ライフサイエンスコーパス: neuromuscular junction

1 receptor (AChR) and inhibit signaling at the neuromuscular junction.

2 ons in the spinal cord or denervation at the neuromuscular junction.

3 ripheral cholinergic synapses, including the neuromuscular junction.

4 llite cells and within motor neurons via the neuromuscular junction.

5 endent synaptic plasticity at the Drosophila neuromuscular junction.

6 r structural modifications at the Drosophila neuromuscular junction.

7 echanism for graded locomotor control at the neuromuscular junction.

8 tamatergic synapses of the Drosophila larval neuromuscular junction.

9 endent synaptic remodeling at the Drosophila neuromuscular junction.

10 citatory synaptic transmission at the larval neuromuscular junction.

11 ue, probably around the nerve endings of the neuromuscular junction.

12 eostatic synaptic compensation at the larval neuromuscular junction.

13 l synaptic-function at the Drosophila larval neuromuscular junction.

14 defects in the structure and function of the neuromuscular junction.

15 locking acetylcholine receptors at the mouse neuromuscular junction.

16 of glutamate receptor clusters at the larval neuromuscular junction.

17 lutamatergic transmission also occurs at the neuromuscular junction.

18 ructural and functional abnormalities at the neuromuscular junction.

19 ise from impaired signal transmission at the neuromuscular junction.

20 teins essential for neurotransmission at the neuromuscular junction.

21 ity-evoked peptide release at the Drosophila neuromuscular junction.

22 presynaptic microtubule stabilization at the neuromuscular junction.

23 rol structure and function of the Drosophila neuromuscular junction.

24 caffolding protein, rapsyn, as at the intact neuromuscular junction.

25 ptic boutons at the axon terminals of larval neuromuscular junction.

26 oline receptor (AChR) is the hallmark of the neuromuscular junction.

27 ctionally validated factors expressed at the neuromuscular junction.

28 dies and immunohistochemical analysis of the neuromuscular junction.

29 holinesterase inhibitors on the postsynaptic neuromuscular junction.

30 e and to long-term structural changes at the neuromuscular junction.

31 growth of synapses at the Drosophila larval neuromuscular junction.

32 r muscle channelopathies and diseases of the neuromuscular junction.

33 f birth when AChRs cluster at the developing neuromuscular junctions.

34 functional defects at a subset of vulnerable neuromuscular junctions.

35 uggesting an effect on synaptogenesis beyond neuromuscular junctions.

36 rupts synaptic structure and function at the neuromuscular junctions.

37 ressive motor neuron loss and denervation of neuromuscular junctions.

38 that inhibits acetylcholine (ACh) release at neuromuscular junctions.

39 autoimmune disorder that selectively targets neuromuscular junctions.

40 he postsynaptic regions of the glutamatergic neuromuscular junctions.

41 ty at active zones of axon terminals at frog neuromuscular junctions.

42 inhibits nerve regeneration and destabilizes neuromuscular junctions.

43 s glutamatergic synaptogenesis in developing neuromuscular junctions.

44 he highly reliable transmission at zebrafish neuromuscular junctions.

45 the sciatic nerve, and axon terminals of the neuromuscular junctions.

46 evoked ACh release at Caenorhabditis elegans neuromuscular junctions.

47 by distinct synaptic scaffolds at C. elegans neuromuscular junctions.

48 ed release at Drosophila melanogaster embryo neuromuscular junctions.

49 ects on synaptic innervation and function at neuromuscular junctions.

50 e A GABA receptors (GABA(A)Rs) at inhibitory neuromuscular junctions.

51 d to support the function of motoneurons and neuromuscular junctions.

52 e, but did improve axonal (re)innervation of neuromuscular junctions.

53 d axonal swellings in their spinal cords and neuromuscular junctions.

54 l muscle by motor neurons occurs through the neuromuscular junction, a cholinergic synapse essential

55 g all Shank proteins and used the Drosophila neuromuscular junction, a model glutamatergic synapse, t

56 At the Drosophila neuromuscular junction, a presynaptic GluR, DKaiR1D, loc

57 eventually resulted in muscle fiber defects, neuromuscular junction abnormalities, compromised motor

58 or may be caused indirectly by neuronal and neuromuscular junction abnormalities.

