ライフサイエンスコーパス: necrotrophic

1 s' expression with or without infection with necrotrophic Alternaria solani, though no adverse effect

2 Both necrotrophic and biotrophic fungi have larger genomes th

3 Necrotrophic and biotrophic pathogens are resisted by di

4 se tomato, Solanum lycopersicum, plants, and necrotrophic and biotrophic pathogens to show that a com

5 the enhanced resistance of cwm plants to the necrotrophic and vascular pathogens negatively impacted

6 s increased susceptibility of plants against necrotrophic attackers by suppressing the jasmonic acid-

7 the increase in PA production in response to necrotrophic B. cinerea and virulent Pst DC3000 infectio

8 tomato DC3000 and for susceptibility to the necrotrophic bacteria Erwinia caratovora pv.

9 Necrotrophic bacteria within the order Enterobacterales

10 ency was most harmful to the host during the necrotrophic colonization phase.

11 With the elucidation of the necrotrophic disease-synergistic role played by miR6024,

12 s inverse gene-for-gene recognition leads to necrotrophic effector-triggered susceptibility and ultim

13 nverse gene-for-gene interactions leading to necrotrophic effector-triggered susceptibility in the wh

14 nvolves the recognition of pathogen-secreted necrotrophic effectors (NEs) by corresponding wheat NE s

15 ated by the recognition of pathogen-produced necrotrophic effectors (NEs) by specific wheat genes.

16 ne interactions involving the recognition of necrotrophic effectors (NEs) by wheat sensitivity genes

17 the US, was evaluated for SNB resistance and necrotrophic effectors (NEs) sensitivity at the seedling

18 es a template for the study of less explored necrotrophic effectors and their host target functions.

19 Parastagonospora nodorum secretes necrotrophic effectors that target wheat susceptibility

20 hormonal network typically activated by both necrotrophic (ET/JA) and biotrophic (SA) pathogens suppo

21 vora is a devastating plant pathogen causing necrotrophic fire blight disease of apple, pear, and oth

22 n the tomato plant's defense response to the necrotrophic foliar pathogen Botrytis cinerea and the bi

23 sucrose and at the expense of starch during necrotrophic fungal growth.

24 thus limiting the defense function of UPI to necrotrophic fungal infection and insect herbivory.

25 cumulated JA in response to infection by the necrotrophic fungal pathogen Alternaria brassicicola.

26 is required for potato defences against the necrotrophic fungal pathogen Alternaria solani.

27 host plant during successful infection by a necrotrophic fungal pathogen and the resistance response

28 ed JA production and plant resistance to the necrotrophic fungal pathogen B. cinerea, but a negative

29 chanisms involved in plant resistance to the necrotrophic fungal pathogen Botrytis cinerea and their

30 nts displayed enhanced susceptibility to the necrotrophic fungal pathogen Botrytis cinerea, but showe

31 dopsis thaliana leaf during infection by the necrotrophic fungal pathogen Botrytis cinerea.

32 wa2 displayed increased tolerance toward the necrotrophic fungal pathogen Botrytis cinerea.

33 phagosomes are induced in Arabidopsis by the necrotrophic fungal pathogen Botrytis cinerea.

34 against herbivorous M. sexta larvae and the necrotrophic fungal pathogen Botrytis cinerea.

35 terial pathogen Pseudomonas syringae and the necrotrophic fungal pathogen Botrytis cinerea.

36 were highly susceptible to infection by the necrotrophic fungal pathogen Botrytis cinerea.

37 mutants impaired in defense responses to the necrotrophic fungal pathogen Botrytis cinerea.

38 tivity is important for the virulence of the necrotrophic fungal pathogen Botrytis cinerea.

39 efense gene expression and resistance to the necrotrophic fungal pathogen Botrytis cinerea.

40 rop facing threats from Alternaria solani, a necrotrophic fungal pathogen causing early blight diseas

41 Southern leaf blight (SLB), caused by the necrotrophic fungal pathogen Cochliobolus heterostrophus

42 e of wheat (Triticum aestivum) leaves to the necrotrophic fungal pathogen Mycosphaerella graminicola

43 nospora nodorum is an economically important necrotrophic fungal pathogen of wheat.

