ライフサイエンスコーパス: neurogenesis

1 age-related alterations in adult hippocampal neurogenesis.

2 gration, which represents a critical step in neurogenesis.

3 epressant properties of ECT, and may reflect neurogenesis.

4 lth span and provide a permissive milieu for neurogenesis.

5 to reveal their effects on prefrontal cortex neurogenesis.

6 sglutaminase 2, which we show is crucial for neurogenesis.

7 hile class 2 mutations (5 of 27) enhance PFC neurogenesis.

8 d -distal elements of negative regulators of neurogenesis.

9 ns in a polyQ length-dependent manner during neurogenesis.

10 the CNS and play a fundamental role in adult neurogenesis.

11 y C-terminal regions, play distinct roles in neurogenesis.

12 y age-dependent decline in proliferation and neurogenesis.

13 in to plain water, with increased markers of neurogenesis.

14 eta2/3-Smad2 pathway in the control of adult neurogenesis.

15 reduce amyloid plaque pathology and promote neurogenesis.

16 irus (ZIKV) causes microcephaly and disrupts neurogenesis.

17 , a process referred to as adult hippocampal neurogenesis.

18 ts hypothesised effects on adult hippocampal neurogenesis.

19 nscriptomic programme associated with active neurogenesis.

20 ent can lost neurons be replenished by adult neurogenesis.

21 mouse cortex and regulates RGP behavior and neurogenesis.

22 ontrolling glial quiescence, reactivity, and neurogenesis.

23 adial glial cells, compatible with a role in neurogenesis.

24 ible by inducing XIST at different stages of neurogenesis.

25 independent of Reelin actions on hippocampal neurogenesis.

26 2Y12 and MerTK/Axl reduces adult hippocampal neurogenesis.

27 evidence exists regarding postnatal enteric neurogenesis.

28 ependent regulation of early events in adult neurogenesis.

29 ex, Notch-depleted astrocytes also initiated neurogenesis.

30 her they are similarly involved during adult neurogenesis.

31 e molecular mechanisms underlying vertebrate neurogenesis.

32 or complex underlying both cardiogenesis and neurogenesis.

33 d to maintain progenitor pools essential for neurogenesis.

34 includes axon regrowth and is accompanied by neurogenesis.

35 ning)-induced anxiolytic effect and adult DG neurogenesis.

36 dels of T2DM display compromised hippocampal neurogenesis.

37 y block the effects of Reelin on hippocampal neurogenesis.

38 negative regulator of postnatal hypothalamic neurogenesis.

39 e long-term maintenance of adult hippocampal neurogenesis.

40 plasticity, nervous system development, and neurogenesis.

41 ated antianxiety-like behaviors and adult DG neurogenesis.

42 ocorticoids (GCs), which are known to impact neurogenesis.

43 mouse neuroblastoma cells, a requirement for neurogenesis.

44 ium base, promoting ciliopathy and premature neurogenesis.

45 emoval but has an active role in maintaining neurogenesis.

46 c-1 as negative regulator of planarian adult neurogenesis.

47 to the brain and stimulate adult hippocampal neurogenesis.

48 ce variants in regulating distinct stages of neurogenesis.

49 ide synthase production, and reduced colonic neurogenesis.

50 ampal cellular proliferation, or hippocampal neurogenesis.

51 y arising from cell proliferation during mid-neurogenesis.

52 t the marginal zones of the VNO: the site of neurogenesis.

53 R4 inhibitor did not have reduced markers of neurogenesis.

54 w it should be applied to the study of adult neurogenesis.

55 cellent animal models for the study of adult neurogenesis.

56 cortex by controlling direct versus indirect neurogenesis.

57 rn cerebrospinal fluid homeostasis [13, 14], neurogenesis [12, 15-18], and embryonic morphogenesis [1

58 justify the comparative examination of adult neurogenesis across mammalian species.

59 ing gene therapies to these cells to enhance neurogenesis after TBI.

60 re-pubertal period impairs adult hippocampal neurogenesis (AHN) and behaviours which rely on this pro

61 ospho-tau accumulation and adult hippocampal neurogenesis (AHN) impairment both contribute importantl

62 ronic stress downregulates adult hippocampal neurogenesis (AHN) in mammals and birds, we employ this

63 cally on the role of human adult hippocampal neurogenesis (AHN) in MDD and AD.

