ライフサイエンスコーパス: nerve growth factor

1 ays 0, 3, 7, and 12 following treatment with nerve growth factor.

2 monoclonal antibody that binds and inhibits nerve growth factor.

3 neurite outgrowth in PC12 cells treated with nerve growth factor.

4 e A (TrkA) is the high-affinity receptor for nerve growth factor.

5 .849, p<0.001), alpha1-ACT (0.638, p<0.001), nerve growth factor (5.475, p<0.005) and visinin-like pr

6 that are also the primary sites of action of nerve growth factor, a powerful modulator of bladder ner

7 at act at receptor tyrosine kinases, such as nerve growth factor, also cause differentiation.

8 ts interaction with proapoptotic ligands pro-nerve growth factor and amyloid-beta peptide.

9 ation around itself, and that high levels of nerve growth factor and axon guidance molecules are reco

10 (2) this interaction is modified by ligands nerve growth factor and beta-amyloid; (3) APP and p75(NT

11 ed in intracellular transport and sorting of nerve growth factor and brain-derived neurotrophic facto

12 ed in intracellular transport and sorting of nerve growth factor and brain-derived neurotrophic facto

13 eutralizing antibodies against brain-derived nerve growth factor and ciliary neurotrophic factor.

14 Nerve growth factor and insulin-like growth factor-1 exp

15 Nerve growth factor and its TrkA receptor were upregulat

16 d the AP1-mediated transcription promoted by nerve growth factor and modulated the expression of seve

17 E7 to E18 corneal expressions of nerve growth factor and neurotrophin-3 genes were unchan

18 otrophins brain derived neurotrophic factor, nerve growth factor and neurotrophin-3 in the supernatan

19 trophin 3, insulin-like growth factor 1, and nerve growth factor and of the nerve growth factor recep

20 F-A-dependent gene transcription elicited by nerve growth factor and serum.

21 on equatorial BM except for higher levels of nerve growth factor and thrombospondin-2 (TSP2) by hES-R

22 tor blockade is independent of brain-derived nerve growth factor and TrkB receptor signaling.

23 diminished levels of inflammatory cytokines, nerve growth factor, and endogenous pruritogens, such as

24 including brain-derived neurotrophic factor, nerve growth factor, and neurotrophin 3.

25 mpathetic axon outgrowth that was blocked by nerve growth factor antibodies.

26 (i) Anti-nerve growth factor antibody accelerated the reactivatio

27 nti-calcitonin gene-related peptide and anti-nerve growth factor) are also discussed.

28 e growth factor-binding protein 2 (IGFBP-2), nerve growth factor (b-NGF), platelet-derived growth fac

29 The plasma concentrations of nerve growth factor (BCAA: 4.0 +/- 1 pg/mL; low-BCAA: 5.

30 day, driven by circadian clock control of a nerve growth factor (BDNF) in the inner ear.

31 e structurally related cystine-knot protein, nerve growth factor beta (NGFbeta), plays an unexpected

32 orcine, bovine, and murine sequences of beta nerve growth factor (beta-NGF).

33 Nerve growth factor-beta (NGF) is essential for the corr

34 ed proteins: a therapeutic IgG1-antibody and nerve growth factor-beta (NGF).

35 ion of Akt and ERK/MAP kinase in response to nerve growth factor-beta (NGF-beta) but not FP6.

36 is was associated with reduced expression of nerve growth factor-beta, indicating less atrial nerve s

37 oclonal antibody, MEDI1912, selected against nerve growth factor binds with picomolar affinity, but u

38 r cells express neurotrophic markers such as nerve growth factor, brain-derived neurotrophic factor,

39 ch a GM-CSF->CCL17 pathway appears critical, nerve growth factor, CGRP, and substance P all appear to

40 ted with the well established pain mediator, nerve growth factor, could also modify macrophage gene t

41 Furthermore, decreased nerve growth factor, decreased c-fos and increased sympa

42 ecal Rab7-siRNA or, indirectly, by reversing nerve growth factor deprivation in peripheral sensory ne

43 le Caspase-6 is activated in axons following nerve growth factor deprivation, microfluidic chamber ex

44 axon-specific but not whole-cell (apoptotic) nerve growth factor deprivation.

