ライフサイエンスコーパス: negative-strand

1 tions in IAV RNA segments (both positive and negative strands).

2 rmutation in the newly synthesized HIV-1 DNA negative strand.

3 an approximately equal ratio of positive and negative strands.

4 rand RNA synthesis but can no longer produce negative strands.

5 eotide during synthesis of both positive and negative strands.

6 s dsDNA, ssDNA, ssRNA positive strand, ssRNA negative strand and retroid and amino acid preference.

7 cap-snatching mechanisms of other segmented, negative-strand and ambisense RNA viruses.

8 rfering (DI) RNA in the positive but not the negative strand, and (iv) as a higher-order structure in

9 the positive strand and -3.0 kcal/mol in the negative strand, and has associated with it beginning at

10 s from an internal site in the DI RNA to the negative-strand antigenome of the helper virus.

11 switch takes place during the generation of negative-strand antileader-containing templates used sub

12 itro replication reactions, using poliovirus negative-strand cloverleaf RNA, led to a decrease in RNA

13 east in part by deaminating cytidines on the negative strand DNA intermediates.

14 into DNA, removes the tRNA used to initiate negative-strand DNA synthesis, and generates and removes

15 Notably, negative-strand DWV RNA, which could indicate viral repl

16 nally contain DWV replication complexes with negative-strand DWV RNA.

17 ses, members of the Bunyaviridae family, are negative-stranded emerging RNA viruses and category A pa

18 his study demonstrates that retroviruses use negative-strand-encoded proteins in the establishment of

19 erpart of the matrix proteins found in other negative strand enveloped RNA viruses.

20 lls by divergent and unrelated positive- and negative-strand-enveloped viruses from the Flaviviridae,

21 virus, family Bunyaviridae, has a tripartite negative-strand genome (S, M, and L segments) and is an

22 virus, family Bunyaviridae) has a tripartite negative-strand genome and causes a mosquito-borne disea

23 The negative-strand genome of tupaia rhabdovirus is composed

24 virus, family Bunyaviridae) has a tripartite negative-strand genome, causes a mosquito-borne disease

25 oup of orthobunyaviruses, has a trisegmented negative-stranded genome comprised of large (L), medium

26 hat positive-strand influenza virus mRNA and negative-strand genomic RNA (gRNA) accumulated to high l

27 replication was detected by the presence of negative strand HCV RNA.

28 consistent with the recent demonstration of negative-strand HCV RNA in brain, and suggest that IRES

29 The presence of negative-strand HCV RNA in PBMCs was evaluated by a stra

30 The presence of negative-strand HCV RNA in PBMCs was significantly posit

31 more likely to have detectable positive- and negative-strand HCV RNA in the PBMC compartment than wer

32 urine nucleotides (ATP and GTP), whereas the negative-strand HCV RNA replication is invariably initia

33 Negative-strand HCV RNA was detected in 78 (25%) of 315

34 n CD68-positive cells in eight patients, and negative-strand HCV RNA, which is a viral replicative fo

35 and tested for the presence of positive- and negative-strand HCV RNA.

36 RNase protection assays showed positive- and negative-strand HCV RNA.

37 found to detect up to 10 pg and 10(-5) pg of negative-strand HEV RNA in first- and second-round PCRs,

38 The ratio of positive to negative strand in macrophages was lower than in control

39 7 h postinfection, the ratio of positive to negative strands in individual cells varies from 5:1 to

40 replication-defective RNAs failed to produce negative strands in transfected cells.

41 plication involves the specific synthesis of negative-strand intermediates followed by an accumulatio

42 mmetric distribution of tags on positive and negative strands is considered.

43 oma and nonhepatoma cells that replicate the negative-strand lymphocytic choriomeningitis virus (LCMV

44 ied by RNA affinity column with biotinylated negative-strand MHV leader RNA and identified by mass sp

45 molecular cloning of the genome of a novel, negative-stranded neurotropic virus, Borna disease virus

46 Most nonsegmented negative strand (NNS) RNA virus genomes have complementa

47 n RNA polymerase L proteins of non-segmented negative strand (NNS) RNA viruses (e.g. rabies, measles,

48 ogenic Ebola virus (EBOV) has a nonsegmented negative-strand (NNS) RNA genome containing seven genes.

