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1 ng organotypic hippocampal slice cultures of neonatal rat.
2 Escherichia coli K1 can be replicated in the neonatal rat.
3 nsights into the E. coli K1 infection of the neonatal rat.
4 role of cytokine regulation following TBI in neonatal rats.
5 udens protein 1 was also better preserved in neonatal rats.
6 was reported to disrupt myelin formation in neonatal rats.
7 I(Kr) in ventricular myocytes isolated from neonatal rats.
8 pulation in the dorsal root ganglia (DRG) of neonatal rats.
9 fused than in glucose-perfused intestines of neonatal rats.
10 in ventricular cardiomyocytes isolated from neonatal rats.
11 ortex and ventral midline thalamic nuclei of neonatal rats.
12 rotavirus strain, CDC-9, in Caco-2 cells and neonatal rats.
13 spontaneous activity in the visual system of neonatal rats.
14 the ventilatory response to hyperthermia in neonatal rats.
16 cholamine secretion and [Ca2+]i responses in neonatal rat adrenal chromaffin cells and involves react
17 ective cyclooxygenase-2 inhibition protected neonatal rats against death, progression of brain injury
20 passage cultures of primary astrocytes from neonatal rats and cultures of the C6 rat glioma cells we
21 l types in brain slices from male and female neonatal rats and mice, including raphe neurons, cortica
23 ort that, in intact hippocampi prepared from neonatal rats and transgenic mice expressing Clomeleon,
24 r was injected subretinally into the eyes of neonatal rats, and this was followed by electroporation.
26 and apoptosis were assessed in (i) cultured neonatal rat astrocytes treated with astrogliosis-induci
31 looked for HI neuroprotection from FBP in a neonatal rat brain slice model, using 14.1 T (1)H/(31)P/
39 Inhibition of endogenous ROCK activity in neonatal rat brainstem slices failed to modulate intrins
42 tion increased promoter activity in cultured neonatal rat cardiac myocytes and human HEK fibroblasts
43 rized mitochondria and rescues cell death in neonatal rat cardiac myocytes subjected to hypoxia/reoxy
44 into SW13 and human smooth muscle cells and neonatal rat cardiac myocytes, and the effects on cytosk
46 ation of depolarized mitochondria in resting neonatal rat cardiac myocytes, as well as in those treat
47 ast and independent nuclear Ca(2+) signal in neonatal rat cardiac myocytes, human embryonic cardiac m
56 cathepsin G (Cat.G) has been shown to induce neonatal rat cardiomyocyte detachment and apoptosis by a
57 cocultures, hMSCs dose-dependently decreased neonatal rat cardiomyocyte excitability, slowed conducti
58 ew preribosomal RNA synthesis occurs in both neonatal rat cardiomyocytes (NRCM) and cardiac progenito
59 ith these findings, adenoviral-induced Pim-1 neonatal rat cardiomyocytes (NRCMs) retain a reticular m
63 DNA breaks associated with DOXO exposure in neonatal rat cardiomyocytes and human cardiomyocytes der
64 nuated cardiomyocyte hypertrophy in cultured neonatal rat cardiomyocytes and in the intact adult hear
66 TA4 in both co-transfected HEK293T cells and neonatal rat cardiomyocytes by co-immunoprecipitation.
73 scent probe Vybrant DiO were cocultured with neonatal rat cardiomyocytes labeled with the red fluores
76 novirus-mediated overexpression of Tsg101 in neonatal rat cardiomyocytes resulted in cell hypertrophy
77 g and overexpression experiments in isolated neonatal rat cardiomyocytes showed that Hsp22 activates
78 Inhibition of OGT or OGA activity within neonatal rat cardiomyocytes significantly affects energy
81 AB(R120G) protein misfolding and aggregates, neonatal rat cardiomyocytes were infected with adenoviru
88 evels were detected in Rnd3-null embryos, in neonatal rat cardiomyocytes, and noncardiac cell lines w
105 Here, we conducted two experiments using neonatal rat cells and an incomplete cervical injury mod
106 which contribute to Bax-related apoptosis in neonatal rat cerebellum at ages of peak ethanol sensitiv
107 in cocultures of A. culbertsoni and primary neonatal rat cerebral cortex microglia, induced apoptosi
108 s inhibitory effect in the nasal passages of neonatal rats, colonization experiments were performed w
109 e used primary OPCs in culture isolated from neonatal rat cortices of both sexes and young male and f
115 lung ECFCs exposed to hyperoxia in vitro and neonatal rat ECFCs isolated from hyperoxic alveolar grow
116 ration in striatal medium spiny neurons from neonatal rats exposed to antiepileptic drugs with proapo
117 he contribution of Tff2 to protection of the neonatal rat from E. coli K1 bacteremia and tissue invas
119 DSLNnT and DS'LNT were shown to protect neonatal rats from necrotizing enterocolitis (NEC) and a
121 has previously been studied by transfecting neonatal rat hair cells in culture with a beta-actin-GFP
124 rdion-like honeycomb scaffolds with cultured neonatal rat heart cells demonstrated utility through: (
125 Here, we investigated the interaction of neonatal rat heart cells with engineered spider silk pro
127 ardial delivery of the progenitor cells into neonatal rat hearts, in vivo incubation and analysis.
