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1  opposite directions can coexist in the same neural tissue.
2 racellular matrix is an integral part of the neural tissue.
3 ting highly unsaturated fatty acid (HUFA) in neural tissue.
4 mode is required for fossilization of labile neural tissue.
5 ryngeal secretions, cerebrospinal fluid, and neural tissue.
6 ole in both the damage and repair of injured neural tissue.
7  comprehensively map synaptic connections in neural tissue.
8 gical phenomena occurring within a volume of neural tissue.
9 livering of multiple signals to and from the neural tissue.
10 mplementations given the constraints of real neural tissue.
11  latter predominantly expresses in postnatal neural tissue.
12 n the adult mouse retina, a disease-relevant neural tissue.
13 an increase in VEGFR2 within the surrounding neural tissue.
14            Fossil arthropods rarely preserve neural tissue.
15 ition of the amyloid beta-peptide (Abeta) in neural tissue.
16 f extracted myelin lipids and those found in neural tissue.
17 educed proliferation, most strikingly in the neural tissue.
18 a basis for semi-automatic reconstruction of neural tissue.
19 t few have applied these techniques to human neural tissue.
20 f many diseases is the unique involvement of neural tissue.
21 irregularities arise at their interface with neural tissue.
22 depletion of the glutathione (GSx) stores in neural tissue.
23 nsion and shape and for autonomous CE of the neural tissue.
24 ng of neuronal structure and function within neural tissue.
25 e electrodes that come into contact with the neural tissue.
26  cells could mediate clearance of HSV-1 from neural tissue.
27 extracellular matrix and disperse within the neural tissue.
28 mportant roles in T cells, muscle, bone, and neural tissue.
29 nhibition of BMP results in the formation of neural tissue.
30 ulture" phenomenon and generalize to complex neural tissue.
31 ne cannot alter the normal fate of posterior neural tissue.
32  microvascular bed that feeds the underlying neural tissue.
33 der, and (c) surrounding spared but reactive neural tissue.
34 d in the cochlea, not in direct contact with neural tissue.
35 ity and the anterior-posterior patterning of neural tissue.
36 iophysical and the biochemical properties of neural tissue.
37 erials are not intrinsically similar to soft neural tissue.
38 iple foci observed at different locations in neural tissue.
39 dissemination by carrying the virus into the neural tissues.
40 ias for circRNA upregulation during aging in neural tissues.
41 oteins in the generation and organization of neural tissues.
42 with its reproducible performance in various neural tissues.
43 n in transcriptomic and proteomic studies of neural tissues.
44  region prevents UBE3A-ATS expression in non-neural tissues.
45 Su(Hw) is a repressor of neural genes in non-neural tissues.
46 re highly stable, and accumulate with age in neural tissues.
47 e broadly distributed in both neural and non-neural tissues.
48 rous proteoglycans found in skin, joints and neural tissues.
49 acity long speculated to reside in mammalian neural tissues.
50  shows IL-1R3 is preferentially expressed in neural tissues.
51 nsferase Vb (GnT-Vb), is highly expressed in neural tissues.
52 essed mainly in the dorsal neuroectoderm and neural tissues.
53 e the development of adjacent neural and non-neural tissues.
54 ndred genes containing long 3' extensions in neural tissues.
55 lating the migration and organization of non-neural tissues.
56 d to vertebrates, as lncRNAs are abundant in neural tissues.
57  modulation of selected cells within complex neural tissues.
58 t fish, accounting for the size reduction of neural tissues.
59 and maintenance of phospholipid membranes in neural tissues.
60         4) Fox-3 expression is restricted to neural tissues.
61 docrine cells as well as other endocrine and neural tissues.
62 uired for clearance of infectious virus from neural tissues.
63 1-enriched proteinaceous inclusions in their neural tissues.
64  heavily expressed in the brain and in human neural tissues.
65 serves metabolic functions in neural and non-neural tissues.
66 us tissue by silencing neuronal genes in non-neural tissues.
67 ment by inactivating murine Brca2 throughout neural tissues.
