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1 for the treatment of C. perfringens-mediated myonecrosis.
2 ine and morphine do not die from clostridial myonecrosis.
3 PCI MI definition and preventing PCI-related myonecrosis.
4 , that cause extensive tissue destruction in myonecrosis.
5 s to reduce tissue damage during clostridial myonecrosis.
6 ction did not correlate with the quantity of myonecrosis.
7 ents strongly associated with periprocedural myonecrosis.
8 GAS interaction may prevent or attenuate GAS myonecrosis.
9 onin T is a sensitive and specific marker of myonecrosis.
10  and burden of CAD and degree of subclinical myonecrosis.
11 educe the extent, but not the occurrence, of myonecrosis.
12 MRI technique allows direct visualization of myonecrosis.
13 e of infection, e.g., skin epithelium during myonecrosis.
14 ence in the muscle, resulting in devastating myonecrosis.
15 ocardial infarction (69%), sepsis (51%), and myonecrosis (66%).
16 re independently associated with severity of myonecrosis after cardiac surgery.
17                                              Myonecrosis after percutaneous intervention is common in
18 lts in greater suppression of periprocedural myonecrosis, although a survival benefit has not been de
19 availability of more sensitive biomarkers of myonecrosis and a new classification system from the uni
20 s strains produce severe diseases, including myonecrosis and enteritis necroticans, in humans and ani
21 ice, histological and biochemical markers of myonecrosis and regeneration are completely suppressed t
22 ronary arteries would reduce peri-procedural myonecrosis and subsequently enhance event-free survival
23 ally used hydroxyl radical trap, reduces the myonecrosis and the mortality caused by an experimental
24 nflammation affects LV function by extending myonecrosis and/or altering LV remodeling remains unknow
25 ease was specific to C. perfringens-mediated myonecrosis; buprenorphine did not protect against disea
26 atients with minimal amounts of recent onset myonecrosis but elevated risk as indicated by CK-MB and
27                              Half of all GAS myonecrosis cases have no known portal of entry; yet, fo
28                    Necrotizing fasciitis and myonecrosis caused by invasive infection with group A st
29 = 10), acute myocardial infarction (n = 10), myonecrosis due either to polymyositis or crush injuries
30                                              Myonecrosis due to group A streptococci (GAS) often deve
31 ng food poisoning, gas gangrene (clostridial myonecrosis), enteritis necroticans, and non-foodborne g
32 iseases, including gas gangrene (clostridial myonecrosis), enteritis necroticans, antibiotic-associat
33 triever model, uUtro non-toxically prevented myonecrosis, even in the most powerful muscles.
34 ormal at 14 days with evidence of persisting myonecrosis, even though gene expression of regulators o
35 gens is the most common cause of clostridial myonecrosis (gas gangrene).
36 only a minor role in preventing the onset of myonecrosis in a mouse animal model of the disease (unpu
37           Ischemic events and biomarkers for myonecrosis, inflammation, and thrombin generation did n
38             Diseases that cause hemolysis or myonecrosis lead to the leakage of large amounts of heme
39                         The PR, TIP-FCS, and myonecrosis markers were serially determined.
40                                              Myonecrosis, noted in 41.4% of the patients who underwen
41  A streptococcal (GAS) necrotizing fasciitis/myonecrosis often necessitates extensive debridement to
42 1), but were not associated with subclinical myonecrosis (OR: 1.14; p = 0.3147).
43 or coated) approaches may be associated with myonecrosis related to atherosclerotic debris plugging t
44  where conventional biomarkers indicative of myonecrosis remain undetected.
45 ptococci in the dorsal muscle, and extensive myonecrosis that occurred far in advance of any systemic
46 MI definition sensitive for small degrees of myonecrosis (the occurrence of which, based on contempor
47 egments with "distal-type" procedure-related myonecrosis using high-resolution quantitative cardiovas
48                                   Infectious myonecrosis virus (IMNV) is an emerging pathogen of pena
49 uses that includes penaeid shrimp infectious myonecrosis virus (IMNV).
50                 The median estimated mass of myonecrosis was 2.0 g (range, 0.7 to 12.2 g), or 1.5% of
51                                    Decreased myonecrosis was also observed in diaphragms and quadrice
52                                     "Distal" myonecrosis was defined as situated at least 10 mm downs
53                                   "Adjacent" myonecrosis was defined as the presence of an area of ne
54 cular lymphocytic infiltrates and rare focal myonecrosis were evident on biopsy at 2 weeks postoperat
55 eria only PLC was necessary for the onset of myonecrosis, while at sublethal doses (10(6)) both PFO a
56 ed in muscle tissues from a patient with GAS myonecrosis who died of streptococcal toxic shock syndro