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1 eased TGF-beta expression and restoration of myelopoiesis.
2 erentiation, whereas its activation enhanced myelopoiesis.
3 a transcription factor that is essential for myelopoiesis.
4 cking this pathway was sufficient to enhance myelopoiesis.
5 n, an miRNA gene known to play a key role in myelopoiesis.
6 ansposition and impaired spermatogenesis and myelopoiesis.
7 ce also drove type I IFN-dependent emergency myelopoiesis.
8 hibition of miR-9 with a miRNA sponge blocks myelopoiesis.
9 p-regulation of FoxO3 inhibits miR-9-induced myelopoiesis.
10 volved in orchestrating inflammation-induced myelopoiesis.
11 ative disease with splenomegaly and aberrant myelopoiesis.
12 lon (C/EBPepsilon) is a critical mediator of myelopoiesis.
13 artment fails to induce LPS-driven emergency myelopoiesis.
14 he sequelae of events that led to MI-induced myelopoiesis.
15 hocyte development and is also implicated in myelopoiesis.
16 pression, enhanced self renewal and expanded myelopoiesis.
17 ral shortcomings, one of which is poor human myelopoiesis.
18 r-binding protein-alpha up-regulation during myelopoiesis.
19 to a loss of Notch-dependent suppression of myelopoiesis.
20 ts and myelocytes, as well as extramedullary myelopoiesis.
21 ease in B lymphopoiesis, and an elevation in myelopoiesis.
22 ced alpha2,6-sialylation results in elevated myelopoiesis.
23 SBP protects against the genotoxic stress of myelopoiesis.
24 liferate and produce progeny for accelerated myelopoiesis.
25 presses HoxA9, Pbx1, and Meis1 during normal myelopoiesis.
26 gulatory T cells regulate the extramedullary myelopoiesis.
27 link between HSPC mobilopathy and excessive myelopoiesis.
28 w insights into regulation of extramedullary myelopoiesis.
29 topoietic stem cells could support long-term myelopoiesis.
30 InsP3KB is also a physiological modulator of myelopoiesis.
31 e progressive growth in our understanding of myelopoiesis.
32 d DUSP4 activation by HoxA10 decrease during myelopoiesis.
33 e for leptin in sustaining lymphopoiesis and myelopoiesis.
34 nding to the DUSP4 promoter decreases during myelopoiesis.
35 show that their interaction also suppressed myelopoiesis.
36 AML, which dephosphorylate HoxA10 throughout myelopoiesis.
37 ge progenitor stage and continues throughout myelopoiesis.
38 ation provides important insight into normal myelopoiesis.
39 cooperates with C/EBPalpha to regulate early myelopoiesis.
40 ibutes to inappropriate cell survival during myelopoiesis.
41 Here, we show a role for Rbm15 in myelopoiesis.
42 d IL-3 also responded to IL-7 with increased myelopoiesis.
43 or (IRF)-8 plays an important role in normal myelopoiesis.
44 n and Mkp2 expression decrease during normal myelopoiesis.
45 gene transcription at various points during myelopoiesis.
46 IL-6, and IL-3) shifted the cultures toward myelopoiesis.
47 ession of CYBB and NCF2 transcription during myelopoiesis.
48 ing proteins (C/EBPs) play critical roles in myelopoiesis.
49 hopoiesis and a corresponding enhancement of myelopoiesis.
50 ulatory element) during normal and leukaemic myelopoiesis.
51 loid leukemia (AML) and results in defective myelopoiesis.
52 icroenvironmental) age-related changes favor myelopoiesis.
53 e results establish a critical role of PS in myelopoiesis.
54 cytic leukemic cells and during normal human myelopoiesis.
55 e stem cell genes, such as scl, in governing myelopoiesis.
56 actor JunB is a transcriptional regulator of myelopoiesis.
57 s by causing imbalances in lymphopoiesis and myelopoiesis.
58 L-RARA, initially has only modest effects on myelopoiesis.
59 ulation of HoxA10 repression activity during myelopoiesis.
60 e is known regarding GSL expression in early myelopoiesis.
61 C/EBPepsilon, have important roles in normal myelopoiesis.
62 expression of PU.1, a critical regulator of myelopoiesis.
63 iation and suggest it is required for normal myelopoiesis.
64 vae, enforcing erythropoiesis and inhibiting myelopoiesis.
65 tation of the hematopoietic system in stress myelopoiesis.
66 they inhibited B lymphopoiesis and enhanced myelopoiesis.
67 wnstream genes and consequently deregulating myelopoiesis.
