ライフサイエンスコーパス: nervous

1 ink between chronically elevated sympathetic nervous activity and hypoxia-induced vascular dysfunctio

2 oxia is associated with elevated sympathetic nervous activity and reduced vascular function in lowlan

3 We describe a lethal combined nervous and reproductive systems disease in three affect

4 nts in physiology and disease, including the nervous, cardiovascular and immune systems, skeletal mus

5 deaths from congenital malformations of the nervous (NO2, 1.525 [1.179, 1.974; p = 0.001]; PM10, 1.4

6 Keywords: Abdomen/GI, Cardiac, Infection, Nervous-Peripheral.

7 , and shortening of long bones), and enteric nervous system (aganglionosis).

8 holds, tone response, compliance), autonomic nervous system (baroreceptor sensitivity and effectivene

9 In the nilotinib group, central nervous system (CNS) amyloid burden was significantly re

10 tion of the immune system within the central nervous system (CNS) and altered the paradigm for succes

11 rebrospinal fluid (CSF) protects the central nervous system (CNS) and analyzing CSF aids the diagnosi

12 ive analyses of CAG expansion in ~50 central nervous system (CNS) and peripheral postmortem tissues f

13 HIV-1 reservoirs in the central nervous system (CNS) are challenging to address due to l

14 chived tissue samples from the human central nervous system (CNS) are currently available in brain ba

15 endocrine hormone synthetized in the central nervous system (CNS) as well as enterochromaffin cells o

16 The central nervous system (CNS) barriers are crucial interfaces bet

17 Central nervous system (CNS) blood vessels contain a functional

18 igand/receptor pair has key roles in central nervous system (CNS) development, mediating axonal, and

19 isability and disease progression in central nervous system (CNS) diseases such as amyotrophic latera

20 discoveries of their involvement in central nervous system (CNS) disorders, and in particular in Alz

21 tic programs that assemble the human central nervous system (CNS) during development and maintain its

22 Loss of central nervous system (CNS) FAO did not result in gross neuroan

23 t virus dissemination throughout the central nervous system (CNS) following many neurotropic virus in

24 Inflammation in the central nervous system (CNS) has been linked to demyelination an

25 The adult human central nervous system (CNS) has very limited regenerative capab

26 the AIM2 inflammasome contributes to central nervous system (CNS) homeostasis specifically through it

27 Disease involved the central nervous system (CNS) in 71 subjects (49%).

28 udy the role of myeloid cells in the central nervous system (CNS) in the pathogenesis of multiple scl

29 apeutic value of mTOR inhibitors for central nervous system (CNS) indications.

30 Central nervous system (CNS) infections cause substantial morbid

31 ver, the transcriptomic landscape of central nervous system (CNS) innate immune cells contributing to

32 Leukocyte entry into the central nervous system (CNS) is essential for immune surveillanc

33 Over time, the central nervous system (CNS) loses function and regenerative cap

34 ived monocytes, other non-microglial central nervous system (CNS) macrophage subtypes include border-

35 owing clinical radiotherapy (RT) for central nervous system (CNS) malignancies are often long-lasting

36 repair-proficient cancers, including central nervous system (CNS) malignancies.

37 therefore a candidate of interest in central nervous system (CNS) myelination and remyelination, and

38 neurons expressing PSST mRNAs in the central nervous system (CNS) of Scyliorhinus canicula using in s

39 IFNs control MuPyV infection in non-central nervous system (CNS) organs, but their relative contribu

40 ary conversion is implemented in the central nervous system (CNS) remains poorly understood.

41 r is a major impediment for targeted central nervous system (CNS) therapeutics, especially with carbo

42 in metazoans, but how they enter the central nervous system (CNS) through the blood-brain barrier (BB

43 (CSF) flow dynamics in children with central nervous system (CNS) tumors before intraventricular ther

44 hibitory neurotransmitter within the central nervous system (CNS) with fast, transsynaptic, and modul

45 nt inflammatory tissue damage in the central nervous system (CNS), and none directly promote repair.

46 d roles that these cells play in the central nervous system (CNS), and this work has been greatly fac

47 e the most abundant cell type in the central nervous system (CNS), performing complex functions in he

48 sted to be present in the cephalopod central nervous system (CNS), Scaros, Croll, and Baratte only re

49 y role in synaptic plasticity in the central nervous system (CNS).

50 curring in the persistently inflamed central nervous system (CNS).

51 a fatal demyelinating disease of the central nervous system (CNS).

52 re the resident myeloid cells in the central nervous system (CNS).

53 interactions with other cells in the central nervous system (CNS).

54 eservoirs in vivo, especially in the central nervous system (CNS).