59 NLP-12 directly modulates neuromuscular junction activity through the cholecystoki

60 ential for normal synaptic plasticity at the neuromuscular junction and for muscle strength, enduranc

61 diate fast chemical neurotransmission at the neuromuscular junction and have diverse signalling roles

62 xons during both postnatal maturation of the neuromuscular junction and myofiber reinnervation after

63 t the postsynaptic membrane of glutamatergic neuromuscular junctions and controls multiple parameters

64 eostasis at the Drosophila, mouse, and human neuromuscular junctions and emerging parallels at synapt

65 nerating across scar tissue, rebuilt healthy neuromuscular junctions and enhanced motor functional re

66 ceptors (AChRs) mediate signalling at mature neuromuscular junctions and fetal-type AChRs are necessa

67 sympathetic neurons make close contact with neuromuscular junctions and form a network in skeletal m

68 er axonal elongation in an in vitro model of neuromuscular junctions and hastened recovery after peri

69 physiological and histological properties of neuromuscular junctions and muscle at P21 and motoric de

70 the age-dependent innervation status of the neuromuscular junctions and mutant tau expression in Tg3

71 cation, caused by the massive elimination of neuromuscular junctions and pruning of axonal branches.

72 in motor units such as spinal motor neurons, neuromuscular junctions and skeletal muscle.

73 evels, the propriospinal projection network, neuromuscular junction, and central pattern generator, p

74 sHB-EGF expression was concentrated at the neuromuscular junction, and Hbegf deletion reduced Galgt

75 gy, and homeostatic plasticity at the larval neuromuscular junction, and impaired olfactory habituati

76 e respiratory rhythm generator and diaphragm neuromuscular junctions appeared normal.

77 th a reduction of AChR protein levels at the neuromuscular junction (approximately 25%) in Dok-7-siRN

78 nation, and gait and also related defects in neuromuscular junction architecture.

79 evelopment and evoked function of the larval neuromuscular junction are surprisingly normal, but the

80 Skeletal muscle and the neuromuscular junction are the earliest sites to manifes

81 eon mutants, postsynaptic specializations of neuromuscular junctions are dramatically expanded, inclu

82 use models of both diseases, suggesting that neuromuscular junctions are highly vulnerable from the v

83 Neuromuscular junctions are primary pathological targets

84 imary drivers of sarcopenia and identify the neuromuscular junction as a focal point of mTORC1-driven

85 Using Drosophila neuromuscular junction as a model synapse, we describe t

86 Using Caenorhabditis elegans neuromuscular junction as a model synapse, we uncovered

87 Using the Drosophila neuromuscular junction as a model, we found that non-enz

88 In conclusion, this study identifies the neuromuscular junction as a target of the sympathetic ne

89 its symptomatic treatment suggests that this neuromuscular junction assay has significant potential f

90 These formed 6618 synapses including 1772 neuromuscular junctions, augmented by 1206 gap junctions

91 hed larval peristaltic contractions, loss of neuromuscular junction bouton structures, impaired olfac

92 role in stabilizing the developing mammalian neuromuscular junction, but MuSK might also be protectiv

93 s that block neurotransmitter release at the neuromuscular junction by cleaving SNAREs (soluble N-eth

94 induces synaptic potentiation at the larval neuromuscular junction by increasing synaptic vesicle (S

95 Prevention of acetylcholine release at the neuromuscular junction causes long-lasting and potential

96 At Caenorhabditis elegans neuromuscular junctions, clustering of levamisole-sensit

97 s by electromyography found that a defective neuromuscular junction component is not always present.

98 aterals to reinnervate previously denervated neuromuscular junctions concurrently with expression of

99 motor neuron soma and maintaining functional neuromuscular junction connections are both essential el

100 cal deficits in synapse growth at the larval neuromuscular junction consistent with a dominant-negati

101 residue (p.Pro308Leu) produced a presynaptic neuromuscular junction defect and a dominant hereditary

102 e lacking miR-218 die neonatally and exhibit neuromuscular junction defects, motoneuron hyperexcitabi

103 ne sulfate was also observed when axonal and neuromuscular junction degeneration have already occurre

104 taline sulfate prevents defects in axons and neuromuscular junction degeneration in a dose-dependent

105 nificant reduction in muscle denervation and neuromuscular junction degeneration in homozygous mutant

106 de preserved ganglioside distribution at the neuromuscular junction, delayed disease onset, improved

107 However, it was not effective at preventing neuromuscular junction denervation in a mutant SOD1(G93A

108 nregulated and gene expression indicators of neuromuscular junction denervation were diminished using

109 uency stimulation, both wild-type and mutant neuromuscular junctions depress to steady-state response

110 In rsu-1 mutants, neuromuscular junctions differentiate as in the wild typ

111 ented with an autosomal-dominant presynaptic neuromuscular junction disorder resembling Lambert-Eaton

112 equirement for MYO9A in the formation of the neuromuscular junction during development.

