ライフサイエンスコーパス: muscle contraction

1 II is the main force-generating motor during muscle contraction.

2 secretion, neuronal excitability, and smooth muscle contraction.

3 on as signaling platforms to regulate smooth muscle contraction.

4 al measurement of 5-HT overflow and circular muscle contraction.

5 sses ranging from intracellular transport to muscle contraction.

6 or processes as diverse as cell division and muscle contraction.

7 s, and whether these residues affect cardiac muscle contraction.

8 eded by long-duration waves of airway smooth muscle contraction.

9 s that reflect the intensity and duration of muscle contraction.

10 rated exercise pressor reflex in response to muscle contraction.

11 AVD vary with the intensity and duration of muscle contraction.

12 s system and are responsible for controlling muscle contraction.

13 ts that appear to be primarily influenced by muscle contraction.

14 cTnC), plays an important role in regulating muscle contraction.

15 with the intensity and duration of skeletal muscle contraction.

16 re dynamics, with important consequences for muscle contraction.

17 It is essential for controlling muscle contraction.

18 ysophosphatidic acid-induced vascular smooth muscle contraction.

19 eracts with troponin I and initiates cardiac muscle contraction.

20 as cell motility, embryonic development, and muscle contraction.

21 a(2+)) release channel required for skeletal muscle contraction.

22 uring a myosin transgene known to facilitate muscle contraction.

23 lved in blood pressure regulation and smooth muscle contraction.

24 nonambulatory postures with higher levels of muscle contraction.

25 three times greater than those possible via muscle contraction.

26 s system and are responsible for controlling muscle contraction.

27 eraction with actin, a process that leads to muscle contraction.

28 on of the muscle action potential crucial to muscle contraction.

29 , reduces the local vasodilatory response to muscle contraction.

30 the fundamental force-generating elements of muscle contraction.

31 ular Ca(2+) release via RyR1 during skeletal muscle contraction.

32 sin light chain, which induces airway smooth muscle contraction.

33 functions such as synaptic transmission and muscle contraction.

34 olvement of ZIPK in the regulation of smooth muscle contraction.

35 ated the receptor, inhibiting nerve-mediated muscle contraction.

36 uencies (50-100 Hz) that would induce strong muscle contraction.

37 ominant-negative effect of mutant protein on muscle contraction.

38 tion of Mb (SmbO2) and PmbO2 kinetics during muscle contraction.

39 lentivirus-mediated shRNA attenuated smooth muscle contraction.

40 ty of only a few motor units active during a muscle contraction.

41 sarcoplasmic reticulum to initiate skeletal muscle contraction.

42 capable of detecting and modifying body wall muscle contraction.

43 molecule in the regulation of human cardiac muscle contraction.

44 function as a regulator of blood flow during muscle contraction.

45 ular signaling, which is required for proper muscle contraction.

46 ose transporter 4 in response to insulin and muscle contraction.

47 tion coupling, which is essential for normal muscle contraction.

48 nderstanding cMyBP-C's modulation of cardiac muscle contraction.

49 gn receptor activity to circadian rhythm for muscle contraction.

50 ctivating RhoA and suppressing airway smooth muscle contraction.

51 ar functions, including cardiac and skeletal muscle contraction.

52 o, indicating that CHI3L1 is secreted during muscle contraction.

53 from motoneurons to muscle fibers to control muscle contraction.

54 hain, which is essential for vascular smooth muscle contraction.

55 eceptors (AChRs) are concentrated to control muscle contraction.

56 embers act elsewhere with roles unrelated to muscle contraction.

57 e troponin I switching mechanism of striated muscle contraction.

58 es and therapeutic interventions that target muscle contraction.

59 te activity in myenteric neurons and produce muscle contraction.

60 uce inflammation and inhibit vascular smooth muscle contraction.

61 ns (in the absence of ATP) and during active muscle contraction.

62 y resulting in twisted postures via abnormal muscle contraction.