49 omain polymerase protein (L) of nonsegmented negative-strand (NNS) RNA viruses catalyzes transcriptio

50 tis virus (VSV), a prototype of nonsegmented negative-strand (NNS) RNA viruses including rabies, meas

51 Nonsegmented negative-strand (NNS) RNA viruses initiate infection by

52 Nonsegmented negative-strand (NNS) RNA viruses possess a ribonucleopr

53 tomatitis virus, a prototype of nonsegmented negative-strand (NNS) RNA viruses, forms a covalent comp

54 IMPORTANCE mRNAs of nonsegmented negative-strand (NNS) RNA viruses, such as VSV, possess

55 The nucleocapsid (N) protein of nonsegmented negative-strand (NNS) RNA viruses, when expressed in euk

56 Nonsegmented negative-stranded (NNS) RNA viruses, among them the viru

57 ins six domains that are conserved among all negative-stranded nonsegmented RNA viruses.

58 Negative-strand (NS) RNA viruses comprise many pathogens

59 e RNA-dependent RNA polymerase L proteins of negative-strand (NS) RNA viruses.

60 The negative strand of HIV-1 encodes a highly hydrophobic an

61 nding is specific for the 3' terminus of the negative strand of the viral genome with a dissociation

62 enomes is transcribed from both positive and negative strands of DNA and thus may generate overlappin

63 required for the elongation of positive- and negative-stranded picornavirus RNA.

64 revented the production of covalently closed negative-strand rcDNA, TOP2 inhibitors reduced the produ

65 (dsDNA, ssDNA, ssRNA positive strand, ssRNA negative strand, retroid) using amino acid distribution.

66 The negative-strand ribonucleic acid (RNA) genome of the vir

67 tion for over a decade, with high titers and negative strand RNA in the liver.

68 ody resulted in significant reduction of HCV-negative strand RNA synthesis.

69 by modeling crystal structures of homologous negative strand RNA virus Ns in NC.

70 MuV is a negative strand RNA virus, similar to rabies virus or Eb

71 In a negative strand RNA virus, the genomic RNA is sequestere

72 eplication and transcription of nonsegmented negative strand RNA viruses (or Mononegavirales) are bel

73 feron induction in cells infected with these negative strand RNA viruses.

74 loop of the PRNTase domain in non-segmented negative strand RNA viruses.

75 erging category A pathogens that carry three negative stranded RNA molecules as their genome.

76 Reverse genetic analyses of negative-strand RNA (NSR) viruses have provided enormous

77 be corresponding to the 5' end of poliovirus negative-strand RNA (the complement of the genomic 3' NC

78 ' gamma-phosphate is a common feature of the negative-strand RNA [(-)RNA] of the packaged dsRNA segme

79 om other viral families, including segmented negative-strand RNA and double-stranded RNA (dsRNA) viru

80 on similar to that of the L domains of other negative-strand RNA and retroviruses.

81 VPg-linked poly(U) products at the 5' end of negative-strand RNA during PV RNA replication.

82 e in the balanced synthesis of positive- and negative-strand RNA for robust viral replication.

83 The expression of the nonsegmented negative-strand RNA genome of respiratory syncytial viru

84 the Mononegavirales order, the nonsegmented negative-strand RNA genome of respiratory syncytial viru

85 ion cycle after primary transcription of the negative-strand RNA genome to mRNA.

86 V) is an enveloped virus with a nonsegmented negative-strand RNA genome whose organization is charact

87 V) is an enveloped virus with a nonsegmented negative-strand RNA genome whose organization is charact

88 ses are enveloped viruses with a bisegmented negative-strand RNA genome whose proteomic capability is

89 Arenaviruses have a bisegmented, negative-strand RNA genome.

90 Arenaviruses have a bisegmented negative-strand RNA genome.

91 riomeningitis virus (LCMV) has a bisegmented negative-strand RNA genome.

92 mily Paramyxoviridae, and has a nonsegmented negative-strand RNA genome.

93 Influenza viruses contain segmented, negative-strand RNA genomes.

94 We report here that the 5' end of poliovirus negative-strand RNA is capable of interacting with endog

95 VPgpUpU(OH) and nascent negative-strand RNA molecules were synthesized coinciden

96 g this difference, the ratio of positive- to negative-strand RNA of 26 was similar to that found with

97 nstrated that poliovirus positive-strand and negative-strand RNA present in cytoplasmic extracts prep

98 [(32)P]UMP incorporated into VPgpUpU(OH) and negative-strand RNA products indicated that 100 to 400 V

99 B3Delta3' lacked the coat protein gene and negative-strand RNA promoter.

100 regulation was correlated with positive- and negative-strand RNA quantitative detection and the relea

101 Vesicular stomatitis virus (VSV), a negative-strand RNA rhabdovirus, preferentially replicat

102 The influenza A virus genome possesses eight negative-strand RNA segments in the form of viral ribonu

103 The CCHFV genome consists of three negative-strand RNA segments, S, M, and L.

104 omes of influenza A viruses consist of eight negative-strand RNA segments.

