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1 t cells were excluded from both the lens and nasal epithelium.
2 d several nonocular sites, such as heart and nasal epithelium.
3 gh expression of asthma-related genes in the nasal epithelium.
4  of TSLP genotype and TSLP expression in the nasal epithelium.
5 kely trigger distinct immune pathways in the nasal epithelium.
6 subsequently confirmed in RNA extracted from nasal epithelium.
7 ating all of the cell types found within the nasal epithelium.
8 man nasal epithelial (HNE) cells and in vivo nasal epithelium.
9 s in the rainbow trout (Oncorhynchus mykiss) nasal epithelium.
10  infant airway transcriptome by sampling the nasal epithelium.
11  and disease can be gained from studying the nasal epithelium.
12 d by a marked paucity of motile cilia in the nasal epithelium.
13 g had a similar effect on gene expression in nasal epithelium.
14 eceptors expressed by sensory neurons in the nasal epithelium.
15 n reported in many human tissues outside the nasal epithelium.
16 tent of CFTR function in the sweat gland and nasal epithelium.
17 ares an apical distribution with CFTR in rat nasal epithelium.
18 trigeminal ganglion (TG) which innervate the nasal epithelium.
19 ponse to short-term smoking-cessation in the nasal epithelium.
20 dicate that bitter substances applied to the nasal epithelium activate the trigeminal nerve and evoke
21 on, infectious virus was recovered only from nasal epithelium; after intratracheal inoculation, it wa
22 l ganglion cells with sensory endings in the nasal epithelium also have branches reaching directly in
23 stem, beginning at embryonic day 11.5 in the nasal epithelium and at embryonic day 16.5 in the olfact
24 id a genome-wide study of DNA methylation in nasal epithelium and atopy or atopic asthma in 483 Puert
25 iptome prediction models for asthma tissues (nasal epithelium and CD4(+) T cells) and apply them in a
26 ed robust biofilms that were adherent to the nasal epithelium and displayed architectural attributes
27 osensory cells that reach the surface of the nasal epithelium and form synaptic contacts with trigemi
28  in the lower airway, they were reflected in nasal epithelium and included IL-1 receptor like 1, pros
29 been implicated in S. aureus colonization of nasal epithelium and is therefore a key virulence factor
30 variants maintained enhanced tropism for the nasal epithelium and large airways but displayed, over t
31 terial rhinosinusitis is an infection of the nasal epithelium and paranasal sinus mucosa, usually cau
32 ive TRP channels in chemesthesis in oral and nasal epithelium and suggest that TRPV3 may be a molecul
33 lial cells and epithelia, and in vivo in the nasal epithelium and sweat gland highlights the complexi
34 ce shows that Pax6 has distinct roles in the nasal epithelium and the principal tissue components of
35  small airways, tumor-distant large airways, nasal epithelium and uninvolved normal lung (collectivel
36  are trapped on the mucous layer coating the nasal epithelium and upper respiratory tract, and are cl
37 ncluding genes with tissue-specific (IL33 in nasal epithelium) and cross-tissue (CCNC and FBXW7) effe
38 diting and in vitro assays in primary mutant nasal epithelium, and gene expression comparisons were m
39  induced at embryonic day 15.5 in epidermis, nasal epithelium, and surface of the tongue.
40        Lipid mediators are detectable in the nasal epithelium, and their levels distinguish asthma ca
41 isrupts miRNA expression profiles within the nasal epithelium, and these alterations likely influence
42  Nuclear Yellow was injected into the dorsal nasal epithelium, and True Blue was injected into the ol
43 fects arise during embryogenesis and, in the nasal epithelium, appear to be independent of any change
44           To determine the usefulness of the nasal epithelium as a pre-screen for lung-directed thera
45                     PLUNC (palate, lung, and nasal epithelium associated) is a 25-kDa secreted protei
46          The temporal interferon response in nasal epithelium associates with viral clearance in the
47  of P. multocida organisms isolated from the nasal epithelium at the end of the experiment (R2 = 0.86
48                Our findings suggest that the nasal epithelium can serve as a minimally-invasive tool
49                      The short palate, lung, nasal epithelium clone (PLUNC) 1 (SPLUNC1) protein is a
50 otid secretory protein (PSP) and palate-lung-nasal epithelium clone (PLUNC) are novel secretory prote
51 nnate immune proteins of the palate lung and nasal epithelium clone (PLUNC) family in patients with C
52 protein is a member of the palate, lung, and nasal epithelium clone (PLUNC) family, also known as bac
53                            Palate, lung, and nasal epithelium clone (Plunc, now renamed Splunc1) is a
54                  The short palate, lung, and nasal epithelium clone (SPLUNC)1 protein is secreted in
55                      Short palate, lung, and nasal epithelium clone 1 (SPLUNC1) protein is a recently
56                      Short palate, lung, and nasal epithelium clone 1 (SPLUNC1) protein is highly exp
57 dies demonstrate that long palate, lung, and nasal epithelium clone 1 protein (LPLUNC1) is involved i
58 lm activity such as PLUNC (palate, lung, and nasal epithelium clone) family proteins, elafin, collect
59 s with similarity to PLUNC (palate, lung and nasal epithelium clone), which is itself related to the
60 , which we call plunc (for palate, lung, and nasal epithelium clone; GenBankTM accession number U6917
61              This cross-sectional study used nasal epithelium collected in 2015-2018 to compare expre
62 n the proportions of epithelial cells in the nasal epithelium, compared with marked neutrophil inflam
63                      We obtained biopsies of nasal epithelium containing ORNs from RTT patients and a
64 n and fluid transport in the sweat gland and nasal epithelium demonstrated the presence of functional
65                  We investigated the role of nasal epithelium-derived extracellular vesicles (EVs) in
66 re BM-derived epithelial cells in the GI and nasal epithelium detected in CFTR-/- transplanted mice p
67  proteomics to a primary cell model of human nasal epithelium differentiated at air-liquid interface.