45 The present study used nerve growth factor differentiated PC12 cells (NGFDPC12

46 c acid (PA)-induced lipotoxicity (PA-LTx) in nerve growth factor-differentiated PC12 (NGFDPC12) cells

47 from cardiac synaptosomes and dopamine from nerve growth factor-differentiated PC12 cells in a conce

48 The homodimer NGF (nerve growth factor) exerts its neuronal activity upon b

49 Choline supplementation increased striatal nerve growth factor expression in wild-type and Mecp2(1l

50 melanocytes and melanogenesis plus FIG4 and nerve growth factor expression, suggesting higher cellul

51 s as well as reduce mast cell activation and nerve growth factor expression.

52 e ratio of pro-nerve growth factor to mature nerve growth factor, favouring p75 receptor expression a

53 including brain-derived neurotrophic factor, nerve growth factor, glial cell-derived neurotrophic fac

54 solate low-affinity antibodies to human beta nerve growth factor (hbetaNGF).

55 Human nerve growth factor (hNGF) is an important pharmaceutica

56 d in chromatin remodeling of the RARbeta and nerve growth factor IB ( NUR77).

57 we have demonstrated increased levels of pro-nerve growth factor in cerebrospinal fluid and increased

58 Increased expression of nerve growth factor in injured or inflamed tissue is ass

59 the plasma concentration of norepinephrine, nerve growth factor in the gastric fundus muscularis ext

60 Overexpression of nerve growth factor in the lumbosacral spinal cord induc

61 trograde model derived from experiments with nerve growth factor in the peripheral nervous system.

62 tor to p75 neurotrophin receptor, blocks pro-nerve growth factor induced apoptosis in cells expressin

63 Nerve growth factor induced mild cold sensitization, con

64 munoprecipitation experiments indicated that nerve growth factor induced the association of endogenou

65 1 transactivates the Skp2 promoter through a nerve growth factor-induced clone B response element (NB

66 ereas USP21 knockdown in PC12 cells inhibits nerve growth factor-induced neurite outgrowth.

67 siRNA improved cell survival in response to nerve growth factor-induced OL apoptosis.

68 hat increased DNA methylation at the NGFI-A (nerve growth factor-induced protein A) binding site of t

69 We find that Vgf nerve growth factor inducible gene up-regulation is a co

70 the transcriptional activity of Nurr1 on an nerve growth factor inducible-B response element reporte

71 hibitory factors (e.g. inhibin alpha and VGF nerve growth factor-inducible), 2) activation of a signa

72 anticipated, long and extensive neuritis on nerve growth factor induction.

73 which show promise in clinical trials, like nerve growth factor inhibitors and p38 kinase inhibitors

74 dy, we determined the interactive effects of nerve growth factor, insulin-like growth factor 1, and e

75 Studies indicate that treatment against nerve growth factor, interleukins, and ischemic-like med

76 onsequence of the increased levels of mature nerve growth factor levels in skin, as revealed by Weste

77 n to motoneurons, depletion of increased pro-nerve growth factor levels or p75 receptor blockade.

78 only known Toll receptor ligand is the human nerve growth factor-like cystine knot protein Spatzle.

79 owed a reduction in neurite outgrowth and in nerve growth factor-mediated neuronal differentiation, t

80 elevated expression of key proteins such as nerve growth factor, myelin protein zero, and brain deri

81 inistration of neurotrophic factors (such as nerve growth factor, neurotrophin3, glial-derived neurot

82 and sympathetic neurons is promoted whether nerve growth factor (NGF) activates TrkA receptors on th

83 rch for neuroactive compounds that mimic the nerve growth factor (NGF) activity for the protection ag

84 the preservation of sufficient expression of nerve growth factor (NGF) and activation of the neurotro

85 ta), tumor necrosis factor alpha (TNFalpha), nerve growth factor (NGF) and brain derived neurotrophic

86 Neurotrophins such as nerve growth factor (NGF) and brain-derived neurotrophic

87 rait loci associated with gene expression of nerve growth factor (NGF) and calneuron 1 (CALN1) genes.

