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1 gnant meningiomas are a rare type of central nervous system tumor.
2 Meningiomas are the most common primary nervous system tumor.
3 and cellular pathogenesis of NF1-associated nervous system tumors.
4 g, and determining the prognosis for central nervous system tumors.
5 aches with radioligand therapies for central nervous system tumors.
6 that causes development of multiple types of nervous system tumors.
7 n the development and progression of central nervous system tumors.
8 nscriptional regulation in pediatric central nervous system tumors.
9 al in neurodegenerative diseases and central nervous system tumors.
10 pruritus is a rare manifestation of central nervous system tumors.
11 ost common and deadly of the primary central nervous system tumors.
12 mors, representing 15% to 25% of all central nervous system tumors.
13 nting for approximately one-fifth of central nervous system tumors.
14 r benefit in patients with malignant central nervous system tumors.
15 genetic changes in the evolution of central nervous system tumors.
16 for malignant rhabdoid and childhood central nervous system tumors.
17 inked to increased aggressiveness in several nervous system tumors.
18 inhibiting prosurvival mechanisms in central nervous system tumors.
19 evaluate the incidence of MSI in 206 central nervous system tumors.
20 ma is one of the most deadly primary central nervous system tumors.
21 d to the development of benign and malignant nervous system tumors.
22 in human gynecological, breast, and central nervous system tumors.
23 on and acquired genetic mutations in central nervous system tumors.
24 biology and management of pediatric central nervous system tumors.
25 romes are associated with the development of nervous system tumors.
26 te individuals), cause of death (eg, central nervous system tumor, 0.48 [SE, 0.08]; P < .001; vs anox
27 recursor [BCP] ALL), 44 (13.3%) with central nervous system tumors, 80 (24.1%) with solid tumors, and
28 CI, 0.39-0.99), but not with risk of central nervous system tumors (AHR, 0.96; 95% CI, 0.51-1.88) or
30 s represent approximately 30% of all central nervous system tumors and 80% of malignant brain tumors.
32 oss of Merlin, patients suffer from multiple nervous system tumors and die on average around age 40.
34 uated primary human NF1-deficient peripheral nervous system tumors and found that MNKs are activated
37 stic leukemia, Hodgkin lymphoma, and central nervous system tumors) and across chronological age grou
38 as constitute the second most common central nervous system tumor, and yet relatively little is known
39 aracterized by multiple benign and malignant nervous system tumors, and by learning defects in 45% of
40 re human hereditary neuropathies, peripheral nervous system tumors, and common degenerative diseases.
41 FI) is a common genetic disorder that causes nervous system tumors, and learning and memory defects i
43 onstrate that VHL disease-associated central nervous system tumors are composed of developmentally ar
48 ghly expressed in a panel of cell lines from nervous system tumors arising from glia, although expres
49 ingiomas are the most common primary central nervous system tumors, arising from arachnoid cap cells
50 uman neurofibromas, schwannomas, and various nervous system tumors associated with NF1 or NF2; midkin
52 As treatment with chemotherapy for central nervous system tumors becomes more successful, new measu
53 e among patients without significant central nervous system tumor burden and to describe the experime
54 ly in familial schwannomas and other central nervous system tumors, but also in malignant tumors unre
59 The 2021 World Health Organization central nervous system tumor classification is a major advance t
60 ty has been established in pediatric central nervous system tumors, epigenomic alterations at the cel
62 However, little is known about how central nervous system tumors guard against immune eradication.
63 as induction of humoral responses by central nervous system tumors has been reported to be minimal, w
65 iencing CRCI, including survivors of central nervous system tumors, Hodgkin lymphoma, testicular, and
66 tumor suppressor found to be mutated in most nervous system tumors; however, it is not mutated in gli
69 une responses against peripheral and central nervous system tumors; however, variable results on the
71 (GBM), the most common and malignant central nervous system tumor in humans, is highly proliferative
74 port that 14% of ptc+/- mice develop central nervous system tumors in the posterior fossa by 10 month
75 ned and novel approaches to modeling central nervous system tumors, including gliomas, meningiomas, m
76 matosis 1 (NF1), are prone to development of nervous system tumors, including neurofibromas and piloc
77 ome are prone to the development of multiple nervous system tumors, including optic pathway gliomas (
78 ion syndrome are prone to the development of nervous system tumors, including schwannomas and meningi
79 vation results in the development of central nervous system tumors, including schwannomas, meningioma
80 ndrome in which affected individuals develop nervous system tumors, including schwannomas, meningioma
81 detected in normal brain, malignant central nervous system tumors, including tumors metastatic to th
83 ess the role of N-myc in neurogenesis and in nervous system tumors, it was conditionally disrupted in
86 red to astrocytomas, the most common central nervous system tumor, little is known about the molecula
88 , -4.0 to 24.2] per 100 000) and sympathetic nervous system tumors (<5 cancer cases; hazard ratio, 7.
89 ns in gene expression found in human central nervous system tumors may actually contribute to the eti
91 Malignant glioma is the most common central nervous system tumor of adults and is associated with a
92 involving >2400 pediatric brain and central nervous system tumors of diverse histologies from the Ch
93 ed not only in schwannomas and other central nervous system tumors of NF2 patients but also in their
94 l age increased risk of leukemia and central nervous system tumors, older paternal age was not associ
95 (OR, 2.1; 95% CI, 1.8-2.5; P<.001), central nervous system tumors (OR, 1.7; 95% CI, 1.5-2.0; P<.001)
96 emia, neuroblastoma, retinoblastoma, central nervous system tumors, or renal or germ-cell tumors.
101 nd oligodendrogliomas, as well as peripheral nervous system tumors such as neurofibromas, schwannomas
104 em gliomas (BSG) are a rare group of central nervous system tumors that arise mostly in children and
108 uman Cancer Consortium convened a meeting on Nervous System Tumors to review recent advances and sugg
109 , which apply broadly to many or all central nervous system tumor types, as well as the addition, eli
111 cytosine modifications in pediatric central nervous system tumors, we utilize a multi-omic approach
112 melanomas, and radiotherapy-related central nervous system tumors, which are associated with excess
115 rade III spinal ependymomas are rare central nervous system tumors with poor prognoses and limited tr
116 ndymoma is a heterogeneous entity of central nervous system tumors with well-established molecular gr
117 an effective antitumor agent for non-central nervous system tumor xenografts with an excellent safety