68                                          Rat nasal epithelium displayed robust immunoreactivity that
69                                     In human nasal epithelium ex vivo, AS-NS had no impact on mucocil
70 CREB regulation in CF is betaarr2-dependent, nasal epithelium excised from wt mice (Cftr +/+; betaarr
71                                       The LC nasal epithelium exhibited increased cytokine production
72 f genes that are differentially expressed in nasal epithelium following lesioning of the olfactory bu
73 y inefficient at initiating infection of the nasal epithelium following vaccination, and therefore, a
74 tigen distribution, being largely limited to nasal epithelium for intranasally infected guinea pigs a
75 m was to examine molecular mechanisms in the nasal epithelium for sensitization to different allergen
76 transcriptome"), we found that bronchial and nasal epithelium from non-smokers were most similar in g
77 nse in asthma, where the association between nasal epithelium gene expression is likely regulated by
78 esting factors necessary for colonization on nasal epithelium, genetic determinants of nasal carriage
79 fferentially expressed between CF and non-CF nasal epithelium (&gt;/=1.5-fold, P</=0.05).
80 mination were examined in rats in which each nasal epithelium had been irrigated with 0.1-0.5 ml 5% z
81                  plunc mRNA was expressed in nasal epithelium, heart, lung, thymus, and salivary glan
82 inal obstruction, bioelectric defects in the nasal epithelium, histopathologic changes in the trachea
83 TP and its metabolite UDP on polarized human nasal epithelium (HNE), and have compared the pharmacolo
84 t UDCA reduces the expression of ACE2 in the nasal epithelium in humans.
85                                              Nasal epithelium in LC exhibits persistent inflammatory
86        Little is known about the role of the nasal epithelium in long COVID (LC).
87  and DNA methylation data are generated from nasal epithelium including cases (current asthma, N = 25
88 dicate that Omicron successfully infects the nasal epithelium including the olfactory epithelium, but
89                                          The nasal epithelium is a major site of viral entry, with ad
90                                          The nasal epithelium is a plausible entry point for SARS-CoV
91                                          The nasal epithelium is an important target site for chemica
92 helium are transcriptionally distinct and CF nasal epithelium is not a good surrogate for the lung re
93                                          The nasal epithelium is richly invested with peptidergic (su
94                                          The nasal epithelium is the initial entry portal and primary
95                               Given that the nasal epithelium is the primary entry and infection site
96 P production in TG neurons projecting to the nasal epithelium is transiently increased after TDI expo
97 ium was inflammatory response, whereas in CF nasal epithelium it was amino acid metabolism.
98  and near-exclusion of mutant cells from the nasal epithelium mirrored the behaviour of mutant cells
99  while a model of fully differentiated human nasal epithelium (MucilAir(TM)) was utilized to evaluate
100 ssociated with SARS-CoV-2 cell entry, in the nasal epithelium of children vs adults.
101 d human airway epithelia in vitro and to the nasal epithelium of cystic fibrosis mice in vivo.
102  This defect is particularly striking in the nasal epithelium of E2f4-/- mice where ciliated cells ar
103  and debris in the respiratory spaces of the nasal epithelium of ferrets inoculated with AT AIVs.
104        When vitamin C was instilled into the nasal epithelium of human subjects, it effectively activ
105        Odorants are detected as smell in the nasal epithelium of mammals by two G-protein-coupled rec
106  AAV5 to efficiently transduce the lungs and nasal epithelium of mice after repeated administration.
107 magnitude of the potential difference across nasal epithelium of mice homozygous for the most common
108  alters miRNA expression profiles within the nasal epithelium of nonhuman primates.