68 ell cycle, apoptosis, and differentiation in neural tissues.
69 domains, ideal for the environments found in neural tissues.
70 d persistence of elevated levels of virus in neural tissues.
71 void of toxins that might harm the sensitive neural tissues.
72 trinsic and microenvironmental properties of neural tissues.
73 , cardiac, skeletal muscle, liver, skin, and neural tissues.
74  RNAs, are known to be enriched in mammalian neural tissues.
75 attern of expression, with highest levels in neural tissues.
76 ssion of the neuronal ankyrin isoform in non-neural tissues.
77 esting that CFI might play a special role in neural tissues.
78 accurately replicate the microenvironment of neural tissues.
79 ion, demonstrating significant enrichment in neural tissues.
80  state-of-the-art approaches for bioprinting neural tissues.
81 atification patterns in the retina and other neural tissues.
82 n have long-term deleterious consequences to neural tissues.
83  via shortening reaction times and saving on neural tissue [8-16].
84   Due to the limited regenerative ability of neural tissue, a diverse set of biochemical and biophysi
85                                 Signals from neural tissue act on endothelial cells to stimulate bloo
86 of irreversible electroporation (IRE) on the neural tissues after ablation in the epidural space of t
87          Conclusion Gadolinium deposition in neural tissues after GBCA administration occurs in the a
88 I to suppress innate immune responses in non-neural tissues against Pseudomonas aeruginosa in Caenorh
89 ylation was defective in Nbs1(DeltaB/DeltaB) neural tissue, although apoptosis occurred normally.
90 nt SOD1 is known to accumulate in the IMS of neural tissue and cause mitochondrial dysfunction.
91 on to compensate for the loss of specialized neural tissue and function.
92 e screened for factors enriched in posterior neural tissue and identify spalt-like 4 (sall4), which i
93 rs the molecular and cellular composition of neural tissue and leads to glial scarring, which inhibit
94 r, the relationship between the integrity of neural tissue and neural function has not been previousl
95 UAS/Gal4 system to deplete CFI proteins from neural tissue and observe that in this condition, multip
96 estin-cre mice showed extensive gammaH2AX in neural tissue and p53 deficiency restored brain histolog
97 ion transmitted, and thus, the properties of neural tissue and principles of its organization into ci
98 eral tissues, which serve to isolate damaged neural tissue and restore barrier functions.
99 ulating neural activity, the regeneration of neural tissue and the delivery of bioactive molecules fo
100 en receptor, in goldfish (Carassius auratus) neural tissue and used reverse-transcription polymerase
101 esized that rs688 modulates LDLR splicing in neural tissues and associates with AD.
102 ' UTR sequences are selectively expressed in neural tissues and contain putative recognition motifs f
103 or both imaging large, cleared, and expanded neural tissues and high-speed, functional imaging in viv
104 nces the kinetics of reovirus replication in neural tissues and highlight a functional role for sialy
105 inimize the mechanical mismatch between soft neural tissues and implants and thereby improve long-ter
106 anscript and protein in the mouse retina and neural tissues and is associated with visual dysfunction
107 ttenuated baseline phosphorylation of Erk in neural tissues and led to growth retardation.
108 derived transcript and SMN protein levels in neural tissues and muscle, which were associated with an
109 s compared with lipids characteristic of non-neural tissues and show further acceleration of change i
110         The mechanical mismatch between soft neural tissues and stiff neural implants hinders the lon
111 stula and gastrula stages and is enriched in neural tissues and the pronephros during later embryogen
112 what are the requirements for Shroom3 in non-neural tissues and what factors control Shroom3 transcri
113                CSPP1 is broadly expressed in neural tissue, and its encoded protein localizes to the
114 t by specifically formulating the bioink for neural tissues, and by spatially patterning cell types a
115 sly assumed, independently of mesodermal and neural tissues, and that Pax7 has a crucial function dur
116 , the mechanisms underlying the formation of neural tissue architectures during development of the ce
117   We generalize connectionist methods to non-neural tissue architectures, showing that a minimal non-
118  During early development, the Otx2-positive neural tissues are patterned anterior-posteriorly to for
119 were found between VFI values and prelaminar neural tissue area (R(2) = 0.20, P = 0.017), average BMO
120 ch is distinguished by bilateral necrosis of neural tissue around the ventricles and a sequela of neu
121 never infiltrate it and instead displace the neural tissue as they grow.