105 to nsp10/11 functions as a single cistron in negative-strand RNA synthesis and analyze recent complem

106 tion in yeast is severely inhibited prior to negative-strand RNA synthesis by a single-amino-acid sub

107 Detection of increased negative-strand RNA synthesis by real time RT-PCR for th

108 A-cleaved TBSV RNAs served as a template for negative-strand RNA synthesis by the TBSV RNA-dependent

109 ontranslated region mutation which inhibited negative-strand RNA synthesis did not inhibit CRE-depend

110 While the tyrosine hydroxyl of VPg can prime negative-strand RNA synthesis in a CRE- and VPgpUpU(OH)-

111 eotides from the 5' terminus of SIN restored negative-strand RNA synthesis in DI genomes but not thei

112 sRNA replication by accelerating the rate of negative-strand RNA synthesis in vitro.

113 viral RNA showed that VPg uridylylation and negative-strand RNA synthesis occurred normally from mut

114 n, low concentrations of UTP did not support negative-strand RNA synthesis when CRE-disrupting mutati

115 VPg inhibited both VPgpUpU(OH) synthesis and negative-strand RNA synthesis, confirming the critical r

116 UpU(OH) synthesis was required for efficient negative-strand RNA synthesis, especially when UTP conce

117 These and other results show that prior to negative-strand RNA synthesis, multiple domains of mitoc

118 s-acting elements required for initiation of negative-strand RNA synthesis, we deleted the entire 3'

119 ce of G3BP and suggest a role of G3BP during negative-strand RNA synthesis.

120 VPgpUpU(OH) synthesis and the initiation of negative-strand RNA synthesis.

121 for CRE-dependent VPgpUpU(OH) synthesis and negative-strand RNA synthesis.

122 ependent VPg uridylylation before and during negative-strand RNA synthesis.

123 replication complexes (RCs) that function in negative-strand RNA synthesis.

124 effects on the magnitude of CRE-independent negative-strand RNA synthesis.

125 st a model for the initiation of coronavirus negative-strand RNA synthesis.

126 nome is the minimal signal for initiation of negative-strand RNA synthesis.

127 A interactions required for positive- versus negative-strand RNA synthesis.

128 g frame or the IRES were required in cis for negative-strand RNA synthesis.

129 sis of VPgpUpU(OH), however, did not inhibit negative-strand RNA synthesis.

130 t required in cis or in trans for poliovirus negative-strand RNA synthesis.

131 the viral replicase during an early step in negative-strand RNA synthesis.

132 ine hydroxyl of VPg in VPg uridylylation and negative-strand RNA synthesis.

133 f VPgpUpU was required for positive- but not negative-strand RNA synthesis.

134 uous elongation of nascent transcript during negative-strand RNA synthesis.

135 g-linked poly(U) sequences at the 5' ends of negative-strand RNA templates were transcribed into poly

136 in the conserved sequence at the 3' ends of negative-strand RNA templates.

137 een documented previously for a nonsegmented negative-strand RNA virus (mononegavirus).

138 irovirus (HAZV) is an enveloped trisegmented negative-strand RNA virus classified within the Nairovir

139 irus, which represent viruses from different negative-strand RNA virus families.

140 encoded by Rice grassy stunt virus (RGSV), a negative-strand RNA virus in the Bunyavirales, causes de

141 The mumps virus is a negative-strand RNA virus in the family Paramyxoviridae.

142 itive-strand RNA virus infections but not in negative-strand RNA virus infections.

143 The nucleocapsid of a negative-strand RNA virus is assembled with a single nuc

144 and reveal the structural organization of a negative-strand RNA virus L protein.

145 The negative-strand RNA virus measles virus (MeV) uses tissu

146 nfluenza virus type 3 (PIV3), a nonsegmented negative-strand RNA virus of the Paramyxoviridae family

147 that both the N- and C-terminal regions of a negative-strand RNA virus P are involved in binding the

148 h has served in the past as a model to study negative-strand RNA virus replication.