63 s because they target upstream regulators of muscle contraction.

64 T are critical for the regulation of cardiac muscle contraction.

65 from mucosal secretion to regulating smooth muscle contraction.

66 tendon organs are most responsive to active muscle contractions.

67 d a particularly active cell associated with muscle contractions.

68 iffening of titin stretched during eccentric muscle contractions.

69 ogether generate appropriate intensities for muscle contractions.

70 aberrant patterning of peristaltic waves of muscle contractions.

71 of motoneuron recruitment to the strength of muscle contractions.

72 atment planning and the induction of intense muscle contractions.

73 ic (e.g., heel pad) mechanisms, or by active muscle contractions.

74 d generate situationally appropriate, graded muscle contractions.

75 n regulates the sensory feedback controlling muscle contractions.

76 rtical stimulations in motor cortex elicited muscle contractions.

77 arges reliably evoke contra-lateral hindlimb muscle contractions.

78 Glucocorticoids increased the heart rate and muscle contractions.

79 o electrically-induced intermittent hindlimb muscle contractions.

80 mately 20 min and consisted of a total of 40 muscle contractions.

81 tumor tissue and the unintended induction of muscle contractions.

82 ary twisting postures arising from sustained muscle contractions.

83 nd is regulated by adrenergic uterine smooth muscle contractions.

84 ing pathway and is independent of adrenergic muscle contractions.

85 bladder but not smooth muscle, and sustained muscle contractions.

86 The frequency of spontaneous muscle contractions (24 hpf) was significantly reduced b

87 is characterized by a weakness in voluntary muscle contraction, a direct consequence of greatly redu

88 o modify motoneuron output to achieve graded muscle contraction (actuation efferent).

89 TMEM16A control neuronal signalling, smooth muscle contraction, airway and exocrine gland secretion,

90 ng transcription, signaling, metabolism, and muscle contraction, all of which oscillate over the cour

91 However, no study has tested whether muscle contractions alone are sufficient to increase adu

92 Results demonstrate that muscle contractions alone, in absence of DG activation,

93 leather or parchment, stresses comparable to muscle contraction already occur at small osmotic pressu

94 eraction, oxidative phosphorylation, cardiac muscle contraction, Alzheimer's disease, Parkinson's dis

95 We found that skeletal muscle contraction amplifies vasodilatation to the endot

96 interneurons disrupted bilaterally symmetric muscle contraction amplitude, without affecting the timi

97 The pressor response to static muscle contraction and alpha,beta-methylene ATP (alphabe

98 precise control of neurotransmitter release, muscle contraction and cell excitability, among many oth

99 in cell metabolism and processes related to muscle contraction and cytoskeleton organization.

100 or neurons slow locomotion during egg-laying muscle contraction and egg release.

101 es with potential regulatory roles in smooth muscle contraction and extracellular matrix-receptor int

102 that are powered by turnover of ATP underlie muscle contraction and have key roles in motility of non

103 m synaptic plasticity, sensory transduction, muscle contraction and hormone release.

104 for mediating motor-based functions such as muscle contraction and intracellular transport.

105 sarcoplasmic reticulum to initiate skeletal muscle contraction and is associated with muscle disease

106 nitiates functional hyperaemia upon skeletal muscle contraction and is attenuated during ageing via a

107 These feedback loops from muscle contraction and mechano-transduction to the Ca(2+

108 variable and complex activity during active muscle contraction and muscle stretch that defy simple e

109 signalling to Ca(2+)-mediated events such as muscle contraction and neuronal excitability.

110 enes, activation of pathways associated with muscle contraction and physiology, and downregulation of

111 d to variable cytoplasmic strain produced by muscle contraction and relaxation, but their morphology

112 for the cooperative switching between heart muscle contraction and relaxation.

113 ) activation of myofilaments during striated muscle contraction and relaxation.

114 esses including epithelial transport, smooth muscle contraction and sensory processing.

