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1 ium (GIK) is a potentially useful adjunct to myocardial protection.
2 ntifying a role for GSNO in angiogenesis and myocardial protection.
3  secondary stroke protection rather than for myocardial protection.
4 tivation is antiapoptotic and contributes to myocardial protection.
5 diomyocytes, which in turn leads to improved myocardial protection.
6 gan (thigh versus arm) would provide further myocardial protection.
7  membrane current, which may be important in myocardial protection.
8 s that may benefit from better postoperative myocardial protection.
9 at-shock protein 70 (HSP 70) plays a role in myocardial protection.
10 ior to ischemia induces preconditioning-like myocardial protection.
11 radykinin B(2) receptor blocker, HOE 140, on myocardial protection.
12 esearch stimulated by interest in endogenous myocardial protection.
13 rane fluxes and metabolic demand and improve myocardial protection.
14  cardioplegia in increasing doses to enhance myocardial protection.
15 d in antithrombotic treatments but little in myocardial protection.
16  cardiopulmonary bypass (CPB), 3) methods of myocardial protection, 4) techniques for aortic occlusio
17 ling a mechanism of metabolic modulation and myocardial protection activated by NO donors.
18 ether these effects could be used to enhance myocardial protection afforded by crystalloid cardiopleg
19 uce hibernation in active animals and afford myocardial protection after ischemia-reperfusion injury.
20  route could be a promising new strategy for myocardial protection against ischemia-reperfusion injur
21 shock protein 72 (HSP72) is known to provide myocardial protection against ischemia-reperfusion injur
22 ls during early myocardial repair, providing myocardial protection against ischemic damage by improvi
23 cule-1 (ICAM-1) has been reported to provide myocardial protection against leukocyte-mediated reperfu
24                                     However, myocardial protection also can be achieved by partial re
25 ort Akt-MSC-mediated paracrine mechanisms of myocardial protection and functional improvement.
26 tant implications for the design of tailored myocardial protection and operative strategies for diabe
27  for the expression of paracrine factors for myocardial protection and regeneration.
28 ding care to high-risk patients and improved myocardial protection and surgical and anesthetic techni
29        With improved cardiopulmonary bypass, myocardial protection, and surgical techniques the morta
30 yrin-B expression as required for vertebrate myocardial protection as hearts deficient in ankyrin-B s
31 perdynamic contractile response and enhanced myocardial protection, as evidenced by a reduction in my
32              L-Arginine afforded significant myocardial protection, as evidenced by increased develop
33 aladaptive myocardial changes and inadequate myocardial protection at the time of surgery.
34                         Strategies to afford myocardial protection both during CABG and in the postop
35 tissue in myocardial injury, contributing to myocardial protection by the mediation of the FGFR1/beta
36 -blood (66:1) cardioplegia provides superior myocardial protection compared with dilute (4:1) cardiop
37 ective chemically-induced cardiac arrest and myocardial protection during global ischaemia.
38 MP and heme, has recently been implicated in myocardial protection during ischemia and reperfusion.
39 KC activity in adult mouse hearts results in myocardial protection during ischemia or reperfusion, he
40 hus, the short-term beneficial role of PC in myocardial protection during MIDCAB may be limited.
41 y bypass surgery and in improving methods of myocardial protection during surgery.
42 , we investigated the potential mechanism of myocardial protection following delta-opioid receptor ac
43  cardioplegia has been the gold standard for myocardial protection for years, but patients undergoing
44 a possible mechanism by which BSJYD provides myocardial protection from hypertension.
45 a cytoprotective gene that confers long-term myocardial protection from ischemia/reperfusion injury.
46 ore, we hypothesized that (1) TNFR2 mediates myocardial protection from ischemia/reperfusion through
47 ed JNK inhibition might also be critical for myocardial protection from ischemia/reperfusion.
48 suggesting the importance of GSHPx-1 gene in myocardial protection from ischemic reperfusion injury.
49  normalization of beta-receptor function and myocardial protection from the effects of catecholamines
50  technically challenging, but new methods of myocardial protection have markedly improved the surgica
51 g with complementary murine studies revealed myocardial protection, improved angiogenesis, inflammato
52                                   Inadequate myocardial protection in patients undergoing coronary ar
53      To unmask whether CXCR2 mediates direct myocardial protection in the intact heart, wild type and
54 rction angina is associated with significant myocardial protection in the setting of primary percutan
55     Pharmacologic blockade of PI3K abrogated myocardial protection in TLR4(-/-) mice following I/R.
56  by adipocytes is adiponectin, which confers myocardial protection, insulin-sensitization, and antiat
57                                     Declined myocardial protection is a major feature of diabetic car
58                                        Local myocardial protection may allow prevention of heart fail
59  The possibility that an innate mechanism of myocardial protection might be inducible in the human he
60 , which could affect the use of aspartate in myocardial protection of hypertrophied hearts.
61 hocardiography, and improved intra-operative myocardial protection techniques have all contributed to
62 ce to presently known principles of surgical myocardial protection using advanced cardioplegic techno
63             Despite optimal current surgical myocardial protection, we found that CA with CBC induced
64      Drugs with delta-opiate activity confer myocardial protection, which is additive to cardioplegia
65                                    Extent of myocardial protection with cold-crystalloid cardioplegia
66 le gas, is known to provide neurological and myocardial protection without side effects.