149 s instead, suggesting that current segmented negative-strand RNA virus taxonomy may need revision.

150 Vesicular stomatitis virus (VSV) is a negative-strand RNA virus with inherent specificity for

151 Newcastle disease virus (NDV) is a negative-strand RNA virus with oncolytic activity agains

152 is required for the entry of the prototypic negative-strand RNA virus, including influenza A virus a

153 rotein encoded by Rice stripe virus (RSV), a negative-strand RNA virus.

154 us, Indiana serotype (VSV(IND)), a prototype negative-strand RNA virus.

155 ratory syncytial virus (RSV), a nonsegmented negative-strand RNA virus.

156 ding is a departure from other nonsegmented, negative-strand RNA viruses (NNSVs) that have been studi

157 The negative-strand RNA viruses (NSRVs) are unique because t

158 What separates negative-strand RNA viruses (NSVs) from the rest of the

159 Negative-strand RNA viruses (NSVs) include some of the m

160 SV genome for viral RNA synthesis.IMPORTANCE Negative-strand RNA viruses (NSVs) include the most path

161 ties in the polymerase proteins of segmented negative-strand RNA viruses and for the search for antiv

162 e viral genome can form during infections of negative-strand RNA viruses and outgrow full-length vira

163 ation strategy should be applicable to other negative-strand RNA viruses and will promote studies int

164 y delineate the evolutionary relationship of negative-strand RNA viruses but also provide insights in

165 that La supports the growth of nonsegmented negative-strand RNA viruses by both IFN suppression and

166 function, against a number of positive- and negative-strand RNA viruses by enhancing type I IFN indu

167 Importance: The paramyxovirus family of negative-strand RNA viruses cause significant disease in

168 mal RNA synthesis machinery of non-segmented negative-strand RNA viruses comprises a genomic RNA enca

169 Negative-strand RNA viruses condense their genome into h

170 verse members of the Paramyxovirus family of negative-strand RNA viruses effectively suppress host in

171 Paramyxoviruses and other negative-strand RNA viruses encode matrix proteins that

172 Negative-strand RNA viruses encode their own polymerases

173 The nucleoprotein of negative-strand RNA viruses forms a major component of t

174 w that the mechanism of RNA encapsidation in negative-strand RNA viruses has many common features.

175 (VSV) is the prototype virus for 75 or more negative-strand RNA viruses in the rhabdovirus family.

176 Negative-strand RNA viruses include a diverse set of vir

177 rnalized viral ribonucleoproteins (vRNPs) of negative-strand RNA viruses induce an early IFN response

178 The genome of nonsegmented negative-strand RNA viruses is tightly embedded within a

179 This suggested that negative-strand RNA viruses produce little, if any, dsRN

180 polymerase complex.IMPORTANCE Replication of negative-strand RNA viruses relies on two components: a

181 However, the impact of TRIM56 on negative-strand RNA viruses remains unclear.

182 Negative-strand RNA viruses represent a significant clas

183 The nucleoprotein (NP) of segmented negative-strand RNA viruses such as Orthomyxo-, Arena-,

184 Paramyxoviruses are enveloped negative-strand RNA viruses that are significant human a

185 Paramyxoviruses are enveloped, nonsegmented, negative-strand RNA viruses that cause a wide spectrum o

186 Arenaviruses are negative-strand RNA viruses that cause human diseases su

187 Arenaviruses are enveloped negative-strand RNA viruses that cause significant human

188 The large (L) proteins of non-segmented, negative-strand RNA viruses, a group that includes Ebola

189 rate vaccine candidates against nonsegmented negative-strand RNA viruses, a large and expanding group

190 this control, HDV behaves similarly to other negative-strand RNA viruses, even though there is no gen

191 ttenuate VSV, and perhaps other nonsegmented negative-strand RNA viruses, for potential application a

192 Nonsegmented negative-strand RNA viruses, including measles virus (Me

193 Negative-strand RNA viruses, including paramyxoviruses,

194 Rabies viruses, negative-strand RNA viruses, infect neurons through axon

195 ing those by ssDNA viruses and positive- and negative-strand RNA viruses, produce dsRNAs detectable b

196 of the RNA than the NP protein of some other negative-strand RNA viruses, reflecting the degree of NP

197 bly diverse family of enveloped nonsegmented negative-strand RNA viruses, some of which are the most