130 orted that neurotrophins induced survival of neonatal rat hippocampal neurons by promoting neural act
131 piratory pre-Botzinger complex (pre-BotC) of neonatal rat in vitro brainstem slice preparations.
133 vitro brainstem-spinal cord preparation from neonatal rat in which the respiratory and the locomotor
134 sion in the normal and the denervated PrV of neonatal rats in an in vitro brainstem preparation.
136 e same sequence is seen with OP exposures in neonatal rats, indicating that direct effects of these a
138 transport of Nanogold-labeled Fc (Au-Fc) in neonatal rat jejunum, focusing on later aspects of trans
139 cal or mechanical irritation of the colon of neonatal rats leads to chronic visceral hypersensitivity
142 D1 inhibition in whole-cell recordings from neonatal rats may be mediated by a change in NMDA recept
143 l of the pre-Botzinger complex (pre-BotC) in neonatal rat medullary slices in vitro, and in the more
144 neuronal current-voltage (I-V) relations in neonatal rat medullary slices in vitro, we demonstrated
146 ble to cause bacteremia or meningitis in the neonatal rat model and was significantly less virulent t
147 reviously that hypoxia-induced seizures in a neonatal rat model induce rapid phosphorylation of serin
148 ipotent Astrocytic Stem Cells (MASCs) into a neonatal rat model of hypoxia-ischemia (HI) and demonstr
154 and after VA supplementation.Sprague-Dawley neonatal rats (n = 104) were nursed by mothers fed a VA-
156 mined the expression and function of REST in neonatal rat oligodendrocyte precursor cells (OPCs).
157 amined in fibrous astrocytes within isolated neonatal rat optic nerve (RON) and in cultured cortical
158 olated by flow cytometry and cocultured with neonatal rat optic nerve cells in separate but media-con
159 A treatment of primary cortical neurons from neonatal rats or mice increases expression of phosphoryl
161 ered vehicle, thyroxine (T4) or metformin to neonatal rats post FAE and rats were tested in the hippo
163 ry and metabolic response to hyperthermia in neonatal rats (postnatal age 2-4 days), pregnant dams we
165 her mild chemical irritation of the colon of neonatal rats produced persistent changes in visceral se
168 n was cleared more rapidly from the lungs of neonatal rat pups compared with the wild-type strain, wh
170 ental emergence of odor fear conditioning in neonatal rat pups, and examined synaptic plasticity of i
172 processing the plasma of the same cohort of neonatal rats revealed no difference in the same cytokin
174 ne (a synthetic glucocorticoid) treatment to neonatal rats showed a temporal decrease in Runx2 with a
176 h frequency in experimental infection of the neonatal rat, significantly reduced the capacity of A192
179 anced by exposing the sacral segments of the neonatal rat spinal cord to the acetylcholinesterase inh
180 ombines patch-clamp recordings in the intact neonatal rat spinal cord with tract-tracing to demonstra
183 that in the whisker-related barrel cortex of neonatal rats, spontaneous whisker movements and passive
184 hat, in the whisker-related barrel cortex of neonatal rats, spontaneous whisker movements and passive
187 cells in vitro and in hippocampal neurons of neonatal rats subjected to cerebral hypoxia-ischemia in
188 etained efficiently in peripheral tissues of neonatal rats, suggesting that a more frequent, lower-do
189 nformatics analyses, we recently developed a neonatal rat system that enables maturation of PSC-deriv
193 els in Vero cells and attenuated and safe in neonatal rats; thus, the study supports clinical develop
194 LCS significantly decrease susceptibility of neonatal rats to oral E. coli K1 infection as reflected
195 blocked in perfused brain preparations from neonatal rats treated with selective antagonists of 5-HT
197 n brain slices obtained from male and female neonatal rats, using voltage-clamp protocols designed to
198 cultures of myocytes and myofibroblasts from neonatal rat ventricles were optically mapped using a vo
199 so that it connected 2 independently beating neonatal rat ventricular cardiomyocyte monolayers; it ac
200 bly, electrophysiology, and contractility of neonatal rat ventricular cardiomyocytes (NRVCMs) culture
203 T-p27) was sufficient to induce autophagy in neonatal rat ventricular cardiomyocytes in vitro, under