68 abolic reprogramming underlies M-CSF-induced myelopoiesis.
69 on and G-CSF- or inflammation-induced stress myelopoiesis.
70 suggesting a mechanism by which NE regulates myelopoiesis.
71 thereby constituting a feed-forward loop for myelopoiesis.
72 viour in mice during emergency and leukaemic myelopoiesis.
73 bolism as a central node in mTORC1-dependent myelopoiesis.
74 nonhematopoietic niche that controls normal myelopoiesis.
75 E or C/EBPalpha are resistant to HFD-induced myelopoiesis.
76 and this pathway was essential for emergency myelopoiesis.
77 the marrow, skewing toward the expansion of myelopoiesis.
78 o rapidly altered stem cells homeostasis and myelopoiesis.
79 tumor-derived soluble factors able to alter myelopoiesis.
80 ic shuttling activity in normal and leukemic myelopoiesis, a mutant defective in nuclear export was e
82 angioblast specification, and for subsequent myelopoiesis, acting as early as cloche and upstream of
83 demonstrated that norepinephrine stimulated myelopoiesis after burn injury and sepsis, but the site
85 -skewing potential from lymphopoiesis toward myelopoiesis, an increase in the long-term-HSC pool, and
86 iferation and expansion, which prevented the myelopoiesis and accelerated atherosclerosis of ApoE(-/-
87 of neutropenia with an intrinsic failure of myelopoiesis and an increase in the incidence of cytogen
89 TLR7.1 mice exhibited hallmarks of emergency myelopoiesis and contained a discrete population of Sca-
91 m a regulatory axis that specifies primitive myelopoiesis and definitive hematopoiesis, but not primi
94 HSC gene expression program is biased toward myelopoiesis and differentiation skewed toward granulocy
95 mammalian hematopoiesis, embryonic zebrafish myelopoiesis and erythropoiesis occur in anatomically se
98 restricted and stage-specific pattern during myelopoiesis and functions to promote monocyte different
100 or a distinct function of IL7Ralpha in fetal myelopoiesis and identify IL7R as a novel regulator of t
104 s HSCs via an IFN-I response that suppresses myelopoiesis and impairs development of protective train
105 ous unrecognized significant role for ACE in myelopoiesis and imply new perspectives for manipulating
106 d to play a role in vascular development and myelopoiesis and in the inflammatory responses of granul
107 ese results reveal a unique role of miR-9 in myelopoiesis and in the pathogenesis of EVI1-induced mye
110 summary, SMARCD2 is a key factor controlling myelopoiesis and is a potential tumor suppressor in leuk
112 n factor C/EBPalpha is a master regulator of myelopoiesis and its inactivation is associated with acu
113 a critical transcription factor involved in myelopoiesis and its inactivation is associated with acu
115 l the SPI1-METTL14-MYB/MYC signaling axis in myelopoiesis and leukemogenesis and highlight the critic
118 core binding factor (CBF) family stimulates myelopoiesis and lymphopoiesis by activating lineage-spe
120 ls does not result in strict separation into myelopoiesis and lymphopoiesis, and that there might be
126 ssion of miR-9 strongly accelerates terminal myelopoiesis and promotes apoptosis in vitro and in vivo
129 usly unknown cytokine-RAR interaction during myelopoiesis and suggest that RAR activation might be a
130 a and systemic exposure to SAA can influence myelopoiesis and susceptibility to amebiasis via epigene
131 (-/-) mice significantly reduced bone marrow myelopoiesis and systemic CD11b(+)Ly6G(+) cell expansion
132 ights an important role for SMARCD2 in human myelopoiesis and the curative effect of hematopoietic st
133 ve a previously unknown function in limiting myelopoiesis and the development of dendritic cells.
134 nsient effects of the monoclonal antibody on myelopoiesis and the more persistent effects on lymphopo
137 that can serve as a model for the failure of myelopoiesis, and dissection of its pathogenesis has yie
138 viability, B lymphopoiesis, and bone marrow myelopoiesis, and is a potent monocyte and T-lymphocyte
139 s known to inhibit lymphopoiesis and elevate myelopoiesis, and its expression was MAPK dependent.
