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1 e diseases, including Huntington disease and myotonic dystrophy.
2 uscle weakness and wasting characteristic of myotonic dystrophy.
3 disease severity and therapeutic response in myotonic dystrophy.
4  facioscapulohumeral muscular dystrophy, and myotonic dystrophy.
5 hogenic feature of the neuromuscular disease myotonic dystrophy.
6 ed and studied with respect to their role in myotonic dystrophy.
7 lular localization is a central component of myotonic dystrophy.
8 g in corrective outcome for a mouse model of myotonic dystrophy.
9 ead to muscle degeneration disorders such as myotonic dystrophy.
10 des to treat Duchenne muscular dystrophy and myotonic dystrophy.
11  skeletal actin, long repeat) mouse model of myotonic dystrophy.
12 on diseases such as Huntington's disease and myotonic dystrophy.
13 ion disorders such as Huntington disease and myotonic dystrophy.
14  gene causes the autosomal dominant disorder myotonic dystrophy.
15 bias seen in expanded CTG triplet repeats in myotonic dystrophy.
16 ssues, including heart failure, diabetes, or myotonic dystrophy.
17 onduction delay, two predominant features of myotonic dystrophy.
18 causes symptoms in the neuromuscular disease myotonic dystrophy.
19 bset of the cardiac dysfunctions observed in myotonic dystrophy.
20 ical diseases such as Huntington disease and myotonic dystrophy.
21  transition, resulting in the development of myotonic dystrophy.
22 a potential drug target for the treatment of myotonic dystrophy.
23 sting a toxic RNA pathogenesis, as occurs in myotonic dystrophy.
24 hat are specific to skeletal muscle, and the myotonic dystrophies.
25 c syndromes, with particular emphasis on the myotonic dystrophies.
26                                              Myotonic dystrophy 1 (DM1) is a multi-system disorder ch
27                                              Myotonic dystrophy 1 (DM1) is a multisystemic disease ca
28                                           In myotonic dystrophy 1 (DM1), aggregation of the mutant DM
29 f skeletal muscle pathology in patients with Myotonic Dystrophy 1 (DM1).
30 ts for Duchenne MD, various limb girdle MDs, myotonic dystrophy 1, facioscapulohumeral MD, dysferlino
31                                              Myotonic dystrophy 2 (DM2) is a multisystem skeletal mus
32                                              Myotonic dystrophy 2 (DM2) is an autosomal dominant, mul
33 es of repeat instability and pathogenesis in myotonic dystrophy, a neurological disorder caused by an
34  disease process raises the possibility that myotonic dystrophy, among genetic disorders, may be unus
35  known RNA-mediated disorders, including the myotonic dystrophies and fragile X tremor ataxia syndrom
36 tor protein that plays a pivotal role in the Myotonic Dystrophies and Huntington's Disease, and sever
37 the results with those of four patients with myotonic dystrophy and 12 healthy individuals.
38 ely short triplet-repeat expansions found in myotonic dystrophy and Friedreich's ataxia confer varieg
39 ween the pathogenic RNA repeat expansions of myotonic dystrophy and MBNL1.
40                Using a cell culture model of myotonic dystrophy and myotonic dystrophy patient tissue
41 ls are key players in both the human disease myotonic dystrophy and the regulation of alternative spl
42 ts in Friedreich's ataxia, (CTG)n repeats in myotonic dystrophy, and (CGG)n repeats in fragile X synd
43 sity in humans as may occur in, for example, myotonic dystrophy, and possibly, the metabolically obes
44 rdiac electrophysiological disease; one with myotonic dystrophy; and one with hypertrophic cardiomyop
45                                              Myotonic dystrophies are the most common, comprising 28.
46 nic mouse model to show that derangements of myotonic dystrophy are reversed by a morpholino antisens
47 leblind-like 1 (MBNL1), a gene implicated in myotonic dystrophy, as a robust suppressor of multiorgan
48              This process is dysregulated in myotonic dystrophy because MBNL proteins are sequestered
49  for a therapeutic strategy for treatment of myotonic dystrophy by ablating or silencing expression o
50 sis might have a clinically relevant role in myotonic dystrophy cardiac conduction defects and pathol
51 e splicing and polyadenylation in congenital myotonic dystrophy (CDM).
