ライフサイエンスコーパス: negative pressure

1 ntilator (20 secs, 14 F catheter, 200 cm H2O negative pressure).

2 gram, where water is both supercooled and at negative pressure.

3 e LMkappaT emerges from the no man's land at negative pressure.

4 ry, and they appear to be unstable except at negative pressure.

5 us, rather than to loss of responsiveness to negative pressure.

6 essure variability by oscillatory lower body negative pressure.

7 G activity and the GG reflex to upper airway negative pressure.

8 during, and immediately after application of negative pressure.

9 t on the XII reflex response to upper airway negative pressure.

10 ined the same throughout different levels of negative pressure.

11 Spaceflight and lower-body negative pressure.

12 -tidal CO(2) and a number of intrapharyngeal negative pressures.

13 in-phonon coupling, and its correlation with negative pressures.

14 liquids, on the formation of cavities under negative pressures.

15 or bubbles emerging from metastable water at negative pressures.

16 So how do plants transport water under negative pressure?

17 nergy short pulses (1 MHz; five cycles; peak negative pressure, 0.35 MPa) of ultrasound emitted at a

18 bilization and flow of liquid water at large negative pressures (-1.0 MPa or lower), continuous heat

19 creased cardiac filling by use of lower-body negative pressure (-15 and -30 mm Hg), and after saline

20 whole nerve reflex response to upper airway negative pressure (-20 cm H2O) at any 5HT concentration

21 plasma norepinephrine response to lower body negative pressure (3.0 +/- 0.3 vs. 2.0 +/- 0.2 nmol/l at

22 negative pressure group (median duration of negative pressure, 4 days) and 802 in the standard dress

23 tral hypovolemia was induced with lower body negative pressure (-60 mm Hg) and pain by the cold press

24 e further reduced by hypovolemia (lower body negative pressure, -60 mm Hg).

25 o2 in these cells, whereas both positive and negative pressure activated piezo1 in a similar manner.

26 This negative pressure acts as thrust forces on the anterior

27 c retardation of OMP folding places a strong negative pressure against spontaneous incorporation of O

28 hese metachronal swimmers rely on generating negative pressure along their surfaces to generate forwa

29 By following the maximum range of negative pressure, along with the observed slight decrea

30 city to mitigate COVID-19 impacts along with negative pressures already present.

31 Negative pressure also evoked single Cl- channel activit

32 In human embryonic kidney 293 cells, negative pressure also increased the window current (250

33 tive stimuli (e.g. rapidly imposed pulses of negative pressure) also activate these muscles.

34 oximately proportional to the square of peak negative pressure amplitude and were statistically signi

35 Ultrasound at 1 MHz, with a peak negative pressure amplitude of 0.75 MPa, was applied to

36 as brisk GG reflex activation in response to negative pressure (amplitude: +78.5 +/- 28.3 % baseline

37 ry baroreflexes were perturbed by lower body negative pressure and head-down tilt.

38 muscles to respond to rising intrapharyngeal negative pressure and increasing Co(2) during sleep, (3)

39 stimulated activity, or TRPV1, which sensed negative pressure and inhibited activity.

40 ile preload was manipulated using lower body negative pressure and rapid saline infusion to define LV

41 ile preload was manipulated using lower body negative pressure and rapid saline infusion.

42 lary wedge pressure and SV during lower body negative pressure and saline loading in 7 men (25+/-2 ye

43 relationship between varying intrapharyngeal negative pressures and genioglossal muscle activation (G

44 ere constructed during decreases (lower body negative pressure) and increases (saline infusion) in ca

45 phy at baseline, -15 and -30 mmHg lower-body negative pressure, and 15 and 30 ml kg(-1) saline infusi

46 echocardiography) at rest, during lower-body negative pressure, and after saline infusion before and

47 sfer with the evaporation of liquid water at negative pressure, and continuous extraction of liquid w

48 ng decreased cardiac filling with lower-body negative pressure, and increased filling with saline inf

49 ers, baseline, with two stages of lower body negative pressure, and repeat baseline with two stages o

50 tracheostomized patient would be exposed to negative pressure, and that high levels of muscle activa

51 us, rather than to loss of responsiveness to negative pressure, and that this wakefulness stimulus ma

52 ith or without concomitant administration of negative pressure, antimicrobials, or photosensitizers,

53 egative pressure application, to the maximum negative pressure application of -20 mmHg (difference, 2

54 One eye of each patient underwent negative pressure application with the MPD.

55 se-dependent increase from baseline, without negative pressure application, to the maximum negative p

56 ye of each subject was randomized to receive negative pressure application; the fellow eye served as

57 Negative pressure applied to on-cell membrane patches ac

58 SDK channels were activated in response to negative pressure applied to patches.

59 of TREK-1 channels by mechanical stretch and negative pressure applied to the cell membrane.

