ライフサイエンスコーパス: nervosa

1 a and higher in women recovered from bulimia nervosa).

2 ium effect for ED, large effect for anorexia nervosa).

3 eatment of eating disorders such as anorexia nervosa.

4 es (0.30%), and 61 males (0.01%) had bulimia nervosa.

5 were found for anorexia nervosa and bulimia nervosa.

6 as a risk factor for arrhythmias in anorexia nervosa.

7 ge 1 and the Genetic Consortium for Anorexia Nervosa.

8 c dysfunction in the pathogenesis of bulimia nervosa.

9 ression, and the psychopathology of anorexia nervosa.

10 of psychoanalytic psychotherapy for bulimia nervosa.

11 al therapy (CBT) in the treatment of bulimia nervosa.

12 patients with, or recovering from, anorexia nervosa.

13 eir first admission to hospital for anorexia nervosa.

14 nd may be efficacious for youth with bulimia nervosa.

15 n anorexia nervosa and overeating in bulimia nervosa.

16 bulimia nervosa with no history of anorexia nervosa.

17 ing raise the risk for mortality in anorexia nervosa.

18 extremely restricted food intake in anorexia nervosa.

19 ental delay, autism, psychosis, and anorexia nervosa.

20 nervosa and promising for adolescent bulimia nervosa.

21 avoid weight gain that characterize bulimia nervosa.

22 flict resolution in adolescents with bulimia nervosa.

23 ty risks, similar to those found in anorexia nervosa.

24 linked to suppression of eating and anorexia nervosa.

25 ng the subtypes anorexia nervosa and bulimia nervosa.

26 ischarge criteria for patients with anorexia nervosa.

27 vosa were unlikely to cross over to anorexia nervosa.

28 a nervosa (AN) and other EDs such as bulimia nervosa.

29 placebo for adult outpatients with anorexia nervosa.

30 HRQoL) in patients with anorexia and bulimia nervosa.

31 eight and duration of amenorrhea in anorexia nervosa.

32 y, dieting-induced weight gain, and anorexia nervosa.

33 ivity to these effects of hunger in anorexia nervosa.

34 desensitized dopaminergic system in bulimia nervosa.

35 neural reward-effort integration in bulimia nervosa.

36 ive diseases, obesity, bulimia, and anorexia nervosa.

37 mong women might be up to 4%, and of bulimia nervosa 2%.

38 Crude mortality rates were 4.0% for anorexia nervosa, 3.9% for bulimia nervosa, and 5.2% for eating d

39 9; n(treatment-resistant depression/anorexia nervosa) = 76) to identify the neuroanatomical substrate

40 o groups: patients with anorexia and bulimia nervosa (ABN; n = 30) and control patients (CN; n = 30).

41 itive dose-response association with bulimia nervosa (adjusted hazard ratio = 1.15, 95% confidence in

42 dless of the solvent, the results suggest M. nervosa aerial extracts present a biological potential d

43 OH) and aqueous extracts from the Micromeria nervosa aerial parts, based on their antioxidant activit

44 Anorexia nervosa affects 1-4% of women in United States and is th

45 white matter fiber organization in anorexia nervosa after recovery could indicate a biological marke

46 Anorexia nervosa also has large and significant genetic correlati

47 The lifetime prevalence of DSM-5 anorexia nervosa among women might be up to 4%, and of bulimia ne

48 steroid metabolism in subjects with anorexia nervosa (AN) after weight gain have not been elucidated.

49 he science, with a primary focus on anorexia nervosa (AN) and binge-eating behavior, and encourages f

50 Patients with anorexia nervosa (AN) and obesity (OB) were investigated in refer

51 Both anorexia nervosa (AN) and obesity are complicated by affective co

52 Anorexia nervosa (AN) and obsessive-compulsive disorder (OCD) are

53 Diagnoses for anorexia nervosa (AN) and other eating disorders (OED: bulimia ne

54 een ADHD and various EDs, including anorexia nervosa (AN) and other EDs such as bulimia nervosa.

55 Anorexia nervosa (AN) and related eating disorders are complex, m

56 ity to reward, yet individuals with anorexia nervosa (AN) are not motivated to eat when starved.

57 rsuit of thinness, individuals with anorexia nervosa (AN) engage in maladaptive behaviors (restrictiv

58 Individuals with anorexia nervosa (AN) engage in relentless restrictive eating and

59 eight is disrupted in patients with anorexia nervosa (AN) for prolonged periods of time.

