ライフサイエンスコーパス: neonatal mouse

1 r maintaining cholesterol homeostasis in the neonatal mouse.

2 rsting in the lumbosacral spinal cord of the neonatal mouse.

3 urally occurring retinal angiogenesis in the neonatal mouse.

4 N region addition, are not available to the neonatal mouse.

5 ired to establish pulmonary infection in the neonatal mouse.

6 s potential peripheral targets of AVP in the neonatal mouse.

7 tor circuitry, are well-characterized in the neonatal mouse.

8 hy of spinal motor neurons in the developing neonatal mouse after axotomy.

9 These studies demonstrate that: 1) normal neonatal mouse airway development entails an IL-4Ralpha-

10 Neonatal mouse AMs retained high expression of some proi

11 tiated keratinocytes in the epidermis of the neonatal mouse and in the bulge area of the adult mouse

12 c neurotransmission was blocked in slices of neonatal mouse and rat hippocampus and neocortex, sPFPs

13 e regulation is recapitulated in vitro using neonatal mouse atrial and ventricular myocytes overexpre

14 9 Wnt genes and Wnt target gene Axin2 in the neonatal mouse bone by in situ hybridization, and demons

15 KCC2 is partially phosphorylated in neonatal mouse brain and dephosphorylated in parallel wi

16 nfect neurons within specific regions of the neonatal mouse brain and produce a lethal meningoencepha

17 that Ets-1 binds to the DOR promoter in the neonatal mouse brain and that overexpressed Ets-1 can si

18 a trans-activator of the DOR promoter in the neonatal mouse brain and thus may contribute to the deve

19 PSCs-derived microglia transplanted into the neonatal mouse brain assume a phenotype and gene express

20 novel in vivo targets of Notch signaling in neonatal mouse brain endothelium, including UNC5B, a mem

21 s into specific regions of the embryonic and neonatal mouse brain in vivo.

22 as a 3.9- and 4.4-kb transcript in adult and neonatal mouse brain total RNA, and in situ hybridizatio

23 t although intraventricular injection of the neonatal mouse brain with adeno-associated virus serotyp

24 ial injection into lateral ventricles of the neonatal mouse brain, a low-affinity AAV4 mutant (AAV4.1

25 After transplantation into the neonatal mouse brain, human ES cell-derived neural precu

26 ociated hMGE cells are transplanted into the neonatal mouse brain, they reform into nests containing

27 be difficult to detect in the embryonic and neonatal mouse brain, we used a new transgenic mouse wit

28 cultured primary human endothelial cells and neonatal mouse brain.

29 functional synapses when transplanted into a neonatal mouse brain.

30 precursors derived in vitro from hiPSCs into neonatal mouse brains and found that the cells acquired

31 1 (NuMA1) is downregulated in AIS-deficient neonatal mouse brains and neurons.

32 ferent dermal origin after implantation into neonatal mouse brains.

33 erived primitive macrophage progenitors into neonatal mouse brains.

34 aptic depression using a rhythmically active neonatal mouse brainstem slice preparation.

35 calpain-mediated mechanisms of cell death of neonatal mouse C17.2 progenitor cells, transplanted at 2

36 leukin-6 by osteoblasts in organ cultures of neonatal mouse calvaria, and in vivo using a mouse model

37 Neonatal mouse calvariae were cultured in acid (Acid; pH

38 oblastic bone formation in organ cultures of neonatal mouse calvariae, and a neutralizing antibody to

39 lls were obtained by sequential digestion of neonatal mouse calvariae, and cultured with fetal calf s

40 of retinoids on bone resorption in cultured neonatal mouse calvarial bones and their interaction wit

41 It is not clear whether B cells in the neonatal mouse can activate the somatic mutation machine

42 to starvation and dehydration well before a neonatal mouse can seek food and water separately.

43 d mechanically integrated ECT using isolated neonatal mouse cardiac cells derived from both wild-type

44 onses in low-density, serum-free cultures of neonatal mouse cardiac myocytes and compared them with r

45 y +8 mV, producing a maximal +34-mV shift in neonatal mouse cardiac myocytes or Chinese hamster ovary

46 ) regulate the intrinsic contraction rate in neonatal mouse cardiac myocytes through distinct signali

47 rising potassium current (I(Kr)) channels in neonatal mouse cardiac myocytes.