113 ATP) levels and a higher fatigability at the neuromuscular junction during high energy demand.

114 expression of genes encoding proteins of the neuromuscular junction, especially acetylcholine recepto

115 n pathway that coordinates muscle growth and neuromuscular junction expansion.

116 in subsynaptic nuclei and remodeling of the neuromuscular junction following ischemia-induced denerv

117 pecification and somitogenesis and abolished neuromuscular junction formation and locomotion.

118 myofiber formation, long-term survival, and neuromuscular junction formation in vitro.

119 mmed cell and tissue death, neuromaturation, neuromuscular junction formation, and neuron cell fate d

120 als between neuron and muscle, which lead to neuromuscular junction formation, dysfunction of which i

121 We found that Agrin, a factor critical for neuromuscular junction formation, is elevated in the hip

122 as reduced motor axon branching and abnormal neuromuscular junction formation.

123 ein 4 (Lrp4), a protein that is critical for neuromuscular junction formation.

124 lcholine receptor (AChR) clustering and NMJ (neuromuscular junction) formation.

125 ction and preserved motor neurons as well as neuromuscular junctions from degeneration.

126 earance occurred at motor nerve terminals of neuromuscular junctions, from where anti-ganglioside ant

127 nervous system synapses and mouse diaphragm neuromuscular junctions fully intoxicated by BoNT seroty

128 sexes for defects in synaptic growth at the neuromuscular junction, identifying 12 mutants with seve

129 With dysfunction at the neuromuscular junction implicated as a key pathological

130 presynaptic neurotransmitter release at the neuromuscular junction in Drosophila.

131 m muscle nuclei to postsynaptic sites at the neuromuscular junction in Drosophila.

132 s unable to rescue structural defects at the neuromuscular junction in fragile x mental retardation 1

133 ed the effect of salbutamol treatment on the neuromuscular junction in the ColQ deficient mouse, a mo

134 Schwann cells cocultured with neurons and at neuromuscular junction in vivo through the MAPK pathway.

135 Furthermore, we compare the structure of neuromuscular junctions in 4- and 8-dpf zebrafish larvae

136 d muscle enables the formation of functional neuromuscular junctions in single organoids.

137 chitecture of larval zebrafish (Danio rerio) neuromuscular junctions in three dimensions.

138 ylcholine receptors for the establishment of neuromuscular junctions in vitro.

139 tive as therapies for human disorders of the neuromuscular junction, in particular many subsets of co

140 ch-dependent morphological phenotypes at the neuromuscular junction including synaptic size and prese

141 Our studies at the Drosophila neuromuscular junction indicate that many synaptic defec

142 At the Drosophila neuromuscular junction, inhibition of postsynaptic gluta

143 Maintenance of neuromuscular junction innervation during the course of

144 nescent mitochondria in their motor axons or neuromuscular junctions; instead, they contain far fewer

145 Skeletal muscle morphology and neuromuscular junction integrity was not different betwe

146 Neuromuscular junction is a synapse between motoneurons

147 f the effect of adrenergic signalling on the neuromuscular junction is essential to facilitate the de

148 the less prominent role of COL13A1 once the neuromuscular junction is mature.

149 hat axon branch loss at the developing mouse neuromuscular junction is mediated by branch-specific mi

150 ynaptic compartment of the Drosophila larval neuromuscular junction is regulated by the conserved RNA

151 n ortholog that is normally localized to the neuromuscular junction, is naturally upregulated in DMD

152 nd on the presynaptic nerve terminals at the neuromuscular junction level, but not on the axonal trac

153 sm underlying impaired neurotransmission and neuromuscular junction maintenance in SMA.

154 iate forms of SMA presenting with a delay in neuromuscular junction maturation and a decrease in the

155 fn1 to rodents promoted axonal regeneration, neuromuscular junction maturation, and functional recove

156 plasticity (PHP) is induced at degenerating neuromuscular junctions, mediated by an evolutionarily c

157 motor function and improved the integrity of neuromuscular junctions, MOE-modified ASO10-29 (MOE10-29

158 ction, and the effect on muscle strength and neuromuscular junction morphology was analysed.