60 Anorexia nervosa (AN) is a complex and heritable eating disorder

61 Anorexia nervosa (AN) is a complex neuropsychiatric disorder pres

62 Anorexia nervosa (AN) is a condition of severe undernutrition ass

63 Anorexia nervosa (AN) is a devastating psychiatric illness that i

64 Anorexia nervosa (AN) is a serious disorder with high rates of mo

65 Anorexia nervosa (AN) is a serious eating disorder characterized

66 Anorexia nervosa (AN) is a serious eating disorder characterized

67 Anorexia nervosa (AN) is a serious mental illness categorized by

68 Anorexia nervosa (AN) is a severe psychiatric disorder associated

69 Anorexia nervosa (AN) is an eating disorder characterized by extr

70 Anorexia nervosa (AN) is an eating disorder observed predominantl

71 Anorexia nervosa (AN) is characterized by a persistent restrictio

72 Anorexia Nervosa (AN) is characterized by Diagnostic and Statisti

73 Anorexia nervosa (AN) is characterized by extremely low body weig

74 included in the dataset as follows: Anorexia Nervosa (AN) n = 171; Bulimia Nervosa (BN) n = 82; Recov

75 Is starvation in anorexia nervosa (AN) or overeating in bulimia nervosa (BN) a for

76 Individuals with anorexia nervosa (AN) override the drive to eat, forgoing immedia

77 been carried out on iron status in anorexia nervosa (AN) patients.

78 Individuals with anorexia nervosa (AN) restrict eating and become emaciated.

79 me-wide association study (GWAS) of anorexia nervosa (AN) using a stringently defined phenotype.

80 ned tolerance to self-starvation in anorexia nervosa (AN), a hypothalamic dysregulation of energy and

81 vely correlate with future onset of anorexia nervosa (AN), bulimia nervosa (BN), binge eating disorde

82 suggested in the pathophysiology of anorexia nervosa (AN), but the role of the endogenous mediators o

83 ations for eating disorders such as anorexia nervosa (AN), in which there is a high prevalence of anx

84 tion has previously been studied in anorexia nervosa (AN), its influence in women with AN on eating d

85 In anorexia nervosa (AN), taste and smell are believed to be anhedon

86 one of the core characteristics of anorexia nervosa (AN), the exact nature of this complex feature i

87 e psychiatric conditions, including anorexia nervosa (AN).

88 ex eating-related behaviors seen in anorexia nervosa (AN).

89 to inpatient medical treatment for anorexia nervosa (AN).

90 e distortions are a core feature of anorexia nervosa (AN).

91 cal activity is a common feature of anorexia nervosa (AN).

92 d genetic correlations (rg) between anorexia nervosa and 159 other phenotypes.

93 sponse in 18 female adolescents with bulimia nervosa and 18 healthy female age-matched subjects durin

94 4.37 (95% CI=2.4-7.3) for lifetime anorexia nervosa and 2.33 (95% CI=0.3-8.4) for bulimia nervosa wi

95 one adolescent female patients with anorexia nervosa and 45 age- and sex-matched healthy volunteers p

96 ssociation study of 16,992 cases of anorexia nervosa and 55,525 controls, identifying eight significa

97 the most established treatments for bulimia nervosa and binge eating disorder, with stepped-care app

98 NA methylation across the genome of anorexia nervosa and binge-eating disorder patients.

99 correlations were observed between anorexia nervosa and body mass index, insulin, glucose, and lipid

100 el, 5-year recovery rates for DSM-5 anorexia nervosa and bulimia nervosa are 69 and 55%, respectively

101 Both anorexia nervosa and bulimia nervosa are associated with increase

102 Anorexia nervosa and bulimia nervosa are common and severe eating

103 ort the longitudinal distinction of anorexia nervosa and bulimia nervosa but do not support the anore

104 lumes were reduced in the recovered anorexia nervosa and bulimia nervosa groups and predicted sensiti

105 Course and outcome studies of both anorexia nervosa and bulimia nervosa show that no significant dif

106 gnostic crossover longitudinally in anorexia nervosa and bulimia nervosa to inform the validity of th

107 Participants who had recovered from anorexia nervosa and bulimia nervosa were studied to avoid confou

108 DNOS, by lowering the threshold for anorexia nervosa and bulimia nervosa, and adding BED as a specifi

109 ected by the diagnostic criteria of anorexia nervosa and bulimia nervosa, which emphasize intrapsychi

110 Similar patterns were found for anorexia nervosa and bulimia nervosa.

111 with an ED, including the subtypes anorexia nervosa and bulimia nervosa.