52 r generation of massive instabilities of the myotonic dystrophy CTG.CAG sequences.
53                                          The myotonic dystrophies (DM) are human diseases in which th
54                                              Myotonic dystrophy (DM) is a genetic disorder caused by
55                                              Myotonic dystrophy (DM) is a multi-system neuromuscular
56                                              Myotonic dystrophy (DM) is a multisystemic disease cause
57                                              Myotonic dystrophy (DM) is caused by a CTG expansion in
58                                              Myotonic dystrophy (DM) is caused by a triplet repeat ex
59                                              Myotonic dystrophy (DM) is caused by either an untransla
60                    The neuromuscular disease myotonic dystrophy (DM) is caused by microsatellite repe
61                                              Myotonic dystrophy (DM) is caused by the expression of m
62                                              Myotonic dystrophy (DM) is caused by two similar noncodi
63  a topic of intense study due to its role in myotonic dystrophy (DM) pathogenesis.
64           The RNA-mediated disease model for myotonic dystrophy (DM) proposes that microsatellite C(C
65                                              Myotonic dystrophy (DM) type 1 is associated with an exp
66 (MBNL) protein family has been implicated in myotonic dystrophy (DM), a specific function for these p
67                                           In myotonic dystrophy (DM), expression of RNA containing ex
68 t roles in muscle and eye development and in myotonic dystrophy (DM), in which expanded CUG or CCUG r
69  foci by C(C)UG microsatellite expansions in myotonic dystrophy (DM), is essential for normal thymus
70                                              Myotonic dystrophy (DM), the most common form of muscula
71                                              Myotonic dystrophy (DM), the most common form of muscula
72 proposed first for the neuromuscular disease myotonic dystrophy (DM), which is associated with the ex
73                                              Myotonic dystrophy (DM)--the most common form of muscula
74  is a key player in the disease mechanism of myotonic dystrophy (DM).
75 tal muscle development and are implicated in myotonic dystrophy (DM).
76 pathogenic event in the RNA-mediated disease myotonic dystrophy (DM).
77                       PURPOSE OF REVIEW: The myotonic dystrophies (DM1 and DM2) are the paradigm for
78 UGn RNA in the induction of stress in type 1 myotonic dystrophy (DM1) cells and in the stress-mediate
79                                              Myotonic dystrophy (DM1) is a highly variable, multi-sys
80                                       Type I myotonic dystrophy (DM1) is caused by a triplet repeat e
81                                              Myotonic dystrophy (DM1) is caused by an expansion of CU
82                                           In myotonic dystrophy (DM1), both inactivation of musclebli
83                                              Myotonic dystrophy (DM1), the most common muscular dystr
84 rections for many genetic diseases including myotonic dystrophy (DM1).
85 ein implicated in the pathogenesis of type I myotonic dystrophy (DM1).
86 ated with the degenerative muscular disease, myotonic dystrophy (DM1).
87 ated with expanded repeat sequences, such as myotonic dystrophy (DM1).
88                        The genetic lesion in myotonic dystrophy does not eliminate an essential muscl
89                                           In myotonic dystrophy (dystrophia myotonica [DM]), an incre
90                                              Myotonic dystrophy (dystrophia myotonica, DM) is a multi
91 s of European origin with PROMM and proximal myotonic dystrophy, from geographically distinct populat
92 ated with multiple human diseases, including myotonic dystrophy, Fuchs endothelial corneal dystrophy,
93                                  Research on myotonic dystrophy has led to the recognition of a novel
94        Many neurological diseases, including myotonic dystrophy, Huntington's disease and several spi
95 AA)n, are associated with diseases including myotonic dystrophy, Huntington's disease, fragile X and
96 unction is a prominent cause of mortality in myotonic dystrophy I (DM1), a disease where expanded CUG
97  in the development of RNA splice defects in myotonic dystrophy I (DM1), we purified RNA-independent
98 s also responsible for the manifestations of myotonic dystrophy in non-muscle tissues.
