplicity of infection (MOI) that, in vitro, results

171 015 to 3.0 for AAV, varying depending on the multiplicity of infection (MOI) used for transduction, a

172 ed depending on the cell line tested and the multiplicity of infection (MOI) used.

173 other approach, +LMN myocytes were infected (multiplicity of infection (MOI), 100; 24 h) with replica

174 phage-adapted chlamydiae correlates with the multiplicity of infection (MOI), and optimal chlamydial

175 f blood-stage parasites affects the observed multiplicity of infection (MOI), as well as the relation

176 efficiency than the wild-type virus at a low multiplicity of infection (MOI), but it grew similarly w

177 IE promoter-dependent transcription at a low multiplicity of infection (MOI), but this increase is no

178 conclude that the frequently used measure of multiplicity of infection (MOI), computed as the ratio o

179 ection with ICP0-null HSV-1 mutants at a low multiplicity of infection (MOI), so that individual plaq

180 are infected with the mutant virus at a low multiplicity of infection (MOI), there is a marked delay

181 ate in noncomplementing cells, even at a low multiplicity of infection (MOI), though a reduction in t

182 ication after infection at a relatively high multiplicity of infection (MOI).

183 saged at an escalating scale by using a high multiplicity of infection (MOI).

184 ly more slowly than wild-type virus at a low multiplicity of infection (MOI).

185 er than that of the wild-type virus at a low multiplicity of infection (MOI).

186 with herpes simplex virus 1 (HSV-1) at high multiplicity of infection (MOI).

187 creased amounts of trehalose with increasing multiplicity of infection (MOI).

188 tomas, we screened 12 parvoviruses at a high multiplicity of infection (MOI).

189 false-negative rates especially with a high multiplicity of infection (MOI).

190 ntegration efficiency as a function of viral multiplicity of infection (MOI); efficiency of site-spec

191 seasonal influenza A (H1N1) viruses at a low multiplicity of infection (MOI; 0.0001 PFU/cell) or with

192 le resistance, even to high titers of virus (multiplicity of infection [MOI] of 100).

193 that the number of viruses infecting a cell (multiplicity of infection [MOI]) influences the magnitud

194 ated and stimulated with live C. pneumoniae (multiplicity of infection [MOI], 5), UVCP (MOI, 5), or c

195 s and perfused human anterior segments using multiplicities of infection (MOIs) 25 or 50.

196 73.Stop) to replicate in cell culture at low multiplicities of infection (MOIs) and found that 73.Sto

197 ARM N and wild-type populations at different multiplicities of infection (MOIs) and initial ratios of

198 were passaged in Vero cells at high and low multiplicities of infection (MOIs) for 11 generations an

199 typhi invasion of INT407 cells at different multiplicities of infection (MOIs) have revealed a stric

200 control neutrophils (without antibiotic) at multiplicities of infection (MOIs) of 30 and 90 bacteria

201 thic effect was observed with macrophages at multiplicities of infection (MOIs) of as low as 20 and w

202 olated rat islets infected with AdCTLA4Ig at multiplicities of infection (MOIs) ranging from 0.1 to 1

203 than HMPV, with the latter requiring higher multiplicities of infection (MOIs) to yield similar leve

204 ght (0-1,000 J/m(2)) and infected at various multiplicities of infection (MOIs) with rAAV containing

205 is dispensable for virus replication at high multiplicities of infection (MOIs), analyses of plaque m

206 to monitor the killing of C. albicans at low multiplicities of infection (MOIs).

207 ult in cell death of most NPCs, even at high multiplicities of infection (MOIs); (ii) limited HSV-1 r

208 nd UV-inactivated C. pneumoniae cultures (at multiplicities of infection [MOIs] of 0.01, 0.1, and 1.0

209 must be grown to a low cell density and the multiplicity of infection must be low.