112 clarifications and modifications to anorexia nervosa and bulimia nervosa; and the inclusion of binge

113 a genome-wide association study of anorexia nervosa and calculated genetic correlations with a serie

114 d mortality ratios were elevated for bulimia nervosa and eating disorder not otherwise specified.

115 tios were significantly elevated for bulimia nervosa and eating disorder not otherwise specified; sui

116 roup (lower in women recovered from anorexia nervosa and higher in women recovered from bulimia nervo

117 re on the development and course of anorexia nervosa and interpreted critical features in light of de

118 stablished treatment for youth with anorexia nervosa and may be efficacious for youth with bulimia ne

119 less likely to exhibit anorexia and bulimia nervosa and more likely to exhibit binge eating disorder

120 f the lumbar spine in patients with anorexia nervosa and normal-weight control subjects and to determ

121 Women with anorexia nervosa and normal-weight control subjects were compared

122 o significantly associated SNPs for anorexia nervosa and only three for educational attainment.

123 contribute to restricted eating in anorexia nervosa and overeating in bulimia nervosa.

124 : stabilization of the incidence of anorexia nervosa and possibly lower incidence rates of bulimia ne

125 first-line treatment for adolescent anorexia nervosa and promising for adolescent bulimia nervosa.

126 increased on the right side in the anorexia nervosa and recovered anorexia nervosa groups and on the

127 nclude genetic correlations between anorexia nervosa and schizophrenia, anorexia and obesity, and edu

128 correlations were observed between anorexia nervosa and schizophrenia, neuroticism, educational atta

129 s are engaged in the development of anorexia nervosa and that stimulus-response learning (that is, ha

130 against schizophrenia, autism, and anorexia nervosa and that these variants may be maintained by new

131 deficit/hyperactivity disorder, and anorexia nervosa) and 17 nonpsychiatric traits in more than 10,00

132 s (0.70%) and 453 males (0.04%) had anorexia nervosa, and 3349 females (0.30%), and 61 males (0.01%)

133 4.0% for anorexia nervosa, 3.9% for bulimia nervosa, and 5.2% for eating disorder not otherwise spec

134 e threshold for anorexia nervosa and bulimia nervosa, and adding BED as a specified eating disorder.

135 lence estimates of anorexia nervosa, bulimia nervosa, and binge-eating disorder were 0.3%, 0.9%, and

136 y 3 variables (any eating disorder, anorexia nervosa, and bulimia nervosa) identified by any lifetime

137 determine whether anorexia nervosa, bulimia nervosa, and eating disorder not otherwise specified are

138 followed them for anorexia nervosa, bulimia nervosa, and eating disorder not otherwise specified unt

139 omization identifies schizophrenia, anorexia nervosa, and higher education as causal for decreased fa

140 ndrome, inflammatory bowel disease, anorexia nervosa, and intestinal pseudo-obstruction.

141 ass procedures, chronic alcoholics, anorexia nervosa, and restrictive diets.

142 odifications to anorexia nervosa and bulimia nervosa; and the inclusion of binge eating disorder as a

143 rates for DSM-5 anorexia nervosa and bulimia nervosa are 69 and 55%, respectively; little is known ab

144 Both anorexia nervosa and bulimia nervosa are associated with increased mortality.

145 Anorexia and bulimia nervosa are associated with poor periodontal condition,

146 Anorexia nervosa and bulimia nervosa are common and severe eating disorders (EDs) of

147 eatment trials for adolescents with anorexia nervosa are few.

148 encourage a reconceptualization of anorexia nervosa as a metabo-psychiatric disorder.

149 dieting behavior characteristic of anorexia nervosa as a well-entrenched habit provides a basis for

150 the same degree in adolescents with bulimia nervosa as in healthy comparison subjects.

151 We also found an enrichment of anorexia nervosa associated genes in the adult and fetal raphe an

152 ivity disorder, alcohol dependence, anorexia nervosa, autism spectrum disorder, bipolar disorder, maj

153 romote eating, yet individuals with anorexia nervosa avoid food despite emaciation.

154 ted eating and weight loss occur in anorexia nervosa because of a failure to accurately recognize hun

155 Women with bulimia nervosa before and during pregnancy and those with binge

156 ew categories are: anorexia nervosa, bulimia nervosa, binge eating disorder, and eating disorder not

157 iagnostic systems: anorexia nervosa, bulimia nervosa, binge eating disorder, avoidant-restrictive foo

158 behaviors (i.e., bulimia nervosa or anorexia nervosa, binge eating/purging type);14 with anorexia ner

159 AN) and other eating disorders (OED: bulimia nervosa, binge-eating disorder, and eating disorder not

160 orexia nervosa (AN) or overeating in bulimia nervosa (BN) a form of addiction?