198 Infection of human dendritic cells (DCs) by negative-strand RNA viruses, such as Newcastle disease v

199 The genomic RNA of negative-strand RNA viruses, such as vesicular stomatiti

200 For the nonsegmented negative-strand RNA viruses, the polymerase is comprised

201 virus (VSV), a prototype of the nonsegmented negative-strand RNA viruses, the two methylase activitie

202 In the replication cycle of nonsegmented negative-strand RNA viruses, the viral RNA-dependent RNA

203 aramyxoviruses, and by analogy for all other negative-strand RNA viruses, we show that directional se

204 es order comprises tick-borne, trisegmented, negative-strand RNA viruses, with several members being

205 rget for developing antivirals against other negative-strand RNA viruses.

206 , with implications for many other segmented negative-strand RNA viruses.

207 Arenaviruses are enveloped, negative-strand RNA viruses.

208 let-shaped rhabdovirus and a model system of negative-strand RNA viruses.

209 information is available about SIE of plant negative-strand RNA viruses.

210 antiviral therapeutics against nonsegmented negative-strand RNA viruses.

211 virus (VSV), a prototype of the nonsegmented negative-strand RNA viruses.

212 of the RNA polymerase (L) of non-segmented, negative-strand RNA viruses.

213 unique GDNQ motif normally characteristic of negative-strand RNA viruses.

214 ts in the design of new therapeutics against negative-strand RNA viruses.

215 ances led to a resurgence in DIP studies for negative-strand RNA viruses.

216 autophagy can play an antiviral role against negative-strand RNA viruses.

217 ire of targets for antiviral therapy against negative-strand RNA viruses.

218 and possibly other families of nonsegmented negative-strand RNA viruses.

219 which are a characteristic hallmark of many negative-strand RNA viruses.

220 HCV negative-strand RNA was not detected in PBMCs from any o

221 found in astrocytes from three patients, but negative-strand RNA was not detected in these cells.

222 titis E virus (HEV) produces an intermediate negative-strand RNA when it replicates.

223 They continuously produced negative-strand RNA, but its synthesis was blocked by th

224 mplexes formed with the 3' end of poliovirus negative-strand RNA, including one that contains a 36-kD

225 cules were made coincident in time with each negative-strand RNA.

226 patocytes expressed HCV core protein and HCV negative-strand RNA.

227 containing the 3' untranslated region of HCV negative-strand RNA.

228 ally interacts with the 3' end of poliovirus negative-strand RNA.

229 , genus Phlebovirus), which has a tripartite negative-stranded RNA genome (consisting of the S, M, an

230 viridae, genus Phlebovirus) has a tripartite negative-stranded RNA genome (L, M, and S segments).

231 They have a nonsegmented negative-stranded RNA genome and can cause a number of d

232 ses are enveloped viruses with a bisegmented negative-stranded RNA genome.

233 Negative-stranded RNA plant rhabdoviruses encode MPs wit

234 virus, representing viruses of the dsDNA and negative-stranded RNA viral groups, were used to infect

235 Vesicular stomatitis virus (VSV) is a negative-stranded RNA virus normally sensitive to the an

236 Influenza virus, a negative-stranded RNA virus that causes severe illness i

237 Vesicular stomatitis virus is a negative-stranded RNA virus.

238 ses are a large family of membrane-enveloped negative-stranded RNA viruses causing important diseases

239 Rhabdoviruses are negative-stranded RNA viruses of the order Mononegaviral

240 r Mononegavirales (comprised of nonsegmented negative-stranded RNA viruses or NNSVs) contains many im

241 Arenaviruses are enveloped, negative-stranded RNA viruses that belong to the family

242 he largest nucleoprotein of the nonsegmented negative-stranded RNA viruses, and like the NPs of other

243 In contrast to most negative-stranded RNA viruses, hantaviruses and other vi

244 contribute to pathogenicity in a variety of negative-stranded RNA viruses.

245 importance for efficient budding of several negative-stranded RNA viruses.

246 habdoviruses that may be applicable to other negative-stranded RNA viruses.

247 hanism of replication of influenza and other negative-stranded RNA viruses.

248 the closest relatives of NYNV and MIDWV are negative-stranded-RNA viruses in the order Mononegaviral

249 uitment to these mitochondrial membranes for negative-strand RNA1 synthesis.