44 odorum blotch (SNB), a disease caused by the necrotrophic fungal pathogen Parastagonospora nodorum, i

45 ants also exhibit enhanced resistance to the necrotrophic fungal pathogen Rhizoctonia solani.

46 e oryzae, but enhanced susceptibility to the necrotrophic fungal pathogen Rhizoctonia solani.

47 culture filtrate elicitor1 (SCFE1) from the necrotrophic fungal pathogen Sclerotinia sclerotiorum th

48 The necrotrophic fungal pathogen Sclerotinia trifoliorum exh

49 Alternaria brassicicola is an important, necrotrophic fungal pathogen that causes black spot dise

50 investigated how Sclerotinia sclerotiorum, a necrotrophic fungal pathogen that causes disease in a ra

51 y, the sma4 mutant was highly resistant to a necrotrophic fungal pathogen, Botrytis cinerea.

52 adian clock influences susceptibility to the necrotrophic fungal pathogen, Botrytis cinerea.

53 es, caterpillars and aphids, and against the necrotrophic fungal pathogen, Botrytis cinerea.

54 tivation of PGN results in susceptibility to necrotrophic fungal pathogens as well as hypersensitivit

55 phagy exhibit enhanced susceptibility to the necrotrophic fungal pathogens B. cinerea and Alternaria

56 sive genes and compromised resistance to the necrotrophic fungal pathogens Botrytis cinerea and Alter

57 cichoracearum but enhanced resistance to the necrotrophic fungal pathogens Botrytis cinerea and Alter

58 of HUB1 show increased susceptibility to the necrotrophic fungal pathogens Botrytis cinerea and Alter

59 gnaling conferred enhanced resistance to the necrotrophic fungal pathogens Botrytis cinerea and Alter

60 factor cause enhanced susceptibility to the necrotrophic fungal pathogens Botrytis cinerea and Alter

61 FENSIN1.2 (PDF1.2) and for resistance to the necrotrophic fungal pathogens Botrytis cinerea and Alter

62 tion of BIK1 causes severe susceptibility to necrotrophic fungal pathogens but enhances resistance to

63 es conferred heritable resistance to several necrotrophic fungal pathogens, suggesting that disease d

64 echanisms of susceptibility, particularly to necrotrophic fungal pathogens.

65 other hand, increases resistance to the two necrotrophic fungal pathogens.

66 f cognate PINE1 proteins in broad host range necrotrophic fungal pathogens.

67 their host targets are largely unexplored in necrotrophic fungal pathogens.

68 to regulate plant defense responses against necrotrophic fungal pathogens.

69 recently proven vital in plant resistance to necrotrophic fungal pathogens.

70 d cell death-eliciting toxin produced by the necrotrophic fungal plant pathogen Fusarium moniliforme,

71 d shared defense mechanism for resistance to necrotrophic fungi and herbivorous insects.

72 the agronomically and economically important necrotrophic fungi B. cinerea, Alternaria brassicicola,

73 ele displayed enhanced susceptibility to the necrotrophic fungi Botrytis cinerea and Alternaria brass

74 plays a pivotal role in the virulence of the necrotrophic fungi Botrytis cinerea and Sclerotinia scle

75 ation of disease responses to biotrophic and necrotrophic fungi in that it antagonizes salicylic acid

76 le of SA-dependent defense responses against necrotrophic fungi is currently unclear.

77 Plant resistance to necrotrophic fungi is regulated by a complex set of sign

78 th regulatory roles in plant defense against necrotrophic fungi most likely through modulation of gen

79 Plants challenged by pathogens, especially necrotrophic fungi such as Botrytis cinerea, produce hig

80 As play in plant defense against insects and necrotrophic fungi, (2) argue for a reassessment of sign

81 nse, generally associated with resistance to necrotrophic fungi, is attenuated in the bik1 mutant bas

82 e modulate the resistance of hub1 mutants to necrotrophic fungi.

83 ant defenses against herbivorous insects and necrotrophic fungi.