161 are presented here and compared with bulimia nervosa (BN) data in the World Health Organization (WHO)

162 Bulimia nervosa (BN) has been associated with dysregulation of t

163 Bulimia nervosa (BN) is characterized by dysregulated intake of

164 lows: Anorexia Nervosa (AN) n = 171; Bulimia Nervosa (BN) n = 82; Recovered AN n = 90; Healthy contro

165 ture onset of anorexia nervosa (AN), bulimia nervosa (BN), binge eating disorder (BED), and purging d

166 cuits in adolescents and adults with bulimia nervosa (BN), but less is known about the microstructure

167 scent and adult female subjects with bulimia nervosa (BN).

168 Lifetime prevalence estimates of anorexia nervosa, bulimia nervosa, and binge-eating disorder were

169 in 1975-1998 and followed them for anorexia nervosa, bulimia nervosa, and eating disorder not otherw

170 this study was to determine whether anorexia nervosa, bulimia nervosa, and eating disorder not otherw

171 e probable four new categories are: anorexia nervosa, bulimia nervosa, binge eating disorder, and eat

172 w recognised in diagnostic systems: anorexia nervosa, bulimia nervosa, binge eating disorder, avoidan

173 d little evidence that the risk for anorexia nervosa, bulimia nervosa, or disordered eating was assoc

174 distinction of anorexia nervosa and bulimia nervosa but do not support the anorexia nervosa subtypin

175 ly-to-severely ill adolescents with anorexia nervosa, but it is costly, and the risks of relapse and

176 on weight in adult outpatients with anorexia nervosa, but no significant benefit for psychological sy

177 nderstanding of the neurobiology of anorexia nervosa by suggesting disturbances in subcortical appeti

178 in which the marked persistence of anorexia nervosa can be usefully understood as a well-ingrained m

179 Anorexia and bulimia nervosa can have significant effects on oral health.

180 se-control cohorts comprising 3,495 anorexia nervosa cases and 10,982 controls, the authors performed

181 from an induced stem cell study of anorexia nervosa cases are expressed at higher levels in the late

182 brain reward learning in adolescent anorexia nervosa changes with weight restoration.

183 unger signals, whereas overeating in bulimia nervosa could represent an exaggerated perception of hun

184 s with chronic treatment-refractory anorexia nervosa, DBS is well tolerated and is associated with si

185 o resolve conflict, adolescents with bulimia nervosa displayed abnormal patterns of activation in fro

186 Patients with anorexia nervosa exhibit abnormal myocardial repolarization and a

187 I=3.2-12.1) for those with lifetime anorexia nervosa for >15 to 30 years (10/67 died).

188 0.9-8.3) for patients with lifetime anorexia nervosa for 0 to 15 years (4/119 died), and 6.6 (95% CI=

189 le patients (aged 11-18 years) with anorexia nervosa from six centres in Germany.

190 Here we combine data from the Anorexia Nervosa Genetics Initiative (ANGI)(8,9) and the Eating D

191 tional connectivity in the remitted anorexia nervosa group compared with the control group.

192 ompared with the control group, the anorexia nervosa group exhibited greater brain response 1) for pr

193 a groups and on the left side in the bulimia nervosa group relative to the healthy comparison group.

194 the control group and the remitted anorexia nervosa group, with an increase and a decrease, respecti

195 among participants in the remitted anorexia nervosa group.

196 the anorexia nervosa and recovered anorexia nervosa groups and on the left side in the bulimia nervo

197 n the recovered anorexia nervosa and bulimia nervosa groups and predicted sensitivity to reward in al

198 Results Women with anorexia nervosa had higher skewness and kurtosis, lower MPP (P <

199 Conclusion Patients with anorexia nervosa had increased skewness and kurtosis and decrease

200 a for DSM-5 binge eating disorder or bulimia nervosa, had 12 months of continuous health care enrollm

201 Although anorexia nervosa has a high mortality rate, our understanding of

202 As bulimia nervosa has been associated with a dysregulated dopamine

203 Anorexia nervosa has been consistently associated with increased

204 Anorexia nervosa has the highest mortality rate of any psychiatri

205 l large population-based studies of anorexia nervosa have been conducted in twins; it is possible tha

206 patients suffering from restrictive anorexia nervosa have enhanced habit formation compared with heal

207 ting disorder, anorexia nervosa, and bulimia nervosa) identified by any lifetime diagnoses recorded i

208 efulness of family interventions for bulimia nervosa in adolescents.