122  applicability to in vivo calcium imaging of neural tissue, as well as other smooth muscle tissue.
123 mated technologies to probe the structure of neural tissue at nanometer resolution and use them to ge
124 M) has become an essential tool for studying neural tissue at resolutions below 10 nm x 10 nm x 10 nm
125 never detected in cerebrospinal fluid nor in neural tissues at necropsy two weeks after infection.
126 matous patients, we found better results for neural tissue-based indexes (compared to LC-derived para
127 for inhibition of Fgf signalling in inducing neural tissue but it is not sufficient to maintain neura
128 te SH2B1(TgKO) mice expressing SH2B1 only in neural tissue but not in other tissues.
129 requires protein O-mannosylation activity in neural tissue but not the meninges.
130 ion, which we show not only marks definitive neural tissue, but also tissue that is not yet committed
131 is involved in cancer, vascular defects, and neural tissue, but is largely unexplored in immune syste
132 th SMN2 transcript as well as SMN protein in neural tissue, but only minimally in peripheral tissue.
133 rP(C)) is widely expressed in neural and non-neural tissues, but its function is unknown.
134 xpression of GPIalpha btx in surrounding non-neural tissues, but not in neurons, does not prevent cel
135  (miRNAs) are highly expressed in vertebrate neural tissues, but the contribution of specific miRNAs
136 rs of oxidative death in both neural and non-neural tissues, but their precise mechanism of action re
137 ons and/or use of cellular grafts to protect neural tissue by local delivery of growth or trophic fac
138 llect EVs from frozen whole murine and human neural tissues by serial centrifugation and purification
139 SYN1 gene transcription is suppressed in non-neural tissues by the RE1-silencing transcription factor
140 ioside GM3, a glycosophingolipid enriched in neural tissue, by adding sialic acid to lactosylceramide
141                                     The ways neural tissue can be stimulated to evoke artificial sens
142                           Deafferentation of neural tissue can result in cell death, morphological ch
143  spite of progressive extracellular edema in neural tissue, capillary endothelial cell tight junction
144 ound that ectopic expression of Gdf11 in the neural tissue causes a rostral displacement of Hoxc prot
145 ka virus (ZikV) is a flavivirus that infects neural tissues, causing congenital microcephaly.
146 tion genomic and gene expression analyses of neural tissue (central brain, optic lobes and ommatidia)
147 ting enzyme-2 (ECE-2), which is expressed in neural tissues, cleaves 'big endothelin' to produce the
148 vels of proinflammatory cytokines within the neural tissue compared with WT littermates.
149 tral cells induces the production of ectopic neural tissue considerably outside the neural field.
150  for human islet isolations contained bovine neural tissue contaminants.
151 medicine require an understanding of how non-neural tissues could process information.
152 several known restorative actions on damaged neural tissue, could play a role.
153 e nervous system and doublesex specified non-neural tissues culminated with claims that fruitless was
154 ission electron microscopy (ssTEM) images of neural tissue currently requires many hours of manual tr
155 nal diseases, results in vasogenic edema and neural tissue damage, causing vision loss.
156 present great promise regarding treatment of neural tissue damage, such as spinal cord injury (SCI).
157 ity, and free-radical release, contribute to neural tissue damage.
158 4 increases SMN expression in neonatal mouse neural tissues, delays motor neuron loss at PND11 and am
159                      Cells in the developing neural tissue demonstrate an exquisite balance between p
160                                No detectable neural tissue deposition or MR imaging signal was observ
161 ed expression of these two IG20-SVs in human neural tissues derived from cerebral cortex, hippocampus
162 , we defined the impact of HCMV infection on neural tissue development and calcium regulation, a crit
163 cts for studying the earliest steps of human neural tissue development and the pathogenesis of brain
164 euronal differentiation, and interruption of neural tissue development.