207 nd increased apoptosis were also observed in neonatal rat ventricular cardiomyocytes overexpressing t
208 from cryptophyte algae expressed in cultured neonatal rat ventricular cardiomyocytes produced inhibit
211 d, in confirmation of the data obtained from neonatal rat ventricular cardiomyocytes, demonstrated in
221 of impulses in monolayers of well-polarized neonatal rat ventricular cardiomyocytes.We traced electr
222 monolayers of cocultured myofibroblasts and neonatal rat ventricular cells by inhibiting myofibrobla
224 We micropatterned cell pairs consisting of a neonatal rat ventricular myocyte (NRVM) coupled to an en
226 ion on reentry frequency, APD, CV, and WL in neonatal rat ventricular myocyte (NRVM) monolayers infec
229 C2C12 skeletal myoblast differentiation and neonatal rat ventricular myocyte hypertrophy are inhibit
236 micropatterned strands (n=152) in which host neonatal rat ventricular myocytes (AP duration=153.2+/-2
238 ne the cellular location of NFAT in cultured neonatal rat ventricular myocytes (NRVMs) and adult feli
240 y, autophagic flux was increased in cultured neonatal rat ventricular myocytes (NRVMs) expressing a m
241 Mechanistically, MANF knockdown in cultured neonatal rat ventricular myocytes (NRVMs) impaired prote
243 growth, PLCepsilon protein was depleted from neonatal rat ventricular myocytes (NRVMs) using siRNA.
244 he level of transcription, HEK 293 cells and neonatal rat ventricular myocytes (NRVMs) were transfect
245 e developed a novel tissue model of cultured neonatal rat ventricular myocytes (NRVMs) with uniform o
246 The sensors were subsequently expressed in neonatal rat ventricular myocytes and acutely isolated a
247 and ROMK mRNA was confirmed to be present in neonatal rat ventricular myocytes and adult hearts.
249 ed IBZ by coculturing skeletal myotubes with neonatal rat ventricular myocytes and performed optical
250 y downregulated in Gq or by Gq expression in neonatal rat ventricular myocytes and reversed by CaMKII
251 GRK5 presented with the opposite results in neonatal rat ventricular myocytes as p65 and p50 were de
254 fness on cardiomyocyte maturation, we plated neonatal rat ventricular myocytes for 7 days on collagen
255 d overexpression of Fstl1 protected cultured neonatal rat ventricular myocytes from hypoxia/reoxygena
256 eduction, action potentials in Wnt3a-treated neonatal rat ventricular myocytes had a lower upstroke a
257 s of GDF11, showed that GDF11 did not reduce neonatal rat ventricular myocytes hypertrophy, but inste
258 culturing Kv1.5-expressing HEK 293 cells and neonatal rat ventricular myocytes in low osmolarity (LO)
259 ential, and cell death were recapitulated in neonatal rat ventricular myocytes infected with constitu
262 cal mapping of V(m) in patterned cultures of neonatal rat ventricular myocytes to assess the relation
267 of-function ETV1 RNA sequencing dataset from neonatal rat ventricular myocytes transduced with Etv1 s
268 agation consisting of monolayers of cultured neonatal rat ventricular myocytes treated with anthopleu
269 lso observed in hERG-HEK cells as well as in neonatal rat ventricular myocytes treated with the musca
270 wn of endogenous Cav3 or Nedd4-2 in cultured neonatal rat ventricular myocytes using siRNA led to an
271 ssion of resistin using adenoviral vector in neonatal rat ventricular myocytes was associated with in
275 model of ACM (expression of JUP(2157del2) in neonatal rat ventricular myocytes) and a robust murine m
276 ioned media exerted antiapoptotic effects on neonatal rat ventricular myocytes, and proangiogenic eff
277 -resolution optical mapping in monolayers of neonatal rat ventricular myocytes, containing approximat
278 In vitro studies using phenylephrine-treated neonatal rat ventricular myocytes, to explore the putati
279 in-like growth factor 1 (IGF-1) treatment in neonatal rat ventricular myocytes, translocates to mitoc
292 THODS AND Patch-clamp recordings of cultured neonatal rat ventricular myofibroblasts revealed that TG
294 vitro brainstem-spinal cord preparation from neonatal rat, we report that the locomotor-related signa
296 ceptors, highly enriched ipRGC cultures from neonatal rats were generated using anti-melanopsin-media
300 ncluding skin, brain, and adipose tissue, in neonatal rats without and after VA supplementation.Sprag