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1 elinating neuropathy and to severely reduced nerve conduction velocity.
2 ative correlation between fat mass and motor nerve conduction velocity.
3 ative correlation between fat mass and motor nerve conduction velocity.
4 ormed association studies of height-adjusted nerve conduction velocity.
5 s an unexpectedly strong predictor of slowed nerve conduction velocity.
6 es myelin outfoldings that impair the normal nerve conduction velocity.
7 x 10(-9)), indicating that INPP4B regulates nerve conduction velocity.
8 e exhibit impaired motor function and slower nerve conduction velocity.
9 al hyperalgesia, cold allodynia, and sciatic nerve conduction velocity.
10 ich, in turn, has a significant influence on nerve conduction velocity.
11 nset of functional changes such as decreased nerve conduction velocity.
12 d progressive demyelination and a slowing of nerve conduction velocity.
13 ore there were any detectable alterations in nerve conduction velocity.
14 o results in a severe decrease in peripheral nerve conduction velocity.
15 did not induce the phenomenon of slowing of nerve-conduction velocity.
16 e population and the horses with the slowest nerve conduction velocities.
17 rological deficit and had profoundly reduced nerve conduction velocities.
18 ents in multiple motor performance tests and nerve conduction velocities.
19 quadriceps muscle contractility, and sciatic nerve conduction velocities.
20 g of axons, delayed myelination, and reduced nerve conduction velocities.
21 xtremities and displayed a mild reduction of nerve conduction velocities.
22 of onset and the degree of slowing of motor nerve conduction velocities.
23 by significant slowing of motor and sensory nerve conduction velocities.
24 human Schwann cells (~5%), and evaluation of nerve conduction velocity (0.13-0.28 m/sec), previously
26 een a ratio of 0.8 and 1.5, histological and nerve conduction velocity abnormalities are observed, bu
27 d were associated with a 25-30% reduction in nerve conduction velocity, all of which were completely
28 a markedly low birth weight, very slow motor nerve conduction velocities and a general decrease in th
29 muscle action potential amplitudes, improved nerve conduction velocities and ameliorated muscle stren
30 thy with hypo- and demyelination, slowing of nerve conduction velocities and disturbed nodal architec
31 ice experienced significant deterioration in nerve conduction velocities and IENF density and develop
32 high-fat diet to a MUFA-rich high-fat diet; nerve conduction velocities and intraepidermal nerve fib
33 on protein fully restored motor- and sensory-nerve conduction velocities and maintained the axonal ca
34 (R98C/R98C) mice develop weakness, abnormal nerve conduction velocities and morphologically abnormal
35 g, mitochondrial DNA deletions, reduction of nerve conduction velocities and of heat sensation, and b
36 al recovery as assessed by motor and sensory nerve conduction velocities and sciatic functional index
37 , resulted in severe hypomyelination, slowed nerve conduction velocity and behaviour dysfunctions in
38 al defects, transgenic mice also have slower nerve conduction velocity and defects in their responses
39 providing critical clinical metrics such as nerve conduction velocity and histomorphometry are neces
40 ical deficits were associated with decreased nerve conduction velocity and increased sensory threshol
41 2 was positively correlated with decreasing nerve conduction velocity and increasing pain severity.
42 arkably ameliorated DPN by improving sciatic nerve conduction velocity and increasing thermal and mec
43 ripheral neuropathy characterized by reduced nerve conduction velocity and myelin outfoldings and inf
46 s of large-diameter myelinated axons, slowed nerve conduction velocity and reduced amplitude of compo
47 s accompanied by decreased motor and sensory nerve conduction velocity and reduced compound muscle ac
48 orphology and impaired transmission, reduced nerve conduction velocities, and a loss of large-diamete
49 rial nutritive blood flow, motor and sensory nerve conduction velocities, and metabolic abnormalities
50 ep-tendon reflexes, sensory impairment, slow nerve conduction velocities, and nerve demyelination.
51 itol supplementation on nerve Na+-K+-ATPase, nerve conduction velocity, and myelinated nerve fiber pa
53 onduction velocity, hindlimb digital sensory nerve conduction velocity, and sciatic nerve concentrati
54 f memory, attention, visuomotor function, or nerve conduction velocities (average z scores were very
55 eripheral neuropathy characterized by slowed nerve conduction velocity, axon loss, and distinctive my
56 Guillain-Barre syndrome, we determined that nerve conduction velocities between the sciatic notch an
57 METHODS AND We found that motor and sensory nerve conduction velocities, blood flow, and capillary d
58 th significant lowering of motor and sensory nerve conduction velocity but no alteration in the mean
59 nerve fibers, specifically sural or sciatic nerve conduction velocities, but significantly improved
60 inhibited a diabetes-induced defect in ulnar nerve conduction velocity, but neither agent was found t
61 TDs, produced significant deficits in caudal nerve conduction velocity, caudal amplitude and digital
62 People with severe PAD have poorer peroneal nerve conduction velocity compared with people with mild
63 by significant slowing of motor and sensory nerve conduction velocities, compared with nondiabetic c
65 DM, we observed that, in addition to reduced nerve conduction velocity, db/db mice also develop PNH.
66 it normalizes polyols and reduces the motor nerve conduction velocity deficit by 59% relative to dia
67 lgesia, tactile allodynia, motor and sensory nerve conduction velocity deficits, and reduction in tib
68 sulted in motor and sensory deficits, slowed nerve conduction velocities, delayed myelination and hyp
69 ropathy that presents in adults, with normal nerve conduction velocities, designated as a 'CMT2' form
71 ytostatic agents and a moderate reduction of nerve conduction velocity for cisplatin and paclitaxel.
