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1 A similar pattern occurred following nasal administration.
2 vivo studies showed mucosal absorption after nasal administration.
3 ung epithelial cells in vivo following intra-nasal administration.
4 induced in DBA/1 mice was studied after the nasal administration (before disease induction) of 1 of
5 variants, exhibits mucosal persistence upon nasal administration, demonstrates enhanced stability co
6 ior of the smart polymers found suitable for nasal administration, including poloxamer, chitosan, EHE
12 e showed that blocking IL-10 concurrent with nasal administration of Ag abolished the disease-suppres
17 t mucosal tolerance in EAG can be induced by nasal administration of an immunodominant peptide from t
26 Collectively, these data suggest that the nasal administration of GAD65 peptides induces a Th2 cel
28 tion against recolonization, suggesting that nasal administration of live attenuated Spn could be an
29 ystemic protective immunity, suggesting that nasal administration of live attenuated Streptococcus pn
33 t mucosal tolerance in EAG can be induced by nasal administration of recombinant rat alpha3(IV)NC1 an
34 tolerance in EAG by examining the effect of nasal administration of recombinant rat alpha3(IV)NC1.
35 Here, we demonstrate that subcutaneous or nasal administration of small-molecule MC activators wit
38 holine receptor (TAChR), to demonstrate that nasal administration of synthetic sequences of the TAChR
43 ificantly different from that seen following nasal administration of these vectors to mouse lung (rAA
44 rance in EAG by examining the effects of the nasal administration of this peptide after the onset of
48 hology studies in rats after 5 days repeated nasal administration showed that Solutol HS15 had no tox
49 ovation warranting further investigation for nasal administration to potentially reduce infection and
51 ed 18-50 years were randomized (1:1:1:1) for nasal administration twice (at a 2-wk interval) with sal