99 r phenotype reflects many of the features of myotonic dystrophy, including muscle histological morpho
100  3' UTR mRNA reproduced cardinal features of myotonic dystrophy, including myotonia, cardiac conducti
101                   The pathomechanism for the myotonic dystrophies is not well understood and the role
102                                              Myotonic dystrophy is a complex neuromuscular disorder a
103                  Therapeutic development for myotonic dystrophy is moving rapidly with the developmen
104             RECENT FINDINGS: RNA toxicity in myotonic dystrophy is now associated with bi-directional
105                   It now appears likely that myotonic dystrophy is the first instance of a genetic di
106                                           In myotonic dystrophy it is the RNA rather than protein pro
107                 In the best studied example, myotonic dystrophy, it appears that the main pathogenic
108 roteins HSP20, HSP25, alphaB-crystallin, and myotonic dystrophy kinase binding protein (MKBP) may reg
109                                        MRCK (myotonic dystrophy kinase-related Cdc42 binding kinase),
110                                              Myotonic dystrophy kinase-related Cdc42-binding kinase (
111 ation and invasion by binding and activating myotonic dystrophy kinase-related CDC42-binding kinase a
112                 Caspase-mediated cleavage of myotonic dystrophy kinase-related CDC42-binding kinase-a
113 ls have been implicated in schizophrenia and myotonic dystrophy (MD), and both conditions carry an in
114 phasis on key updates in muscular dystrophy, myotonic dystrophy, mitochondrial myopathy, spinal muscu
115 hronic progressive external ophthalmoplegia, myotonic dystrophy, neurofibromatosis type 2, and basal
116 an skeletal actin (HSA)(LR) mice (a model of myotonic dystrophy) of various ages.
117 cell culture model of myotonic dystrophy and myotonic dystrophy patient tissue, we have evidence that
118 L1, a splicing factor that is sequestered in myotonic dystrophy patients by binding to expanded r(CUG
119                          In human cells from myotonic dystrophy patients, treatment with 5-aza-CdR st
120 e variant CaV1.1e in the skeletal muscles of myotonic dystrophy patients.
121 sed Znf9 and Clc1 expression and rescued the myotonic dystrophy phenotype in Znf9+/- mice.
122 e Znf9 haploinsufficiency contributes to the myotonic dystrophy phenotype in Znf9+/- mice.
123 muscle cross-sectional area in patients with myotonic dystrophy, preferentially in healthy-appearing
124 e basis for a new type of instability of the myotonic dystrophy protein kinase (DMPK) gene in patient
125                                              Myotonic dystrophy protein kinase (DMPK), a muscle- and
126 G)n tract in the 3' UTR of the gene encoding myotonic dystrophy protein kinase (DMPK), which results
127 ' untranslated region of the gene coding for myotonic dystrophy protein kinase (DMPK).