159 such as lethality, wing and eye morphology, neuromuscular junction morphology, bang sensitivity and

160 12 d to a mean of 456 d, with improvement in neuromuscular junction morphology, down-regulation of tr

161 e of exocytosis in isolated nerve terminals, neuromuscular junctions, neuroendocrine cells and in hip

162 ments, anxiety, hippocampal LTP deficits and neuromuscular junction (NMJ) abnormalities, characterize

163 aladaptive changes within motor neurons, the neuromuscular junction (NMJ) and muscle fibres.

164 Using the Drosophila larval neuromuscular junction (NMJ) as a model, we provide evid

165 inal Schwann cells (tSCs) at the adult mouse neuromuscular junction (NMJ) by using mice expressing di

166 The neuromuscular junction (NMJ) consists of a tripartite sy

167 t GlyRS leads to motor deficits, progressive neuromuscular junction (NMJ) denervation and pre-synapti

168 sive loss of motor neurons (MNs) preceded by neuromuscular junction (NMJ) denervation.

169 hR clustering, a complete reversal of normal neuromuscular junction (NMJ) development where AChR clus

170 heir application to the study of adult human neuromuscular junction (NMJ) development, a process requ

171 anifesting deficits in coordinated movement, neuromuscular junction (NMJ) development, synaptic glyco

172 Neuromuscular junction (NMJ) disruption is an early path

173 al of Motor Neuron (SMN) protein, leading to neuromuscular junction (NMJ) dysfunction and spinal moto

174 mulate the LRP4-MuSK receptor in muscles for neuromuscular junction (NMJ) formation.

175 SK is one of the key molecules orchestrating neuromuscular junction (NMJ) formation.

176 Although the frog neuromuscular junction (NMJ) has long been a model synap

177 complex and regulates its expression at the neuromuscular junction (NMJ) in Drosophila.

178 ptobrevin-2, are expressed at the developing neuromuscular junction (NMJ) in mice, but their specific

179 ia, the terminal Schwann cells (SCs), at the neuromuscular junction (NMJ) in mice.

180 ve recently reported on the pathology of the neuromuscular junction (NMJ) in Pompe disease, reflectin

181 ed and retained at postsynaptic sites at the neuromuscular junction (NMJ) in vivo remains largely unk

182 ces motor end plate volume without affecting neuromuscular junction (NMJ) integrity.

183 ctions requires neural activity; because the neuromuscular junction (NMJ) is a cholinergic synapse, a

184 The Drosophila melanogaster larval neuromuscular junction (NMJ) is a model synapse with rob

185 The neuromuscular junction (NMJ) is a specialized synapse th

186 The neuromuscular junction (NMJ) is a synapse formed between

187 The neuromuscular junction (NMJ) is a tripartite synapse tha

188 hether weakened synaptic transmission at the neuromuscular junction (NMJ) is an aspect of CMT2D.

189 SIGNIFICANCE STATEMENT: The neuromuscular junction (NMJ) is critical for all volunta

190 ectrical response.SIGNIFICANCE STATEMENT The neuromuscular junction (NMJ) is designed to faithfully e

191 The neuromuscular junction (NMJ) is one of the best-studied

192 A hallmark of the neuromuscular junction (NMJ) is the high density of acet

193 The neuromuscular junction (NMJ) is the site of a number of

194 While the morphology of the neuromuscular junction (NMJ) is typically affected by ne

195 s suggest that pathological targeting of the neuromuscular junction (NMJ) may play a key role in cach

196 Here, we test the roles of Mmp at the neuromuscular junction (NMJ) model synapse in the reduct

197 ling stabilizes synaptic transmission at the neuromuscular junction (NMJ) of Drosophila, mice, and hu

198 electron microscopy at the adult Drosophila neuromuscular junction (NMJ) of ventral abdominal muscle

199 Denervation of the neuromuscular junction (NMJ) precedes the loss of motor

200 The neuromuscular junction (NMJ) provides the interface betw

201 utions of a macrophage-mediated response for neuromuscular junction (NMJ) reinnervation following ner

