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1 cidosis is being re-evaluated as a factor in muscle fatigue.
2 cal processes is causal to the use-dependent muscle fatigue.
3 n limb blood flow also accompany inspiratory muscle fatigue.
4 along with oxidative phosphorylation without muscle fatigue.
5 ource of nitric oxide and can delay skeletal muscle fatigue.
6 to mean frequency, a conventional measure of muscle fatigue.
7 a low-cost alternative method to measure hip muscle fatigue.
8 cise capacity and decreased time to skeletal muscle fatigue.
9  used an exerting squat-based task to induce muscle fatigue.
10 tion in conditions of weakness and premature muscle fatigue.
11 k of breathing-related changes in quadriceps muscle fatigue.
12 ts when attempting to understand and predict muscle fatigue.
13 nd can lead to muscle weakness and premature muscle fatigue.
14  response to 50 Hz stimulation and increased muscle fatigue.
15  experimental studies into the mechanisms of muscle fatigue.
16 cle pain and an inability to exercise due to muscle fatigue.
17 lactic acid accumulation are major causes of muscle fatigue.
18 D) is limited by both breathlessness and leg muscle fatigue.
19 equent prey movement by inducing involuntary muscle fatigue.
20 nce, membrane potentials, contractility, and muscle fatigue.
21 the thermal reaction norm of limpet adductor muscle fatigue.
22 tressors, from dystrophy to heart failure to muscle fatigue.
23 ractions can be sustained after the onset of muscle fatigue.
24 the physiological impairments that can cause muscle fatigue.
25 there is no global mechanism responsible for muscle fatigue.
26 it task failure rather than those that cause muscle fatigue.
27 ometric contraction would be expected as the muscle fatigued.
28  by reducing impairments in peripheral (i.e. muscle) fatigue.
29       Fast-timescale disturbances occur when muscles fatigue.
30 r exercise on: (1) exercise performance, (2) muscle fatigue, (3) capillarity, and (4) mitochondrial b
31  condition of fishlike body odor and chronic muscle fatigue, accompanied by elevated levels of the mu
32 of muscle has recently been shown to prevent muscle fatigue after exercise.
33                                              Muscle fatigue after ligation was related to the extent
34  be a major neural mechanism contributing to muscle fatigue and associated performance impairment.
35 CaMKII) and Ca(2+) store refilling to reduce muscle fatigue and atrophy.
36 ovements in objective measures of peripheral muscle fatigue and autonomic function, bringing them clo
37 provide a therapeutic opportunity to improve muscle fatigue and dysfunction in this population.
38  accumulation are two of the major causes of muscle fatigue and exhaustion.
39 en with acute severe asthma with respiratory muscle fatigue and failure of medical treatment.
40     Shorter-term treatment protected against muscle fatigue and increased mdx hindlimb muscle force b
41 luate: (1) the overall relationships between muscle fatigue and inorganic phosphate (Pi) and hydrogen
42 luated (1) the overall relationships between muscle fatigue and inorganic phosphate (Pi) and hydrogen
43 dvance our understanding of exercise-induced muscle fatigue and its role in muscle disease.
44  prey muscle preparations result in profound muscle fatigue and loss of contractile force.
45                 By describing the effects of muscle fatigue and recovery in terms of two phenomenolog
46 reased central motor output and end-exercise muscle fatigue and reduced endurance performance.
47                                              Muscle fatigue and subsequent recruitment of poorly effi
48 in but this strategy could lead to increased muscle fatigue and symptom aggravation in the long term.
49 the pivotal role of amino acid catabolism in muscle fatigue and type 2 diabetes pathogenesis.
50 ury, understanding the mechanisms underlying muscle fatigue and weakness has been the focus of much i
51 uscles, myofibrils, and myofibers identified muscle fatigue and weakness phenotypes, an increased rat
52 Ss) are increasingly recognized as causes of muscle fatigue and weakness.
53 O2 debt following exercise, a major cause of muscle fatigue, and a key factor in acidosis-induced tis
54 lactate] comparable to those observed during muscle fatigue, and accounted for this paradoxical conse
55  reliance on carbohydrate fuels, exaggerated muscle fatigue, and impaired endurance exercise capacity
56 f sarcoplasmic reticulum Ca(2+) stores, slow muscle fatigue, and increase running endurance without n
57 examining the relationship between IMU data, muscle fatigue, and multi-limb dynamics should be explor
58 uated by 73% (P = 0.012) in line with higher muscle fatigue by 26% (P = 0.079).
