1 hannel subunit KCNQ2 lead to benign familial neonatal convulsions, a dominantly inherited form of gen

2  (adjusted HR, 23.6; 95% CI, 20.6-27.1), and neonatal convulsions (adjusted HR, 33.5; 95% CI, 30.1-37

3            Analogies between benign familial neonatal convulsions and other channelopathies of skelet

4 and composite severe neonatal morbidity (eg, neonatal convulsions) and perinatal mortality (ie, still

5 ildhood epilepsy syndrome of benign familial neonatal convulsions (BFNC) exhibits the remarkable feat

6                              Benign familial neonatal convulsions (BFNC) is a rare autosomal dominant

7                              Benign familial neonatal convulsions (BFNC), a class of idiopathic gener

8                  One type is benign familial neonatal convulsions (BFNC), a dominantly inherited diso

9  form of inherited epilepsy, benign familial neonatal convulsions (BFNC), has also been localized to

10 e the human seizure disorder benign familial neonatal convulsions (BFNC), presumably by reducing IK(M

11 ant mode of inheritance: the benign familial neonatal convulsions (BFNC; refs 2,3).

12                              Benign familial neonatal convulsion (BNFC) is a neurological disorder ca

13 ed form of epilepsy known as benign familial neonatal convulsions, for the first time enabled insight

14         There were 12 perinatal deaths and 5 neonatal convulsions in the control group compared with

15 roup compared with 3 perinatal deaths and no neonatal convulsions in the DHA group (P = 0.03 in both

16 ominantly inherited, such as benign familial neonatal convulsions, juvenile myoclonic epilepsy, as we

17 birth asphyxia (n=3), septicaemia (n=1), and neonatal convulsion (n=1).

18 channels that are mutated in benign familial neonatal convulsions represent an important new target f

19 tion reduces the risk of perinatal death and neonatal convulsions requires further investigation.