64 Understanding why adult hippocampal neurogenesis (AHN) is impaired in Alzheimer's disease (A

65 e found microglomeruli-like complexes, adult neurogenesis, aminergic innervation, and elevated expres

66 social behaviors, sleep function, and adult neurogenesis, among many fundamental themes.

67 bgroups of autism mutations that perturb PFC neurogenesis and are correlated to abnormal WNT/betacate

68 ptophan-kynurenine metabolism, the HPA axis, neurogenesis and BDNF, epigenetics, and obesity.

69 oked serotonin release, stimulation of adult neurogenesis and behavioral improvement.

70 on cycle 42 (CDC42) plays essential roles in neurogenesis and brain development.

71 l to map the 3D genome architecture in human neurogenesis and brain tissues and also demonstrated tha

72 from 10 tissue or cell types with a focus on neurogenesis and brain tissues.

73 ophagy and trafficking protein with roles in neurogenesis and cancer cell invasion.

74 ' phenotype with early lethality, defects in neurogenesis and cardiac dilation.

75 he SWI/SNF epigenetic complex, controls both neurogenesis and cardiogenesis from human embryonic stem

76 ied p120-catenin as an upstream regulator of neurogenesis and cell cycle pathways and a predictor of

77 depletion reduced stroke-induced hippocampal neurogenesis and cell survival.

78 transferred the effects of exercise on adult neurogenesis and cognition to sedentary aged mice.

79 dentate gyrus, with sex-dependent effects on neurogenesis and cognition.

80 he potential impact of immune homeostasis on neurogenesis and cognitive decline during brain aging ha

81 NK cells leads to sustained improvements in neurogenesis and cognitive function during normal aging.

82 ckout (KO) zebrafish model exhibits abnormal neurogenesis and craniofacial patterning, and in vivo co

83 f WS, with the top-scoring module related to neurogenesis and development of the central nervous syst

84 This method leads to sustained neurogenesis and formation of an expanded cortical plate

85 he short and long-term effects of colitis on neurogenesis and glia responses in the hippocampus.

86 permitted the parallel study of the ongoing neurogenesis and gliogenesis.

87 tive potential play important roles in human neurogenesis and gray matter expansion, the mechanisms o

88 everse transcribed into cDNA) and found that neurogenesis and haematopoiesis dominate at both the gen

89 These proteins are involved in neurogenesis and have been associated with various neuro

90 long-term potentiation, synaptic plasticity, neurogenesis and hippocampal-dependent spatial memory in

91 are sufficient to increase adult hippocampal neurogenesis and improve behavioral performance.

92 SC O-GlcNAc levels coincident with decreased neurogenesis and increased gliogenesis in the mature hip

93 ic experimental colitis on adult hippocampal neurogenesis and innate immune cell responses, highlight

94 e cerebral cortex, which is not conducive to neurogenesis and integration of new neurons.

95 st that cdnf has broad effects on regulating neurogenesis and maturation of transmitter-specific neur

96 ffective dose of IVIg to promote hippocampal neurogenesis and modulate the inflammatory milieu.

97 l aging and impacts the homeostasis of adult neurogenesis and mood regulation.

98 studied the role of Zeb2 and its domains in neurogenesis and neural differentiation in the young pos

99 oding for LIS1) results in the disruption of neurogenesis and neuronal migration via dysregulation of

100 t role in brain development, particularly in neurogenesis and neuronal migration.

101 portional differences were maintained during neurogenesis and over 18 weeks of growth.

102 y regulator of NSPC activity, disrupts human neurogenesis and potentially contributes to cognitive de

103 of postmitotic neurons but also impairs RGC neurogenesis and radial glia processes.

104 ect amyloid beta concentrations but enhanced neurogenesis and reduced neuroinflammation.

105 Loss of ET-1 signaling increases neurogenesis and reduces oligodendrocyte progenitor cell

106 fic deletion of Ars2 compromises hippocampal neurogenesis and results in specific behavioral defects.

107 of MS patients, we investigated hippocampal neurogenesis and structural development of adult-born ne

108 pp5e leads to a transient increase in direct neurogenesis and subsequently to an overproduction of la

109 deficits in MS, we investigated hippocampal neurogenesis and synaptic connectivity of adult-born neu

110 Interestingly, the altered neurogenesis and synaptic connectivity of newborn neuron

111 mice also display impairments in hippocampal neurogenesis and synaptic plasticity.