55 lia, the resident macrophages of the central nervous system (CNS).

56 and during axon regeneration in the central nervous system (CNS).

57 n development and pathologies of the Central Nervous System (CNS); however, their coordinating mechan

58 ne results in diffuse central and peripheral nervous system (CNS, PNS) demyelination.

59 The enteric nervous system (ENS) coordinates essential intestinal fu

60 The enteric nervous system (ENS) exists in close proximity to lumina

61 f two main human tau isoforms in the enteric nervous system (ENS) in CD but not in UC.

62 The enteric nervous system (ENS) is a complex network constituted of

63 equilibrium, here, we show that the enteric nervous system (ENS) plays an essential and non-redundan

64 an important role in the development of the nervous system (p = 4.29 x 10(-11) with neuroticism), as

65 w cholesterol/lipid metabolism in peripheral nervous system (PNS) contributes to the pathogenesis of

66 lerosis, and Parkinson's disease, peripheral nervous system (PNS) disorders such as chemotherapy-indu

67 ) is used to describe the sensory peripheral nervous system (PNS) in the tail of a cephalochordate, A

68 ation on axon regeneration in the peripheral nervous system (PNS) is the slow rate of regrowth.

69 Previous studies using cultured peripheral nervous system (PNS) neurons have demonstrated that KIF1

70 derstanding of other cells in the peripheral nervous system (PNS) remains limited.

71 the formation and function of the peripheral nervous system (PNS).

72 ological conditions affecting the peripheral nervous system (PNS).

73 nerve pathway between the colon and central nervous system (spinal cord and brain) that underlies th

74 , 0.5 to 0.9 per 100 000 persons for central nervous system abscesses, and 24.4 to 32.9 per 100 000 p

75 at have previously been proposed to modulate nervous system activity and behaviours of their hosts(2,

76 c chemoreceptor-driven levels of sympathetic nervous system activity and respiratory drive.

77 ral hemodynamics and the effect of autonomic nervous system activity on these signals has received le

78 s included fatigue, subjective muscular, and nervous system AEs.

79 which the immune system affects the central nervous system after surgical trauma.

80 ker is released immediately from the central nervous system after TBI.

81 in-2 that is highly expressed in the central nervous system and associated with vesicles in neurons.

82 oreflex, thereby stimulating the sympathetic nervous system and causing hypertension.

83 s, our understanding of how neurons form the nervous system and enable function is very fragmentary,

84 ng the function of the sensory and autonomic nervous system and endo- and exocrine glands.

85 Recent evidence has implicated the nervous system and glial family ligands (GFLs) as potent

86 F's role in translational homeostasis in the nervous system and implicate RQC dysfunction in causing

87 l type-specific alternative exons across the nervous system and leverage ENCODE and GTEx data sets to

88 ly resulting in rapid necrosis of the entire nervous system and muscle cells throughout the entire or

89 c studies of the role of nitric oxide in the nervous system and other organs.

90 ch is clinically manifested by damage to the nervous system and retinal degeneration.

91 n signals from the peripheral to the central nervous system and that exerts its effects on neurons by

92 nal communication system between the central nervous system and the gastrointestinal tract, especiall

93 Neural circuits in the adult nervous system are characterized by stable, cell type-sp

94 sms affecting the development of the central nervous system are poorly understood.

95 icity, but the effects on other cells of the nervous system are unknown.

96 is imperative for the robust function of the nervous system as a whole, we tested whether activity-de

97 hesis must be finely tuned in the developing nervous system as the final essential step of gene expre

98 i35-Bregs suppress central nervous system autoimmune diseases by inducing infectiou

99 Adult mammalian central nervous system axons have intrinsically poor regenerativ

100 Central nervous system B cells have several potential roles in m

101 ncompetent sensory neurons in the peripheral nervous system but also allowed temporal tuning and prop

102 g animal Trichoplax adhaerens, which lacks a nervous system but possesses single gene homologues for

103 N-glycan alterations in the nervous system can result in different neuropathological

104 ffective interventions for adult non-central nervous system cancer patients to manage cancer-related

105 wing cranial radiotherapy to control central nervous system cancers.

106 ing (scRNA-seq) data from 727 peripheral and nervous system cell types spanning 17 mouse organs with

107 soforms that are highly expressed in central nervous system cells.