48 on were further tested in cultured adult and neonatal mouse cardiac myocytes.

49 Molecular and functional maturation of neonatal mouse cardiomyocytes and human embryonic stem c

50 R-199a-3p and hsa-miR-590-3p both in primary neonatal mouse cardiomyocytes and in vivo.

51 gested that fusion between WB F344 cells and neonatal mouse cardiomyocytes did not take place.

52 00000117266 led to a significant increase of neonatal mouse cardiomyocytes in G0/G1 phase and reducti

53 Neonatal mouse cardiomyocytes undergo a metabolic switch

54 BMP signaling also rescues neonatal mouse cardiomyocytes, human fibroblasts and hum

55 -induced cardiac cell death were examined in neonatal mouse cardiomyocytes.

56 nslational stability of Na/Ca exchanger 1 in neonatal mouse cardiomyocytes.

57 was further revealed in primary cultures of neonatal mouse cardiomyocytes.

58 and augments oxidative capacity in cultured neonatal mouse cardiomyocytes.

59 he proximal ANF promoter by ChIP assay using neonatal mouse cardiomyocytes.

60 Here, we show that neonatal mouse CD43(-) non-B-1 cells also produce substa

61 Finally, IL-10 secreted from neonatal mouse CD43(-) non-B-1 cells was sufficient to i

62 In neonatal mouse CD43(-) non-B-1 cells, BCR engagement act

63 We have utilized primary cultures from neonatal mouse cerebella in order to determine (i) wheth

64 GRIP infection of neonatal mouse cerebellum in vivo enhances granule cell

65 Sumoylation of FOXP2 in neonatal mouse cerebellum regulates Purkinje cell develo

66 The neonatal mouse cerebellum shows remarkable regenerative

67 ce tags in heterogeneous primary cultures of neonatal mouse cerebellum that respond to the mitogen So

68 nestin-expressing progenitors (NEPs) in the neonatal mouse cerebellum.

69 neous waves of activity propagate across the neonatal mouse cerebral cortex and that these waves are

70 (CreERT2) mice to delete Gata3 in SCs of the neonatal mouse cochlea and showed that loss of Gata3 res

71 Our findings demonstrate that the neonatal mouse cochlea is capable of spontaneous hair ce

72 taneously regenerate hair cells (HCs) in the neonatal mouse cochlea, yet little is known about the re

73 urified Atoh1-expressing hair cells from the neonatal mouse cochlea.

74 e developed two genetic strategies to ablate neonatal mouse cochlear hair cells in vivo.

75 ly unknown connections of motoneurons in the neonatal mouse cord that are likely to play important ro

76 C and SerpinE1 prior to transplantation into neonatal mouse cortex enhanced their migration and morph

77 Using mixed neuronal and glial cultures from neonatal mouse cortex of both sexes, we show that SPARCL

78 Here, we describe a transient window in neonatal mouse development during which the spatial prop

79 Here, we use neonatal mouse endometrial organoids and assembloid cocu

80 ther epithelial proteins was detected in the neonatal mouse epidermis lacking periplakin.

81 c cell lines XS52-4D and XS106 (derived from neonatal mouse epidermis), bone marrow-derived dendritic

82 luripotent neural crest-like stem cells from neonatal mouse epidermis, with different potencies, isol

83 as naturally occurring hyperproliferation in neonatal mouse epidermis.

84 sin, is a protective antigen, using a lethal neonatal mouse ETEC challenge model and passive dam vacc

85 MNs) in the thoracolumbar spinal cord of the neonatal mouse exclusively via axons descending ipsilate

86 When Lin(-) BM cells were injected into neonatal mouse eyes, they extensively and stably incorpo

87 es increased cellular uptake of BCAAs in the neonatal mouse forebrain, and membrane mediated transpor

88 Normal neonatal mouse forepaws were imaged by micro-computed to

89 , to mice markedly prolonged the survival of neonatal mouse heart allografts.

90 diac endothelial cells (CECs) and CMs in the neonatal mouse heart and find that they are spatiotempor

91 Regenerative capacity is robust in the neonatal mouse heart but is lost during postnatal develo

92 one major unresolved question is whether the neonatal mouse heart can also regenerate in response to

93 In contrast, the neonatal mouse heart can efficiently regenerate during t

94 The neonatal mouse heart can regenerate, but only during the

95 Using the neonatal mouse heart cryoinjury and apical resection mod

96 sive analysis of transcriptomes derived from neonatal mouse heart left and right ventricles, a total

97 mulation of Agrin, an essential regulator of neonatal mouse heart regeneration.