72 syndrome, but not healthy controls, sensory nerve conduction velocity for Digits 2 and 3 was slower
75 rh10 alone or in addition to HSCT normalized nerve conduction velocity, improved ultrastructure, and
76 rmore, treated mice showed increased sciatic nerve conduction velocities, improvement of myelination
78 yses also reveal that the reduction of motor nerve conduction velocities in affected patients is prop
79 d axonal degeneration, which does not impact nerve conduction velocities in E365del mice, but it does
80 g the formation of compact myelin and normal nerve conduction velocities in the homozygous state.
84 nd 21b,d all produce a reversible slowing of nerve-conduction velocity in the mice, the onset of whic
85 obands from all families presented with slow nerve conduction velocities, matching the diagnostic cat
86 ct) techniques, such as electromyography and nerve conduction velocity measurement, do not reliably p
87 as determined by abnormal motor and sensory nerve conduction velocity, mechanical allodynia, and los
88 vascular relaxation precede slowing of motor nerve conduction velocity (MNCV) and decreased sciatic n
89 ld) clearly developed manifest sciatic motor nerve conduction velocity (MNCV) and hind-limb digital s
90 and axonal loss, which underlie slowed motor nerve conduction velocity (MNCV) and reduced compound mu
92 ic with streptozotocin, and changes in motor nerve conduction velocity (MNCV), mechanical and thermal
93 al abnormalities of EDN, i.e., reduced motor nerve conduction velocity (MNCV), total and endoneurial
94 voluntary locomotor activity, reduced motor nerve conduction velocities (MNCVs) and muscle atrophy.
95 vere PMD, increased myelination and restored nerve conduction velocity, motor function and lifespan o
96 ozygous R98C animals, but it did not improve nerve conduction velocity, myelin thickness, G-ratios or
97 Transplantation significantly improved tail nerve conduction velocity, Na(+)-K(+)-ATPase activity, a
100 used on either behaviors or acute changes in nerve conduction velocity (NCV) and amplitude, but great
102 sion of peripheral neuropathy as assessed by nerve conduction velocity (NCV) in individuals affected
105 in transducing high glucose into deficits in nerve conduction velocity (NCV) that are characteristic
106 on nerve laser Doppler blood flow (NLDF) and nerve conduction velocity (NCV) were assessed after 1-16
107 gait disturbances, foot deformities, reduced nerve conduction velocity (NCV) without demyelination an
108 heral neuropathy (DPN) is marked by impaired nerve conduction velocity (NCV), reduced nerve blood flo
110 Negative correlations were found with sural nerve conduction velocities (NCVs) (r = -0.65, P < 0.001
111 and FA values were correlated to electrical nerve conduction velocities (NCVs) with Pearson correlat
112 Patients with CMT1A typically have slowed nerve conduction velocities (NCVs), reduced compound mot
113 Sural nerve myelinated fiber density (MFD), nerve conduction velocities (NCVs), vibration perception
114 Effects of each drug on caudal and digital nerve conduction velocity, nerve amplitude, and sciatic
119 = 0.04), sural (p = 0.02) and peroneal motor nerve conduction velocity (p = 0.03) deteriorated signif
121 ow, significantly improved motor and sensory nerve conduction velocity, prevented the development of
122 ative correlations were found for the tibial nerve conduction velocity (r = -0.23; 95% CI: -0.44, -0.
123 ative correlations were found for the tibial nerve conduction velocity (r = -0.24; 95% CI: -0.45, -0.
124 ents from six pedigrees (n = 38) had reduced nerve conduction velocities ranging from 7.5 to 27.0m/se
125 l content was elevated (P < 0.001) and motor nerve conduction velocity reduced (P < 0.05) in galactos
126 layed maturation of nodes of Ranvier, slowed nerve conduction velocity, reduced muscle mass and reduc
129 rt internodal lengths and associated reduced nerve conduction velocity seen in the absence of periaxi
131 (R/S)-1 and (R)-1 partially prevented motor nerve conduction velocity slowing in a mouse model of ty
132 elocity (MNCV) and hind-limb digital sensory nerve conduction velocity (SNCV) deficits, thermal hypoa
134 rats fed high-fat diets caused a decrease in nerve conduction velocity, thermal hypoalgesia, and intr
135 an extracellular matrix protein involved in nerve conduction velocity, use of this alternative splic
138 young mice with short mutant Schwann cells, nerve conduction velocity was reduced and motor function
142 erences in neurobehavioral assessments or in nerve conduction velocity when compared with children wh
143 tions in sciatic motor and saphenous sensory nerve conduction velocity, which were 62 and 78% correct