88 The nerve growth factor (NGF) and glial cell line-derived ne

89 The effect of nerve growth factor (NGF) and its receptor (NGFR) in inf

90 ends, can be modulated by luminar release of nerve growth factor (NGF) and neurotrophin-3 (NT-3), res

91 Nerve growth factor (NGF) and neurotrophin-3 serve as at

92 h cone collapse and loss of actin filaments, nerve growth factor (NGF) and neurotrophin-3 still induc

93 SorCS3 binds the nerve growth factor (NGF) and platelet-derived growth fa

94 Nerve growth factor (NGF) antagonism is on the verge of

95 brain-derived neurotrophic factor (BDNF) and nerve growth factor (NGF) are crucial modulators in the

96 Levels of nerve growth factor (NGF) are elevated in inflamed tissu

97 In this paper, we identify nerve growth factor (NGF) as a binding partner for MOG a

98 In this study, we test nerve growth factor (NGF) as an understudied therapeutic

99 satetraenoic acid (12[S] or 15[S]-HETE), and nerve growth factor (NGF) as positive control.

100 aglandin E(2) (PGE(2)), bradykinin (BK), and nerve growth factor (NGF) as well as multiple kinases, i

101 in animals and humans show that blockade of nerve growth factor (NGF) attenuates both malignant and

102 tal apoptotic pathway that is activated when nerve growth factor (NGF) becomes limiting for neuronal

103 TrkA activation by nerve growth factor (NGF) binding the second extracellul

104 esis of SCG neurons at a level comparable to nerve growth factor (NGF) but also doubled the neurite l

105 PC12 pheochromocytoma cell line responds to nerve growth factor (NGF) by exiting from the cell cycle

106 ated the output activity of Ngf encoding the nerve growth factor (NGF) by increasing histone H4 acety

107 d the selective translation and secretion of nerve growth factor (NGF) by PDAC cells to promote tumor

108 odels of MM to show significant induction of nerve growth factor (NGF) by the tumour-bearing bone mic

109 Elevating levels of nerve growth factor (NGF) can have pronounced effects on

110 SIGNIFICANCE STATEMENT The R100W mutation in nerve growth factor (NGF) causes Hereditary Sensory and

111 Nerve growth factor (NGF) contributes to the development

112 Although nerve growth factor (NGF) controls survival, maturation

113 Sertad1 is also induced in neurons by nerve growth factor (NGF) deprivation and Abeta (beta-am

114 the painful arthritic knee joint and whether nerve growth factor (NGF) drives this pathologic reorgan

115 ent a detailed motion analysis of retrograde nerve growth factor (NGF) endosomes in axons to show tha

116 Nerve growth factor (NGF) exerts protective effects on t

117 We show that PC12 cells stimulated with nerve growth factor (NGF) exhibit statistically signific

118 elta), two transcription factors involved in nerve growth factor (NGF) expression and downregulated s

119 timulation of the gastric epithelium induced nerve growth factor (NGF) expression, and in turn NGF ov

120 linergic neurons in both regions depend upon nerve growth factor (NGF) for their survival and maturat

121 ciated haplotype blocks were intronic to the Nerve Growth Factor (NGF) gene (P=0.001, 0.001, 0.002),

122 In 2001, we initiated a clinical trial of nerve growth factor (NGF) gene therapy in AD, the first

123 Nerve growth factor (NGF) has an important role in regul

124 ) at the cell surface, and administration of nerve growth factor (NGF) has been considered and attemp

125 The neurotrophin nerve growth factor (NGF) has been implicated as a key m

126 ng mode of Spz to Toll is similar to that of nerve growth factor (NGF) in complex with the p75 neurot

127 +) BMCs expressed neurotrophins and secreted nerve growth factor (NGF) in conditioned medium.