115 erms of estimation, the proteins involved in muscle contraction and structure were preferentially enz

116 hat mirrors the well-known effects of PZQ on muscle contraction and tegumental disruption.

117 l-wide release of Ca2+, which in turn causes muscle contraction and the generation of the mechanical

118 e Z-band will help us understand its role in muscle contraction and the role of these proteins in the

119 l mechanism for the control of airway smooth muscle contraction and thus are a critical factor in air

120 l mechanism for the control of airway smooth muscle contraction and thus are a critical factor in air

121 ogether with the troponin complex, regulates muscle contraction and, along with tropomodulin and leio

122 rmal sensory feedback to control coordinated muscle contractions and body posture.

123 T mutations in Drosophila result in abnormal muscle contractions and cause embryo torsion.

124 creases the interval between systemic smooth muscle contractions and increases the rate of morphogene

125 r characterized by sustained or intermittent muscle contractions and its pathophysiological mechanism

126 The H-FIRE protocol substantially reduces muscle contractions and the therapy can be delivered wit

127 enriched with muscle-related terms such as 'muscle contraction' and 'muscle system process'.

128 responses, including dilatation to skeletal muscle contraction) and in relation to the spread of vas

129 gulation of myosin interaction with actin in muscle contraction, and in turn, promote better understa

130 e proliferator-activated receptor signaling, muscle contraction, and ion transport were already prese

131 es a highly conserved protein that regulates muscle contraction, and is a tumour-suppressor gene.

132 sin, the molecular motor that powers cardiac muscle contraction, and its accessory protein, cardiac m

133 ptor signaling, circadian exercise, striated muscle contraction, and lipid and carbohydrate oxidative

134 llular transport, signaling, cell migration, muscle contraction, and sensory perception.

135 sforming growth factor-beta, vascular smooth muscle contraction, and the hedgehog and Wnt signaling p

136 orphogenesis, the frequency of airway smooth muscle contraction, and the rate of developmental matura

137 mino acids from urine, periodically activate muscle contraction, and their adipocytes differentially

138 reflecting conserved functions in regulating muscle contraction, and they also contain significantly

139 hways, induce inhibition of the ileal smooth muscle contractions, and affect distinct physiological v

140 ological processes, including fertilization, muscle contraction, apoptosis, secretion, and synaptic p

141 results suggest that the Mb dynamics during muscle contraction are contributing factors to faster VO

142 Skeletal muscle contractions are initiated by an increase in Ca(2

143 Given the complexity by which muscle contractions are naturally controlled, we approac

144 Our findings demonstrate that muscle contractions, as a component of exercise, can dir

145 These data demonstrate that muscle contractions, as part of exercise, are sufficient

146 uce more predictable ablations and alleviate muscle contractions associated with IRE.

147 ment to understanding the connection between muscle contraction at the molecular and macro scale.

148 near nature of musculotendon systems, cyclic muscle contractions at the passive natural frequency of

149 ) channel that underlies skeletal and smooth muscle contraction, audition, hormone secretion and neur

150 he fact that single nonmuscle cell and whole-muscle contraction both follow a Hill-like force-velocit

151 sthetized and exposed to bilateral hind limb muscle contractions (both concentric and eccentric) via

152 Muscle contractions brought about by the excitation of m

153 al circuits that direct precise sequences of muscle contraction, but the identity and organizational

154 several methods to measure cardiomyocyte and muscle contraction, but these require customized hardwar

155 findings suggest that Plk1 regulates smooth muscle contraction by modulating vimentin phosphorylatio

156 e alpha2 subunit plays a key role in cardiac muscle contraction by regulating intracellular Ca(2+), w

157 Exercise/muscle contraction can enhance whole-body insulin sensit

158 activation of the motor neurons that induce muscle contraction can facilitate quantitative studies o

159 at increased shear stress, in the absence of muscle contraction, can acutely render the vascular endo

160 r characterized by sustained or intermittent muscle contractions causing abnormal movements and postu

161 ical phenomena, such as synaptic plasticity, muscle contraction, cell cycle, and circadian rhythms.