108 ng protein of 43 kDa (TDP-43) in the central nervous system characterizes frontotemporal dementia and

109 DHA is a key central nervous system constituent and the precursor of several

110 ssue types present in metazoans, the central nervous system contains some of the highest levels of al

111 el below 1 nM, may lead to liver and central nervous system damages in humans and animals, while exis

112 Genes encoding cell-surface proteins control nervous system development and are implicated in neurolo

113 se observations indicate a role of CG4266 in nervous system development and function and support the

114 hat STRAP preferably targets transcripts for nervous system development and regulates AS through pref

115 However, the mechanisms of CHD7 function in nervous system development are not well understood.

116 higher or lower in PVEC vs. ABEC identified "Nervous system development" and "Response to Stress" as

117 DARs in vertebrates, and hence their role in nervous system development, due to experimental limitati

118 eft-right axis specification; components for nervous system development, including a suite of G-prote

119 ed laminin-related protein, is essential for nervous system development.

120 though Alzheimer's disease (AD) is a central nervous system disease and type 2 diabetes MELLITUS (T2D

121 dverse events, the most common of which were nervous system disorders (four [3%]) and cardiac disorde

122 erious treatment-related adverse events were nervous system disorders (three [4%] of 68 patients and

123 1-propargyl-4-styrylpiperidines for central nervous system disorders.

124 onutrient underlie several diseases, notably nervous system disorders.

125 that aberrant expression of fusogens in the nervous system disrupts neuronal individuality, which, i

126 eful for in vivo characterization of central nervous system drug candidates, neurodegenerative diseas

127 lia results in an overall compression of the nervous system due to integrin dysregulation, which caus

128 cell phenotypes that infiltrate the central nervous system during human immunodeficiency virus (HIV)

129 ses sympathetic neuron death and sympathetic nervous system dysfunction (dysautonomia).

130 al beta1 and beta4 spectrin show exacerbated nervous system dysfunction compared to mice lacking beta

131 sensory neurons in the Drosophila peripheral nervous system exhibit organ sparing at the level of arb

132 Despite the relevance of the peripheral nervous system for normal sensory and motor capabilities

133 gnaling, altered neurotransmitter control of nervous system function, and fundamental changes in beha

134 previously implicated in IL-17 signaling or nervous system function.

135 in the deep brain would advance the study of nervous system function.

136 ly shared between the peripheral and central nervous system glia, indicating common immunological fea

137 eminal advances in itch circuitry within the nervous system have intersected with discoveries in immu

138 The central nervous system hosts parenchymal macrophages, known as m

139 Astrocytes control multiple processes in the nervous system in health and disease.

140 Determining the cellular content of the nervous system in terms of cell types and the rules of t

141 ed with generating numbers that describe the nervous system in terms of the volumes, surfaces, length

142 ithout hearing loss, implicating the central nervous system in the generation of hyperacusis.

143 19 is also anticipated to take a toll on the nervous system in the long term.

144 ll other studied groups, is expressed in the nervous system in the sea anemone Nematostella vectensis

145 for development of the brain and peripheral nervous system in this vertebrate embryo model.

146 The sympathetic nervous system innervates peripheral organs to regulate

147 Autonomic nervous system involvement precedes the motor features o

148 te that the peripherally distributed octopus nervous system is a key site for signal processing and h

149 t these receptors' expression in the central nervous system is crucial for T cell recruitment and rea

150 The Drosophila nervous system is ensheathed by a layer of outer glial c

151 The sympathetic nervous system is essential for maintenance of cardiac f

152 The interaction of the immune system and the nervous system is known to play an important role in the

153 The developing nervous system is remarkably sensitive to environmental

154 against pathogens and tumours in the central nervous system is thought to be limited owing to the lac

155 We find that the nervous system learns from the relative speed difference

156 to provide a detailed description of central nervous system lesions in Leigh syndrome and their biolo

157 shape and reshape many regions of the adult nervous system long after the neurodevelopmental period.

158 in cancer progression, especially in central nervous system metastases.

159 of behavior, a complete understanding of the nervous system must incorporate glial cells.

160 for the fusion of cells-are expressed in the nervous system of different species under conditions of

161 tribution of Pax6 cells in the adult central nervous system of lungfishes, the closest living relativ

162 from individual cell clusters in the central nervous system of the marine mollusk Aplysia californica

163 iple organs, with the central and peripheral nervous system often affected.

164 s here on one direction - the effects of the nervous system on immunity.

165 become drug candidates for distinct central nervous system pathologies and possessing accentuated th

166 ronmental information processing, immune and nervous system pathways and up-regulation of genetic inf

167 in E- embryos were characterized by improper nervous system patterning of the usually carefully progr

168 landmark reports have demonstrated that the nervous system plays an active role in cancer initiation

169 hosphamide, systemic cytarabine, and central nervous system radiotherapy were not used.

170 d neurotrophic factor (GDNF) induces enteric nervous system regeneration in mouse models of HSCR.