140 sulted in enhanced embryonic erythropoiesis, myelopoiesis, and lymphopoiesis, including a 2- 3-fold i
141 ntifies IL-3 as an orchestrator of emergency myelopoiesis, and reveals a new therapeutic target for t
142 emonstrate that Tet2 is important for normal myelopoiesis, and suggest that disruption of TET2 enzyma
143 program that underlies both human and murine myelopoiesis, and that is central to the pathogenesis of
151 factors that have important roles in normal myelopoiesis as well as associated with myeloid disorder
152 ignaling was essential for microbiota-driven myelopoiesis, as microbiota colonization or transferring
153 asma cells (PCs) to age-related increases in myelopoiesis, as PCs exhibit immune regulatory function
155 macrophages are essential to extramedullary myelopoiesis because these macrophages use the adhesion
156 icroRNAs whose regulation is required during myelopoiesis, but also provide an example of synergy in
159 y ST6Gal-1, mostly of hepatic origin, limits myelopoiesis by a mechanism independent of hepatic sialy
161 n negatively regulate splenic extramedullary myelopoiesis by suppressing the naive T cell differentia
162 We also show that MBT-1 appears to influence myelopoiesis by transiently enhancing p57(KIP2) expressi
163 llowed by specification to hematopoiesis and myelopoiesis by vascular endothelial growth factor and h
164 gest that DC differentiation, during induced myelopoiesis, can be regulated by the nature of the Notc
165 effects result from trained immunity-induced myelopoiesis caused by epigenetic rewiring of multipoten
166 hat Kit(W-sh) causes aberrant extramedullary myelopoiesis characterized by the expansion of immature
168 nt (TR) in the zebrafish results in impaired myelopoiesis, despite normal development of haematopoiet
169 o bias hematopoietic progenitor cells toward myelopoiesis directly by replacing cytokine and differen
170 the suppressive effects of Notch ligands on myelopoiesis, do not transcribe Notch1 target genes when
172 r Erk2 disruption did not grossly compromise myelopoiesis, dual Erk1/2 disruption rapidly ablated gra
174 idence that LAL is an important regulator of myelopoiesis during hematopoietic development, different
175 addition to stimulatory effects of G-CSF on myelopoiesis, endogenous G-CSF suppressed development of
176 c mice displayed enhanced marrow and splenic myelopoiesis: greatly increased progenitor cell cycling
178 tivities for these C/EBP-epsilon isoforms in myelopoiesis, human CD34(+) progenitors were transduced
179 eloid cell autonomous defect led to abnormal myelopoiesis, immune suppression, and lung adenocarcinom
181 n of several genes activated during terminal myelopoiesis in BM cells and identified a group of them
183 myeloid cell-intrinsic PD1 signaling limits myelopoiesis in cancer pertinent to anti-PD1 therapies.
188 with NF-kappaB p65, we have now investigated myelopoiesis in nfkb1(-/-) mice lacking NF-kappaB p50.
191 pectedly, in vivo ablation of pDCs increased myelopoiesis in the bone marrow and specifically induced
192 ced chronic arthritis, severe bone loss, and myelopoiesis in the bone marrow and spleen, which result
193 oid cells of lal(-/-) mice reversed abnormal myelopoiesis in the bone marrow starting at the granuloc
195 e data identify BCAP as a novel inhibitor of myelopoiesis in the steady state and of emergency myelop
196 il strategies to reprogram immunosuppressive myelopoiesis in tumors to boost cancer immunotherapy.
198 ght important regulatory effects of IL-33 on myelopoiesis in vitro and in vivo, where excessive IL-33
199 cate that Notch-dependent signaling controls myelopoiesis in vivo and in vitro and identifies a requi
200 t to Dll1, Jag1, in vitro and during induced myelopoiesis in vivo, prevented DC differentiation by pr
202 g lymphoid development and suppressing overt myelopoiesis, in part through processes controlled by O-
206 L-ENL-induced AML but dispensable for normal myelopoiesis, indicating a specific requirement for Hhex
207 essive AML in vivo without preventing normal myelopoiesis, indicating that strategies to inhibit Myb-
208 myeloid differentiation, known as emergency myelopoiesis, involves recognition of pathogen-associate
213 idence for their participation in modulating myelopoiesis is much less clear, and roles for posttrans
221 ause IFN-gamma-deficient mice also increased myelopoiesis, it was suggested that IL-7 induced product
222 hrombocytosis, and a marked dysregulation of myelopoiesis, leading to an important increase in myeloi
223 On transfer to IL-3, IL-6, and SCF to induce myelopoiesis, levels of granulocytic RNAs are reduced an
225 considered the alternative possibility that myelopoiesis may be regulated not by the hepatic sialyl
226 etic nervous system-induced up-regulation of myelopoiesis mediates the proinflammatory component of t
227 and myeloid progenitor cells toward enhanced myelopoiesis might also perpetuate inflammation in chron
228 ence of JAK2(V617F)) and putative pattern of myelopoiesis (monoclonal versus polyclonal), it is yet t
233 w that IFN-gamma-dependent infection-induced myelopoiesis occurs via the direct effect of the cytokin
234 ctivator B cells produce IL-3, which induces myelopoiesis of Ly-6C(high) monocytes and neutrophils an
235 tem cells and at more moderate levels during myelopoiesis of murine cell lines and primary murine cel
237 evelopment in the thymus but did not inhibit myelopoiesis or natural killer (NK) cell development in
238 ocytes (Dmp1-Gsalpha(KO) mice) have abnormal myelopoiesis, osteopenia, and reduced adipose tissue.