59 ase in cell pH) was thought to contribute to muscle fatigue by direct inhibition of the cross-bridge
60 norganic phosphate (P(i) ) may contribute to muscle fatigue by precipitating calcium salts inside the
61 n activator (FSTA), CK-2066260, can mitigate muscle fatigue by reducing the cytosolic free [Ca(2+) ]
62  hypothesized that CK-2066260 could mitigate muscle fatigue by reducing the energetic cost of muscle
63  that the FSTA CK-2066260 mitigates skeletal muscle fatigue by reducing the metabolic cost of force g
64  work was to re-evaluate the role of H(+) in muscle fatigue by studying the effect of low pH (6.2) on
65 e glycogen content in in situ stimulated rat muscles fatigued by repeated contractions at matching fo
66                                              Muscle fatigue can be defined as the transient decrease
67 t or obese, populations in which respiratory muscle fatigue can be limiting.
68  a visuomotor rotation, or internal, such as muscle fatigue, can create a difference between the moto
69 muscular Pi was more consistently related to muscle fatigue compared with H(+).
70  In a series of experiments, we describe how muscle fatigue, defined as degradation of maximum force
71 with a known level of pre-existing locomotor muscle fatigue (DeltaQ(tw,pot) -20%) (PFT(67%)-TT).
72 with a known level of pre-existing locomotor muscle fatigue (DeltaQ(tw,pot) -36%) (PFT(83%)-TT).
73 arkedly enhances endurance and resistance to muscle fatigue, despite reducing muscle force.
74 ion (OCC) to determine relationships between muscle fatigue development and motor unit activation dur
75                                 The roles of muscle fatigue development and motor unit activation in
76 ents limits CMD but also minimizes locomotor muscle fatigue development by stimulating adequate venti
77 sed NO(2) (-) exposure can mitigate skeletal muscle fatigue development.
78 ion, has been reported to attenuate skeletal muscle fatigue development.
79 nfluence on the rate of peripheral locomotor muscle fatigue development.
80 y," characterized by joint and muscle pains, muscle fatigue, difficulty lifting, and extremity parest
81  the working muscle in order that peripheral muscle fatigue does not exceed a critical threshold.
82 ns into the mechanisms underlying peripheral muscle fatigue due to energetic supply/demand mismatch a
83 a low-profile elastic exosuit to reduce back muscle fatigue during leaning, which may improve enduran
84 action analysis to assess loads on bones and muscle fatigue during simulation of surgical interventio
85 r the central effects of fatiguing locomotor muscle fatigue exert an inhibitory influence on central
86          However, the magnitude of locomotor muscle fatigue following various TTs was not different (
87 unit fatigue as a tractable means to predict muscle fatigue for a variety of tasks and to illustrate
88 pretations render a renewed understanding of muscle fatigue from a more unified motor control perspec
89          Increases in pre-existing locomotor muscle fatigue from control TT to PFT(83%)-TT resulted i
90 rlying the P(i)-induced loss of force during muscle fatigue from intense contractile activity.
91       These data demonstrate in humans, that muscle fatigue, generated in the initial minutes of exer
92  Taken together, in the absence of locomotor muscle fatigue, group III/IV-mediated leg muscle afferen
93 he fatigue task, suggesting that significant muscle fatigue had occurred.
94                                              Muscle fatigue has been known to differentially affect t
95 eceptor deficiency, and demonstrate improved muscle fatigue, improved neuromuscular transmission and
96 own to impair contractile function and cause muscle fatigue in dystrophic (mdx) mice.
97                              Despite greater muscle fatigue in individuals with spinal cord injury (S
98 nform interventions for mitigating lymphatic muscle fatigue in patients with dysfunctional lymphatics
99  between healthy controls and FM patients in muscle fatigue in response to exercise.
100  the FSTA CK-2066260 effectively counteracts muscle fatigue in rodent skeletal muscle in vitro, in si
101 sed as simple, static exercise to elicit hip muscle fatigue in the clinic, and that assessment of kne
102 s most consistently and effectively elicited muscle fatigue in the gluteus maximus, gluteus medius, a
103 ring ISC, despite adjustment for the greater muscle fatigue in this condition (P < 0.001).
104 reat mild to moderate anxiety, insomnia, and muscle fatigue in Western countries, leading to its emer
105       Additionally, JTV519 improved skeletal muscle fatigue in WT and calstabin-2-/- mice with HF by
106 or all limb muscles in 2 of 8 sparing bulbar muscles, fatigue in 9 of 10, mild proximal weakness in 3
107     The purpose of this study was to examine muscle fatigue-induced resting-state interhemispheric mo
108                                          How muscle fatigue influences these two types of cell popula
109 nce during physical exercise and exacerbated muscle fatigue is a prominent symptom among a broad spec
110                                              Muscle fatigue is a temporary decline in the force and p
111  present study investigated whether skeletal muscle fatigue is affected by the fast skeletal muscle t
112 utput, so that the development of peripheral muscle fatigue is confined to a certain level.