60 n the NPR, we quantified GG EMG responses to negative pressure applied to the isolated upper airway i

61 embrane deformation in response to a step in negative pressure applied to the membrane by a micropipe

62 tric coupling in single-phase oxides using a negative pressure approach.

63 Only when the negative pressures approach -100 bar does cavitation occ

64 logical processes that involve liquids under negative pressure are vulnerable to the formation of cav

65 order to encourage admission before force or negative pressures are used.

66 l upper airway respiratory stimuli, possibly negative pressure, are important in mediating the increa

67 Lung volume was altered with continuous negative pressure as applied to the chest wall with a po

68 em surfactants support water transport under negative pressure as explained by the cohesion-tension t

69 Relying on negative pressure, as opposed to high pushing pressure,

70 Negative pressures associated with multiaxial strain and

71 s were acquired after 2 minutes of sustained negative pressure at each target pressure to allow for s

72 no systematic reduction in the GG reflex to negative pressure at sleep onset.

73 rm stabilizes interfacial binding due to the negative pressure at the hydrocarbon-water interface.

74 igid head-out shell equipped with a positive/negative pressure attachment for manipulation of extrath

75 We define a negative pressure between the skeleton/membrane connecti

76 non-invasively by externally applying focal negative pressure bilaterally to the neck.

77 uilliform swimmers-which produce thrust with negative pressure but do so through undulatory mechanics

78 able to repair embolized xylem vessels under negative pressure, but its hydraulic vulnerability segme

79 During LHR treatment, two 6-L negative pressure canisters were used to capture 30 seco

80 Application of lower body negative pressure caused progressive reductions of R-R i

81 uscle, ventilatory, and arousal responses to negative-pressure challenges during sleep in 19 healthy

82 th supine treadmill exercise in a lower-body negative pressure chamber (EX group).

83 e line relating SV to PCWP during lower-body negative pressure characterized the steepness of the Sta

84 99Mo/99mTc generators were placed in the negative-pressure clean room to ensure a more efficient

85 bodies of these carangiform swimmers through negative pressure comprises 28% of the total thrust prod

86 However, a negative pressure control of spin-phonon coupling for en

87 g multiple FUS-MB treatments with fixed peak-negative pressures, de novo CCM formation was reduced by

88 t significant portions of inspiration, i.e., negative pressure dependence (Level 4).

89 -S) S-S molecular mode has an unconventional negative pressure dependence, whereas other peaks stiffe

90 a large pressure-driven exchange rate and a negative pressure-dependent activation volume, reflectin

91 By placing the lower body in a negative pressure device (LBNP) that pulls fluid away fr

92 ssure wound therapy, with application of the negative pressure device immediately after repair of the

93 Brief exposure to lower-body negative pressure did not affect these parameters.

94 Acute exposure to 25-mm Hg lower-body negative pressure did not alter optic nerve head or reti

95 t for at least nine indoor air exchanges for negative pressure difference testing and four indoor air

96 binet opposite OR 2, which was maintained at negative pressure differentials, then was poured into bo

97 gth of stay was significantly reduced in the negative pressure dressing group [6.1 vs 14.7 days, P =

98 included, with 25 patients randomized to the negative pressure dressing group and 25 to the standard

99 The effect of prophylactic negative pressure dressing of closed incisional wounds o

100 the abdominal wall are being developed from negative pressure dressing therapies to acellular allogr

101 Prophylactic use of negative pressure dressings for closed laparotomy wounds

102 en to investigate the effect of prophylactic negative pressure dressings on postoperative surgical si

103 ally more collapsible upper airway with more negative pressure driven muscle activation.