165                   Mice ablated for NMII-B in neural tissues die between postnatal day 12 and 22 witho
166 ld-type Gfr gene restored the HRP epitope in neural tissues, directly demonstrating that the Gfr muta
167  We present example data sets from mammalian neural tissue, Drosophila brain, and Chlamydomonas reinh
168  of this gene is predominately restricted to neural tissue during embryogenesis and is expressed in a
169 hots of neural activity in a large volume of neural tissue, e.g., a complete mouse brain, by circumve
170  of hMSCs and hence, would be beneficial for neural tissue engineering scaffolds.
171 ctive scaffolds or supporting substrates for neural tissue engineering.
172 e diverse facets of the challenging field of neural tissue engineering.
173 C differentiation and are thus important for neural tissue engineering.
174 ogel biomaterials-an attractive approach for neural tissue engineering.
175 s a novel regulator of T. gondii invasion of neural tissue, enhancing invasion likely by promoting su
176 didate genes are preferentially expressed in neural tissue, especially during the prenatal period, an
177 luding a reduction in the amount of anterior neural tissue, especially in the telencephalic, optic an
178                                      How can neural tissue exhibit both type I and type II excitabili
179 nitor cell from the 8-cell embryo formed the neural tissue fated clone through divisions to the 32-ce
180  cell of the 16-cell embryo gave rise to its neural-tissue-fated clone in the embryo developing to th
181 erface between the blood circulation and the neural tissue features unique characteristics that are e
182 onent of a long-term immune cell presence in neural tissues following genital HSV-1 infection and pla
183 phobic fluorescent dye are retained in local neural tissue for up to 5 days and that PgP can efficien
184                                              Neural tissue formation is induced by growth factors tha
185  procedure to use a GMP-manufactured, bovine neural tissue-free collagenase blend.
186             Ectopic Sox3 caused induction of neural tissue from a very early stage of cell specificat
187 gies to attenuate neuroinflammation, protect neural tissue from collateral injury, and enhance endoge
188 1 or Nbs1 hypomorphic mutations, we analyzed neural tissue from Mre11(ATLD1/ATLD1) and Nbs1(DeltaB/De
189  to improve the coverage of neuropeptides in neural tissue from the model organism C. borealis.
190  patients with temporal lobe epilepsy and in neural tissues from animal models of epilepsy.
191 conducted in three brain regions and two non-neural tissues from humans, chimpanzees, macaque monkeys
192 tral nervous system (CNS) barriers partition neural tissues from the blood, providing a homeostatic e
193 of all other cells tested, including retina, neural tissue, glial cells, and a cancer cell line.
194                                 We find that neural tissue has marked non-ohmic and frequency-filteri
195       The extracellular ionic environment in neural tissue has the capacity to influence, and be infl
196 ar behaviors underlying CE in the epithelial neural tissue, have not been identified.
197  strategy for improving collateral function, neural tissue health, and functional recovery following
198  mutants reveals that Pikfyve is critical in neural tissues, heart, lung, kidney, thymus, and spleen.
199            The poor regenerative capacity of neural tissue highlights the need for the development of
200                               When acting on neural tissue, however, testosterone can be metabolized
201 Several studies have conducted MSI of insect neural tissues; however, these studies did not detect ne
202 ids would specify the positional identity of neural tissue in a distance-dependent manner.
203 DAC11 and neural cells in vitro, we examined neural tissue in a previously uncharacterised Hdac11 kno
204 that can potentially access large volumes of neural tissue in a single treatment is intra-arterial (I
205 ression of NKCC1 results in the formation of neural tissue in ectodermal explants.
206  Rat SH2B1beta was specifically expressed in neural tissue in SH2B1-transgenic (SH2B1(Tg)) mice.
207    Here we use the structural context of the neural tissue in which dendritic trees exist to drive th
208  long-term repression of target genes in non-neural tissues in adult zebrafish.