128                 Abnormal expression of human myotonic dystrophy protein kinase (hDMPK) gene products
129 let-repeat expansion region from a truncated myotonic dystrophy protein kinase transcript mimic in vi
130 -coil domain reminiscent of eukaryotic DMPK (Myotonic Dystrophy Protein Kinase) family kinases such a
131                      In mammalian cells, the myotonic dystrophy-related Cdc42-binding kinase possesse
132                By this mechanism, effects of myotonic dystrophy repeat expansions impact many differe
133 d the pathobiology of disease mechanisms for myotonic dystrophy, spinal muscular atrophy, and fragile
134 ar ataxia, amyotrophic lateral sclerosis and myotonic dystrophy) that involve mutations within the an
135                             In both types of myotonic dystrophy the expanded repeat is transcribed an
136 c mechanisms that have been proposed for the myotonic dystrophies, the clinical and molecular feature
137                                           In myotonic dystrophy, the expression of expanded CUG repea
138                                           In myotonic dystrophy, the lack of properly localized MBNL1
139 rnative splicing and have been implicated in myotonic dystrophy, the most common form of adult onset
140 ansions of noncoding CUG and CCUG repeats in myotonic dystrophies type 1 (DM1) and DM2 cause complex
141                                              Myotonic dystrophies type 1 (DM1) and type 2 (DM2) are n
142                                              Myotonic dystrophies type 1 and type 2 are progressive m
143 tive approach to screening and management of myotonic dystrophies type 1 and type 2 requires a multid
144  human samples from patients with congenital myotonic dystrophy type 1 (CDM1) and spinal muscular atr
145  with Huntington's disease (CAG repeats) and myotonic dystrophy type 1 (CTG repeats).
146 anded rCUG and rCAG repeat RNAs expressed in myotonic dystrophy type 1 (DM1) and spinocerebellar atax
147                                              Myotonic dystrophy type 1 (DM1) and type 2 (DM2) are cau
148 d CCUG are the underlying genetic causes for myotonic dystrophy type 1 (DM1) and type 2 (DM2), respec
149  sequence is considered a causative agent of myotonic dystrophy type 1 (DM1) because of its ability t
150 nscript (CUG(exp)) is the causative agent of myotonic dystrophy type 1 (DM1) by sequestering musclebl
151 man spinocerebellar ataxia type 8 (SCA8) and myotonic dystrophy type 1 (DM1) CAG expansion transcript
152  disease (HD) FEN1 +/- heterozygous mice and myotonic dystrophy type 1 (DM1) FEN1 +/- heterozygous mi
153                     Background Patients with myotonic dystrophy type 1 (DM1) increased their physical
154 A working hypothesis for the pathogenesis of myotonic dystrophy type 1 (DM1) involves the aberrant se
155                                              Myotonic dystrophy type 1 (DM1) is a complex neuromuscul
156                         The genetic basis of myotonic dystrophy type 1 (DM1) is a CTG expansion in th
157                                              Myotonic dystrophy type 1 (DM1) is a CTG microsatellite
158                                              Myotonic Dystrophy type 1 (DM1) is a dominant neuromuscu
159                                              Myotonic dystrophy type 1 (DM1) is a dominantly inherite
160                                              Myotonic dystrophy type 1 (DM1) is a genetic disorder in
161                                              Myotonic dystrophy type 1 (DM1) is a genetic disorder li
162                                              Myotonic dystrophy type 1 (DM1) is a life-threatening an
163                                              Myotonic dystrophy type 1 (DM1) is a microsatellite expa
164                                              Myotonic dystrophy type 1 (DM1) is a multisystem neuromu
165                                              Myotonic dystrophy type 1 (DM1) is a multisystemic genet
166                                              Myotonic dystrophy type 1 (DM1) is a neuromuscular disor
167                                              Myotonic dystrophy Type 1 (DM1) is a rare genetic diseas
168                                              Myotonic dystrophy type 1 (DM1) is a triplet repeating d
169                                              Myotonic dystrophy type 1 (DM1) is an autosomal dominant
170                                              Myotonic dystrophy type 1 (DM1) is an autosomal dominant
