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1 omic diversity among strains of Helicobacter mustelae.
2 s to 1.67 Mb for H. pylori and 1.7 Mb for H. mustelae.
3 ans was observed in ferrets infected with H. mustelae.
4 ization and persistence of infection with H. mustelae, 19 ferrets, specific pathogen free for H. must
5                                 Helicobacter mustelae, a gastric pathogen of ferrets, synthesizes a d
6 alpha2-6-sialyltransferase, and Helicobacter mustelae alpha1-2-fucosyltransferase, as efficient tools
7 ever, in the absence of serum, H. pylori, H. mustelae, and H. muridarum became sensitive to polymyxin
8  the 1.2:1 kappa lambda ratio observed in H. mustelae-associated chronic gastritis.
9 ed with Helicobacter pylori and Helicobacter mustelae by direct analysis of individual cultured colon
10 oduced by infection with these strains of H. mustelae correlated with the number of organisms present
11  for colonization and pathogenesis in the H. mustelae ferret model.
12   Sequencing of these enlarged genes from H. mustelae, H. canis (two strains) and H. muridarum identi
13  the gastric helicobacters, H. pylori and H. mustelae, H. hepaticus possesses a high level of urease
14 obacters (H. canis, H. cineadi, H. felis, H. mustelae, H. nemestrinae, H. pullorum, H. pylori, and H.
15                Isogenic mutant strains of H. mustelae have been constructed by disruption of the flaA
16 corresponding to the predominant focus of H. mustelae induced gastritis in ferrets.
17           The wild-type parent strains of H. mustelae infected all ferrets at all time points.
18                                           H. mustelae infection in ferrets has been used as a model f
19                                           H. mustelae infection status was determined by culture, his
20 ion with Helicobacter pylori or Helicobacter mustelae is associated with an increased risk of gastric
21                                 Helicobacter mustelae, like Helicobacter pylori, possesses two flagel
22    Scant colonization by the Hsr-negative H. mustelae strain at the end of the 18-week study, despite
23 n in the animals that received the mutant H. mustelae strain, a finding consistent with its waning in
24 -deficient mutant strain or the wild-type H. mustelae strain.
25                                 Helicobacter mustelae, the gastric pathogen of ferrets, produces an a
26 an the nickel-enzyme, its activity allows H. mustelae to survive the carnivore's low-nickel gastric e
27 ylori was centered around m/z 58,268, and H. mustelae was distinguished from H. pylori by its ions at
28 omach, ferrets specific pathogen free for H. mustelae were inoculated with an Hsr-deficient mutant st
29 e, 19 ferrets, specific pathogen free for H. mustelae, were given either the HMF1 flaA::km (weakly mo