109 dministration of an adenovirus vector to the nasal epithelium of patients with CF with five escalatin
110 ane conductance regulator (CFTR) gene to the nasal epithelium of patients with cystic fibrosis.
111 sion, function, and regulation of TJs in the nasal epithelium of patients with house dust mite (HDM)-
112 utes to an exacerbated inflammatory state in nasal epithelium of patients with radiological abnormali
113 an provide CFTR activity in the GI tract and nasal epithelium of recipient cystic fibrosis mice.
114 is related to JSRV but induces tumors in the nasal epithelium of sheep and goats.
115  length that is expressed in the presumptive nasal epithelium of the mouse embryo.
116          We identified host responses in the nasal epithelium of the upper respiratory tract differen
117 nea induced by electrical stimulation of the nasal epithelium or delivery of ammonia vapors to the no
118 45365 (ZBTB16) was cross-tissue validated in nasal epithelium (p= 0.003) and associated with five ind
119 particles in CF mice produces changes in the nasal epithelium potential difference assay, consistent
120            The addition of CD4(+) T cell and nasal epithelium prediction databases to the public sphe
121                  Elevated IL-1beta levels in nasal epithelium promoted ILC activation and plasticity
122 e model, recapitulating the structure of the nasal epithelium, provides a useful model to bridge in v
123 tion of non-neuronal cells and damage in the nasal epithelium rather than direct infection of olfacto
124 er of mucus and periciliary mucins to infect nasal epithelium remains unclear.
125 exclusively on free nerve endings within the nasal epithelium, requiring that trigeminal irritants di
126                   Furthermore, injury to the nasal epithelium resulted in increased peripheral nerve
127 l cells evoked by weak (i.e., perithreshold) nasal epithelium shocks (1.0 Hz) in 17/18 cells (mean In
128 e inactivation of olfactory receptors in the nasal epithelium significantly reduced responses to intr
129  Olfrs localized to overlapping zones of the nasal epithelium, suggesting regional biases, but not to
130 bundantly and heterogeneously present in the nasal epithelium, suggesting specialized subtypes.
131  saturable binding of [18F]GV1-57 in primate nasal epithelium, supporting its translational potential
132 leptin administered directly to the brain or nasal epithelium suppresses seizures via direct effects
133 ell-documented, it is less clear whether the nasal epithelium sustains prolonged HRV infections along
134 ine release and morphological changes in the nasal epithelium that are characteristic of AR.
135 d with AIV infection and transmission in the nasal epithelium, the determinant anatomical site of inf
136           By extending to the surface of the nasal epithelium, these chemosensory cells serve to expa
137                       plunc was expressed in nasal epithelium, thymus, and salivary gland during embr
138                Using responses in the ferret nasal epithelium to a panel of H1N1 AIVs, we describe ho
139 er airway diseases, whereby reactions of the nasal epithelium to diverse chemical and physical stimul
140 nating others, despite leaving the path from nasal epithelium to PC intact.
141 scription of the continuum existing from the nasal epithelium to successive divisions of the airways
142 cociliary clearance functions of the in vivo nasal epithelium, to compare lethal [Severe acute respir
143 at is statistically mediated by clinical and nasal epithelium transcriptomic biomarkers of asthma.
144 he ability of S. salivarius to adhere to the nasal epithelium under AR conditions.
145 atabases for unstimulated CD4(+) T cells and nasal epithelium using an elastic net framework.
146  by blocking odorant passage to one half the nasal epithelium via unilateral naris closure, a manipul
147 ingle-cell RNA sequencing (scRNA-Seq) of the nasal epithelium was performed on nonallergic and house
148                    SP nerve fiber density in nasal epithelium was significantly increased 12, 24, and
149                  The order of efficiency for nasal epithelium was: Ba2+ > clofilium >>> TEA = azimili
150 IL-19 is highly expressed in the metaplastic nasal epithelium when compared to normal or hyperplastic
151 tein-coupled receptors, are expressed in the nasal epithelium where they mediate the sense of smell.
152  viruses establish primary infections in the nasal epithelium, where efficient innate immune inductio
153  Similarly, fused is highly expressed in the nasal epithelium, where fused knockouts display bilatera
154 iversity among HCoVs during infection of the nasal epithelium, which is likely to influence downstrea
155 ograde transport by olfactory neurons in the nasal epithelium, which may limit the utility of this ro
156 zyme 2 (ACE2) receptors, highly expressed in nasal epithelium with parallel high infectivity.(1,2) Th
157 tigen redirection occurred mainly across the nasal epithelium without subsequent transport along olfa

 
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