171                                              Myotonic dystrophy type 1 (DM1) is an autosomal dominant
172                                              Myotonic dystrophy type 1 (DM1) is an incurable neuromus
173                                              Myotonic dystrophy type 1 (DM1) is an inherited dominant
174                                              Myotonic dystrophy type 1 (DM1) is an RNA dominant disea
175                                              Myotonic dystrophy type 1 (DM1) is an RNA-dominant disea
176                                              Myotonic dystrophy type 1 (DM1) is associated with expan
177                                              Myotonic dystrophy type 1 (DM1) is caused by a CTG expan
178                                              Myotonic dystrophy type 1 (DM1) is caused by a CTG expan
179                                              Myotonic dystrophy type 1 (DM1) is caused by a CTG trinu
180                                              Myotonic dystrophy type 1 (DM1) is caused by a CUGn expa
181                                              Myotonic dystrophy type 1 (DM1) is caused by an expanded
182                                              Myotonic dystrophy type 1 (DM1) is caused by expansion o
183                                              Myotonic dystrophy type 1 (DM1) is caused by expansion o
184                                              Myotonic dystrophy type 1 (DM1) is caused by the expansi
185                                              Myotonic dystrophy type 1 (DM1) is one of the most varia
186                                              Myotonic dystrophy type 1 (DM1) is one such disorder tha
187                                              Myotonic dystrophy type 1 (DM1) is the most common form
188 ion hypothesis for the CTG expansion causing myotonic dystrophy type 1 (DM1) located in the 3' noncod
189             The disease mechanism underlying myotonic dystrophy type 1 (DM1) pathogenesis in skeletal
190           Accumulation of RNA CUG repeats in myotonic dystrophy type 1 (DM1) patients leads to the in
191                                           In myotonic dystrophy type 1 (DM1), dystrophia myotonica pr
192  splicing has become a molecular hallmark of myotonic dystrophy type 1 (DM1), in which neonatal splic
193 ough cataract is a characteristic feature of myotonic dystrophy type 1 (DM1), little is known of the
194  and GAA.TTC are integral to the etiology of myotonic dystrophy type 1 (DM1), myotonic dystrophy type
195                                           In myotonic dystrophy type 1 (DM1), somatic mosaicism of th
196                                              Myotonic dystrophy type 1 (DM1), the most common adult m
197                                Among them is myotonic dystrophy type 1 (DM1), the most common form of
198                                              Myotonic dystrophy type 1 (DM1), the most common form of
199                                              Myotonic dystrophy type 1 (DM1), the most common form of
200                                              Myotonic dystrophy type 1 (DM1), the most common muscula
201                                              Myotonic dystrophy type 1 (DM1), the most prevalent musc
202       In the hereditary degenerative disease myotonic dystrophy type 1 (DM1), transcripts from the mu
203                                           In myotonic dystrophy type 1 (DM1), triplet repeat expansio
204 date disease for RNAi therapy application is myotonic dystrophy type 1 (DM1), which results from toxi
205 (hDMPK) gene products has been implicated in myotonic dystrophy type 1 (DM1), yet the impact of distr
206 rophia myotonica protein kinase (DMPK) cause myotonic dystrophy type 1 (DM1).
207 k CTG expansion (CTG(exp)) knockin models of myotonic dystrophy type 1 (DM1).
208 S; trisomy 21) and the dementia component of myotonic dystrophy type 1 (DM1).
209 als with the inherited multisystemic disease myotonic dystrophy type 1 (DM1).
210 ting symptom experienced by individuals with myotonic dystrophy type 1 (DM1).
211 d pathogenic role of expanded CUG repeats in myotonic dystrophy type 1 (DM1).
212 ocess is impaired in patients afflicted with myotonic dystrophy type 1 (DM1).
213 rationally designed, multi-target agents for myotonic dystrophy type 1 (DM1).
214 h are disrupted on loss of MBNL1 function in myotonic dystrophy type 1 (DM1).