210 and 0.5% survival of LNCaP cells was seen at multiplicities of infection of 2, 10, and 25, respective

211 In contrast, the identical multiplicities of infection of Ad5-hTERT-E1 had no effec

212 Infection of HPMECs with increasing multiplicities of infection of an adenovirus encoding fo

213 lated neutrophils were infected with varying multiplicities of infection of both viruses, and opsonop

214 ffective concentration (EC50) ranging from a multiplicity of infection of 0.01 to 0.1.

215 days after exposure to all HIV-1 clades at a multiplicity of infection of 0.01.

216 On the other hand, G207 at a multiplicity of infection of 0.1 was able to purge bone

217 endent manner and showed 100% cytolysis at a multiplicity of infection of 0.1.

218 ing 1x10(9) plaque-forming units of Ad-LacZ (multiplicity of infection of 0.4), transduction rate in

219 ctosidase (Ad-LacZ) or E2F-1 (Ad-E2F-1) at a multiplicity of infection of 1 in vitro.

220 pe p53 gene, showed near complete lysis at a multiplicity of infection of 1.

221 on 5-40 h after infection of 9L cells with a multiplicity of infection of 1.5.

222 This response required a minimum multiplicity of infection of 10 and was not prevented by

223 e-positive after adenovirus infection with a multiplicity of infection of 10 for 60 minutes.

224 Furthermore, at a multiplicity of infection of 10, extraordinary sensitivi

225 The parietal cells were transduced with a multiplicity of infection of 100 of the adenoviral vecto

226 Transduction of the parietal cells with a multiplicity of infection of 100 of the adenoviral vecto

227 Infection of cultured rat aortic SMCs at a multiplicity of infection of 100 with AdCMV.hTIMP-2 resu

228 m C57BL/6 mice were infected with KA274 at a multiplicity of infection of 100, and transplanted into

229 rs after infection with AdCat or AdmCat at a multiplicity of infection of 100, intracellular catalase

230 5, which efficiently transduced islets at a multiplicity of infection of 100.

231 tion of 50, and a 71% decrease observed at a multiplicity of infection of 100.

232 heat-killed virulent M. tuberculosis H37Rv (multiplicity of infection of 1:100).

233 immature DCs with the lentiviral vector at a multiplicity of infection of 20 resulted in stable gene

234 f Cos1 cells with plaque-purified virus at a multiplicity of infection of 20-40 induced high expressi

235 miRNAs was modulated 3 h postinfection (at a multiplicity of infection of 25).

236 fluorescent protein) or Ad.betagal.GFP at a multiplicity of infection of 250 for 48 hours.

237 fluorescent protein (EGFP), or AdLMP2B at a multiplicity of infection of 250.

238 ry adapted) and TR (a clinical strain), at a multiplicity of infection of 3.

239 Adv-TGF-beta-R (4T1+Adv-TGF-beta-R) using a multiplicity of infection of 300.

240 re infected with C. pneumoniae (TW-183) at a multiplicity of infection of 3:1, and at 2 h postinfecti

241 ecin or to infection with influenza A virus (multiplicity of infection of 5).

242 The data show that an adenoviral multiplicity of infection of 50 transduces 100% of cultu

243 dependent, with a 49% decrease observed at a multiplicity of infection of 50, and a 71% decrease obse

244 2 gene transfer were obtained in IEC using a multiplicity of infection of 50.

245 Infection of tumor cells with 30 multiplicity of infection of AdCIFN-beta (but not contro

246 Cells infected with 10 or 100 multiplicity of infection of AdCIFN-beta, an adenoviral

247 after a 15-min exposure to H. ducreyi, and a multiplicity of infection of approximately 1 CFU per mac

248 HIT-LZ vector (multiplicity of infection of approximately 10) instilled

249 wo BmNPVs increased only marginally when the multiplicity of infection of each virus was increased 10

250 RNA levels when cells were stimulated with a multiplicity of infection of greater than 1 virulent M.

251 s has been optimized for incubation time and multiplicity of infection of invasive E.coli with HeLa c

252 ERK1/2 induction was observed even with low multiplicity of infection of live and UV-inactivated KSH

253 cle seed stocks could be amplified after low multiplicity of infection of PCLs, again without generat

254 pression of FFHA and GFP consistent with the multiplicity of infection of the adenovirus.