239 ages of the host inflammatory response (from myelopoiesis over leukocyte recruitment to efferocytosis
240 ; cell cycle arrest is required for terminal myelopoiesis, perhaps due to expression of p53 or hypo-p
243 The overexpression of Osm resulting from myelopoiesis prevented HSPC mobilization after granulocy
246 potential, but enhanced megakaryopoiesis and myelopoiesis, recapitulating the major phenotypes of the
247 ut there is increasing evidence that altered myelopoiesis, reduced effector T-cell function, and expa
249 neutrophil elastase acts as an inhibitor of myelopoiesis, substantiating a chalone hypothesis propos
252 syndrome neonates for the transient abnormal myelopoiesis (TAM) disorder based on blood cell morpholo
253 DS) clonally evolves from transient abnormal myelopoiesis (TAM), a preleukemic condition in DS newbor
255 particular miRNAs and the unique features of myelopoiesis that are being uncovered by experimental ma
257 jury and sepsis results from an imbalance in myelopoiesis that is driven by the increased expression
258 tive erythropoiesis and, to a lesser extent, myelopoiesis that is independent of effects on vasculatu
259 (W-sh) mutation broadly affects key steps in myelopoiesis that may have an impact on mast cell resear
262 e cells regulate hematopoiesis, constraining myelopoiesis through a Gsalpha-mediated mechanism that a
263 s reveal a novel mechanism for inhibition of myelopoiesis through defective mitosis and cytokinesis d
264 gs suggest that HOTAIRM1 plays a role in the myelopoiesis through modulation of gene expression in th
265 omatous plaques of ApoE(-/-) mice with their myelopoiesis through regulation of hematopoietic stem an
266 usly to negatively regulate HSC function and myelopoiesis through Tet2-dependent and Tet2-independent
267 s, HFD feeding rapidly activates bone marrow myelopoiesis through the NE-dependent C/EBPalpha-GFI-1 p
268 we show that GM-CSF promotes extramedullary myelopoiesis, tissue-toxic neutrophil accumulation in ta
269 hanistically, we discovered that OSM couples myelopoiesis to mobilopathy by inducing Cxcl12 in BM str
271 place in LT-HSC, and not at later stages of myelopoiesis, to induce MPD and that only junB-deficient
273 liferation and infringed preferentially upon myelopoiesis under both steady-state and stressful condi
274 function provides insight into regulation of myelopoiesis under normal conditions and in myeloprolife
279 le of Janus kinase-2 (JAK2) in regulation of myelopoiesis was established 2 decades ago, but identifi
281 edian, 50% vs 18%; P < .0001), and wild-type myelopoiesis was suppressed in CALR(MUT) but not JAK2(V6
284 genic mice (line 215), resulting in aberrant myelopoiesis, we analyzed 17 cytokines in the peripheral
287 e the role of this novel enhancer further in myelopoiesis, we generated mice with deletion of this re
288 of creating a resource for in-depth study of myelopoiesis, we have executed a 2-pronged strategy to o
289 n that MDSCs are a manifestation of aberrant myelopoiesis, we hypothesized that MDSCs arise from pert
290 nesis screen to identify genes essential for myelopoiesis, we identified an insertional allele hi1727
291 Although best known as a potent inducer of myelopoiesis, we previously reported that G-CSF also pro
294 -p66Shc pathway leads to diabetes-associated myelopoiesis, whereas its transcellular hematostromal ac
295 gs provide new insights into early events in myelopoiesis, whereby Trib1 functions at 2 distinct stag
296 uction of red blood cells and an increase in myelopoiesis, which contributes to the immunosuppressive
298 mily member IRF-8 is a critical regulator of myelopoiesis, which when deleted in mice results in a sy
299 of MN1-TEL-expressing mice developed altered myelopoiesis with severe anemia after long latency.
300 th interleukin-3 (IL-3) resulted in expanded myelopoiesis without a block in differentiation, PML-RAR