202 The Drosophila larval glutamatergic neuromuscular junction (NMJ) represents a powerful synap

203 At the glutamatergic neuromuscular junction (NMJ) synapse, we find that Notum

204 The postsynaptic apparatus of the neuromuscular junction (NMJ) traps and anchors acetylcho

205 pled to skeletal muscles interacting via the neuromuscular junction (NMJ) within a microfluidic devic

206 Aging can also impact the neuromuscular junction (NMJ), a synapse that transmits s

207 The Drosophila larval neuromuscular junction (NMJ), at which glutamate acts as

208 At the neuromuscular junction (NMJ), early alterations in peris

209 of NAMPT on synaptic vesicle cycling in the neuromuscular junction (NMJ), end-plate structure of NMJ

210 n to be post-translationally modified at the neuromuscular junction (NMJ), hence increasing their sta

211 lized, non-myelinating, synaptic glia of the neuromuscular junction (NMJ), that participate in synaps

212 Here we show that in the Drosophila neuromuscular junction (NMJ), the endocytic scaffolding

213 on of glial cell activity takes place at the neuromuscular junction (NMJ), the output of motor neuron

214 le specific receptor tyrosine kinase) at the neuromuscular junction (NMJ), thereby preventing fragmen

215 ed y+z+ agrin regulates the formation of the neuromuscular junction (NMJ), while y-z- agrin is widely

216 naptic growth, structure and function at the neuromuscular junction (NMJ).

217 dysfunction and progressive weakening of the neuromuscular junction (NMJ).

218 is crucial for vesicle fusion at presynaptic neuromuscular junction (NMJ).

219 nd intensity of ACh receptor labeling at the neuromuscular junction (NMJ).

220 e morphogenic protein (BMP) signaling at the neuromuscular junction (NMJ).

221 c receptor function at the Drosophila larval neuromuscular junction (NMJ).

222 motoneurons and motor nerve terminals of the neuromuscular junction (NMJ).

223 maintenance of the synaptic structure of the neuromuscular junction (NMJ).

224 (SSR) at the Drosophila melanogaster larval neuromuscular junction (NMJ).

225 ators of synapse morphogenesis at the larval neuromuscular junction (NMJ).

226 in (BMP) signaling pathway at the Drosophila neuromuscular junction (NMJ).

227 n has been extensively studied at the rodent neuromuscular junction (NMJ).

228 le degeneration, denervation, neuromuscular [neuromuscular junction (NMJ)] abnormalities, and axonal

229 We show that at larval neuromuscular junctions (NMJ), motor neuron expression o

230 nges in mouse models of fALS are seen in the neuromuscular junctions (NMJs) and lower motor neurons,

231 Neuromuscular junctions (NMJs) are critical for survival

232 ar architecture of active zones in mammalian neuromuscular junctions (NMJs) at sub-diffraction limite

233 As mammals age, their neuromuscular junctions (NMJs) gradually change their fo

234 rate significant pathological alterations at neuromuscular junctions (NMJs) of the diaphragm and tibi

235 Schwann cells (SCs) at neuromuscular junctions (NMJs) play active roles in syna

236 tical for the development and maintenance of neuromuscular junctions (NMJs) remains largely unknown.

237 By studying Drosophila neuromuscular junctions (NMJs) we show that AZs consist

238 CMNs form anatomically mature and functional neuromuscular junctions (NMJs) when cocultured with chic

239 At larval neuromuscular junctions (NMJs), DCAF12 is expressed pres

240 ility of motor neurons and their output, the neuromuscular junctions (NMJs), has been considered a ke

241 At mammalian neuromuscular junctions (NMJs), prolonged inactivity lea

242 e the morphology of their previously damaged neuromuscular junctions (NMJs), suggesting that the bene

243 pruning also occurs at embryonic Drosophila neuromuscular junctions (NMJs), where low-frequency Ca(2

244 ive disorders resulting from degeneration of neuromuscular junctions (NMJs), which form the connectio

245 on and number of proprioceptive synapses and neuromuscular junctions (NMJs), while having no effects

246 f SMN deficiency are profound defects of the neuromuscular junctions (NMJs).

247 king of a pHluorin-tagged Syt4 at Drosophila neuromuscular junctions (NMJs).

248 maintenance depends on motor innervation at neuromuscular junctions (NMJs).

249 vious loss of boutons and synapses at larval neuromuscular junctions (NMJs).

