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1 ial dystonic seizures, Morvan's syndrome and neuromyotonia.
2 toms, electrophysiologically confirmed to be neuromyotonia.
3 s sampled, which suggests the possibility of neuromyotonia.
4  had a history of episodic ataxia and 19 had neuromyotonia.
5 halitis and seizures, Morvan's syndrome, and neuromyotonia.
6 current trains of axonal firing described in neuromyotonia.
7 re similar to the clinical manifestations of neuromyotonia.
8 encephalitis, Morvan's syndrome and acquired neuromyotonia.
9 eatures and outcomes of patients with ocular neuromyotonia.
10 ls in a large series of patients with ocular neuromyotonia.
11 sms of cerebellar dysfunction and persistent neuromyotonia.
12 ravenous immunoglobulin, considering primary neuromyotonia.
13                                              Neuromyotonia (100%), neuropsychiatric features (insomni
14 adiation is the most common cause for ocular neuromyotonia, affecting the sixth cranial nerve most of
15 associated with a broad range of phenotypes: neuromyotonia alone or with seizures, EA1 with seizures,
16 on of voltage-gated K(+) channels (VGKCs) in neuromyotonia and demyelination in peripheral neuropathi
17 des of cerebellar dysfunction and persistent neuromyotonia and is associated with an increased incide
18 l seizures, prominent amnesia, dysautonomia, neuromyotonia and neuropathic pain.
19 ied antibody-mediated mechanisms in acquired neuromyotonia, and began the molecular work that identif
20 roup of patients with undulating myokymia or neuromyotonia, and EMG doublet or multiplet ('myokymic')
21 s case highlights the overlap of myasthenia, neuromyotonia, and thymoma, emphasizing the importance o
22 mbert Eaton myasthenic syndrome and acquired neuromyotonia are caused by antibodies to voltage-gated
23 uch as Lambert-Eaton myasthenic syndrome and neuromyotonia are clearly mediated by autoantibodies.
24 mplex proteins.We present a case of acquired neuromyotonia as the heralding symptom of recurrent thym
25         Although neither patient had obvious neuromyotonia at presentation, both showed excessive sec
26 ed protein, or glycine receptor antibodies), neuromyotonia (Caspr2 antibodies), and opsoclonus--myocl
27               Patients diagnosed with ocular neuromyotonia from January 1, 2004, through January 1, 2
28                                     Acquired neuromyotonia is increasingly recognized as an autoimmun
29 se many terms including undulating myokymia, neuromyotonia, Isaacs' syndrome and Cramp-Fasciculation
30                 Distinctive "tornado-shaped" neuromyotonia-like discharges may be seen in patients wi
31 ases of fetal arthrogryposis and in acquired neuromyotonia, Morvan's syndrome and Miller-Fisher syndr
32 tissue have been identified in patients with neuromyotonia, Morvan's syndrome, limbic encephalitis an
33 essed Shaker-type K+ channels cause acquired neuromyotonia, MoS and LE, and suggest that future assay
34 antibody with limbic encephalitis (n = 1) or neuromyotonia (n = 1), and collapsin response-mediator p
35  neural tissues with sera from patients with neuromyotonia (n = 10), MoS (n = 2) or LE (n = 5), compa
36 in several neurological syndromes, including neuromyotonia (NMT), Morvan syndrome (MoS), and limbic e
37 tactin-associated protein-antibody-2, 10 had neuromyotonia or Morvan's syndrome, compared with only 3
38                         In the rare acquired neuromyotonia, reduced repolarization of the nerve termi
39                                 In contrast, neuromyotonia sera bound more strongly to Kv1.2 subunits
40 .1 subunits more prominently than did MoS or neuromyotonia sera, suggesting an association between hi
41                                              Neuromyotonia should be considered in the differential d
42 ility is the chief manifestation of acquired neuromyotonia; the combination of neuromyotonia with aut
43          We ascertained patients with ocular neuromyotonia through a search using the medical records
44  patients with an observed episode of ocular neuromyotonia were included and the medical records were
45 -two patients who were diagnosed with ocular neuromyotonia were included.
46                         Only one patient had neuromyotonia, which was excluded by electromyography in
47 f acquired neuromyotonia; the combination of neuromyotonia with autonomic and CNS involvement is call