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1 ch may cause remodeling or reconnectivity of neuroglia.
2 oteins (mGluR2/3) are mainly associated with neuroglia.
3 o perturbations within neurons or supporting neuroglia.
4 +) gradient-dependent transport processes in neuroglia, a reduced affinity for K(+) or Na(+) is not a
5                      Microglia are a type of neuroglia accounting for 10 to 12% of all cells in the b
6 d the viability and morphological changes in neuroglia and neurons located in different brain regions
7            UNC-6 is expressed in 12 types of neuroglia and neurons, creating a hierarchy of netrin cu
8                                              Neuroglia are critical for the life-long shaping of syna
9 oring the more inclusive view, positing that neuroglia are fundamental for defining the cognitive res
10  We conclude that S1P receptors expressed by neuroglia are involved in regulating the response to inj
11 study provides direct evidence for targeting neuroglia by antisense DNA-based SPION-gfap that enables
12 hins may represent a novel mechanism whereby neuroglia can regulate the local concentration of these
13  to microglial activity underlying essential neuroglia developmental processes that may deeply influe
14  T lymphocytes and cell death of neurons and neuroglia in allo-HSCT recipients with GVHD.
15                  How amphetamine affects the neuroglia in living brains is not well understood.
16 rt to elucidate this effect, we investigated neuroglia in response to amphetamine exposure using anti
17 onclude that amphetamine induces toxicity to neuroglia in vivo, which may cause remodeling or reconne
18                                    Targeting neuroglia may represent an untrodden path for prolonging
19              Here we show that the amorphous neuroglia networks of the mouse ENS are composed of over
20 y are well understood, the role of intrinsic neuroglia networks remains largely unknown.
21 led electron-dense nanoparticles only in the neuroglia of normal or transgenic mice [B6;DBA-Tg(Fos-tT
22 ansformed from fetal OPCs, are idiosyncratic neuroglia of the central nervous system (CNS) that are d
23 -d reaction product in the blood vessels and neuroglia of the unmyelinated optic nerve head and myeli
24 fine brain maintenance and resilience, while neuroglia regenerative capabilities are critical for bra
25 d FIV lentiviral vectors for hepatocyte- and neuroglia-related disease applications.
26  show that microglia regulate the process of neuroglia remodeling during ocular injury, and their dep
27     This mechanism of retinal damage through neuroglia remodeling may be clinically important for the
28 ion, and their depletion results in abnormal neuroglia remodeling that exacerbates neuroretinal tissu
29 s an antiviral type I interferon response in neuroglia, resulting in complement-mediated synapse elim
30        Experimental advances in the study of neuroglia signaling have been greatly accelerated by the
31               Terminal Schwann cells (TSCs), neuroglia that cover motoneuron terminals, play a role i
32 c glia are a unique population of peripheral neuroglia that regulate homeostasis in the enteric nervo
33                           In cocultures with neuroglia, the frequency and amplitude of spontaneous po
34 ioneer growth cones initially navigate along neuroglia to establish an axon scaffold that guides late
35  and tumor growth factor-beta1, derived from neuroglia, were the most prevalent cytokines in brain ti
36 al studies show reduced glutathione (GSH) in neuroglia, whereas immunocytochemistry of glutaraldehyde
37 mmunohistochemistry, a reduction in striatal neuroglia, with gliogenesis in the subventricular zone a