112 nes and pathways disrupted in ASD, including neurogenesis and synaptic processes, underlying the obse

113 correlation between training regimens during neurogenesis and synaptogenesis and the resulting plasti

114 using optical stimulation implemented during neurogenesis and synaptogenesis.

115 illustrated that vOrganoids exhibited robust neurogenesis and that cells of vOrganoids differentially

116 nctional investigations of the patterning of neurogenesis and the transition of progenitors to neuron

117 proliferation-differentiation switch during neurogenesis and the upstream epigenetic triggers remain

118 targets for the inhibition of tumor-induced neurogenesis and tumor progression.

119 e and the switch between direct and indirect neurogenesis and ultimately distinct neuronal lineage ou

120 Our results confirmed an outside-in trend of neurogenesis and uncovered a rostrocaudal progression.

121 expression affecting pathways necessary for neurogenesis and upregulation of gliogenic genes.

122 ed anxiety-like behaviors, impaired adult DG neurogenesis, and abolished activity (e.g., voluntary wh

123 in damage, neuroinflammation, stroke-induced neurogenesis, and atrophy of parvalbumin-positive (PV(+)

124 nxiety-like behaviors but promoting adult DG neurogenesis, and both functions are likely through acti

125 red proteins are critical for embryogenesis, neurogenesis, and cell signaling.

126 GDNF prolonged survival, induced enteric neurogenesis, and improved colon structure and function

127 es, alleviates neuroinflammation, stimulates neurogenesis, and improves cognitive function in a tripl

128 e that CX3CL1 is a strong activator of adult neurogenesis, and that it reduces neuronal loss and impr

129 t the specific roles of CDC42u and CDC42b in neurogenesis are due to their opposing effects on mTORC1

130 emporal interactions posited to occur during neurogenesis are necessary for the formation of the char

131 Negative regulators of adult neurogenesis are of particular interest as targets to en

132 patial and temporal features of adult canine neurogenesis are similar to that of other gyrencephalic

133 elinated hippocampus, suggesting hippocampal neurogenesis as a potential target to normalize cognitiv

134 providing critical insight into hippocampal neurogenesis as a potential therapeutic target to treat

135 ovide activity-dependent regulation of adult neurogenesis as well as maintain inhibitory control of m

136 allels to recent findings on mammalian adult neurogenesis, as both systems seem to exhibit a similar

137 duced ipsilateral projection and altered RGC neurogenesis associated with perturbed melanogenesis in

138 global transcriptome changes associated with neurogenesis, axonogenesis, neuronal migration and neuro

139 rogram that alters their secretome, limiting neurogenesis both in vivo and in vitro In addition, chro

140 We show that acute DSS colitis enhanced neurogenesis but with deficits in cell cycle kinetics of

141 olitis was characterized by normal levels of neurogenesis but with deficits in the migration and inte

142 and centriolar satellites have functions in neurogenesis, but little is known about their roles in t

143 ptoRAF1 did not have a significant effect on neurogenesis, but was able to promote neuronal neurite g

144 1 and 8, significantly increased hippocampal neurogenesis by 4-, 3-, and 1.5-fold in comparison to sa

145 Treatment with PDD005 prevents impairment of neurogenesis by increasing sex-determining region Y-box

146 d single-cell RNA sequencing to characterize neurogenesis by Notch-depleted striatal astrocytes in vi

147 ll be the first to demonstrate that enhanced neurogenesis by overexpressed CX3CL1 is mitigated by dis

148 e balance between progenitor maintenance and neurogenesis by physically interacting with Ascl1, inter

149 neurons per ganglion, and reduced markers of neurogenesis compared with mice given saline.

150 Neurogenesis comprises many highly regulated processes i

151 le-age rats, linked to synaptic function and neurogenesis, correlated with beacon discrimination perf

152 This enhanced neurogenesis correlates well with elevated expression of

153 l interactions between the time and place of neurogenesis could underlie multiple features of empiric

154 However, since neurogenesis declines dramatically with age, it is uncle

155 Genes contributing to neurogenesis, dendritic development and synaptogenesis o

156 to be touchscreen-, radiation particle-, or neurogenesis-dependent, as (56)Fe and (28)Si irradiation

157 the niche that trigger astrogliogenesis over neurogenesis during CNS disease are unclear.