128 A role for nerve growth factor (NGF) in contributing to increased v

129 taRII-deficient pups showed a lower level of nerve growth factor (NGF) in its mRNA; however, higher l

130 n channel subfamily M, member 8 (TRPM8); and nerve growth factor (NGF) in nasal biopsy specimens.

131 s and becomes functional by the neurotrophin nerve growth factor (NGF) in native brainstem neurons.

132 on (DRG) neurons by increasing expression of nerve growth factor (NGF) in the colon wall.

133 se it is supported by decades of research on nerve growth factor (NGF) in the peripheral nervous syst

134 The endogenous nerve growth factor (NGF) in the urinary bladder regulat

135 Previously, we demonstrated that nerve growth factor (NGF) increased the excitability of

136 Nerve growth factor (NGF) induces collateral branching a

137 Nerve growth factor (NGF) influences the key pathologica

138 Nerve growth factor (NGF) influences the survival and di

139 The microinjection of nerve growth factor (NGF) into the cat pontine tegmentum

140 Nerve growth factor (NGF) is a key mediator of nocicepti

141 Nerve growth factor (NGF) is a key regulator of chronic

142 Nerve growth factor (NGF) is a neuropeptide essential fo

143 Nerve growth factor (NGF) is a neurotrophin that activat

144 Nerve growth factor (NGF) is a neurotrophin that is impl

145 Nerve growth factor (NGF) is a potent survival and axon

146 Nerve growth factor (NGF) is a target-derived neurotroph

147 Nerve growth factor (NGF) is elevated in certain chronic

148 Nerve growth factor (NGF) is initially synthesized as a

149 that retrograde signaling by target-derived nerve growth factor (NGF) is necessary for soma-to-axon

150 Nerve growth factor (NGF) is produced in the hippocampus

151 r mechanism of TRKA activation by its ligand nerve growth factor (NGF) is still unsolved.

152 Nerve growth factor (NGF) is the neurotrophin responsibl

153 Nerve growth factor (NGF) levels are highly increased in

154 Nerve growth factor (NGF) monoclonal antibody therapy ha

155 eactivation in medium containing antibody to nerve growth factor (NGF) or delayed reactivation in med

156 We hypothesized that nerve growth factor (NGF) plays a key role in this proce

157 they innervate is regulated by the supply of nerve growth factor (NGF) produced by these tissues.

158 Target-derived nerve growth factor (NGF) promoted expression of its own

159 Here, we report that Nerve Growth Factor (NGF) promotes endocytosis of its Tr

160 Nerve growth factor (NGF) promotes growth, differentiati

161 on that leads to cell proliferation, whereas nerve growth factor (NGF) promotes sustained ERK activat

162 In sensory neurons, nerve growth factor (NGF) promotes the formation of axon

163 Nerve growth factor (NGF) protein expression and secreti

164 Activation of the high-affinity nerve growth factor (NGF) receptor Trk occurs through mu

165 Activation of the nerve growth factor (NGF) receptor trkA and tissue acido

166 as' disease parasite Trypanosoma cruzi binds nerve growth factor (NGF) receptor TrkA, increasing rece

167 The Tp53inp2 transcript interacts with the nerve growth factor (NGF) receptor TrkA, regulating TrkA

168 Notably, we identified the nerve growth factor (NGF) receptor tyrosine kinase (NTRK

169 The nerve growth factor (NGF) receptor, trkA, the tumour sup

170 tic neurons by inhibiting endocytosis of the nerve growth factor (NGF) receptor, TrkA.

171 he administration of neuregulin 1 (NRG1) and nerve growth factor (NGF) recombinant proteins.