209 evidence base for effectiveness for anorexia nervosa in adolescents.

210 nd possibly lower incidence rates of bulimia nervosa in Caucasian North American and Northern Europea

211 increasing rates of binge eating and bulimia nervosa in Hispanic and Black American minority groups i

212 subtype predicted fatal outcome for anorexia nervosa in males.

213 had an independent association with anorexia nervosa in males.

214 s (n = 13), focusing on anorexia and bulimia nervosa in very small samples with considerable sample o

215 sses two trials of individuals with anorexia nervosa in which deep brain stimulation of different bra

216 primarily by a low body-mass index, anorexia nervosa is a complex and serious illness(1), affecting 0

217 Anorexia nervosa is a complex eating disorder with genetic, metab

218 Anorexia nervosa is a complex heritable phenotype for which this

219 Anorexia nervosa is a life-threatening illness.

220 Anorexia nervosa is a psychiatric disorder of unknown etiology.

221 Anorexia nervosa is a severe psychiatric disorder associated with

222 Anorexia nervosa is a well characterized disorder with remarkable

223 Anorexia nervosa is an important cause of physical and psychosoci

224 A core feature of anorexia nervosa is an over-estimation of body size.

225 Anorexia nervosa is prevalent in adolescents and young adults, an

226 ng the anorexia nervosa subtypes and bulimia nervosa may reflect problems with the validity of the cu

227 The study included 30 women with anorexia nervosa (mean age +/- standard deviation, 26 years +/- 6

228 depression, suicide, schizophrenia, anorexia nervosa, migraine, dementia, and PD.

229 The genetic architecture of anorexia nervosa mirrors its clinical presentation, showing signi

230 and conference abstracts addressing anorexia nervosa (n = 13), bulimia nervosa (n = 6), and binge-eat

231 ddressing anorexia nervosa (n = 13), bulimia nervosa (n = 6), and binge-eating disorder (n = 1), publ

232 women (N=14), women recovered from anorexia nervosa (N=14) had significantly diminished and women re

233 diminished and women recovered from bulimia nervosa (N=14) had significantly elevated hemodynamic re

234 led trial of adult outpatients with anorexia nervosa (N=152, 96% of whom were women; the sample's mea

235 25 years in 1,885 individuals with anorexia nervosa (N=177), bulimia nervosa (N=906), or eating diso

236 anorexia nervosa (N=24), women with bulimia nervosa (N=19), and healthy comparison women (N=24).

237 women with current restricting-type anorexia nervosa (N=19), women recovered from restricting-type an

238 METHOD: Female adolescents with anorexia nervosa (N=21; mean age, 16.4 years [SD=1.9]) underwent

239 men recovered from restricting-type anorexia nervosa (N=24), women with bulimia nervosa (N=19), and h

240 duals with anorexia nervosa (N=177), bulimia nervosa (N=906), or eating disorder not otherwise specif

241 aths (6.5%) were recorded (lifetime anorexia nervosa, N=14; bulimia nervosa with no history of anorex

242 bulimia nervosa with no history of anorexia nervosa, N=2).

243 For adults with anorexia nervosa, no one specialist treatment has been shown to b

244 rrelation between the Eyes Test and anorexia nervosa, openness (NEO-Five Factor Inventory), and diffe

245 eating and purging behaviors (i.e., bulimia nervosa or anorexia nervosa, binge eating/purging type);

246 dies of adolescents and adults with anorexia nervosa or bulimia nervosa that, together, implicate dys

247 al of 216 women with a diagnosis of anorexia nervosa or bulimia nervosa were followed for 7 years; we

248 atment-seeking female patients with anorexia nervosa or bulimia nervosa were interviewed every 6 mont

249 In humans with anorexia nervosa or kwashiorkor, ghrelin and growth hormone are k

250 , 0.8% had partial- or full-criteria bulimia nervosa or purging disorder and 2.9% had partial or full

251 that the risk for anorexia nervosa, bulimia nervosa, or disordered eating was associated with zygosi

252 EDs: OR = 4.66, 95% CI = 4.47, 4.87; bulimia nervosa: OR = 5.01, 95% CI = 4.63, 5.41) and their relat

253 death among patients with lifetime anorexia nervosa peaked within the first 10 years of follow-up, r