158 regulating motor and cognitive functions and neurogenesis (e.g., dentate gyrus, hypothalamus, olfacto

159 ontrast, Ascl-1(null) mutants showed reduced neurogenesis for both vomeronasal and GnRH-1ns but less

160 ct at the circuit level to ensure continuous neurogenesis from NSCs remains unknown.

161 entate gyrus expressed synaptic function and neurogenesis genes correlated with beacon discrimination

162 Here we show that somatosensory neurogenesis gives rise to neurons in a transcriptionall

163 age.SIGNIFICANCE STATEMENT Adult hippocampal neurogenesis has been extensively studied in the context

164 serve of plasticity that persists even after neurogenesis has declined to low rates.

165 bute to hippocampal function in old age when neurogenesis has declined.

166 critical for maintenance of pluripotency and neurogenesis, has been found associated with several lnc

167 During neurogenesis, Her6 expression transitions from fluctuati

168 rexpression promotes only the first stage of neurogenesis (i.e. the formation of neuroprogenitors) an

169 mice and found that they exhibited cortical neurogenesis impairment, macrocephaly, and hallmark ASD

170 e neuronal loss, increased inflammation, and neurogenesis impairment.

171 current study tested the effects of aberrant neurogenesis in a mouse model of repeated mild traumatic

172 Therefore, morphine and naloxone promote neurogenesis in a receptor-independent manner at least d

173 ovide a further rationale for maintaining DG neurogenesis in adult life.

174 MPP5 is essential for neurogenesis in animal models, but human variants leadin

175 clear whether PRDM16 plays a similar role in neurogenesis in both dorsal and ventral progenitor linea

176 Tg-CX3CL1 mice exhibit enhanced neurogenesis in both subgranular and subventricular zone

177 li3 repressor rescues the decreased indirect neurogenesis in Inpp5e mutants.

178 stroke, however, striatal astrocytes undergo neurogenesis in mice, triggered by decreased Notch signa

179 eptor agonist prucalopride increases enteric neurogenesis in normal development and after injury.

180 Putative factors promoting neurogenesis in response to IVIg-FUS include the down-re

181 Adult neurogenesis in rodents is modulated by dopaminergic sig

182 nge transcriptional repressor that regulates neurogenesis in several developmental contexts.

183 ion into the mechanisms underlying decreased neurogenesis in T2DM and its link to the cognitive decli

184 adictory findings concerning the function of neurogenesis in the adult dentate gyrus.

185 Neurogenesis in the adult hippocampus declines with age,

186 molecular basis of metaplasia and declining neurogenesis in the aging olfactory epithelium (OE) rema

187 cally-quiescent state dramatically decreased neurogenesis in the demyelinated hippocampus.

188 In the animal analog of ECT, neurogenesis in the dentate gyrus (DG) of the hippocampu

189 vely regulates RGP behavior to ensure proper neurogenesis in the mammalian cortex.

190 ress this by examining the origin of de novo neurogenesis in the post-embryonic zebrafish ENS.

191 ve-like behavior, cognition, and hippocampal neurogenesis in the repeated-corticosterone (CORT) parad

192 ve-like behavior and enhanced the endogenous neurogenesis in the subventricular zone (SVZ) and the de

193 Neurogenesis in the zebrafish olfactory epithelium requi

194 the mechanisms controlling morphogenesis and neurogenesis in these structures has grown, how these pr

195 ognitive deterioration, however, the fate of neurogenesis in these studies is highly controversial.

196 intrinsic factors that control the timing of neurogenesis in this region are poorly characterized.

197 V-H41R is less virulent and does not inhibit neurogenesis in vitro or corticogenesis in utero.

198 In zebrafish, there is spatial patterning of neurogenesis in which non-neurogenic zones form at bound

199 endpoints thought to be dependent on dentate neurogenesis, including memory acquisition, short-term a

200 showed complex, subtype-specific patterns of neurogenesis, including some patterns of development con

201 of NSC O-GlcNAcylation in young mice reduces neurogenesis, increases astrocyte differentiation, and i

202 ion maintaining neural stem cells (NCSs) and neurogenesis into adulthood.