104 A novel, simple, negative pressure-driven device with manually operated m

105 First, we updated the model for negative pressure-driven inflation that allowed incorpor

106 mming direction, such an airfoil experiences negative pressure due to both its shape and pitching mov

107 ditions and 10 to 20 minutes into lower-body negative pressure exposure.

108 A negative pressure face shield (NPFS) was developed to co

109 By contrast, application of a negative pressure failed to activate piezo2 in these cel

110 Understanding the role of negative pressure fields in metachronal swimmers may pro

111 A striking result from our simulations is negative pressure-flow correlations observed in several

112 fiber was carried out by applying 11 kPa of negative pressure for 3 s.

113 Maximal negative pressures found in plants are around -100 bar,

114 tained a bullet and responded to positive or negative pressure from the recording pipette were consid

115 y, dissociating the influences of pharyngeal negative pressure, from inspiratory airflow, resistance,

116 during slow (physiological) oscillations in negative pressure generated spontaneously and passively

117 ation of the muscle, combined with increased negative-pressure generation during inspiration.

118 ions were significantly more frequent in the negative pressure group (7.0% vs 0.6%; difference, 6.95%

119 , 1608 (99%) completed the study: 806 in the negative pressure group (median duration of negative pre

120 apnea based on pharyngeal closing pressure: negative pressure group (pharyngeal closing pressure les

121 s diagnosed in 29 participants (3.6%) in the negative pressure group and 27 (3.4%) in the standard dr

122 lysis showed increased adverse events in the negative pressure group and futility for the primary out

123 ng the contraction of the vortex core as the negative pressure grows back to positive values, the vor

124 Negative pressure has emerged as a powerful tool to tail

125 activity and reflex response to upper airway negative pressure in 15 decerebrated, vagotomized, paral

126 ing uniform three-dimensional strain-induced negative pressure in nanocomposite films of (EuTiO(3))(0

127 n coefficient, and we use it here to achieve negative pressure in perovskite-phase CsPbI(3) embedded

128 bility of the actuator is utilized to create negative pressure in the ampoule and collect ISF.

129 tein effects these changes by decreasing the negative pressure in the headgroup region of the outer l

130 ditionally, specific conditions, such as the negative pressure in water transporting xylem vessels, m

131 on is what generally limits the magnitude of negative pressures in liquids that contain lipid bilayer

132 saturation in the vapour phase of water into negative pressures in the liquid phase, stabilization an

133 expressing human embryonic kidney 293 cells, negative pressure increased Na(V) peak currents by 27+/-

134 Furthermore, ultrasound exposure (1 MPa peak negative pressure) increased the distance at which Dox c

135 gioVue) of the optic nerve head at different negative pressure increments of -5 mmHg, starting from 0

136 solution micro-CT imaging, revealed a higher negative pressure inside the airway of Dp16 mice compare

137 epiglottic pressures during basal breathing, negative-pressure (iron-lung) ventilation, heliox breath

138 The channel is only active after negative pressure is applied to cell attached patches, c

139 Negative pressure is one potential stimulus for this neu

140 s range of blood gases: (1) the GG reflex to negative pressure is unchanged; (2) slow airway pressure

141 ical Examiner's Office (Everett, WA, USA) in negative-pressure isolation suites during February and M

142 pattern generator activity, intrapharyngeal negative pressure itself modulates genioglossus activity

143 three 5 min stages of progressive lower body negative pressure (LBNP) (-15, -30 and -45 mmHg) before

144 Volume redistributions with lower body negative pressure (LBNP) are similar to those that occur

145 0 min at rest and during 5 min of lower body negative pressure (LBNP) at -10 and -40 mmHg (n = 11).

146 mpathetic activation, produced by lower body negative pressure (LBNP) at -40 mmHg, on cerebrovascular

147 ic nerve activity (SNA) evoked by lower body negative pressure (LBNP) at rest and during moderate-int

148 isengagement of baroreflexes with lower body negative pressure (LBNP) can engage the sympathetic nerv

149 ts undergoing monitoring with the lower body negative pressure (LBNP) device.

150 tral blood volume was reduced via lower-body negative pressure (LBNP) during normothermia, whole-body

151 lex sympathetic activation during lower body negative pressure (LBNP) evoked decreases in muscle oxyg

152 achieved by stepwise increases in lower-body negative pressure (LBNP) in 14 healthy young volunteers.

