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1  on combinatorial cell approaches to support myocardial regeneration.
2 l non-coding RNAs called microRNAs (miRs) in myocardial regeneration.
3  is feasible, safe or can provide sufficient myocardial regeneration.
4 cover molecular mechanisms that will promote myocardial regeneration.
5  novel off-the-shelf microtissue product for myocardial regeneration.
6  in neonatal mice, which, in turn, decreased myocardial regeneration.
7 d have been unthinkable only a few years ago-myocardial regeneration.
8  would represent the most powerful cells for myocardial regeneration.
9 ulate cardiomyocyte expansion in therapeutic myocardial regeneration.
10 ecovery was due to the cytokines rather than myocardial regeneration.
11  may provide a molecular strategy to promote myocardial regeneration.
12 cally to stimulate resident CPCs and promote myocardial regeneration.
13 egy is to stimulate endogenous mechanisms of myocardial regeneration.
14 protein appears to have no obvious effect on myocardial regeneration.
15             Here we review efforts to induce myocardial regeneration.
16  from mechanisms that are distinct from true myocardial regeneration.
17 rdiomyocyte dysfunction, and even to promote myocardial regeneration.
18 sses hold enormous potential for therapeutic myocardial regeneration.
19 he use of BM-derived cardiac progenitors for myocardial regeneration.
20 sample to develop new insights for achieving myocardial regeneration.
21 rdiac stem/progenitor cells (CSCs) and adult myocardial regeneration.
22 disease and conceptual strategies to promote myocardial regeneration.
23 defects and conceptual strategies to promote myocardial regeneration.
24 e validated and were important in explaining myocardial regeneration.
25 rdium provides a path to realize sustainable myocardial regeneration.
26 c growth factors has been shown to result in myocardial regeneration after acute myocardial infarctio
27 SF) and stem cell factor (SCF) could promote myocardial regeneration after coronary artery occlusion
28 tenins might be a useful strategy to promote myocardial regeneration after injury.
29  cell cycle regulatory mechanisms to enhance myocardial regeneration after myocardial infarction.
30                                   To achieve myocardial regeneration and foster development of effici
31          Transplanted hCDCs promoted greater myocardial regeneration and functional improvement in in
32 nd 6) explore the potential of stem cells in myocardial regeneration and repair in hearts damaged by
33 dings shed new light on our understanding of myocardial regeneration and suggest potential novel ther
34 duce pathological cardiac remodeling, induce myocardial regeneration, and improve ventricular functio
35 lial cell transplantation into the heart for myocardial regeneration, and induction of angiogenesis f
36 ed cardiomyocyte, its antagonistic effect on myocardial regeneration, and its potential contribution
37        This beneficial effect, together with myocardial regeneration attenuated postinfarction dilate
38            The recruited cells contribute to myocardial regeneration both as a structural component o
39  recently shown to play an essential role in myocardial regeneration by regulating different processe
40 the lack of unequivocal evidence in favor of myocardial regeneration by the injection of BMCs in the
41                                 CSCs induced myocardial regeneration, decreasing infarct size by 29%.
42               Again, we found no evidence of myocardial regeneration from blood-borne partner-derived
43 kable effect cannot be readily attributed to myocardial regeneration from the donor cells.
44                             Documentation of myocardial regeneration in acute stress has challenged t
45      Human cardiac stem cells (CSCs) promote myocardial regeneration in adult ischemic myocardium.
46 cardiac progenitor cells (hCPCs) may promote myocardial regeneration in adult ischemic myocardium.
47  scRNAseq datasets generated from studies of myocardial regeneration in mice and pigs, our AI-based t
48 n and cytokinesis and improved indicators of myocardial regeneration in neonatal mice.
49  evolving appreciation for the complexity of myocardial regeneration in the adult context.
50 ings define a mechanistic basis for enhanced myocardial regeneration in transgenic mice overexpressin
51                                              Myocardial regeneration in zebrafish involves the dediff
52                                              Myocardial regeneration induced increased survival and r
53                                     Biologic myocardial regeneration is a new and rapidly evolving ar
54 he mechanism of benefit is unclear, although myocardial regeneration is not a significant factor.
55                                              Myocardial regeneration is observed in amphibians, where
56                                              Myocardial regeneration is perhaps the most widely studi
57                                              Myocardial regeneration is restricted to early postnatal
58 rdiomyocyte cell-cycle activity and promotes myocardial regeneration, leading to improved function af
59                Importantly, the magnitude of myocardial regeneration obtained in this study is signif
60 ll-mediated regeneration to promote enhanced myocardial regeneration, persistence of donated cells, a
61 hanged since our survey of the landscape for myocardial regeneration powered by adult stem cells 4 ye
62 f cellular adoptive transfer for therapeutic myocardial regeneration, problems remain for donated cel
63         This approach of inducing endogenous myocardial regeneration provides proof-of-concept eviden
64                           Because definitive myocardial regeneration remains undemonstrated, we propo
65 ell reprogramming represents a promising new myocardial regeneration strategy involving in situ trans
66 In seven acute infarcts, foci of spontaneous myocardial regeneration that did not involve cell fusion
67 rstand the role of fusion in stem cell-based myocardial regeneration, the present study was designed
68                         Our study shows that myocardial regeneration through targeted peptides is pos
69 blast reprogramming offers the potential for myocardial regeneration via in situ cell transdifferenti
70                                              Myocardial regeneration via stem-cell mobilisation at th
71                                              Myocardial regeneration was detected in all treated infa
72 peutic strategy, the induction of endogenous myocardial regeneration was investigated by initiating c
73 ion of miR-133 levels after injury inhibited myocardial regeneration, while transgenic miR-133 deplet
74                                              Myocardial regeneration with stem-cell transplantation i