172 Nerve growth factor (NGF) regulates many aspects of neur

173 The neurotrophin nerve growth factor (NGF) regulates neuronal growth, dif

174 Here we demonstrate that nerve growth factor (NGF) regulates the levels of IP(5)

175 performing candidate mediates high levels of nerve growth factor (NGF) secretion from astrocytes, cau

176 s promoted ADRB2-dependent PDAC development, nerve growth factor (NGF) secretion, and pancreatic nerv

177 gic deficit is associated with alteration in nerve growth factor (NGF) signaling and its relation to

178 ic neurons that was essential for retrograde nerve growth factor (NGF) signaling and neuron target ti

179 Facilitation of nerve growth factor (NGF) signaling by the p75 neurotrop

180 This study reports that Rab22 promotes nerve growth factor (NGF) signaling-dependent neurite ou

181 MO7e express TrkA, the primary receptor for Nerve Growth Factor (NGF) signaling.

182 to disrupt retrograde axonal trafficking of nerve growth factor (NGF) signals.

183 anges in living colonies of PC12 cells under nerve growth factor (NGF) stimulation for up to 7 days u

184 RK) activity and cell proliferation, whereas nerve growth factor (NGF) stimulation leads to sustained

185 ng in cholesterol-rich membrane regions upon nerve growth factor (NGF) stimulation: We argue that thi

186 Retrograde trophic signaling of nerve growth factor (NGF) supports neuronal survival and

187 Numerous studies have indicated that nerve growth factor (NGF) supports survival and phenotyp

188 Studies also show that peroxynitrite impairs nerve growth factor (NGF) survival signaling in sensory

189 n ongoing discussion is whether signaling of nerve growth factor (NGF) through its high-affinity rece

190 reported that enables intravenously injected nerve growth factor (NGF) to enter the CNS in healthy mi

191 and impairs neurite outgrowth in response to nerve growth factor (NGF) treatment.

192 egeneration in regards to local secretion of nerve growth factor (NGF) upon carious injury.

193 Nerve growth factor (NGF) was discovered because of its

194 The death of sympathetic neurons after nerve growth factor (NGF) withdrawal requires de novo ge

195 Nerve growth factor (NGF), a classical trophic factor fo

196 asthma control by stimulating expression of nerve growth factor (NGF), a neurotrophin associated wit

197 ytosis into axon growth cones is enhanced by nerve growth factor (NGF), acting locally on distal axon

198 e treatment with an antibody that sequesters nerve growth factor (NGF), administered when the pain an

199 ptase (TPS1) and mastin, neuromedin-B (NMB), nerve growth factor (NGF), and leukotriene-synthesis enz

200 x8 expression and function were regulated by nerve growth factor (NGF), and the effect of NGF was pot

201 brain derived neurotrophic factor (BDNF) and nerve growth factor (NGF), and this increase is accompan

202 MCF2L regulates a nerve growth factor (NGF), and treatment with a humanize

203 cific enolase, growth-associated protein 43, nerve growth factor (NGF), and tyrosine kinase receptor

204 ated by hormones of the neurotrophin family: nerve growth factor (NGF), brain derived neurotrophic fa

205 ophin family of growth factors, comprised of nerve growth factor (NGF), brain derived neurotrophic fa

206 free DPSC cultures and neurotrophic factors; nerve growth factor (NGF), brain-derived neurotrophic fa

207 sence of fibroblast growth factor 2 (FGF-2), nerve growth factor (NGF), brain-derived neurotropic fac

208 d protein expression increase in response to nerve growth factor (NGF), concomitant with differentiat

209 pared routes of delivery of the neurotrophin nerve growth factor (NGF), either through a multisynapti

210 ine distinct monoclonal antibodies targeting nerve growth factor (NGF), for which we compare the pred

211 (d) in culture and this can be prevented by nerve growth factor (NGF), GDNF and ARTN, but not NRTN.

