戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 hat could be antagonized by cotreatment with muscarine.
2 tential (EPP) amplitude normally produced by muscarine.
3 beling techniques, are profoundly excited by muscarine.
4 amide to inhibit the contractile response to muscarine.
5 lar to that seen in response to bath-applied muscarine.
6 n sympathetic neurones was its modulation by muscarine.
7 ation of ACh to B neurones were unchanged by muscarine.
8 prevented the modulation of Ca2+ currents by muscarine.
9 d by CaN420 was different from the effect of muscarine.
10                                              Muscarine (10 microM) did not affect the SOR or the RD.
11                                              Muscarine (10 microM) inhibited N- and Q- but not R-, T-
12                                              Muscarine (10 microM) inhibited release by 70 +/- 3% (n
13 ogue 8-bromo-cAMP (1 mM), and application of muscarine (100 microM).
14                From dose-response curves for muscarine a mean dissociation constant of K(D) = 1.95 +/
15 neuromodulation was significantly altered as muscarine abolished respiratory network activity in alph
16                Immunocytochemistry utilizing muscarine acetylcholine receptor (mAChR) subtype-specifi
17  and CNQX, MK-801, or AP-7; ACh, nicotine or muscarine; ACh and alpha-bungarotoxin (Bgt) or methyllyc
18                         It is concluded that muscarine acts on M3 muscarinic acetylcholine receptors
19            The formal total synthesis of the muscarine alkaloids (-)-allomuscarine and (+)-epimuscari
20                                              Muscarine also depolarizes dentate granule cells and ele
21                                              Muscarine also increased Mn2+ quenching of intracellular
22 diated fast excitation, but were mimicked by muscarine and antagonized by atropine, indicating that i
23 ummary, we present a novel biphasic role for muscarine and implicate M3 receptors in the inhibition a
24 nd two muscarinic receptor (mAChR) agonists, muscarine and McN-A-343 (M1-selective), but not by nicot
25 tion between any changes in V(m) response to muscarine and morphology, as determined by reconstructio
26 s and classical muscarinic agonists, such as muscarine and oxotremorine, had higher affinity versus o
27         Efferent-mediated slow excitation or muscarine application enhanced the sensitivity of CD aff
28  channels.Cholinergic agents (ACh, nicotine, muscarine, bethanechol and pilocarpine) increased [Ca2+]
29                                 Dopamine and muscarine both increased the paired-pulse ratio of GABAA
30 ured KCs to PV IgG or the muscarinic agonist muscarine both induced changes in the expression of kera
31 e (by up to 29%; IC50 = 16 +/- 3 microM) and muscarine (by up to 41%; IC50 = 1.0 +/- 0.4 microM).
32            We found that bath application of muscarine caused a direct depolarization in approximatel
33                               Muscarine ((+)-muscarine chloride) reversibly inhibited the current wit
34 h, at a concentration of 100 nM, shifted the muscarine concentration-response curve to the right by a
35 evident under current clamp, where 10 microM muscarine depolarized granule neurons such that action p
36                                              Muscarine elevates the firing rate of inhibitory cells,
37 ne, serotonin, and the acetylcholine agonist muscarine, evoked direct inhibitory actions.
38                                         Peak muscarine-evoked rises of [Ca2+]i were variable, but the
39                          The SHNs excited by muscarine had a broad range of conduction velocities (0.
40 ethanechol depressed this excitation whereas muscarine had inconsistent effects.Atropine and C6 depre
41                                              Muscarine had no effect on EPSCs in C neurones.
42  the most significant effect of dopamine and muscarine in the STN is to reduce inhibitory synaptic in
43                                              Muscarine increased the coefficient of variation (c.v.)
44                                              Muscarine-induced activation of M3 receptors (0-12 min)
45 se C (PLC) inhibitor U-73122 all blocked the muscarine-induced Ca2+ signal but had little or no effec
46 ion of 2-AG to PGE2-G by COX-2 underlies the muscarine-induced enhancement of neurotransmitter releas
47                                          The muscarine-induced excitations in SHNs were found not to
48 eptor antagonist 4-DAMP effectively reversed muscarine-induced inhibition of IPSCs with an IC50 of 0.
49 ections of cholinergic agonists carbachol or muscarine into HVc strongly affected discharge rates and
50 ]i elevations arising out of Ca2+ influx and muscarine-mediated release of Ca2+ from intracellular st
51                                              Muscarine, methacholine, and arecaidine propargyl ester
52 this pathway specificity, the mAChR agonist, muscarine more potently suppressed transmission at input
53                                              Muscarine ((+)-muscarine chloride) reversibly inhibited
54 physiological and pharmacological effects of muscarine on antidromically identified septohippocampal
55                                The action of muscarine on IK(SO) was unaffected by the M2 receptor an
56 y type of hippocampal neuron, the effects of muscarine on SHNs would also have a profound effect on h
57 ive and antiallodynic actions of intrathecal muscarine or neostigmine in normal rats and in a rat mod
58  significantly attenuated the effect of both muscarine or neostigmine in normal rats.
59                     Intrathecal injection of muscarine or neostigmine significantly increased the wit
60                    The effect of intrathecal muscarine or neostigmine was examined through pretreatme
61                     Application of 20 microM muscarine produced a mean 78 +/- 1.4% inhibition of the
62  PEA), psychedelics (psilocybin, 5-MeO-DMT), muscarine receptor antagonists (scopolamine), and kappa
63 ocytotic vesicle fraction may have a role in muscarine receptor endocytosis.
64                    The data suggest that the muscarine receptor stimulated release of the alpha-synuc
65                         Targeted blockade of muscarine receptors over these neurons profoundly reduce
66                                 Bath-applied muscarine reduced the amplitude of EPSCs recorded at < 0
67  that in vitro gamma oscillations induced by muscarine require activation of M1 receptors on hippocam
68                                              Muscarine reversibly reduced the amplitude of GABAA IPSC
69 Central muscarinic cholinergic activation by muscarine, the M1 receptor agonist McN-A-343, and the M2
70 ine also reversed the inhibition of IPSCs by muscarine, this effect was only observed at relatively h
71 rent recordings revealed that application of muscarine to NB neurons induced a GIRK current, and this
72 ) receptors in the antinociceptive action of muscarine was also determined.
73                       Inhibition of IPSCs by muscarine was completely blocked by scopolamine (10 micr
74 odynic effect of intrathecal neostigmine and muscarine was largely eliminated by CGP55845 in diabetic
75 e and himbacine indicated that the effect of muscarine was mediated by the M(1) class of muscarinic r
76 tinociceptive action produced by intrathecal muscarine was significantly reduced by CGP55845 pretreat
77  in 0.5 mM Ca2+, but inhibition by 10 microM muscarine was significantly reduced.
78                            The inhibition by muscarine was unaffected by pre-incubation for 17-20 h w
79            The current and its inhibition by muscarine were unaffected by 100 microM Cd2+.
80                     These cells responded to muscarine with extended periods of excitation, received