113 sible relevance of these effects to skeletal muscle fatigue is considered.
114 paired by the absence of GLUT4, and onset of muscle fatigue is hastened.
115 supports that the in vivo metabolic basis of muscle fatigue is similar across sexes, and that differe
116 n conclusion, in vivo the metabolic basis of muscle fatigue is similar between sexes.
117                           The development of muscle fatigue is typically quantified as a decline in t
118 unction during high-intensity exercise (i.e. muscle fatigue) is generally less in women than in men.
119 ry in the field, that lactic acidosis causes muscle fatigue, is unlikely to tell the whole story.
120                                     Skeletal muscle fatigue limits performance during physical exerci
121                                     Skeletal muscle fatigue limits performance in various physical ac
122 se due to dyspnea (n = 16) (as compared with muscle fatigue, n = 11) displayed weaker respiratory mus
123  When the mapping is perturbed, e.g., due to muscle fatigue or optical distortions, we are quickly ab
124 gnal changes of the brain and muscles during muscle fatigue processes induced by maximal voluntary co
125 IRL) exacerbated exercise-induced quadriceps muscle fatigue (Q(tw) = -12 +/- 8% IRL-CTRL versus-20 +/
126 1 with muscle power (r = 0.60, p = 0.02) and muscle fatigue (r = 0.57, p = 0.03).
127  co- contraction (R(2) = 0.63, p = 0.02) and muscle fatigue (R(2) = 0.64, p = 0.04).
128                 Surprisingly, however, while muscle fatigue reached a plateau, oxygen uptake continue
129 ce ( approximately 50%) and enhanced in situ muscle fatigue resistance ( approximately 30%) were obse
130 ncided with restoration to control levels of muscle fatigue resistance (P > 0.999), although overload
131 cute microsphere dose-dependent reduction in muscle fatigue resistance (P < 0.001), despite preserved
132         Smoking cessation increased skeletal muscle fatigue resistance (p < 0.001).
133 g cessation was accompanied with an improved muscle fatigue resistance and a reduction in low-grade s
134 CK-2066260 treatment also increased skeletal muscle fatigue resistance and exercise performance in a
135 SK3 inhibition in C57 mdx mice also improves muscle fatigue resistance and increases cage ambulation.
136 anical overload for, restoration of hindlimb muscle fatigue resistance and microvascular impairment i
137                          We demonstrate that muscle fatigue resistance is closely coupled with functi
138                                     Impaired muscle fatigue resistance was found after development of
139                                   Changes in muscle fatigue resistance were closely related to functi
140 ification of training responses, and altered muscle fatigue resistance.
141 ruvate lyase (NPL) was associated to in vivo muscle fatigue resistance.
142 ular metabolites differs between sexes (i.e. muscle fatigue sensitivity).
143                        Diaphragm and forearm muscle fatigue showed very similar time-dependent effect
144  The model provides a possible mechanism for muscle fatigue, suggesting that at low but nonzero glyco
145                                              Muscle fatigue tests and plasma analysis of biomarkers w
146  be the primary contributor to the premature muscle fatigue that is a hallmark of PVD.
147 ease the voltage delivered to prey, inducing muscle fatigue that turns challenging prey items into ea
148 wer for both the EEG and EMG activities with muscle fatigue, the fatigue weakens strength of brain-mu
149                                         With muscle fatigue, the locus of flow control resides in dis
150 that more knee wobble may be an indicator of muscle fatigue, this single IMU is not capable of reliab
151  measures could potentially be used to infer muscle fatigue under controlled conditions.
152 s and (2) determine the relationship between muscle fatigue using sEMG sensors and knee wobble using
153                                              Muscle fatigue was higher in both sexes following DEX, b
154                                Unexpectedly, muscle fatigue was unaffected by nNOS depletion, reveali
155  microcirculation and contribute to enhanced muscle fatigue, whereas formation of oxygen free radical
156 in brain and muscle signals during voluntary muscle fatigue, which may suggest weakening of functiona
157 ndgrip contractions that induced significant muscle fatigue, with resting state fMRI data collected b
158                We tested the hypothesis that muscle fatigue would attenuate vasodilatory responsivene
159 d oxidative ATP synthesis, and (2) ischaemic muscle fatigue would be related to the accumulation of i
160                         We hypothesized that muscle fatigue would create a temporary "disrupted state

 
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