212 We show that nerve growth factor (NGF), implicated in the morphogenes

213 Neurotrophins, including nerve growth factor (NGF), increase neurite outgrowth in

214 directly binds acinus, which is regulated by nerve growth factor (NGF), inhibiting its stimulatory ef

215 ociceptive and inflammatory factors, such as nerve growth factor (NGF), interleukin-6 (IL-6), and car

216 However, the prototypic neurotrophin, nerve growth factor (NGF), is not thought to be anterogr

217 by receptor tyrosine kinase ligands such as nerve growth factor (NGF), that involves both Rap1 and P

218 a (TNFalpha), interleukin-1 beta (IL1 beta), nerve growth factor (NGF), the neuronal nuclear protein

219 Pro-nerve growth factor (NGF), the precursor of NGF, is a we

220 ne interleukin 6 (IL-6) and the neurotrophin nerve growth factor (NGF), which are intimately linked t

221 to ischemia, retinal neuronal cells express nerve growth factor (NGF), which can be proangiogenic.

222 oietic cytokine ligands of gp130 but also to nerve growth factor (NGF), which does not bind to gp130-

223 In this study we determined the number of nerve growth factor (NGF)- and interleukin-1beta (IL1bet

224 During this process, nerve growth factor (NGF)- TrkA signaling in axons commu

225 tors tropomyosin-related kinase A (TrkA) for nerve growth factor (NGF)-beta, TrkB for brain-derived n

226 We discovered that KIF1Bbeta is required for nerve growth factor (NGF)-dependent neuronal differentia

227 This affects the development of nerve growth factor (NGF)-dependent neurons including sy

228 Drp1-mediated fission is required for nerve growth factor (NGF)-induced collateral branching i

229 f human CCF astrocytoma cells but stimulated nerve growth factor (NGF)-induced neurite outgrowth from

230 terference RNA methodology strongly enhanced nerve growth factor (NGF)-induced neurite outgrowth in P

231 ative mutants of ATL1 in PC-12 cells inhibit nerve growth factor (NGF)-induced neurite outgrowth.

232 s study, we investigated the effect of ES on nerve growth factor (NGF)-induced neuronal differentiati

233 Egr3 is a nerve growth factor (NGF)-induced transcriptional regula

234 Nerve growth factor (NGF)-induced transport of large ves

235 NF-alpha-TNFR1 forward signaling to suppress nerve growth factor (NGF)-mediated neurite growth, survi

236 oter under basal conditions and also enhance nerve growth factor (NGF)-mediated trkA promoter activat

237 This is reversed by inhibition of nerve growth factor (NGF)-mediated tropomyosin receptor

238 This retention is mediated by a nerve growth factor (NGF)-regulated checkpoint that dela

239 Here, using single-cell image analysis of nerve growth factor (NGF)-stimulated PC12 cells, we iden

240 ouse sympathetic neurons, the target-derived nerve growth factor (NGF)-tropomyosin-related kinase typ

241 s mediate extension of sympathetic axons via nerve growth factor (NGF).

242 onal growth in response to the neurotrophin, nerve growth factor (NGF).

243 e transmembrane receptor tyrosine kinase for nerve growth factor (NGF).

244 ll neurite extension in response to released nerve growth factor (NGF).

245 ng leptin, insulin, growth hormone (GH), and nerve growth factor (NGF).

246 , basic fibroblast growth factor (bFGF), and nerve growth factor (NGF).

247 anscription factor activation in response to nerve growth factor (NGF).

248 lated local IMPA1 translation in response to nerve growth factor (NGF).

249 at involves depriving sympathetic neurons of nerve growth factor (NGF).

250 the cell surface, similar to treatment with nerve growth factor (NGF).

251 p-regulation of the growth-promoting factor, nerve growth factor (NGF).

252 found to express the following receptors for nerve growth factor (NGF): neurotrophic receptor tyrosin

253 (P<0.0001) and cells positively staining for nerve growth factor (NGF; P<0.0001) in the infarcted bra

254 of brain-derived neurotrophic factor (BDNF), nerve-growth factor (NGF), neurotrophin-3 (NT-3), fibrob

255 brain-derived neurotrophic factor (BDNF) and nerve-growth factor (NGF).