153 earm vasodilator responses during lower body negative pressure (LBNP) in 21 non-obstructive hypertrop

154 application of -20 and -30 mm Hg lower-body negative pressure (LBNP) in 24 patients with chronic hea

155 A validated lower body negative pressure (LBNP) model was used to induce progre

156 ans (n = 20) during a progressive lower body negative pressure (LBNP) protocol designed to cause pres

157 er carotid sinus massage (CSM) or lower body negative pressure (LBNP) received Paxil (20 mg/d) or pla

158 Lower body negative pressure (LBNP) simulates the effects of gravit

159 omen underwent an initial maximal lower body negative pressure (LBNP) test to place them into a low (

160 Progressive lower body negative pressure (LBNP) to onset of cardiovascular coll

161 nce was determined by progressive lower-body negative pressure (LBNP) to presyncope.

162 articipants underwent progressive lower-body negative pressure (LBNP) until pre-syncope; end-tidal ca

163 lerance was assessed using graded lower-body negative pressure (LBNP) until the onset of symptoms ass

164 hen a cold pressor test (CPT) and lower body negative pressure (LBNP) were superimposed upon heating.

165 ary wedge pressure (PCWP), during lower-body negative pressure (LBNP) while subjects are normothermic

166 ctions in were accomplished using lower body negative pressure (LBNP), while increases in were accomp

167 aemorrhagic challenge imposed via lower-body negative pressure (LBNP).

168 n of sympathetic nerves evoked by lower body negative pressure (LBNP).

169 sympathetic activation evoked by lower body negative pressure (LBNP).

170 re recorded during nonhypotensive lower body negative pressure (LBNP; -10 mm Hg) and nonhypertensive

171 C, followed by the application of lower body negative pressure (LBNP; -30 mmHg).

172 efore and after the intervention, lower body negative pressure (LBNP; 3 min at -15, -30 and -45 mmHg)

173 pre-syncopal limited progressive lower-body negative pressure (LBNP; a validated model for simulatin

174 were also obtained during graded lower body negative pressure (LBNP; activates baroreflex-mediated s

175 Subjects experienced lower-body negative-pressure (LBNP) of 0, 15 and 30 mmHg during nor

176 % NaCl) on endogenously mediated (lower body negative pressure [LBNP]) and exogenously mediated (brac

177 o hypotensive hypovolemic stress (lower body negative pressure [LBNP]) in healthy human males.

178 lk modulus, kappa = 140 +/- 20 kPa; applying negative pressures leads to volumetric expansion of the

179 rodes prevented the shift in kinetics, while negative pressure led to an abrupt shift to fast inactiv

180 ormula: see text] monolayer ice (LD-48MI) at negative pressures (<-0.3 GPa).

181 sap is well known to operate under absolute negative pressure, many terrestrial, vascular plants sho

182 (GG) activation in response to upper airway negative pressure may be an important mechanism in the m

183 airway pressure (CPAP) was applied to reduce negative pressure mediated muscle activation.

184 airway pressure (CPAP) was applied to reduce negative pressure-mediated muscle activation).

185 nd, vs standard wound dressing not involving negative pressure (n = 763).

186 red with control nights before and after the negative pressure nights.

187 nd 37.8 +/- 29.1 events/h during each of two negative pressure nights; p < 0.001) that were associate

188 d pulses of 6-microsecond duration at a peak negative pressure of 15 MPa and a pulse repetition frequ

189 Circumpapillary CD measurements at target negative pressures of -10 mmHg, -15 mmHg, and -20 mmHg w

190 ent loading conditions: baseline, lower-body negative pressures of -15 and -30 mm Hg, and rapid salin

191 before and after the application of topical negative pressures of -50, -70, and -120 mmHg, using las

192 Oscillatory lower body negative pressure (OLBNP) was used to create consistent

193 ressure fluctuations (oscillatory lower body negative pressure, OLBNP) across a range of frequencies

194 The effects of negative pressure on open probability were graded as a f

195 type CaAg(5) phase is found to exhibit large negative pressures on each Ca atom, which are concentrat

196 in pure bulk water only occurs at much more negative pressures on the relevant timescales.

197 te emphasis on scientific outputs, and other negative pressures on the scientific enterprise.