171 ergic inputs originating from the peripheral nervous system regulate the inflammatory immune response

172 assessment of the emotional and sympathetic nervous system responding to real-life social experience

173 phenotype in key compartments of the central nervous system responsible for regulating feeding behavi

174 cine seeks to decode and modulate peripheral nervous system signals to obtain therapeutic control of

175 t DRG findings may be generalizable to other nervous system structures.

176 yn has acute systemic effects on the central nervous system such that structural and resting-state fu

177 ic processes are an integral part of central nervous system synapses(1,2); however, the molecular mec

178 ease (CMT) is a neuropathy of the peripheral nervous system that afflicts ~1:2500 people.

179 thin the extracellular matrix of the central nervous system that have generated an explosion of inter

180 malignant embryonal tumor of the sympathetic nervous system that is most commonly diagnosed in the ab

181 luorescence map for the entire hermaphrodite nervous system that resolves all neuronal identities.

182 .IMPORTANCE HSV-1 is a pathogen of the human nervous system that uses its own virus-encoded proteins

183 lay this information to areas of the central nervous system that, in turn, regulate gut physiology(4)

184 In many parts of the nervous system these patterns are established during dev

185 ll transcriptomic data from the entire mouse nervous system to systematically identify cell types und

186 rodites; rather, tra-1 also acts in the male nervous system to transiently suppress a sexual dimorphi

187 er axon outgrowth and synapse formation, the nervous system transitions to a stable architecture.

188 melanomas, and radiotherapy-related central nervous system tumors, which are associated with excess

189 or expressed within the developing and adult nervous system where it mostly functions to help maintai

190 s, a daunting task in the developing central nervous system where thousands of cell types are generat

191 ) pathology is confined within the autonomic nervous system with no motor features, but mouse models

192 (without clinical symptoms of damage to the nervous system) during the search for the cause of hepat

193 he blood-brain barrier and enter the central nervous system, activating neuroinflammatory responses a

194 ch signal that is provided by the adrenergic nervous system, and demonstrate that cell-intrinsic adre

195 reservoirs, with an emphasis on the central nervous system, and describe relevant new work in functi

196 ALT-1 is expressed broadly in the C. elegans nervous system, and neuronal IL-17-MALT-1 signaling regu

197 Their molluscan heritage, innovative nervous system, and specialized behaviors create a uniqu

198 the principal glial cells of the peripheral nervous system, are now considered to be important playe

199 ation of different structures of the central nervous system, as compared to rodents.

200 In the developing nervous system, axons navigate through complex terrains

201 regeneration following injury to the central nervous system, but the mechanism by which the membrane-

202 s communicate nutritional information to the nervous system, but whether they also relay signals from

203 n with a self-contained, autonomously acting nervous system, composed of 20 neurons that fall into 14

204 In a developing nervous system, endogenous electric field (EF) influence

205 ia, the resident immune cells of the central nervous system, engulf surplus neurons and synapses.

206 RV) emerges from influences of the autonomic nervous system, fetal body and breathing movements, and

207 merous cell types and regions of the central nervous system, has been difficult to study due to limit

208 major cell type found throughout the central nervous system, have general roles in the modulation of

209 lthough ammonia plays essential roles in the nervous system, in particular at glutamatergic synapses,

210 In the vertebrate nervous system, ions accumulate in diffusion-limited syn

211 ver, heart, skeletal muscle, and the central nervous system, its use in adipose tissue has been limit

212 d gene expression in, central and peripheral nervous system, liver, kidney and skeletal muscle.

213 Gap junctions are ubiquitous throughout the nervous system, mediating critical signal transmission a

214 ociated mRNA for three broad tissue classes (nervous system, muscle, and intestine) and two neuronal

215 cted delivery of SMN-enhancing agents to the nervous system, or stems from broader defects of the mot

216 Compared with the somatic nervous system, pharyngeal neurons both physically assoc

217 aptic activity in the central and peripheral nervous system, presynaptically and postsynaptically.

218 wann cells (SCs), the glia of the peripheral nervous system, protect injured axons by virtue of a dra

219 nd, given their widespread expression in the nervous system, raise the possibility of a general role

220 tion of kidney, vasculature, and sympathetic nervous system, recent experimental data suggest that im

221 in neurons, especially those of the central nervous system, remains unclear.

222 expression throughout the brain and central nervous system, strongest during early development, and

223 e preferentially expressed in the peripheral nervous system, suggesting that their inhibition might t

224 In the isolated nervous system, T neurons were inhibited during the cont

225 likely gene expression profiles for muscle, nervous system, tegument, oesophageal gland, parenchymal

226 The internalization of the central nervous system, termed neurulation in vertebrates, is a

227 organs: disseminated skin sites, the central nervous system, the heart and large joints.

