ライフサイエンスコーパス: muscle pain

1 erish, two headache, one joint pain, and one muscle pain).

2 ns, prevents development of activity-induced muscle pain.

3 y systemic symptoms such as fever, bone, and muscle pain.

4 in modulation can also contribute to chronic muscle pain.

5 target for potential drug therapies to treat muscle pain.

6 els of peripheral neuropathy to evaluate for muscle pain.

7 nd transient, mild lower-extremity edema and muscle pain.

8 ot appear to modulate acute exercise-induced muscle pain.

9 nd was free of muscle weakness, and bone and muscle pain.

10 y accepted as a causal mechanism for chronic muscle pain.

11 tions of the CeA in a mouse model of chronic muscle pain.

12 to summarize neural pathways of craniofacial muscle pain.

13 area) and higher sensitivity of the masseter muscle pain.

14 ted acid injections also affect craniofacial muscle pain.

15 fibrosis (2.41, 1.37 to 4.25, P=0.002), and muscle pain (1.89, 1.63 to 2.20, P<0.001); and also for

16 (47.6% vs. 35.6%), fatigue (18.4% vs. 6.3%), muscle pain (23.1% vs. 10.1%), memory loss (29.2% vs. 4.

17 d fever (temperature >38.5 degrees C), 7 had muscle pain, 6 had sinusitis, and 6 had biopsy evidence

18 (1832 [25.4%]), fatigue (1361 [18.9%]), and muscle pain (942 [13.1%]) were the most commonly reporte

19 Thus, induction of muscle pain activates the immune system with sex-specifi

20 I signaling was activated after induction of muscle pain; activation of this pathway was blocked by p

21 atients with PCC had greater exacerbation of muscle pain after HIIT (mean [SD] VAS ranks, 33.4 [17.7]

22 al underlying reasons for the comorbidity of muscle pain and altered sympathetic reflexes in disease

23 rome (CFS) patients often suffer from severe muscle pain and an inability to exercise due to muscle f

24 usible that this finding partly explains the muscle pain and exercise intolerance that many patients

25 us infection can cause mild fever, rash, and muscle pain and in rare cases can lead to brain or nervo

26 Results could uncover new ways to treat muscle pain and inflammation in neonates.

27 development of alternative therapeutics for muscle pain and may be especially relevant to pain cause

28 Muscle pain and weakness are the cardinal symptoms and o

29 Recovery occurred within 24 hrs, although muscle pain and weakness persisted for 2 months after st

30 wn statin-like lowering of LDL-C without the muscle pain and weakness responsible for discontinuation

31 the frequency of less-serious events such as muscle pain and weakness, which may affect 1% to 5% of p

32 ducing extravascular manifestations, such as muscle pains and stiffness diagnosed as polymyalgia rheu

33 derate levels of experimental sustained deep muscle pain, and a baseline, control state.

34 elm, Lethargy, Increased Appetite, Joint and Muscle Pain, and Breast Tenderness, reaching 50% of peak

35 ndition (PCC) experience persistent fatigue, muscle pain, and cognitive problems that worsen after ex

36 Gastrointestinal symptoms, muscle pain, and fever markedly positively correlated wi

37 adache, nausea and vomiting, abdominal pain, muscle pain, and joint pain.

38 malaise, liver disease, acute kidney injury, muscle pain, and neurocognitive impairment.

39 ne pathways in both sexes after induction of muscle pain, and these pathways were attenuated in physi

40 A prevalent side effect of statin therapy is muscle pain, and yet the basic mechanism behind it remai

41 trategies behind the control of force during muscle pain are not well understood as previous research

42 ates motor unit behaviour in the presence of muscle pain at high forces.

43 Many clinicians believe that statins cause muscle pain, but this has not been observed in clinical

44 le of the cAMP pathway in a model of chronic muscle pain by assessing associated behavioral changes a

45 ical role in development of activity-induced muscle pain by modulating release of inflammatory cytoki

46 ecessary for development of activity-induced muscle pain by promoting release of pro-inflammatory cyt

47 resolve within a few days, chronic joint and muscle pain can be long lasting.

48 was found at the ankle and on the arm during muscle pain compared to the control condition.

49 e TA muscle were found during saline-induced muscle pain compared to the infusion of isotonic saline.

50 symptoms of post-traumatic stress disorder, muscle pain, concentration problems, and headache.

51 ia potentially enhances treatment of chronic muscle pain conditions.

52 ns in blood flow/oxygenation and a number of muscle pain disorders are based in problems of periphera

53 central command modulates the perception of muscle pain during exercise.

54 nemius was harvested 24hr after induction of muscle pain for RNA sequencing or flow cytometry.