84 defence against insect herbivores and foliar necrotrophic fungi.

85 of MAMP-triggered immunity in resistance to necrotrophic fungi.

86 y regulating SA/JA/ET-mediated resistance to necrotrophic fungi.

87 s (ROS) is critical for pathogenicity in the necrotrophic fungus Alternaria alternata.

88 Alternaria leaf blight (ALB), caused by a necrotrophic fungus Alternaria brassicae is a serious di

89 ingly, the elevated resistance of gai to the necrotrophic fungus Alternaria brassicicola and suscepti

90 iggered immunity and immune responses to the necrotrophic fungus Alternaria brassicicola and the bact

91 ble genes and enhanced susceptibility to the necrotrophic fungus Alternaria brassicicola.

92 Early blight, caused by the necrotrophic fungus Alternaria solani, is an increasing

93 Ascochyta blight, caused by the necrotrophic fungus Ascochyta rabiei, is a major threat

94 production and Arabidopsis resistance to the necrotrophic fungus B. cinerea.

95 ) of sorghum, a foliar disease caused by the necrotrophic fungus Bipolaris cookei (also known as Bipo

96 Target Leaf Spot (TLS) disease caused by the necrotrophic fungus Bipolaris cookei in the SCP.

97 ty to the isolate ND90Pr (Bs(ND90Pr)) of the necrotrophic fungus Bipolaris sorokiniana.

98 s present an increased susceptibility to the necrotrophic fungus Botrytis cinerea and an increased to

99 ence (BOI RNAi) were more susceptible to the necrotrophic fungus Botrytis cinerea and less tolerant t

100 e expression and increased resistance to the necrotrophic fungus Botrytis cinerea and the caterpillar

101 RNAi) increases tomato susceptibility to the necrotrophic fungus Botrytis cinerea and to feeding by l

102 eudomonas syringae pv. tomato DC3000 and the necrotrophic fungus Botrytis cinerea As pldgamma1 mutant

103 Atdpl1 mutants exhibit tolerance to the necrotrophic fungus Botrytis cinerea but susceptibility

104 In Arabidopsis, resistance to the necrotrophic fungus Botrytis cinerea is conferred by eth

105 of lipid species altered in response to the necrotrophic fungus Botrytis cinerea revealed decreases

106 Nup88/MOS7 is essential for immunity to the necrotrophic fungus Botrytis cinerea The mos7-1 mutation

107 interaction of Arabidopsis thaliana with the necrotrophic fungus Botrytis cinerea using millicell cul

108 sion show an increased susceptibility to the necrotrophic fungus Botrytis cinerea, and increased sens

109 of PS improves Arabidopsis resistance to the necrotrophic fungus Botrytis cinerea, consistent with su

110 expression of defense markers induced by the necrotrophic fungus Botrytis cinerea, including the gene

111 cete Hyaloperonospora arabidopsidis, and the necrotrophic fungus Botrytis cinerea.

112 r WRKY33 is essential for defense toward the necrotrophic fungus Botrytis cinerea.

113 defense responses and protection against the necrotrophic fungus Botrytis cinerea.

114 Southern leaf blight (SLB), caused by the necrotrophic fungus Cochliobolus heterostrophus, is a ma

115 among victorin (an effector produced by the necrotrophic fungus Cochliobolus victoriae), TRX-h5 (a d

116 findings suggest an explanation for why the necrotrophic fungus Gibberella fujikuroi, causal agent o

117 We observe that the lesions produced by this necrotrophic fungus on Arabidopsis leaves are smaller wh

118 ically important wheat disease caused by the necrotrophic fungus Parastagonospora nodorum.

119 the enhanced susceptibility of agb1-2 to the necrotrophic fungus Plectosphaerella cucumerina BMM (PcB

120 sis (Arabidopsis thaliana) resistance to the necrotrophic fungus Plectosphaerella cucumerina.

