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1 and the responses underlying lithium-induced nephrogenic diabetes insipidus.
2 in renal handling of water in health and in nephrogenic diabetes insipidus.
3 and trafficking, the absence of which causes nephrogenic diabetes insipidus.
4 alcitonin has a potential therapeutic use in nephrogenic diabetes insipidus.
5 VP, a vasopressin analog), characteristic of nephrogenic diabetes insipidus.
6 nt selected alleles responsible for X-linked nephrogenic diabetes insipidus.
7 otypes--ranging from congenital cataracts to nephrogenic diabetes insipidus.
8 deletion in mice produces distinct forms of nephrogenic diabetes insipidus.
9 ility; mutations or downregulation can cause nephrogenic diabetes insipidus.
10 were compatible with a diagnosis of partial nephrogenic diabetes insipidus.
11 MIP underlie an autosomal recessive form of nephrogenic diabetes insipidus and absence of the Colton
12 -of-function mutations in aquaporins include nephrogenic diabetes insipidus and congenital cataracts.
13 nstay treatment for bipolar disorder, causes nephrogenic diabetes insipidus and hypercalcemia in abou
15 these results identify a novel mechanism of nephrogenic diabetes insipidus and uncover a role of SOC
16 familiar diseases such as Gaucher's disease, nephrogenic diabetes insipidus, and Creutzfeldt-Jakob di
17 Finally, renal distal tubular acidosis or nephrogenic diabetes insipidus can be caused by autoanti
18 e for retinitis pigmentosa, color blindness, nephrogenic diabetes insipidus, familial ACTH resistance
19 hium, a potent inhibitor of GSK3beta, causes nephrogenic diabetes insipidus, GSK3beta may play a cruc
20 vasopressin 2 receptor and aquaporin 2 cause nephrogenic diabetes insipidus; however, expression of t
23 forms of diabetes insipidus (e.g., X-linked nephrogenic diabetes insipidus) in which the kidney resp
24 ion R137H, which is associated with familial nephrogenic diabetes insipidus, induces constitutive arr
27 with tolvaptan, a VR2 blocker that causes a nephrogenic diabetes insipidus-like excessive loss of hy
29 chronic experimental iron overload leads to nephrogenic diabetes insipidus marked by AVP-resistant u
30 aquaporin-2 (AQP2) point mutants that cause nephrogenic diabetes insipidus (NDI) are retained in the
35 wn in a variety of animal models of acquired nephrogenic diabetes insipidus (NDI) that loss of the aq
37 n-2 (AQP2) mutations that cause non-X-linked nephrogenic diabetes insipidus (NDI) were characterized
38 20% of patients treated with lithium develop nephrogenic diabetes insipidus (NDI), a disorder charact
39 fect in urinary concentrating ability, i.e., nephrogenic diabetes insipidus (NDI), but the molecular
40 ep in generating a transgenic mouse model of nephrogenic diabetes insipidus (NDI), we have analyzed t
48 have shown that a molecular determinant for nephrogenic diabetes insipidus, the vasopressin receptor
49 ceptors known to be responsible for X-linked nephrogenic diabetes insipidus were used as model system
50 hat Foxa1(-/-) mice represent a new model of nephrogenic diabetes insipidus with unique molecular eti
51 a nonsense mutation known to cause X-linked nephrogenic diabetes insipidus (XNDI) in humans (Glu242s