162 are key players in cytoskeletal remodeling, muscle contraction, cell migration and differentiation p

163 ering the coupling between hydrodynamics and muscle contraction characteristics, as well as the trade

164 s with increasing body size, consistent with muscle contraction characteristics; (3) the long-standin

165 more than 2,000 J/kg of specific work during muscle contraction, compared with just 40 J/kg for natur

166 nectin-dependent component of the integrated muscle contraction-dependent arteriolar vasodilatation i

167 Muscle contraction depends on release of Ca(2+) from the

168 Muscle contraction depends on the cyclical interaction o

169 n phosphorylation (RLC) necessary for smooth muscle contraction depends on the respective activities

170 Although the PmbO2 during muscle contraction did not affect the increased mVO2 in

171 ed as a negative control to confirm that the muscle contractions did not activate the hippocampus, an

172 that increased BP but changed PEP and smooth muscle contraction differently.

173 nisms to release energy more rapidly than by muscle contraction directly, thus amplifying power outpu

174 In response to skeletal muscle contraction during exercise, paracrine factors co

175 h generates propagating peristaltic waves of muscle contractions during crawling.

176 Recent evidence suggests muscle contractions during exercise release factors into

177 Muscle contraction, especially of lengthened muscle, dis

178 bid with altered cardiovascular responses to muscle contraction/exercise.

179 in regulating neuronal excitability, smooth muscle contraction, fluid secretion and gut motility.

180 of 1947 mRNAs encoding proteins involved in muscle contraction, focal adhesion, integrin, JAK/STAT,

181 eurologic disorder characterized by abnormal muscle contractions for which standard medical therapy i

182 fined and it may involve positive effects on muscle contraction force, microvascular O(2) delivery an

183 in's Ca(2+)-triggered regulation of striated muscle contraction has advanced greatly, particularly vi

184 The role of Plk1 in smooth muscle contraction has not been investigated.

185 that exogenously added carnosine can improve muscle contraction, has antioxidant activity, and can qu

186 unique functions of skeletal muscle such as muscle contraction have not been well examined.

187 ) regulate many physiological functions like muscle contraction, hormone secretion, gene expression,

188 Most encode proteins involved in muscle contraction; however, mutations in ion channel-en

189 SOCE) plays key roles in cell proliferation, muscle contraction, immune responses, and memory formati

190 ion, growth, and division in single cells to muscle contraction in animals.

191 M3-mAChR phosphorylation in bronchial smooth muscle contraction in health and in a disease state with

192 gia can regain volitional, graded control of muscle contraction in his paralyzed limb.

193 ected to most efficiently induce involuntary muscle contraction in nearby animals.

194 drenoreceptor, a GPCR that stimulates smooth muscle contraction in response to binding noradrenaline.

195 tal disorder characterized by loss of smooth muscle contraction in the bladder and intestine.

196 d in muscles and directly mediates sphincter muscle contraction in the defecation cycle in hermaphrod

197 Skeletal muscle contraction in these mice, however, was unaltered

198 This result supports a model for muscle contraction in which power output and efficiency

199 ly closed at rest and opened actively during muscle contractions in a process he termed 'capillary re

200 are generally linked with reduced levels of muscle contractions in chair-sitting postures and associ

201 skeletal muscle clock in vivo and a bout of muscle contractions in vitro.

202 showed a concentration-dependent decrease in muscle contraction, increase in heart rate, and accelera

203 equence decreased the dilatation to skeletal muscle contraction, indicating that there is a physiolog

204 activity in myenteric neurons during smooth muscle contractions induced by application of capsaicin,

205 Cyclic muscle contractions, induced by neural stimulation, driv

206 dentified distinct pathways linked to smooth muscle contraction, inflammatory cytokines, immune media

207 Smooth muscle contraction initiated by myosin regulatory light

208 Prior to muscle contraction, insulin action was compromised in my

209 Striated muscle contraction involves sliding of actin thin filame

210 ntinuous days), the amplified BP response to muscle contraction is alleviated in PAD.