203 Adult hippocampal neurogenesis is considered to play an important role in

204 Murine modeling shows that neurogenesis is likely altered in these individuals, wit

205 pothesized that the main function of dentate neurogenesis is long-term memory formation because we as

206 Age-related decline of neurogenesis is paralleled by a progressive decline of r

207 However, lactate's ability to stimulate neurogenesis is reversed by blocking MAPK.

208 Adult hippocampal neurogenesis is sensitive to experiences, suggesting tha

209 dicate that Ascl-1, while crucial for GnRH-1 neurogenesis, is not required for normal OEC development

210 On the basis of its role in neurogenesis, it has been postulated that LMNB1 variants

211 the hippocampus in humans is attributable to neurogenesis, it is expected to be exclusively present i

212 s PI3K-AKT-mTOR activity and alters cortical neurogenesis, leading to macrocephaly and autistic-like

213 in, gene expression, cell types, birthdates, neurogenesis, lineage and migration, circuit connectivit

214 These SMN-specific mRNAs are associated with neurogenesis, lipid metabolism, ubiquitination, chromati

215 mechanisms and functional relevance of adult neurogenesis, many studies rely on quantification of adu

216 ling and double smFISH of her6 and the early neurogenesis marker, elavl3, suggest that the change in

217 dies suggest that alterations in hippocampal neurogenesis may contribute to cognitive deterioration,

218 rinsic and cell-extrinsic cues that organize neurogenesis, migration, and axo-dendritic specification

219 ific risk genes disrupts cell proliferation, neurogenesis, migration, and cell fate, while in trimest

220 etic resonance (MR) imaging, reduced dentate neurogenesis moderately correlated with deficits in memo

221 During adult hippocampal neurogenesis, most newborn cells undergo apoptosis and a

222 nd inflammatory markers (N = 118), and adult neurogenesis (N = 6).

223 phic factors which play an important role in neurogenesis, neural maintenance and repair.

224 e3 and upregulation of genes associated with neurogenesis, neuronal differentiation, and function.

225 er, cilia have been described as crucial for neurogenesis, neuronal maturation, and survival in the c

226 bstrates, loss of AMOT negates the rescue of neurogenesis normally induced by pharmacologic inhibitio

227 ter they have matured.SIGNIFICANCE STATEMENT Neurogenesis occurs in the hippocampus throughout adult

228 ogenitors in the gut, post-embryonic enteric neurogenesis occurs via gut-extrinsic Schwann cell precu

229 Hippocampus is a brain region where adult neurogenesis occurs with functional implications in mood

230 sity of laminar compartments, a delay in the neurogenesis of infragranular layers relative to layer 1

231 data shed new light on mechanisms underlying neurogenesis of inhibitory neurons.

232 that SUMOylation may stimulate survival and neurogenesis of neuronal stem cells.

233 ttern of abnormalities may reflect disrupted neurogenesis of projection neurons in outer cortical lay

234 yers relative to layer 1, and a delay in the neurogenesis of supragranular layers relative to infragr

235 and long-term effects of GC exposure during neurogenesis on messenger RNA (mRNA) expression and DNA

236 increases beta-catenin activity and rescues neurogenesis on stiff substrates.

237 adult hippocampal astrogliogenesis, but not neurogenesis or behavioral performance in mice.

238 lecular mechanism of LRRK2 in the control of neurogenesis or gliogenesis was investigated.

239 e Eif2 signaling pathway, synaptogenesis and neurogenesis pathways.

240 has the unique characteristic of exhibiting neurogenesis persisting into adulthood.

241 A limited amount of stem cell-driven neurogenesis persists in postnatal life and is reduced i

242 Hippocampal neurogenesis plays an important role in learning and mem

243 The gut microbiome is fundamental in neurogenesis processes.

244 we systematically investigated the temporal neurogenesis profiles of V3 INs in the mouse spinal cord

245 physiological activator of adult hippocampal neurogenesis, recruited hiROS NPCs into proliferation vi

246 the adult mouse colon, TLR2 promotes colonic neurogenesis, regulated by intestinal bacteria.

247 ved in metabolism, chromatin remodeling, and neurogenesis-related functions.

248 ogenous regulators of postnatal hypothalamic neurogenesis remain elusive.