198 the perovskite cage, equivalent to exerting "negative pressure" on the perovskite structures.

199 c changes in airway mechanoreceptor stimuli (negative pressure or flow) were highly correlated with w

200 2 fashion with the combination of lower body negative pressure or not (normovolemia), and ice water o

201 If negative pressure (or another local airway stimulus) wer

202 volved a specialized dressing used to create negative pressure over the closed wound vs the surgeon's

203 volved a specialized dressing used to create negative pressure over the wound, vs standard wound dres

204 Hz) decreased progressively with lower body negative pressure (p < .001).

205 hasic and tonic GG EMG, and the GG reflex to negative pressure (Pchoa = -12.5 cm H(2)O).

206 w that islets treated with pFUS at low (peak negative pressure (PNP): 106kPa, spatial peak temporal p

207 ad reduction using a standardized lower-body negative pressure protocol.

208 based on acoustic parameters, including peak negative pressure, pulse length, and pulse repetition fr

209 ), the correlation with GGEMG was robust for negative pressure (R(2) = 0.98) and less strong for othe

210 onging effects of a sustained square wave of negative pressure (range, -4.0 to -14.9 cmH2O) sufficien

211 traluminal EVT) and supplied with continuous negative pressure (ranging between 75 and 150 mmHg).

212 of the central circuitry that mediates this negative pressure reflex (NPR).

213 Substantial data support the role of a local negative pressure reflex in modifying genioglossal activ

214 These data suggest that while the negative pressure reflex is able to maintain GGEMG durin

215 ct plane is introduced into one of these two negative-pressure regions, breaking the symmetry equival

216 s to determine which GG premotoneurons relay negative pressure-related information to the hypoglossal

217 rtificial muscles can be driven by fluids at negative pressures (relative to ambient).

218 activity on and off by applying positive and negative pressures, respectively.

219 ly increased their open probability, whereas negative pressure reversibly decreased it.

220 Testing was conducted in a negative-pressure room.

221 th no active pulmonary disease measured in a negative-pressure room.

222 volunteers were quarantined in functionally negative pressure rooms (Oxford, UK) for 14 days and unt

223 intranasal drops and remained in individual negative pressure rooms for a minimum of 14 days.

224 Negative pressure (suction) applied through the pipette

225 th the same properties as those activated by negative pressure, suggesting that the channels were str

226 After completing a maximal lower body negative pressure test to determine level of orthostatic

227 ritical information to assess impacts during negative pressure testing, adding room-specific indoor a

228 At negative pressure, the fluorescence images are consisten

229 Endoscopic negative pressure therapy is a novel and successful trea

230 nts were treated with endoluminal endoscopic negative pressure therapy with simultaneous feeding opti

231 sure ulcers: wound cleansers, repositioning, negative pressure therapy, debridement, enteral and pare

232 erial and achieving the exotic condition of "negative pressure." This approach is hypothetically gene

233 en decreased symmetrically when lowering the negative pressure to baseline.

234 Application of negative pressure to cell-attached patches (-20 mmHg) ca

235 s over three isochores at high, ambient, and negative pressures to determine the thermodynamic stabil

236 Progressive lower-body negative pressure (to -50 mm Hg; LBNP) was used to exami

237 e was associated with 23% greater lower body negative pressure tolerance using an active impedance th

238 Lower body negative pressure tolerance was reduced after both condi

239 ngle-atom chain, linked by a tension-driven (negative-pressure) transformation.

240 olunteers by applying progressive lower body negative pressure (under two experimental conditions: a)

241 lled hydrophilic nanochannels under enormous negative pressures up to -7 MPa.

242 e VILI compared to physiologically-analogous negative pressure ventilation (NPV) devices.

243 d GGEMG during both basal breathing (BB) and negative pressure ventilation (NPV) during wakefulness (

244 itive pressure ventilation (IPPV) to cuirass negative pressure ventilation (NPV) was investigated in

245 Using a model of passive negative pressure ventilation, we have previously report

246 fic personalized ventilation strategies; and negative pressure ventilation.

247 y of positive-pressure ventilation (PPV) and negative-pressure ventilation (NPV).