55 ling feverish, repeated shaking with chills, muscle pain, headache, sore throat, and new loss of tast

56 Symptoms of headache, fatigue, joint pain, muscle pain, hearing loss, visual loss, numbness of hand

57 nemius muscle strongly enhanced acid-induced muscle pain in mice that was abolished by genetic inacti

58 bubble gum for 6 min) increases masticatory muscle pain in patients, but not in asymptomatic control

59 a pro-inflammatory state after induction of muscle pain in sedentary mice (M1 + M1/2) but toward an

60 males and seven females, with no history of muscle pain in the masticatory system participated in th

61 ), headache (in 25.1% and 31.9%, vs. 16.9%), muscle pain (in 22.3% and 26.9%, vs. 13.3%), feverishnes

62 Induction of muscle pain increased the number of macrophages and T-ce

63 Activity-induced muscle pain increases interleukin-1beta (IL-1beta) relea

64 Craniofacial muscle pain is highly prevalent in temporomandibular dis

65 ns, motor unit discharge rates decrease when muscle pain is induced by injecting nociceptive substanc

66 t study was to determine if exercise-induced muscle pain is modulated by central neural mechanisms (i

67 y include fever, headache, nausea, vomiting, muscle pain, lack of appetite, and rash.

68 ults support the suggestion that neuropathic muscle pain may be a greater clinical problem than gener

69 ce for the involvement of Vi in craniofacial muscle pain mechanisms.

70 Prior to induction of muscle pain, mice (C57/BL6) were sedentary or physically

71 Evidence of muscle disease was indicated by muscle pain, muscle weakness and wasting, significant fa

72 -myo-neuropathy," characterized by joint and muscle pains, muscle fatigue, difficulty lifting, and ex

73 s, the placebo-corrected incidences of minor muscle pain, myopathy (with significant elevations in cr

74 , fatigue and malaise, acute kidney disease, muscle pain, neurologic system (neurocognitive impairmen

75 CWI did not improve chronic perceived muscle pain nor reduce circulating markers of muscle dam

76 The effect of experimental muscle pain on the amplitude and velocity sensitivity of

77 Muscle pain or discomfort lasting several days (primary

78 ur hypothesis, CWI did not improve perceived muscle pain or reduce circulating markers of muscle dama

79 fects of the statin-are myopathy (defined as muscle pain or weakness combined with large increases in

80 Myopathy--muscle pain or weakness with blood creatine kinase level

81 py may cause symptomatic adverse events (eg, muscle pain or weakness) in up to about 50-100 patients

82 elling, erythema, fatigue/malaise, headache, muscle pain, or fever was higher in individuals boosted

83 ay result in widespread deep hyperalgesia in muscle pain patients.

84 Ratings of muscle pain perception (MPP) and perceived exertion (RPE

85 with symptoms that include severe joint and muscle pain, rashes, and fever, as well as prolonged per

86 afferent function, leading to modulation of muscle pain-related behaviors and EPRs after BAO.

87 ll described in skeletal muscle, neuropathic muscle pain remains to be clarified.

88 Nonetheless, the role of SP in muscle pain remains unknown.

89 headache, reported by 14 (58%), followed by muscle pain reported by 13 (54%).

90 Muscle pain scores were higher at baseline in symptomati

91 nding of neurobiology unique to craniofacial muscle pain should lead to the development of novel mech

92 ponse pattern may be associated with chronic muscle pain states.

93 were neutropenia (38 [35%] of 110 patients), muscle pain (ten [9%]), rash (eight [7%]), cough, dyspno

94 HWI induced a lower perceived muscle pain than TWI (P = 0.035) and lower levels of cir

95 The ability of muscle pain to generate somatosensory sensibility change

96 Induction of muscle pain triggers a local immune response to produce

97 ed by mild muscle weakness, exercise-induced muscle pain, variable susceptibility to episodes of rhab

98 In contrast, carrageenan-induced muscle pain was prevented in both Cx3cr1(Cre+)/ASIC3(fl/

99 Muscle pain was produced via an activity-induced pain mo

100 The inhibitory mechanism during muscle pain was shown to be effective for the deep tissu

101 Because chewing-induced masticatory muscle pain was significantly greater in female control

102 Patients experience muscle pain, weakness, fatigue, and atrophy, but the und

103 notable for cognitive deficits, depression, muscle pain, weakness, intolerance to exercise, and fati

104 Mild cold exacerbation and significant muscle pain were equally common features in dominant and

105 hyperalgesia, and susceptibility to chronic muscle pain, which is unveiled by exposure to stress in

106 sh, fever episodes, arthralgia, and bone and muscle pain with cryopyrin-associated periodic syndromes

107 purposes, such as the fever, skin diseases, muscle pains, wounds and stomach problems.