209     Some family members are also enriched in neural tissues in both vertebrates and invertebrates.
210 crophages do not damage and may even protect neural tissues in EAN.
211 ion of BMP signaling is sufficient to induce neural tissues in vivo remains controversial.
212 l tetramers are prevalent in lymphocytes and neural tissues, in which octamers are abundant but hexam
213 Here we report that both melanoma and normal neural tissues including dorsal root ganglion (DRG) prod
214                   Biophysical stimulation of neural tissue induced demyelination and Schwann cell pro
215                                    The first neural tissue induced is anterior and subsequent neural
216  that gene therapy should be directed to the neural tissue instead of the meninges.
217   This study reveals the location of FAAH in neural tissue involved in peripheral nociceptive transmi
218        Delayed revascularization of ischemic neural tissue is a major impediment to preservation of f
219  is known about whether pathogen invasion of neural tissue is affected by immune-based mechanisms in
220             Somatic transposon expression in neural tissue is commonly considered as a measure of mob
221 hat the amount of pathological aggregates in neural tissue is exceedingly low, precluding examination
222               The propagation of activity in neural tissue is generally associated with synaptic tran
223              The delivery of therapeutics to neural tissue is greatly hindered by the blood brain bar
224 al tissue induced is anterior and subsequent neural tissue is posteriorized to form the midbrain, hin
225           We investigated how glutamate in a neural tissue is protected from catabolism.
226 humans, but the role of PtdIns(3,5)P2 in non-neural tissues is poorly understood.
227 iest step in this process - the induction of neural tissue - is intimately linked to patterning of th
228 localized in cytoplasmic puncta in zebrafish neural tissue, it is on the plasma membrane in mib1 muta
229 ng and histopathologic examination showed no neural tissue lesions within the spinal cord; however, f
230 two-photon imaging, we show that neurons and neural tissue maintain basal stores of loosely bound cop
231 tion, bone-marrow derived macrophages invade neural tissue, making it difficult to distinguish betwee
232 hough these cell lines were not derived from neural tissue, many neurobiologically relevant genes wer
233 temporal spectra nor processed by registered neural tissue maps.
234 ctive action of perisynaptic CSPGs in mature neural tissue may account for the therapeutic effects of
235   However, genes with essential roles in non-neural tissues may be missed in traditional loss-of-func
236 sent an approach to form functional in vitro neural tissue mimic (NTM) of different shapes using stem
237              We review a range of bioprinted neural tissue models and discuss how they can be used to
238 ility for development of in vitro functional neural tissue models of varying forms for therapeutic bi
239 eural plate during neurula stages and in the neural tissue of adult frogs.
240                               Microarrays of neural tissue of animal models of the disease showed dec
241 ple functions in the development of anterior neural tissue of vertebrate embryos.
242 t performs these functions in epithelial and neural tissues of both insects and mammals, as well as i
243 s involved in the regulation of apoptosis in neural tissues of both WKY and SHRSP rats.
244 -to-I editing was recently discovered in the neural tissues of coleoids (octopuses, squids, and cuttl
245  To determine whether gadolinium deposits in neural tissues of patients with intracranial abnormaliti
246 f the brain and spinal cord without damaging neural tissues or triggering foreign body reactions.
247 y, reduced transmigration of leukocytes into neural tissue, or reduced invasion by extracellular para
248               Due to limited access to human neural tissue, pathogenetic studies have, so far, mostly
249 hanical properties of the LC (PC2) or of the neural tissue (PC4), rotation of the peripapillary scler
250                                           In neural tissue, PCDH-gamma, together with PCDH-alpha, for
251 e connective tissue (laminar) and prelaminar neural tissue (prelaminar) components of optic nerve hea
252             It is impossible to consider how neural tissue processes these numbers without first cons
253  studies have shown that during development, neural tissues produce mRNAs with particularly long 3'UT
254 avage site within the hevin sequence for the neural tissue proteinase ADAMTS4.