121 Sheath blight (ShB), caused by necrotrophic fungus Rhizoctonia solani, is one of the mo

122 ed global gene expression in response to the necrotrophic fungus Sclerotinia sclerotiorum in 23 Arabi

123 Botrytis cinerea is a well-studied necrotrophic fungus taken as a model organism in fungal

124 S. sclerotiorum is a necrotrophic fungus that causes stem rot diseases on mor

125 nt parasitic styles: a vascular bacterium, a necrotrophic fungus, and a biotrophic oomycete.

126 allenge by a hemi-biotrophic bacterium and a necrotrophic fungus, as well as in the growth response t

127 pretreatment induced resistance against the necrotrophic fungus, Botrytis cinerea The induced resist

128 a pathogen stress response, specifically for necrotrophic fungus, is poorly understood.

129 ve to aos, opr3 has enhanced resistance to a necrotrophic fungus.

130 ty against avirulent bacteria and a virulent necrotrophic fungus.

131 ng fructan metabolites, during the switch to necrotrophic growth and reproduction.

132 We hypothesize that the switch to necrotrophic growth enables the fungus to evade the effe

133 ase in the penetrated epidermis cell, before necrotrophic growth is initiated upon further host colon

134 le during the transition from symptomless to necrotrophic growth of Septoria.

135 m Phytophthora that are expressed during the necrotrophic growth phase, as well as programmed cell de

136 host cells followed by a rapid transition to necrotrophic growth resulting in disease lesions.

137 s indicate that this bacterium is capable of necrotrophic growth, is able to metabolize halogenated c

138 g of host transcription marks this switch to necrotrophic growth.

139 pressorium on the cuticle and biotrophic and necrotrophic hyphae in its host.

140 ophic hyphae was unaffected in RNAi strains, necrotrophic hyphae showed severe distortions.

141 all rigidity in appressoria and fast-growing necrotrophic hyphae, its rigorous downregulation during

142 re detected in cell walls of appressoria and necrotrophic hyphae.

143 er, the AtECS plants exhibited resistance to necrotrophic infection and salt stress, while the pad2-1

144 es can result in impaired plant tolerance to necrotrophic infection or abiotic stress.

145 iption is downregulated by methyl JA (MeJA), necrotrophic infection or mechanical injury.

146 nses by switching from a hemibiotrophic to a necrotrophic infection program, thereby gaining an advan

147 -HAC boosts JA-dependent defenses during the necrotrophic infection stage of F. graminearum but suppr

148 and senescence, the pathogens switch to the necrotrophic lifestyle and cause decay.

149 ls during the transition from biotrophy to a necrotrophic lifestyle.

150 host plant and later switch to a destructive necrotrophic lifestyle.

151 ate the transition from the quiescent to the necrotrophic lifestyle.

152 merina alternates between hemibiotrophic and necrotrophic lifestyles, depending on initial spore dens

153 abidopsis thaliana) immune responses against necrotrophic microorganisms via a SA-independent mechani

154 n plants and biotrophic, hemibiotrophic, and necrotrophic oomycetes affect disease progression.

155 ions, such as the marine carbon pump through necrotrophic-parasitism, facilitating the export of diat

156 is required for potato defences against the necrotrophic pathogen A. solani.

157 ant immunity, and facilitates disease by the necrotrophic pathogen A. solani.

158 nse, rendered plants more susceptible to the necrotrophic pathogen Alternaria brassicicola by suppres

159 stress as well as in the defense against the necrotrophic pathogen Alternaria brassicicola.

160 novel components of plant immunity toward a necrotrophic pathogen and provides mechanistic insights

161 However, upon necrotrophic pathogen attack, how JA-mediated defense re

162 ols broad-spectrum disease resistance to the necrotrophic pathogen Botrytis cinerea and contributes t

163 equired for JA-mediated defenses against the necrotrophic pathogen Botrytis cinerea and for the shade

164 tudy suggests that plant defense against the necrotrophic pathogen Botrytis cinerea is primarily quan

165 SIB2 are rapidly and strongly induced by the necrotrophic pathogen Botrytis cinerea.

166 ociated with increased susceptibility to the necrotrophic pathogen Botrytis cinerea.

167 ponsive gene PDF1.2 and in resistance to the necrotrophic pathogen Botrytis cinerea.

168 th plant growth and defense responses to the necrotrophic pathogen Botrytis cinerea.