98 c transcription factor GATA4 is required for neonatal mouse heart regeneration.

99 Here, we find that apical neonatal mouse heart resection surgery led to rapid and

100 tnatal coronary vessels arise de novo in the neonatal mouse heart, rather than expanding from preexis

101 schemic reperfusion in the isolated perfused neonatal mouse heart.

102 metabolism promotes cardiac regeneration in neonatal mouse heart.

103 o drive angiogenesis and regeneration of the neonatal mouse heart.

104 increase in miR-378 expression in 1-week-old neonatal mouse hearts compared with 16-day-old fetal hea

105 Fetal or neonatal mouse hearts containing proliferating cardiac m

106 Pitx2-deficient neonatal mouse hearts failed to repair after apex resect

107 The introduction of injury models for neonatal mouse hearts has accelerated research on the me

108 We demonstrate that neonatal mouse hearts use a novel mechanism to build col

109 solve the entire architecture of large-scale neonatal mouse hearts, revealing the helical orientation

110 cardial angiogenesis in vitro and in injured neonatal mouse hearts.

111 quired for the full regenerative capacity of neonatal mouse hearts.

112 CN) channel subunits in pyramidal neurons of neonatal mouse hippocampus using electrophysiological an

113 Herein, a neonatal mouse hyperoxic model of coincident BPD and ret

114 xpressing dorsal-medial (mpd) neurons of the neonatal mouse hypothalamus.

115 ype I IFN elicits a marginal ISG response in neonatal mouse IECs and does not inhibit rotavirus repli

116 elicits robust and uniform ISG expression in neonatal mouse IECs and inhibits the replication of IEC-

117 Using a medullary slice preparation from a neonatal mouse, including the site of the neural network

118 Line scan images (2 ms repetition rate) of neonatal mouse inner hair cells filled with the fluoresc

119 t Cd11b(+)Ly6c(-) macrophages in the dam-fed neonatal mouse intestine.

120 We also demonstrate that although the neonatal mouse is susceptible to thrombocytopenia-induce

121 Extracts from neonatal mouse lenses contained strong VEID-7-amino-4-tr

122 duction of C/EBPbeta protein isoforms in the neonatal mouse liver is regulated by C/EBPalpha.

123 nd poor lipid nanoparticle (LNP) uptake into neonatal mouse liver.

124 eroxia-induced biomolecular modifications in neonatal mouse lung fibroblasts (nMLFs).

125 thelial cell ingestion of P. aeruginosa in a neonatal mouse lung infection model led to increased lev

126 reference genes in studies investigating the neonatal mouse lung.

127 e transfer of maternal IgG into the prenatal/neonatal mouse made possible by the beta 2m-dependent Fc

128 ry drive potentials in preBotC neurons using neonatal mouse medullary slice preparations that generat

129 o were expressed by both primary cultures of neonatal mouse microglia and astrocytes exposed to heat-

130 nal injury in the oxygen-induced retinopathy neonatal mouse model (see the related article beginning

131 This report introduces a neonatal mouse model for active protection studies with

132 First, we used a neonatal mouse model of asthma to identify age-related m

133 Using a neonatal mouse model of BPD, we show that hyperoxia incr

134 Using a neonatal mouse model of CCM disease, we show that expres

135 Conscious bioimaging was applied to a neonatal mouse model of cerebral palsy (Hypoxic-Ischaemi

136 We previously described a neonatal mouse model of coxsackievirus B3 (CVB3) infecti

137 In this study, a neonatal mouse model of critical pertussis is characteri

138 well as reduced morbidity and mortality in a neonatal mouse model of disease.

