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1 NG2 cells in a mouse model of DWMI (chronic neonatal hypoxia).
2 hreshold, thus mimicking clinical aspects of neonatal hypoxia.
3 studies demonstrate for the first time that neonatal hypoxia affects the Foxo1/p27(Kip1) pathway dur
7 ther evaluated as a treatment strategy after neonatal hypoxia and leaders in the field of neonatology
11 rocyte (OL) regeneration in a mouse model of neonatal hypoxia (HX) that reproduces diffuse white matt
15 n response to 12% O(2), male rats exposed to neonatal hypoxia increased ventilation less than untreat
16 the SDF-1/CXCR4 axis in the pathogenesis of neonatal hypoxia-induced cardiopulmonary remodeling and
17 ts of either sex (P > 0.05), suggesting that neonatal hypoxia-induced plasticity occurs between the p
18 on, exosomes derived from neonatal normoxia, neonatal hypoxia, infant hypoxia, and child hypoxia sign
19 It was shown that the combination of MIA and neonatal hypoxia ischemia produces autistic-like behavio
21 ion and NMDAR tyrosine phosphorylation after neonatal hypoxia-ischemia (HI) and investigated the neur
22 lamide in both severe and moderate models of neonatal hypoxia-ischemia (HI) in postnatal day 10 Sprag
25 delayed and ongoing neurodegeneration after neonatal hypoxia-ischemia (HI), but the mechanisms and t
29 ental adult rat focal ischemia and in a mild neonatal hypoxia-ischemia (HI, 90 min hypoxia) rat model
31 S) shRNA into the hippocampus of rats before neonatal hypoxia-ischemia decreased vulnerability to inj
37 oubles surviving tissue in a severe model of neonatal hypoxia-ischemia, a major cause of neonatal dea
38 r protein expression increased rapidly after neonatal hypoxia-ischemia, in concert with cleavage of p
43 rotection in instances of cardiac arrest and neonatal hypoxia/ischemia but faces different challenges
49 developmental and behavioral recovery after neonatal hypoxia, while promoting myelin repair followin