248 esistances were reduced significantly during negative-pressure ventilation (P < .05 and P < .03, resp

249 Negative-pressure ventilation improved the cardiac outpu

250 Negative-pressure ventilation improves cardiac output in

251 We investigated the effect of negative-pressure ventilation on cardiac output in 11 ch

252 ermittent positive-pressure ventilation, and negative-pressure ventilation was delivered with the Hay

253 pressure ventilation and after 15 minutes of negative-pressure ventilation.

254 ssure generated spontaneously and passively (negative pressure ventilator).

255 Oscillatory lower body negative pressure was applied at six frequencies from 0.

256 Progressive lower body negative pressure was applied in 5-min stages until the

257 l area of the lining flap were measured when negative pressure was applied.

258 We found that the application of negative pressure was associated with the development of

259 device overnight for 1 year and the applied negative pressure was programmed by subtracting a refere

260 he relationship between GGEMG and epiglottic negative pressure was tight across all conditions in bot

261 Lower body negative pressure was utilized to induce a range of symp

262 nerve activity during progressive lower-body negative pressure were not different between trials.

263 ffer when a cold pressor test and lower body negative pressure were superimposed upon heating.

264 me of the units included patient rooms under negative pressure, while most were maintained at a neutr

265 ose-dependent increase with the induction of negative pressure, while RNFL thickness measurements rem

266 Negative pressure with the ocular pressure adjusting pum

267 merged: a current activated in hair cells by negative pressure, with some similarity to the transduct

268 Vascular plants transport water under negative pressure without constantly creating gas bubble

269 vs 16% with glucose >150 mg/dL); and use of negative pressure wound therapy (9.7% with vs 15% withou

270 It is not known if incisional negative pressure wound therapy (iNPWT) can reduce SSI r

271 Incisional negative pressure wound therapy (n = 785), which involve

272 e aim of this study was to determine whether negative pressure wound therapy (NPWT) applied to primar

273 Negative pressure wound therapy (NPWT) has become a popu

274 Negative pressure wound therapy (NPWT) has emerged as an

275 Negative pressure wound therapy (NPWT) has increasingly

276 We investigated whether negative pressure wound therapy (NPWT) improves the prop

277 manufactured using 3D printing for use with negative pressure wound therapy (NPWT) in the management

278 T) was undertaken to determine the effect of negative pressure wound therapy (NPWT) on closed incisio

279 e of this study is to evaluate the effect of Negative Pressure Wound Therapy (NPWT) on closed surgica

280 Determine if negative pressure wound therapy (NPWT) reduces surgical

281 hanges in the intestines during conventional negative pressure wound therapy (NPWT), and NPWT using a

282 risk [RR], 1.58 [95% CI, 1.20 to 2.08]) and negative pressure wound therapy (RR, 1.49 [CI, 1.11 to 2

283 ctive studies have suggested that the use of negative pressure wound therapy can potentially prevent

284 It is not known if negative pressure wound therapy can reduce infection rat

285 ioperative glucose concentrations, and using negative pressure wound therapy can reduce the rate of s

286 This study seeks to evaluate the efficacy of negative pressure wound therapy for surgical-site infect

287 I occurred in 9.7% (6/62) of patients in the negative pressure wound therapy group and in 31.1% (19/6

288 5.84% (45 of 770 patients) of the incisional negative pressure wound therapy group and in 6.68% (50 o

289 11.4% [72 of 629 patients] in the incisional negative pressure wound therapy group vs 13.2% [78 of 59

290 s do not support routine use of prophylactic negative pressure wound therapy in obese women after ces

291 closure during pancreaticoduodenectomy using negative pressure wound therapy in patients at high risk

292 indings do not support the use of incisional negative pressure wound therapy in this setting, althoug

293 We randomly assigned patients to receive negative pressure wound therapy or a standard wound clos

294 read use, it is unclear whether prophylactic negative pressure wound therapy reduces surgical-site in

295 The use of negative pressure wound therapy resulted in a significan

296 n undergoing cesarean delivery, prophylactic negative pressure wound therapy, compared with standard

297 ated lower limb fractures, use of incisional negative pressure wound therapy, compared with standard

298 In a real-world setup of negative pressure wound therapy, the optimized PVAc memb

299 omly assigned to either undergo prophylactic negative pressure wound therapy, with application of the

300 r objective was to determine if prophylactic negative-pressure wound therapy (pNPWT) allows preventin