139 Here, we establish a neonatal mouse model of echovirus-induced aseptic mening

140 In a neonatal mouse model of EV-D68 infection, Jun6504 signif

141 Using a neonatal mouse model of functional tendon healing compar

142 otent and substantially more protective in a neonatal mouse model of group B Streptococcus infection

143 In the BMP9/10-immunoblocked (BMP9/10ib) neonatal mouse model of HHT, we report here that the mTO

144 Using a neonatal mouse model of HI, mRNA, and protein expression

145 ee of brain damage sustained by animals in a neonatal mouse model of hypoxia-ischemia depends on the

146 antly less virulent than PAO1 in a BALBc/ByJ neonatal mouse model of infection as measured by their a

147 To address this question, we established a neonatal mouse model of influenza infection to test the

148 SVE was replicated in a neonatal mouse model of intestinal perforation.

149 ves: To investigate whether a multifactorial neonatal mouse model of lung injury perturbs neural prog

150 In this study, we used a neonatal mouse model of ovalbumin (OVA)-induced allergic

151 l neovascularization were determined using a neonatal mouse model of oxygen-induced retinopathy (OIR)

152 munoglobulin G to induce acantholysis in the neonatal mouse model of pemphigus.

153 A neonatal mouse model of pneumonia was used to determine

154 comparing the virulence of fliC mutants in a neonatal mouse model of pneumonia.

155 The neonatal mouse model of retinopathy of prematurity (ROP)

156 We used the neonatal mouse model of rotavirus infection and virus st

157 We used the neonatal mouse model of rotavirus infection to study ext

158 Lastly, apoptotic inhibitor treatment of a neonatal mouse model of RSV infection mitigated infectio

159 Here we review the use of a neonatal mouse model of RSV infection to mimic severe in

160 In summary, our neonatal mouse model of ZIKV infection suggests that ZIK

161 BO Journal, Chandler et al, demonstrate in a neonatal mouse model that maternal antibodies interfere

162 Here, we developed an in vivo neonatal mouse model that recapitulates key aspects of e

163 Our results demonstrate the potential of our neonatal mouse model to characterize viral and host dete

164 accharide inhibited bacterial ingestion in a neonatal mouse model, resulting in increased amounts of

165 In the oxygen-induced ROP neonatal mouse model, retinal neovascularization was dec

166 Using a neonatal mouse model, we previously determined that coxs

167 Using a neonatal mouse model, we previously revealed that mice i

168 Using a neonatal mouse model, we tested the hypothesis that pass

169 dy described in the present paper, we used a neonatal mouse model, which more closely mimics human in

170 nd is required for mucosal colonization in a neonatal mouse model.

171 ant exfoliative toxin A (rETA) was used in a neonatal mouse model.

172 to elicit protection from GBS infection in a neonatal mouse model.

173 ting EV-D68 infection-induced paralysis in a neonatal mouse model.

174 otection against lethal CVA16 infection in a neonatal mouse model.

175 efficacy against C. parvum infection in the neonatal mouse model.

176 dust mite and A alternata were compared in a neonatal mouse model.

177 an inactivated influenza virus vaccine in a neonatal mouse model.

178 lanocyte proliferation and migration using a neonatal mouse model.

179 Here, using adult and neonatal mouse models of DM1, we show that intramuscular

180 Neonatal mouse models of RSV were employed, wherein mice

181 owed by mechanistic studies in humans and in neonatal mouse models provided evidence that environment

182 lted in a more severe microcephalic brain in neonatal mouse models.

183 erize FNPB progenitor cell-derived colonies, neonatal mouse mononuclear cells were cultured directly

184 ne transfer of the E83K-GPD1-L mutation into neonatal mouse myocytes markedly attenuated the sodium c

185 ) regulate the intrinsic contraction rate in neonatal mouse myocytes through distinct signaling pathw

186 In > 60 % of neonatal mouse myocytes, a sizable IKs could be measured

187 Using a neonatal mouse NEC model, we examined the changes in int

188 also reveals inhibitory GABA actions in the neonatal mouse neocortex and hippocampus in vivo.

189 rivative D156844 increases SMN expression in neonatal mouse neural tissues, delays motor neuron loss

190 nhance the expression of DOR mRNA in primary neonatal mouse neuronal cells.

191 e that the E glycan loop deletions represent neonatal mouse neurovirulence markers of ZIKV.

192 r, resulting in a significant enhancement of neonatal mouse neurovirulence.