255 ction and by the materials properties of the neural tissue, recent advances in neural interrogation a
256  the molecular cascades through which target neural tissues regulate vessel stabilization and pattern
257 sophila have revealed molecular pathways and neural tissues regulating sleep; however, genes that mai
258 mal phenotype with a decreased proportion of neural tissue relative to nascent mesoderm.
259 o their innate ability to enhance endogenous neural tissue repair and promote functional recovery.
260 sions to recruit endogenous NPCs and enhance neural tissue repair/regeneration.
261 entially grow as compared to neighboring non-neural tissues, resulting in dendrite overgrowth.
262              Intriguingly, analysis of T32KO neural tissue revealed a decreased concentration of neur
263                                              Neural tissue samples dissected from rat brain and the c
264  unfolded protein response was identified in neural tissues (sciatic nerve, spinal cord) of streptozo
265 embrane, a process that requires neurabin (a neural tissue-specific protein).
266 egulation of adenosine-evoked responses by a neural tissue-specific protein, neurabin.
267 changes point to a dynamic modulation of the neural tissue structure upon activation, which remains t
268                     CircRNAs are enriched in neural tissues, suggesting that they might have neural f
269 owing ischemic stroke, the penumbra, at-risk neural tissue surrounding the core infarct, survives for
270 nd reveal extensive movements of the cranial neural tissue that are independent of neural fold zippin
271 mucosa, the organ of smell in the nose, is a neural tissue that regenerates new sensory neurons throu
272                  The retina is a specialized neural tissue that senses light and initiates image proc
273 otrophin-3 (NT-3) is highly expressed in non-neural tissues that receive peripheral innervation, we i
274 onal properties of Nell-1 in oral-dental and neural tissues that will be the frontiers of future Nell
275 l canal enlargement, cupping, and prelaminar neural tissue thickening.
276 cribrosa (LC) thickness and area, prelaminar neural tissue thickness and area, anterior LC depth, Bru
277 action, CD40 restricts T. gondii invasion of neural tissue through a mechanism that appears mediated
278 o serous retinal detachment or herniation of neural tissue through the LC defect.
279 y, histology analysis revealed filling-in of neural tissue through the macroporous network and attrac
280 mouse embryo inhibits BMP signaling to allow neural tissue to form as a default fate-in the absence o
281 d either by stimulating or by recording from neural tissue to treat or assist people with sensory, mo
282                             Yet, unlike real neural tissue, traditional computing architectures physi
283 genetically normal and unrelated allografted neural tissue transplanted into the brain of affected HD
284 o that tau pathology can manifest in healthy neural tissue transplanted into the brains of patients w
285                                   Developing neural tissue undergoes a period of neurogenesis followe
286 genes that encode heat shock proteins in non-neural tissues upon exposure to heat.
287 aset (>100,000 spikes) recorded from varying neural tissue (V1 and retina) using different calcium in
288 ons to complete pyritization, revealing that neural tissue was initially preserved as carbonaceous fi
289 arance of infectious HSV type 1 (HSV-1) from neural tissues was also detected.
290           Clearance of infectious virus from neural tissues was not significantly different in perfor
291 neutrophils during murine peritonitis and by neural tissues was separated from natural isomers and su
292 ble transcriptional targets of PGC-1alpha in neural tissue, we conducted a microarray on neuroblastom
293       Histopathologic characteristics of the neural tissues were assessed and used to select a voltag
294                              HSV-1 titers in neural tissues were greatly reduced over time in CD8(+)
295                               The prelaminar neural tissues were thickened inferiorly, inferonasally,
296 n Opa1 transcript and protein in retinal and neural tissue, which manifests as visual dysfunction in
297 " neurotransmitters allow optical control of neural tissue with high spatial and temporal precision.
298 (CDK5) is expressed at high levels in MM and neural tissues with relatively low expression in other o
299 larged vascular structures located in benign neural tissues within the cerebellum and spinal cord of
300 ascribed a mechanical role in the support of neural tissues, yet this idea has not been specifically

 
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