211 ng of sensory-motor control, the dynamics of muscle contraction is an important determinant of moveme

212 ay hyper-responsiveness, and the strength of muscle contraction is determined by the frequency of osc

213 ay hyper-responsiveness, and the strength of muscle contraction is determined by the frequency of osc

214 The local vasodilatory response to muscle contraction is due in part to the activation of i

215 Muscle contraction is governed by tropomyosin (Tpm) shif

216 Together, these findings suggest that smooth muscle contraction is mediated by the recruitment of bet

217 umans, the vasodilatory response to skeletal muscle contraction is mediated in part by activation of

218 Heart muscle contraction is normally activated by a synchroniz

219 C (cMyBP-C) cause heart disease, its role in muscle contraction is not well understood.

220 Muscle contraction is powered by actin-myosin interactio

221 Myocardial depolarization leading to cardiac muscle contraction is reflected by the amplitude and dur

222 Striated muscle contraction is regulated by Ca(2+) -dependent mod

223 Muscle contraction is regulated by the movement of end-t

224 Striated muscle contraction is regulated by the translocation of

225 Striated muscle contraction is the result of sarcomeres, the basi

226 Skeletal muscle contraction is triggered by Ca(2+) release from t

227 Cardiac muscle contraction is triggered by calcium binding to tr

228 ABSTRACT: Airway smooth muscle contraction is typically the key mechanism underl

229 Airway smooth muscle contraction is typically the key mechanism underl

230 The sarcomere, the fundamental unit of muscle contraction, is a highly-ordered complex of hundr

231 The actin-myosin system, responsible for muscle contraction, is also the force-generating element

232 Increases in plasma insulin and exercise/muscle contraction lead to recruitment of additional MVU

233 us pathophysiology, which causes involuntary muscle contractions leading to abnormal movements and po

234 mplex and involves the mechanical effects of muscle contraction; local metabolic, red blood cell and

235 oth oxidant cascades have complex effects on muscle contraction, metabolic function and tissue perfus

236 However, whether the change in PmbO2 during muscle contraction modulates mVO2 and whether the O2 rel

237 Forces generated by heart muscle contraction must be balanced by adhesion to the e

238 At onset of muscle contraction, myoglobin (Mb) immediately releases

239 nsport, olfaction, phototransduction, smooth muscle contraction, nociception, cell proliferation and

240 During isometric tasks, muscle contractions occur without an actual movement and

241 Striated muscle contraction occurs when myosin thick filaments bi

242 both hyperreflexia and abnormal involuntary muscle contractions on hindlimbs.

243 ferential growth of tissues or forces due to muscle contraction or gravity.

244 retain proper tension and sensitivity during muscle contraction or relaxation.

245 eneral anesthesia and paralytics to minimize muscle contraction, or biphasic with sedation because th

246 pears to be attributable to the reduction in muscle contraction per se.

247 ta indicates that activation of TRPV4 during muscle contraction plays an important role in triggering

248 The mechanism, which is independent of muscle contractions, produces heat based on the activity

249 activity to generate appropriate patterns of muscle contractions, providing animals with the ability

250 ze and many acoustic characteristics because muscle contraction rate dictates the fundamental frequen

251 Functional vasodilatation in response to muscle contraction recovered at 35 days.

252 Inhibition of muscle contraction reduces growth in both day and night,

253 Vascular smooth muscle contraction-related genes were enriched in VR-PAH

254 epidermal microfilaments that are induced by muscle contractions relayed by actin-severing proteins a

255 Muscle contraction relies on the interaction of myosin m

256 adhesion, morphogenesis, mechanosensing, and muscle contraction rely on contractile actomyosin bundle

257 w a reversible reduction in the amplitude of muscle contractions, representing a surrogate marker for

258 s involved in metabolism, cardiac disorders, muscle contraction, reproduction, behavior, and signalin

259 These findings indicate that skeletal muscle contractions selectively amplify endothelium-depe

260 ralinear summation occurs and increases with muscle contraction speed.