255 Phage were evolved by serial transfer at low multiplicity of infection on rapidly dividing bacteria t

256 In head-to-head infections at fixed multiplicities of infection, one SA13/JFH1orig mutant te

257 We observed that even at a very low multiplicity of infection, only 70% of the input virus s

258 en this mutant virus was delivered at a high multiplicity of infection or in the presence of the cell

259 bone marrow-derived in vitro cultures, high multiplicity of infection or the use of opsonized bacter

260 (v) High-multiplicity-of-infection passage of TR339 on BHK cell c

261 High multiplicity of infection passaging of both NiV clinical

262 when cells were infected at a relatively low multiplicity of infection, presumably due to the compoun

263 xpressing the human CAR cDNA (AdCAR) at high multiplicity of infection, primary fibroblasts were conv

264 rated by serial passage of the virus at high multiplicities of infection, provide important insight i

265 nfection with our recombinant virus at a low multiplicity of infection resulted in a substantial decr

266 npermissive phenotype is most evident at low multiplicities of infection, results in reduced accumula

267 g caution regarding the substantially higher multiplicity of infection seen in organ transplantation-

268 iated gene targeting increases with time and multiplicity of infection, similar to AAV-mediated gene

269 ion with a control Ad vector (Ad-LacZ) at 50 multiplicity of infection slightly increased CD40 and CD

270 g-passaged populations was reduced at higher multiplicities of infection, suggesting the presence of

271 ion only when the cells were infected at low multiplicity of infection, suggesting a defect in spread

272 and human embryonic lung (HEL) cells at all multiplicities of infection tested and was capable of es

273 harper decrease in viability with increasing multiplicities of infection than do cells treated with A

274 d normalized by the PreS1*-MOI, which is the multiplicity of infection that reflects the number of Pr

275 tudied in the RNA phage phi6 at high and low multiplicities of infection (that is, at high and low ra

276 At a high multiplicity of infection, the IE1 deletion mutant is co

277 irus propagation, such as time of infection, multiplicity of infection, the length of replication cyc

278 At a high multiplicity of infection, the UL21a deletion virus synt

279 hybrid viruses could be applied at very high multiplicities of infection to increase transduction rat

280 Using HeLa cells and conditions of low multiplicity of infection to favor use of the most avid

281 When applied at low multiplicity of infection to the apical surface of diffe

282 e marrow-derived murine macrophages at a low multiplicity of infection under conditions in which the

283 e and irradiated M. leprae, as a function of multiplicity of infection under permissive (33 degrees C

284 e of one strain (W3) in Vero cells at a high multiplicity of infection, under conditions of competiti

285 ession levels obtained were dependent on the multiplicity of infection used and the incubation time a

286 At low multiplicities of infection, viral gene expression in ra

287 ssays detected HDV replication even when the multiplicity of infection was 0.2 GE/cell.

288 12 months and at sick visits were assessed; multiplicity of infection was evaluated by PCR that targ

289 assortants, suggesting that a uniformly high multiplicity of infection was not achieved in vivo.

290 on normal mouse fibroblasts even when a high multiplicity of infection was used.

291 At a low multiplicity of infection, we observed only a small perc

292 High fungal:epithelial cell multiplicities of infection were able to rescue the cell

293 was detected in dl-L-infected mouse cells as multiplicities of infection were increased and approache

294 COS-7 monkey kidney cells, but higher viral multiplicities of infection were required.

295 In contrast to multiplicity of infection, which is based on titer and i

296 At low multiplicities of infection, wild-type virus yields are

297 y infected cell is concordant with the input multiplicity of infection, with each structure containin

298 lysis or latency, depending on the cellular multiplicity of infection within a broad class of gene r

299 yping demonstrated a significant decrease in multiplicity of infection within the first 2 days in bot

300 days of growth following adsorption at a low multiplicity of infection, yields of T3C84-MA and T3C84