215    METHODS AND We selected 855 patients with myotonic dystrophy type 1 (women, 51%; median age, 37 ye
216  cause dominantly inherited diseases such as myotonic dystrophy type 1 and 2 (DM1/2), Huntington's di
217                           Friedreich ataxia, myotonic dystrophy type 1 and 3 forms of intellectual di
218 detected in mouse models with DCM, including myotonic dystrophy type 1 and CELF1 overexpression model
219 s previously characterized in the context of myotonic dystrophy type 1 and epithelial-to-mesenchymal
220 se sequences are involved in the etiology of myotonic dystrophy type 1 and Friedreich's ataxia, respe
221 xias and the initial clinical application in myotonic dystrophy type 1 and Huntington's disease.
222                                              Myotonic dystrophy type 1 and type 2 (DM1 and DM2) are g
223 ress in elucidating the disease mechanism in myotonic dystrophy type 1 and type 2.
224                          Patients with adult myotonic dystrophy type 1 are at high risk for arrhythmi
225 ng in the molecular and clinical features of myotonic dystrophy type 1 as well as the screening of cl
226 epeats (CUG(exp)) are the causative agent of myotonic dystrophy type 1 by sequestering MBNL1.
227             Up to one-third of patients with myotonic dystrophy type 1 die suddenly.
228                  Historically, patients with myotonic dystrophy type 1 have not received the medical
229                                              Myotonic dystrophy type 1 is an autosomal dominant disor
230                                              Myotonic dystrophy type 1 is caused by the dysregulation
231             Previous studies have shown that myotonic dystrophy type 1 is caused by the expansion of
232                  The average age of death in myotonic dystrophy type 1 is in the fifth decade.
233                                              Myotonic dystrophy type 1 is the most prevalent muscular
234 he size of the CTG expansion in the blood of myotonic dystrophy type 1 patients is associated with to
235 n contrast to the CUG-RNA hairpins formed by myotonic dystrophy type 1 repeats, we found no evidence
236 umina sequencing in Huntington's disease and myotonic dystrophy type 1 subjects, we show that rs55787
237 der than 18 years with genetically confirmed myotonic dystrophy type 1 who were admitted to the Neuro
238   How this untranslated CTG expansion causes myotonic dystrophy type 1(DM1) has been controversial.
239  been implicated in human diseases including myotonic dystrophy type 1, Alzheimer's disease and multi
240                          Among patients with myotonic dystrophy type 1, an invasive strategy was asso
241  nine had myotonic dystrophy type 2, one had myotonic dystrophy type 1, and 17 had no identified muta
242 (P = 0.003) in both Huntington's disease and myotonic dystrophy type 1, and slower progression (P = 3
243 isease phenotype in Huntington's disease and myotonic dystrophy type 1, and suggests a common DNA rep
244 le for causing neurological diseases such as myotonic dystrophy type 1, but its binding mechanism rem
245 ples from individuals with one such disease, myotonic dystrophy type 1, provides an opportunity to pa
246 f Friedreich ataxia, fragile X syndrome, and myotonic dystrophy type 1, respectively.
247  dementia, fragile X tremor ataxia syndrome, myotonic dystrophy type 1, spinocerebellar ataxia type 8
248                                           In myotonic dystrophy type 1, the association between mutat
249                                   Similar to myotonic dystrophy type 1, the poly(CUG)n RNA co-localiz
250  atrophy, amyotrophic lateral sclerosis, and myotonic dystrophy type 1, were also reviewed.
251           Exon 2 is significantly reduced in myotonic dystrophy type 1, whose symptoms include dement
252 uation, out of 1014 patients included in the Myotonic Dystrophy Type 1-Heart Registry between January
253  myotonica protein kinase (DMPK) gene causes myotonic dystrophy type 1.
254 able therapeutic target for the treatment of myotonic dystrophy type 1.
255 ive diseases, such as Huntington disease and myotonic dystrophy type 1.
256 tracts in the size range that is typical for myotonic dystrophy type 1.
257 n abnormalities in the neuromuscular disease myotonic dystrophy type 1.
258 06 adult patients with genetically confirmed myotonic dystrophy type 1.