261 Cyclic muscle contractions, spontaneous or electrically stimula

262 ograms that require multiple precisely timed muscle contractions, such as, for example, playing the p

263 mble motor neuron activity that induces fast muscle contraction, suggesting that eel high-voltage vol

264 monstrates that cilia activity combined with muscle contractions sustain the long-range transport of

265 essential biological processes that include; muscle contraction, synaptic transmission, hormone secre

266 eal a simple circuit of neurons controls the muscle contractions that allow fruit fly larvae to crawl

267 blood through the heart is due to a wave of muscle contractions that are in turn due to a wave of el

268 rd injury (SCI) are debilitating involuntary muscle contractions that have been associated with incre

269 is involved in regulation of vascular smooth muscle contraction through direct phosphorylation of LC2

270 used a recently discovered drug to eliminate muscle contraction, thus allowing prolonged intracellula

271 niches, regulating processes that range from muscle contraction to acidification of intracellular org

272 Perhaps MVT allows the load of muscle contraction to act as a signal to reorganize acti

273 number of biological processes, ranging from muscle contraction to co-translational folding.

274 PK is also a prerequisite for the ability of muscle contraction to increase insulin sensitivity.

275 ial for many cellular processes ranging from muscle contraction to learning and memory.

276 lls of Cajal (ICC) control intestinal smooth muscle contraction to regulate gut motility.

277 to drive essential cellular processes, from muscle contraction to vesicular transport.

278 synchronization of motor neuron activity and muscle contraction under optogenetic control for the stu

279 Flexibility in the bilateral coordination of muscle contraction underpins variable locomotor movement

280 MYLK, and PRKG1), a group called the smooth muscle contraction vasculopathies.

281 egulate myenteric neuron activity and smooth muscle contraction via a parasympathetic spinal circuit,

282 indicate that IL-17A promotes airway smooth muscle contraction via direct recruitment of Rab35 to IL

283 acetylation, especially Lys(328), modulates muscle contraction via disrupting inhibitory Tpm positio

284 optic nerve ultimately drive iris-sphincter-muscle contraction via excitatory cholinergic parasympat

285 The role of Pak in airway smooth muscle contraction was evaluated by inhibiting acetylcho

286 Electrical stimulation induced muscle contraction was significantly impacted in the cKO

287 17, a key player in the regulation of smooth muscle contraction, was downregulated by lipopolysacchar

288 light chain-20, a key regulator of lymphatic muscle contraction, was observed in insulin-resistant LM

289 ur experiments demonstrated that coordinated muscle contraction waves are associated with asymmetric

290 dings in humans maintaining steady isometric muscle contractions, we found evidence that the cortex s

291 ning period, deoxygenated Mb kinetics during muscle contraction were measured using near-infrared spe

292 genes related to the "immune response" and "muscle contraction" were altered in ATHs.

293 ntify a novel mechanism acting downstream of muscle contraction, where YAP activates JAG2 expression

294 regulates speed of plantarflexor (i.e., calf muscle) contraction, which influences economy of force p

295 This model predicts that the onset of muscle contraction will lead to the cessation of O2(-) p

296 Proteomics revealed one protein related to muscle contraction with significantly different relative

297 light chain (RLC) phosphorylation for smooth muscle contraction with subsequent dephosphorylation for

298 ics were estimated by combining the model of muscle contraction with validated models of lower limb m

299 production, storage of sperm, and changes in muscle contraction within the reproductive tract (RT).

300 , unidirectional lymphatic flow is driven by muscle contraction working in conjunction with valves.