250 e BMV replication factors 1a and 2a, and use negative-strand RNA3 as a template for genomic RNA3 and

251 ed RNA3 replication at a step or steps after negative-strand RNA3 synthesis, implying competition wit

252 tion with positive-strand RNA3 synthesis for negative-strand RNA3 templates, viral replication factor

253 and the higher levels of both positive- and negative-strand RNAs for the chimeras than for the H77 p

254 f HCV proteins as well as both positive- and negative-strand RNAs in the stable Huh7 cell lines.

255 te switch during the synthesis of subgenomic negative-strand RNAs to add a copy of the leader sequenc

256 initiation nucleotides of both positive- and negative-strand RNAs were found to be either an adenylat

257 U) portions of poliovirus (PV) positive- and negative-strand RNAs were used as reciprocal templates d

258 3'-dCTP inhibited the elongation of nascent negative-strand RNAs without affecting CRE-dependent VPg

259 ction of HCV proteins and both positive- and negative-strand RNAs.

260 Influenza A virus is a negative-strand segmented RNA virus in which antigenical

261 Hantaviruses, similarly to other negative-strand segmented RNA viruses, initiate the synt

262 The objective of this study was to develop a negative-strand-specific reverse transcription-PCR (RT-P

263 In addition to the liver, the negative-strand-specific RT-PCR assay identified replica

264 The standardized negative-strand-specific RT-PCR assay was subsequently u

265 sponse to respiratory syncytial virus (RSV), negative strand ssRNA virus, depends upon the ability to

266 ] synthesis on an ectopically expressed RNA3 negative strand [(-) strand] and faithfully complete the

267 nal sequences that were highly efficient for negative-strand synthesis and replication.

268 cre(2C) hairpin had no significant effect on negative-strand synthesis but completely inhibited posit

269 nal end of stem a had little or no effect on negative-strand synthesis but dramatically reduced posit

270 ting from apparent template switching during negative-strand synthesis of subgenomic RNA 7.

271 egion of the gRNA, contains the promoter for negative-strand synthesis, and influences several infect

272 equences were required for RNA1 recruitment, negative-strand synthesis, and subsequent positive-stran

273 Thus, after negative-strand synthesis, the ns proteins appeared to i

274 0/11 ( approximately 150 kDa) is involved in negative-strand synthesis.

275 cteriophage replication, likely by targeting negative-strand synthesis.

276 ng nsp10 as being a cofactor in positive- or negative-strand synthesis.

277 stabilized viral RNA and inhibited efficient negative-strand synthesis.

278 is thought to occur during the synthesis of negative-strand templates for sgmRNA production and to b

279 the 3'AAUUUUGUC5' sequence at the 3' ends of negative-strand templates.

280 t the association of hnRNP C with poliovirus negative-strand termini acts to stabilize or otherwise p

281 p70 was cross-linked to the MHV positive- or negative-strand UTR in vitro and in vivo.

282 cross-linking to bind both the positive- and negative-strand UTRs of MHV RNA specifically.

283 d an occult infection, with the detection of negative strand viral genome, indicating viral replicati

284 anning 4,254 km of the coterminous USA, with negative strand viral RNA demonstrating active replicati

285 ide new evidence that the 3' terminus of the negative-strand viral genome in the double-stranded RNA

286 Thus, the detection of negative-strand viral RNA is indicative of HEV replicati

287 lling template selection for translation and negative-strand viral RNA synthesis, two processes that

288 Encapsidated negative-strand viral RNA was detected using CsCl-purifi

289 A synthetic negative-strand viral RNA was generated from the plasmid

290 Both positive- and negative-strand viral RNA were detected by real-time qua

291 designed to amplify a 232-bp fragment of the negative-strand viral RNA.

292 luorescence, and PCR for positive-strand and negative-strand viral RNA.

293 efficient replication of both positive- and negative-strand viral RNAs as well as enzymes capable of

294 g CsCl-purified CVB3/TD virions, although no negative-strand virion RNA was detected in similarly tre

295 bryonic fibroblasts extremely susceptible to negative-stranded virus infection, including vesicular s

296 r vesicular stomatitis virus (VSV) and other negative-strand viruses is the RNA genome in association

297 Nucleocapsids of nonsegmented negative-strand viruses like VSV are assembled in the cy

298 nfection with HCV; in particular, an HCV RNA-negative strand was detectable almost exclusively in the

299 reaction, and concentration of positive and negative strands was determined by a novel quantitative

300 ss the viral genome on both the positive and negative strands, with clusters of miRNAs at a number of