256 er, culture in serum-free media, presence of nerve growth factor, or addition of pituitary adenylate

257 t or the inhibition of adrenergic receptors, nerve growth factor, or brain-derived neurotrophic facto

258 Reactivation can be induced by depletion of nerve growth factor; other commonly used reactivation st

259 Significant examples include the nerve growth factor (P = 7.86 x 10(-33)), epidermal grow

260 t from lactate release and activation of pro-nerve growth factor-p75 receptor signalling are key comp

261 When treated with nerve growth factor, PC12 cells will differentiate over

262 Second, there was evidence of increased nerve growth factor production and dysregulation of the

263 orward signaling loop in which tumor-derived nerve growth factor promotes enteric tumor innervation,

264 The precursor of nerve growth factor (proNGF) has been described as a bio

265 We report that the precursor of nerve growth factor (proNGF) is overexpressed in prostat

266 pathway entails the proteins that mature pro-nerve growth factor (proNGF) to NGF and those that degra

267 ells showed intense immunoreactivity for pro-nerve growth factor (proNGF), neurotrophin receptor p75

268 lates expression of numerous genes including nerve growth factor receptor (NGFR), which becomes trans

269 asolateral expression of the apically sorted nerve growth factor receptor (NGFR, p75; extracellular a

270 antage of their restricted expression of the nerve growth factor receptor (p75) in conjunction with f

271 ell sorting based on their expression of the nerve growth factor receptor (p75), a surface marker for

272 he NTRK1 gene that encodes the high-affinity nerve growth factor receptor (TRKA protein).

273 hese (AG879) is a selective inhibitor of the nerve growth factor receptor and human epidermal growth

274 gy domain transcription factor RUNX1 and the nerve growth factor receptor TrkA.

275 re initially marked by the expression of the nerve growth factor receptor TrkA.

276 r hierarchical expression of CD271/p75/NGFR (nerve growth factor receptor) marks cells with enriched

277 sine phosphorylation sequences of either the nerve growth factor receptor, TrkA (tropomyosin receptor

278 We found that the high-affinity nerve growth factor receptor, TrkA, is down-regulated by

279 DHEA was previously shown to bind to the nerve growth factor receptor, tropomyosin-related kinase

280 Adult C57BL/6N and nerve growth factor receptor-deficient mice.

281 neurotrophic receptor tyrosine kinase 1 and nerve growth factor receptor.

282 rker PGP 9.5 and the Schwann cell marker p75 nerve growth factor receptor.

283 otic properties via mechanisms involving the nerve growth factor receptors (tropomyosin-related kinas

284 factor 1, and nerve growth factor and of the nerve growth factor receptors, tyrosine kinase receptor

285 Ephrins (EFNs), 8 semaphorins (SEMAs), and 2 nerve growth factor receptors.

286 Bone is richly innervated by nerve growth factor-responsive (NGF-responsive) tropomyo

287 neuroprotective treatment (recombinant human nerve growth factor, rh-NGF) predominantly targeting sec

288 nalyses implicated p75 neurotrophin receptor/nerve growth factor signaling and innate immune toll-lik

289 c convergence on gene mutations that disrupt nerve growth factor signaling, upon which sympathetic an

290 sary for PP2A/B'beta-mediated enhancement of nerve growth factor signaling.

291 cells, and enhanced neurite outgrowth after nerve growth factor stimulation.

292 that this death is not due to a reduction in nerve growth factor synthesis.

293 ons of brain-derived neurotrophic factor and nerve growth factor than NCSC-SCs.

294 ncluding prostaglandin E(2), bradykinin, and nerve growth factor, that reduce the threshold and incre

295 of the Trk receptors, which are activated by nerve growth factor, there are only two established phos

296 uction and dysregulation of the ratio of pro-nerve growth factor to mature nerve growth factor, favou

297 we report that EVT901 reduces binding of pro-nerve growth factor to p75 neurotrophin receptor, blocks

298 ers indicate that PLCbeta induced soon after nerve growth factor treatment associates with TRAX rathe

299 cts neuroprotection by neurotrophins such as nerve growth factor, which bind to p75NTR receptors on M

300 After nerve growth factor withdrawal, sympathetic axons derive