169 Golovinomyces cichoracearum, but not to the necrotrophic pathogen Botrytis cinerea.

170 rophic pathogen Pseudomonas syringae and the necrotrophic pathogen Botrytis cinerea.

171 was sufficient to change the host range of a necrotrophic pathogen but not a hemibiotroph or saprotro

172 How QDR against a generalist necrotrophic pathogen evolved and whether it is driven b

173 ce to pitch canker, a disease incited by the necrotrophic pathogen Fusarium circinatum.

174 t be advantageous to the plant by preventing necrotrophic pathogen growth in tissues undergoing PCD.

175 ay, during JA-mediated defense response upon necrotrophic pathogen interaction.

176 PR1 during JA mediated defense response upon necrotrophic pathogen interaction.

177 ns of wheat (Triticum aestivum), including a necrotrophic pathogen of barley, a hemibiotrophic pathog

178 Stagonospora nodorum is a major necrotrophic pathogen of wheat that causes the diseases

179 Against infection by the necrotrophic pathogen Plectosphaerella cucumerina, nrpe1

180 Botrytis cinerea is a major necrotrophic pathogen responsible for significant crop l

181 s required to restrict the spread of another necrotrophic pathogen, Alternaria brassicicola, suggesti

182 developmental stages and in response to the necrotrophic pathogen, Sclerotinia sclerotiorum.

183 tibility to Alternaria brassicicola, another necrotrophic pathogen, suggesting a broader role for the

184 trates that CMNs transfer jasmonic acid from necrotrophic pathogen-infected to uninfected tomato plan

185 ced resistance to Cochliobolus miyabeanus, a necrotrophic pathogen.

186 by necrosis that occurred in response to the necrotrophic pathogen.

187 1 protein in plant immunization against this necrotrophic pathogen.

188 in shaping resistance against a cosmopolitan necrotrophic pathogen.

189 the outcome of NLR-miRNA interaction during necrotrophic pathogenesis is a hindrance to the deployme

190 atively impacts plant immune response during necrotrophic pathogenesis.

191 or envelopment of the diatom nuclei infers a necrotrophic-pathogenic interaction.

192 rmones critical for plant resistance against necrotrophic pathogens and chewing herbivores.

193 way that enables the plant to defend against necrotrophic pathogens and herbivorous insects apparentl

194 The fungal genus Armillaria contains necrotrophic pathogens and some of the largest terrestri

195 Necrotrophic pathogens are important plant pathogens tha

196 Necrotrophic pathogens are notorious for their aggressiv

197 While necrotrophic pathogens are sensitive to jasmonic acid (J

198 Because wound-invading necrotrophic pathogens are vulnerable to biocontrol, ant

199 Biotrophic and necrotrophic pathogens attack plants using different str

200 In contrast, necrotrophic pathogens benefit from host cell death, so

201 branch of immunity and are more resistant to necrotrophic pathogens but not insect herbivores.

202 ospora nodorum, to review the means by which necrotrophic pathogens circumvent, or outright hijack, t

203 between defence against (hemi)biotrophic and necrotrophic pathogens has been widely described across

204 ts of host resistance against biotrophic and necrotrophic pathogens have been documented in various p

205 Few studies of quantitative resistance to necrotrophic pathogens have used large plant mapping pop

206 nhance understanding of host manipulation by necrotrophic pathogens in causing disease.

207 -dependent defence responses engaged against necrotrophic pathogens in root tissue.

208 f mechanisms of resistance to biotrophic and necrotrophic pathogens is crucial for activating or supp

209 derstanding of the plant defense response to necrotrophic pathogens is limited.

210 Genetic resistance to disease incited by necrotrophic pathogens is not well understood in plants.

211 This work suggests that these necrotrophic pathogens may thrive by subverting the resi

212 Necrotrophic pathogens produce toxins, necrosis-inducing

213 cells, whereas JA activates defense against necrotrophic pathogens that kill host cells for nutritio

214 ssociated PCD could leave them vulnerable to necrotrophic pathogens that thrive on dead host cells.