228 g amyloid and tau pathology from the central nervous system, we hypothesized that cholinergic input o

229 s, mCH is found at CAC trinucleotides in the nervous system, where it is associated with transcriptio

230 acrophages and nociceptors in the peripheral nervous system, which may contribute to the sensitizatio

231 gene exert its effect on CAD through central nervous system-lifestyle risk factors.

232 aging ligand for mGluR2 in different central nervous system-related conditions.

233 on in distinct Cre driver lines designed for nervous system-specific recombination.

234 for structural and functional support of the nervous system.

235 ble phases of dSarm signaling in the injured nervous system.

236 tability is proposed to be widespread in the nervous system.

237 iles among various cell types of the central nervous system.

238 is the main metabolic pathway in the central nervous system.

239 ate scar-free healing in the adult mammalian nervous system.

240 are crucial steps in the development of the nervous system.

241 mediating regeneration in the adult central nervous system.

242 s critical for the normal functioning of the nervous system.

243 Nogo-A may exert its effects in the central nervous system.

244 ant adipose depots, skeletal muscle, and the nervous system.

245 r control and pain processing in the central nervous system.

246 on of this major component of the Drosophila nervous system.

247 he gastrointestinal tract, platelets and the nervous system.

248 terest in the anatomy of the mouse autonomic nervous system.

249 representation of information throughout the nervous system.

250 (NMDARs) play critical roles in the central nervous system.

251 nts showed metastatic disease in the central nervous system.

252 tes to temporal accumulation of TRA-1 in the nervous system.

253 lved in the early development of the central nervous system.

254 ed in heart rate regulation by the autonomic nervous system.

255 tigating speciation and the evolution of the nervous system.

256 d phosphor-tau, respectively, in the central nervous system.

257 omical features of the whip spiders' central nervous system.

258 gical features across different parts of the nervous system.

259 ed, given their critical role in the central nervous system.

260 lopment of autoimmune disease in the central nervous system.

261 COVID-19 poses a global threat to the entire nervous system.

262 bitory motor neurotransmitter in the enteric nervous system.

263 communication across different parts of the nervous system.

264 hood from primitive cells of the sympathetic nervous system.

265 (Syt2a) during development of the zebrafish nervous system.

266 ocytes in autoimmune diseases of the central nervous system.

267 the regenerative potential in the mammalian nervous system.

268 currently known about their function in the nervous system.

269 uidance and synaptic organisation within the nervous system.

270 tical in the development and function of the nervous system.

271 and executing experiments within the central nervous system.

272 m, gastro-intestinal system, skin as well as nervous system.

273 fast excitatory synaptic transmission in the nervous system.

274 cing all of the neurons and macroglia in the nervous system.

275 THIK-1 is strongly expressed in the central nervous system.

276 Ks) are essential for the development of the nervous system.

277 or assessing microbial interactions with the nervous system.

278 meostasis and characteristically afflict the nervous system.

279 into adulthood, leading to a highly ramified nervous system.

280 log as a suppressor of proteotoxicity in the nervous system.

281 Hospitalizations for endocarditis, central nervous system/spine infections, osteomyelitis, and sept

282 additional important function in the central nervous system: acting as a sensory network, they detect

283 MPH-220 provides a potential nervous-system-independent option to treat spasticity an

284 The extent to which nervous systems are sexually dimorphic and the cellular

285 ontribution of each somatostatin to specific nervous systems circuits and behaviors in this important

286 Nervous systems contain sensory neurons, local neurons,

287 Several features of the adult nervous systems develop in a "critical period" (CP), dur

288 ogically complex cells that exist throughout nervous systems of multiple species.

289 Insect nervous systems offer unique advantages for studying int

290 nt and function of the immune, metabolic and nervous systems through dynamic bidirectional communicat

291 e tissues of both the central and peripheral nervous systems was normalized, and the neurodegeneratio

292 nue toward functional restoration of injured nervous systems.

293 citatory responses in peripheral and central nervous systems.

294 the organization and evolution of vertebrate nervous systems.

295 abolism, and function of both the immune and nervous systems.

296 r targets in both the central and peripheral nervous systems.

297 within the vertebral canal, despite evident nervous tissue deterioration after Thiel embalming.

298 several lipids were significantly altered in nervous tissue during oxaliplatin-induced acute pain.

299 dolinium retention in central and peripheral nervous tissues (1.8-333.2 nmol Gd/g tissue).

300 se of protein recruitment from the cnidarian nervous to venom system.