249 cture of ribosomes from the neocortex during neurogenesis, revealing Ebp1 as a high-occupancy 60S pep

250 Although proliferation of NSCs and neurogenesis seen in Ccn1 knockout mice eventually retur

251 temporal progression of tuberal hypothalamic neurogenesis.SIGNIFICANCE STATEMENT Here, we investigate

252 e long-term maintenance of adult hippocampal neurogenesis.SIGNIFICANCE STATEMENT Microglia are the br

253 receptor (IL-6R) and robustly support adult neurogenesis, specifically by augmenting the survival of

254 In the absence of Neurog2, neurogenesis stalls, with a significant reduction in ear

255 e MerTK downregulation transiently increases neurogenesis, suggesting that microglial phagocytosis pr

256 After neurogenesis, suppression of apoptosis is needed to ensu

257 downstream processes such as proliferation, neurogenesis, synaptogenesis, and neural activity.

258 t of cell ageing on gene networks related to neurogenesis, telomere maintenance, cell senescence and

259 The spatial and temporal features of RGC neurogenesis that give rise to ipsilateral and contralat

260 shes a link between the length of gestation, neurogenesis, the maternal environment, and key features

261 other HDAC complexes have been implicated in neurogenesis, the physiological role of MiDAC remains un

262 natural age-related decline in the levels of neurogenesis, the regenerative capacity of the hippocamp

263 As astrocytes initiated neurogenesis, they became transcriptionally very similar

264 Thus, our work demonstrates neurogenesis timing as a developmental mechanism underly

265 Thus, our current work indicates neurogenesis timing can modulate the functions of early

266 In specific cases of other IN classes, neurogenesis timing can play a role in determining the u

267 To examine whether neurogenesis timing contributes to V3 diversity, we syst

268 not late-born, V3 INs, which indicates that neurogenesis timing may enable newborn V3 INs to interac

269 3 IN population, we investigated the role of neurogenesis timing on IN diversity.

270 to better understand the consequences of V3 neurogenesis timing, we investigated the time-dependent

271 tebrates has pleotropic effects ranging from neurogenesis to modulation of behaviors.

272 ouse model of endogenously deficient dentate neurogenesis to test this hypothesis.

273 sed RT-PCR gene arrays ("wound healing" and "neurogenesis") to evaluate tissue samples from the C2-C6

274 During neurogenesis, tuberal hypothalamic neurons are thought t

275 across the life span, we estimate that adult neurogenesis ultimately produces half of the cells and t

276 genitors per field, indicating that impaired neurogenesis underlies the neuronal decrease in OSB fetu

277 x, consistent with an impairment of cortical neurogenesis underlying the neuronal decrease in OSB fet

278 ation, morphine, but not naloxone, inhibited neurogenesis via traditional receptor-dependent and miR1

279 strogliogenesis by 75%, but no difference in neurogenesis was detected and no improvement in behavior

280 First, we found that neurogenesis was disrupted in male and female mice chron

281 While adult hippocampal neurogenesis was extensively studied with regard to its

282 Surprisingly, GnRH-1 neurogenesis was intact in Gli3(Xt/Xt) mice but they dis

283 Intriguingly, the enhanced adult neurogenesis was mitigated when Smad2 expression was del

284 In the demyelinated hippocampus, neurogenesis was nearly absent in the dentate gyrus, whi

285 In addition, adult hippocampal neurogenesis was observed in both species, occurring at

286 arly stage of NSC differentiation, increased neurogenesis was observed.

287 In contrast, neurogenesis was transiently increased in mice in which

288 In the absence of Nox2, baseline neurogenesis was unaffected, but the activity-induced in

289 To further understand the control of neurogenesis, we have carried out single cell RNA sequen

290 ities of morphine and naloxone to facilitate neurogenesis were also observed in opioid receptor-knock

291 nxiety-like behaviors and increased adult DG neurogenesis, whereas ablating or chronically inhibiting

292 bstrates, loss of AMOT substantially reduces neurogenesis, whereas on stiff substrates, loss of AMOT

293 A production, including key miRNAs linked to neurogenesis, which is not observed after ZIKV-H41R infe

294 the CX3CL1 C-terminal domain can upregulate neurogenesis, which may ameliorate neurodegeneration.

295 cell accumulation in the aging brain impairs neurogenesis, which may serve as a therapeutic target to

296 ife span of PS19 tau mice by enhancing adult neurogenesis while having minimal effect on tau patholog

297 derepressed mRNA isoforms during neocortical neurogenesis whose orthologs include risk genes for neur

298 mir-934 is a novel regulator of early human neurogenesis with potential implications for a species-s

299 nterfering with its DNA binding and limiting neurogenesis within LGE progenitors.

300 vestigated the general timing and pattern of neurogenesis within the tuberal hypothalamus.