250 Neuromuscular junction number was unaltered in the media

251 ere alterations in synaptic growth using the neuromuscular junction of Drosophila melanogaster as a m

252 At the embryonic neuromuscular junction of Drosophila melanogaster, mutat

253 At the neuromuscular junction of female Drosophila larvae, we o

254 local concentration of acetylcholine at the neuromuscular junction of frog cutaneous pectoris muscle

255 gnificant decrease in the denervation of the neuromuscular junction of the tibialis anterior muscle i

256 In addition, the neuromuscular junctions of salbutamol treated mice showe

257 We show for the first time that neuromuscular junctions of the extraocular muscles (resp

258 Conversely, neuromuscular junctions of the lumbrical muscles of the

259 Neuromuscular junctions on slow fibers became multiply i

260 roved neuromuscular function and ameliorated neuromuscular junction pathology in SMA mice.

261 cluding increased motor neuron size, reduced neuromuscular junction pathology, increased muscle fiber

262 essing defects induced by SMN deficiency and neuromuscular junction pathology.

263 complement proteins that target muscle, the neuromuscular junction, peripheral nerves, the spinal co

264 y, we examined the relative contributions of neuromuscular junction physiology and the motor program

265 on and motor defects, but not the defects in neuromuscular junction physiology.

266 y autoantibodies that target proteins at the neuromuscular junction, primarily the acetylcholine rece

267 is an autoimmune disease in which Abs target neuromuscular junction proteins, in particular the acety

268 es motor neuron regeneration and accelerates neuromuscular junction re-innervation.

269 flexor digitorum superficialis atrophy, and neuromuscular junction reinnervation.

270 targeted to synaptic sites at the vertebrate neuromuscular junction remains unclear.

271 n increased number of active zones in larval neuromuscular junctions, representing large glutamatergi

272 Uptake at the neuromuscular junction represents a major unexpected pat

273 e overextension of axons and mistargeting of neuromuscular junctions, resulting in uncoordinated loco

274 well as the need to look outside traditional neuromuscular junction-specific proteins for further con

275 Moreover, compared with the neuromuscular junction, steps regulating the synaptogeni

276 changes to the structure and function of the neuromuscular junction, suggesting novel roles for MuSK

277 in SMA changes appeared concomitantly at the neuromuscular junction, suggesting that mechanisms of ne

278 P2B(Intron5), at the Drosophila melanogaster neuromuscular junction synapse revealed synaptic overgro

279 sted synaptic facilitation and depression in neuromuscular junction synapses that use exclusively CaV

280 Salbutamol enhances neuromuscular junction synaptic structure by counteracti

281 keletal muscle builds a functional all-human neuromuscular junction that can be triggered to twitch u

282 Loss of TMEM184b causes swellings at neuromuscular junctions that become more numerous with a

283 At GABAergic neuromuscular junctions, the short isoform MADD-4B binds

284 ation of the actin cytoskeleton in axons and neuromuscular junctions to protect motoneurons in SMA.

285 orms of homeostatic plasticity at Drosophila neuromuscular junctions to stabilize synaptic communicat

286 n was slackened mechanically by bringing the neuromuscular junction toward the central nervous system

287 of surviving motor units and instability of neuromuscular junction transmission.

288 ophysiological and morphological deficits of neuromuscular junctions upon sympathectomy and in myasth

289 ChE) are anchored in the basal lamina of the neuromuscular junction using a collagen-like tail subuni

290 c proteoglycan with critical function at the neuromuscular junction was previously found mutated in m

291 Green) uptake in the presynaptic terminal of neuromuscular junctions was restored to control levels i

292 uper-resolution microscopy at the Drosophila neuromuscular junction we quantitatively map vesicle:Ca(

293 At the neuromuscular junction, we showed mEPP amplitudes and fr

294 ed postsynaptic scaffold proteins within the neuromuscular junction were completely eliminated.

295 Tongue muscle neuromuscular junctions were also spared in both animal

296 The development and maturation of neuromuscular junctions were not disrupted in Tg30 mice,

297 annel is highly expressed at the presynaptic neuromuscular junction where it contributes to action po

298 Here experiments at the Drosophila neuromuscular junction, where DCVs contain neuropeptides

299 vesicle recycling pathways at complexin null neuromuscular junctions, where spontaneous release is dr

300 with kinesin, only on nuclei associated with neuromuscular junctions, whereas all adult cardiomyocyte