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1 tr(-/-) mice the structural integrity of the myotendinous and neuromuscular junctions are maintained.
4 n skin deep; it extends to the depths of the myotendinous architecture and the mechanical basis for p
7 colemma localization, SPN is enriched at the myotendinous junction (MTJ) and neuromuscular junction (
9 responding tendon cells to form a functional myotendinous junction (MTJ) that allows for the force ge
14 last subtypes, including tenocytes along the myotendinous junction and fin mesenchymal cells in the f
15 of the muscle cell, including markers of the myotendinous junction and functionally validated factors
16 toward and subsequent differentiation of the myotendinous junction by priming formation of a protein
21 , thus suggesting the important influence of myotendinous junction integrity on the severity of muscu
25 alpha7 integrin and utrophin in maintaining myotendinous junction structure and enabling force trans
26 ndary of fibroblast-derived myonuclei at the myotendinous junction that controls limb muscle patterni
27 d at the sarcolemma, neuromuscular junction, myotendinous junction, and in peripheral nerve, but not
28 n, loss of tension-mediating proteins at the myotendinous junction, and misshaped and internalized nu
29 revealed that electrical stimulation to the myotendinous junction, where palisade endings are locate
35 ole for dystrophin in maintaining vertebrate myotendinous junctions (MTJs) has been postulated previo
36 l muscle, talin 1 regulates the stability of myotendinous junctions (MTJs), but the function of talin
40 of those that underlie synapse formation at myotendinous junctions and that the outgrowth of seconda
41 unctions, which supports the hypothesis that myotendinous junctions are distinct domains in which the
42 underlying muscle guidance and formation of myotendinous junctions are poorly understood both in ver
45 accumulates at the neuromuscular synapse and myotendinous junctions in adult skeletal muscle, and is
47 is enriched at intersomitic junctions and at myotendinous junctions in somites and the myotome, where
49 ss tenocytes at multiple tendon entheses and myotendinous junctions reveals that their responses depe
51 keletal proteins that are highly enriched at myotendinous junctions that we hypothesize to be subject
53 Ultrastructural analysis revealed abnormal myotendinous junctions, and functional tests showed a ni
54 confined in skeletal muscle to synapses and myotendinous junctions, and in kidney to the glomerular
55 zed tendon cells, resembling tendon cells at myotendinous junctions, form at the ends of these muscle
56 erentially concentrated at neuromuscular and myotendinous junctions, suggesting a role at sarcolemmal
57 hese territories were further specialized at myotendinous junctions, where both Dmd transcripts and d
58 hows that the mRNA is highly concentrated at myotendinous junctions, which supports the hypothesis th
67 We also found a positive correlation between myotendinous strain injury and ringed fibers in the HSA(
69 olecular and cellular adaptations to chronic myotendinous strain injury in mdx mice expressing a micr
72 nd the formation of rings are adaptations to myotendinous strain injury that help to prevent muscle n