215 No elevated resistance toward herbivores or necrotrophic pathogens was detected for cpk28 plants, ei

216 Plant resistance against necrotrophic pathogens with a broad host range is comple

217 ant role in plant defense, especially toward necrotrophic pathogens, and highlight a novel connection

218 by a dramatic increase in susceptibility to necrotrophic pathogens, such as Botrytis cinerea.

219 rsely, ros1 displayed enhanced resistance to necrotrophic pathogens, which was not associated with in

220 n resistance of Arabidopsis thaliana against necrotrophic pathogens.

221 dentified a role for TGA3 in defense against necrotrophic pathogens.

222 rky33 mutant, which is highly susceptible to necrotrophic pathogens.

223 plays a critical role in plant resistance to necrotrophic pathogens.

224 on factor that is required for resistance to necrotrophic pathogens.

225 ctivators of WRKY33 in plant defense against necrotrophic pathogens.

226 hance our understanding of plant immunity to necrotrophic pathogens.

227 the regulation of plant defense responses to necrotrophic pathogens.

228 hways mediating responses to P. syringae and necrotrophic pathogens.

229 nses to resistance to several biotrophic and necrotrophic pathogens.

230 plex roles in defense against biotrophic and necrotrophic pathogens.

231 in response to wounding, feeding insects, or necrotrophic pathogens.

232 that are less vulnerable to modification by necrotrophic pathogens.

233 isms of plant defense against biotrophic and necrotrophic pathogens.

234 te-based defense responses to herbivores and necrotrophic pathogens.

235 teraction with biotrophic, hemibiotrophic or necrotrophic pathogens.

236 phic pathogen without becoming vulnerable to necrotrophic pathogens.

237 ar from being understood, especially against necrotrophic pathogens.

238 timise defences against (hemi)biotrophic and necrotrophic pathogens.

239 required for defense against biotrophic and necrotrophic pathogens; however, the upstream and downst

240 factor is important for plant resistance to necrotrophic pathogens; therefore, elucidation of its fu

241 hemibiotrophic Pseudomonas syringae and the necrotrophic Pectobacterium carotovorum bacteria.

242 host tissue asymptomatically, followed by a necrotrophic phase, during which host-cell death is indu

243 ere highly expressed by the mycobiota at the necrotrophic phase, showing an active pathogen response

244 at the switch between the biotrophic and the necrotrophic phases.

245 Here we characterized a CP (SsCP1) from the necrotrophic phytopathogen Sclerotinia sclerotiorum.

246 In the case of Sclerotinia sclerotiorum, a necrotrophic phytopathogen, a secreted protein named SsP

247 ynthase, encoded by the BcBOT2 gene from the necrotrophic plant pathogen Botrytis cinerea, catalyzes

248 pecies and diversifying selection within the necrotrophic plant pathogen ecological niche.

249 brassicicola is a successful saprophyte and necrotrophic plant pathogen.

250 l death as a component of diseases caused by necrotrophic plant pathogens.

251 re caused by biotrophic, hemibiotrophic, and necrotrophic plant pathogens.

252 The necrotrophic plant-pathogen fungus Botrytis cinerea prod

253 e resistance toward (hemi)biotrophic but not necrotrophic rice pathogens.

254 As a necrotrophic specialist, it deploys effector proteins th

255 The diversity of virulence strategies in necrotrophic species corresponds to multifaceted host im

256 ession of effector-triggered necrosis at the necrotrophic stage by an NLR receptor in plants.

257 transition from the biotrophic stage to the necrotrophic stage in disease symptom expression are mai

258 thways associated with senescence during the necrotrophic stage of fungal development.

259 stabilizes APIP5 to prevent necrosis at the necrotrophic stage.

260 nal activity and protein accumulation at the necrotrophic stage.

261 or enhanced JA-dependent defenses during the necrotrophic stages of infection.

262 mechanism includes sequential biotrophic and necrotrophic stages.

263 SsPINE1 enhances S. sclerotiorum necrotrophic virulence by specifically interacting with

264 ic vs. non-pathogenic) and pathogenic niche (necrotrophic vs. biotrophic).