193 ished for the first time primary cultures of neonatal mouse olfactory bulb expressing TH and tested w

194 In a neonatal mouse, only strains with intact agr and sarA lo

195 ct of drug-induced fictive locomotion in the neonatal mouse or change gait, motor coordination, or gr

196 ptide, we found that short-term treatment of neonatal mouse ovaries increased nuclear exclusion of Fo

197 Neonatal mouse pathogenesis of virus (TR339) generated f

198 ic AVPR1A ligand binding was observed in the neonatal mouse periphery in sensory tissues of the head

199 Here, using neonatal mouse primary sympathetic neurons, we investiga

200 We showed that neonatal mouse pup plasma contains A1AT fragments that i

201 roduced into the medial prefrontal cortex of neonatal mouse pups by electroporation, and the regulati

202 s in neocortical and hippocampal slices from neonatal mouse pups in vitro, but also reveals inhibitor

203 Neonatal mouse pups were exposed to >90% hyperoxia or ro

204 g promotes developmental angiogenesis in the neonatal mouse retina assessed at postnatal day 6.

205 A neonatal mouse retina developmental model was used to st

206 eceptors, we examined the development of the neonatal mouse retina in an organotypic culture system.

207 d terminal processes of cholinergic cells in neonatal mouse retina.

208 ed to test whether such angioblasts exist in neonatal mouse retina.

209 development in dissociated-cell cultures of neonatal mouse retina.

210 Ankyrin-G depletion in neonatal mouse retinas markedly reduced CNG channel expr

211 acetylases (HDACs) in rod differentiation in neonatal mouse retinas, we used a pharmacological approa

212 We used ATAC-seq on sorted neonatal mouse SAN to compare regions of accessible chro

213 en together, these results indicate that the neonatal mouse SCN has its full complement of cells, som

214 ly decreased the extent of acantholysis in a neonatal mouse skin explant model.

215 electron tomography of plastic sections from neonatal mouse skin to visualize the organization of des

216 In neonatal mouse skin, two types of dermal papilla (DP) ar

217 elayed transplantation of SCs generated from neonatal mouse skin-derived precursors (SKP-SCs) promote

218 In neonatal mouse slice preparations that retain the preBot

219 tonic destruction of Dbx1 preBotC neurons in neonatal mouse slices impairs respiratory rhythm but sur

220 ecordings of olfactory receptor neurons from neonatal mouse slices revealed that ATP reduced cyclic n

221 recordings were made from dually innervated neonatal mouse soleus muscle fibers, and quantal content

222 nclude that lumbar locomotor networks in the neonatal mouse spinal cord are targets for modulation by

223 lcium imaging in the in vitro isolated whole neonatal mouse spinal cord preparation to record the act

224 between motoneurons in the L6 segment of the neonatal mouse spinal cord that contains limb-innervatin

225 ng during fictive locomotion in the isolated neonatal mouse spinal cord, following earlier work on lo

226 In the neonatal mouse spinal cord, we studied the firing proper

227 ize left-right locomotor coordination in the neonatal mouse spinal cord.

228 l evidence that Mef2c inhibition by Foxp2 in neonatal mouse striatum controls synaptogenesis of corti

229 S127A variant caused robust proliferation of neonatal mouse supporting cells, which produced progeny

230 Using a neonatal mouse system as an appropriate model for human

231 ntine reticulospinal (pRS) projection in the neonatal mouse that mediates synaptic effects on spinal

232 ese data suggest that in the orally infected neonatal mouse, the extraintestinal spread of rotavirus

233 element could activate expression in injured neonatal mouse tissues and was divisible into tissue-spe

234 identification of regenerative potential in neonatal mouse tissues that normally heal poorly in adul

235 totransferrin Cre (Ltf-Cre) model and in the neonatal mouse uterus using the progesterone receptor Cr

236 Here, the epithelium of the neonatal mouse uterus was isolated and subjected to sing

237 Here we show that HC damage in neonatal mouse utricle activates the Wnt target gene Lgr

238 g reperfusion by acutely knocking out MCU in neonatal mouse ventricular myocyte (NMVM) monolayers sub

239 Neonatal mouse ventricular myocytes were treated with a

240 In neonatal mouse ventricular myocytes, overexpression of C

241 In HeLa cells and neonatal mouse ventricular myocytes, peroxide exposure d

242 a,L) was examined in pertussis toxin-treated neonatal mouse ventricular myocytes.

243 Here, we describe rhythmically timed neonatal mouse vocalizations that occur within single br

244 strogen-regulated genes in the uterus of the neonatal mouse, we have isolated a murine homologue of t