254 Adolescents with and without bulimia nervosa performed similarly on the task.

255 ularly for adolescent patients with anorexia nervosa, point to the benefits of specialised family-bas

256 Of the five male anorexia nervosa probands, only one was from an opposite-sex twin

257 conducted in which 70 patients with bulimia nervosa received either 2 years of weekly psychoanalytic

258 cerebral glucose metabolism in key anorexia nervosa-related structures at both 6 months and 12 month

259 The pathophysiology of anorexia nervosa remains obscure, but structural brain alteration

260 20-60 years and had a diagnosis of anorexia nervosa (restricting or binge-purging subtype) and a dem

261 binge eating/purging type);14 with anorexia nervosa, restricting type; and 13 healthy comparison sub

262 Research in Anorexia Nervosa (RIAN) is a 2-group (FBT and SyFT) randomized tr

263 -based interventions for addiction, anorexia nervosa, schizophrenia, and depression.

264 Two patterns emerge: (1) anorexia nervosa, schizophrenia, obsessive-compulsive disorder, a

265 dolescent patients with non-chronic anorexia nervosa seems no less effective than IP for weight resto

266 studies of both anorexia nervosa and bulimia nervosa show that no significant differences exist betwe

267 Anorexia nervosa shows a stronger genetic correlation with body f

268 anxiety, affective regulation, and anorexia nervosa-specific behaviours at 12 months after surgery,

269 a combination of re-nourishment and anorexia nervosa-specific psychotherapy is most effective.

270 r BMI at admission, and restrictive anorexia nervosa subtype predicted fatal outcome for anorexia ner

271 of diagnostic "crossover" among the anorexia nervosa subtypes and bulimia nervosa may reflect problem

272 imia nervosa but do not support the anorexia nervosa subtyping schema.

273 Similar results were found for anorexia nervosa (suicide attempts: crude, 4.42 [4.12-4.74] vs ad

274 ] vs adjusted, 2.67 [1.78-4.01]) and bulimia nervosa (suicide attempts: crude, 6.26 [5.73-6.85] vs ad

275 Brain circuits believed to drive anorexia nervosa symptoms can be accessed with surgical technique

276 represent a phenotype of adolescent anorexia nervosa that does not respond well to treatment.

277 te information about bone health in anorexia nervosa that is independent of that provided with bone m

278 and adults with anorexia nervosa or bulimia nervosa that, together, implicate dysregulation of front

279 eat is crucial for survival, but in anorexia nervosa, the brain persistently supports reduced food in

280 ward-processing deficits in remitted bulimia nervosa, the purpose of this study was to identify the r

281 s from the largest genetic study of anorexia nervosa to date were enriched for expression in the arcu

282 ngitudinally in anorexia nervosa and bulimia nervosa to inform the validity of the DSM-IV-TR eating d

283 del helps explain the resistance of anorexia nervosa to interventions that have established efficacy

284 26 women who were in remission from anorexia nervosa (to avoid the confounding effects of malnutritio

285 Adults with anorexia nervosa too have a realistic chance of achieving recover

286 The h(2)SNP of anorexia nervosa was 0.20 (SE=0.02), suggesting that a substantia

287 Anorexia nervosa was independently predicted by multiple birth (a

288 , the prevalence of DSM-IV or broad anorexia nervosa was not significantly different than that of wom

289 patients with treatment-refractory anorexia nervosa were enrolled between September, 2011, and Janua

290 h a diagnosis of anorexia nervosa or bulimia nervosa were followed for 7 years; weekly eating disorde

291 le patients with anorexia nervosa or bulimia nervosa were interviewed every 6 months for a median of

292 recovered from anorexia nervosa and bulimia nervosa were studied to avoid confounding effects of alt

293 Women with bulimia nervosa were unlikely to cross over to anorexia nervosa.

294 nsiveness is elevated in adolescent anorexia nervosa when underweight and after weight restoration.

295 tic criteria of anorexia nervosa and bulimia nervosa, which emphasize intrapsychic conflicts such as

296 o understanding the neurobiology of anorexia nervosa, which still remains a mystery and poses a chall

297 authors found that individuals with anorexia nervosa, who make maladaptive food choices to the point

298 ed (lifetime anorexia nervosa, N=14; bulimia nervosa with no history of anorexia nervosa, N=2).

299 ervosa and 2.33 (95% CI=0.3-8.4) for bulimia nervosa with no history of anorexia nervosa.

300 he progress of genomic discovery in anorexia nervosa, with the identification of the first genome-wid