259 expansion disorders Huntington's disease and myotonic dystrophy type 1.
260 xpansions observed in human diseases such as myotonic dystrophy type 1.
261 en implicated in the cardiac pathogenesis of myotonic dystrophy type 1.
262  atrophy, amyotrophic lateral sclerosis, and myotonic dystrophy type 1.
263  atrophy, amyotrophic lateral sclerosis, and myotonic dystrophy type 1.
264  disorders, including Huntington disease and myotonic dystrophy type 1.
265 er onset of symptoms and is less common than myotonic dystrophy type 1.
266 ranslated CCTG expansion in an intron causes myotonic dystrophy type 2 (DM2) have uncovered a new typ
267                                              Myotonic dystrophy type 2 (DM2) is a genetic disorder ch
268                                              Myotonic dystrophy type 2 (DM2) is a multisystemic disor
269                                              Myotonic dystrophy type 2 (DM2) is an incurable neuromus
270                                              Myotonic dystrophy type 2 (DM2) is caused by a CCTG expa
271                    The recent discovery that myotonic dystrophy type 2 (DM2) is caused by an untransl
272                                              Myotonic dystrophy type 2 (DM2) is caused by the extreme
273 ponsible for the massive expansions found in myotonic dystrophy type 2 (DM2) patients.
274  At some sites of repeat expansion, e.g. the myotonic dystrophy type 2 (DM2) tetranucleotide repeat e
275 ribed but untranslated CCTG expansion causes myotonic dystrophy type 2 (DM2), along with other discov
276 oops in r(CCUG)(exp), the causative agent of myotonic dystrophy type 2 (DM2), and are transformed int
277 etiology of myotonic dystrophy type 1 (DM1), myotonic dystrophy type 2 (DM2), and Friedreich's ataxia
278 so examined the similarly slowly progressing myotonic dystrophy type 2 (DM2).
279             Recently, it was discovered that myotonic dystrophy type 2 (proximal myotonic myopathy) i
280 ofilm formation in Staphylococcus aureus and myotonic dystrophy type 2 in human, respectively.
281                                              Myotonic dystrophy type 2 is a genetic neuromuscular dis
282                                              Myotonic dystrophy type 2 is caused by a (CCTG)/(CCUG)n
283  not well understood and the role of ZNF9 in myotonic dystrophy type 2 pathogenesis has not been full
284                               In comparison, myotonic dystrophy type 2 tends to cause a milder phenot
285              We explored this question using myotonic dystrophy type 2, a multisystemic disease thoug
286 irst intron of the ZNF9 gene associated with myotonic dystrophy type 2, form slipped-strand DNA struc
287 s, 34 had sodium channel mutations, nine had myotonic dystrophy type 2, one had myotonic dystrophy ty
288 l mutations, chloride channel mutations, and myotonic dystrophy type 2.
289                    The neuromuscular disease myotonic dystrophy type I (DM1) affects multiple organ s
290                                              Myotonic dystrophy type I (DM1) exhibits distinctive dis
291                                              Myotonic dystrophy type I (DM1) is a disabling multisyst
292                                              Myotonic dystrophy type I (DM1) is a disabling neuromusc
293                                              Myotonic dystrophy type I (DM1) is a multisystemic autos
294                                              Myotonic dystrophy type I (DM1) is an RNA-mediated disea
295        They contain CUG repeats, relevant to myotonic dystrophy type I, and CAG repeats associated wi
296 muscleblind function and the pathogenesis of myotonic dystrophy, we generated Drosophila incorporatin
297 eases, including spinal muscular atrophy and myotonic dystrophy, where defects of splicing or alterna
298 operties of potential therapeutic agents for myotonic dystrophy, which is caused by sequestration of
299                   Molecular therapeutics for myotonic dystrophy will probably bridge the translationa
300 ment for clinical screening of patients with myotonic dystrophy with proactive and systematic managem

 
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