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1 rrelate with alterations in the formation of neuronal intranuclear inclusions.
2 d characteristic lentiform ub-immunoreactive neuronal intranuclear inclusions.
3 PGRN) gene, particularly in those cases with neuronal intranuclear inclusions.
4 ressive, gender-specific motor deficits, and neuronal intranuclear inclusions.
5 n Huntington's disease, with the presence of neuronal intranuclear inclusions.
6 neuronal loss and gliosis and the absence of neuronal intranuclear inclusions.
7 rites in both grey and white matter and also neuronal intranuclear inclusions.
8 ion, particularly visceral neuropathies with neuronal intranuclear inclusions.
9 e disorders, including some characterized by neuronal intranuclear inclusions.
10                                              Neuronal intranuclear inclusions and cytoplasmic aggrega
11 ma-positive neuronal cytoplasmic inclusions, neuronal intranuclear inclusions and neurites were recor
12 d limb ataxia, weight loss, premature death, neuronal intranuclear inclusions, and decreased tyrosine
13                                              Neuronal intranuclear inclusions are found in the brains
14                                              Neuronal intranuclear inclusions are present in affected
15                              We suggest that neuronal intranuclear inclusions are the common neuropat
16                                     Striatal neuronal intranuclear inclusion burden was similar betwe
17            Given that MSP is associated with neuronal intranuclear inclusions comprised of TDP-43 pro
18                                              Neuronal intranuclear inclusion disease (NIID) is a mult
19                                              Neuronal intranuclear inclusion disease (NIID) is a neur
20                                              Neuronal intranuclear inclusion disease (NIID) is a slow
21 have been identified as the genetic cause of neuronal intranuclear inclusion disease (NIID).
22 fragile X-associated tremor/ataxia syndrome, neuronal intranuclear inclusion disease, oculopharyngeal
23                                              Neuronal intranuclear inclusions have been found in the
24 ein (CBP) and mSin3a, and CBP to localize to neuronal intranuclear inclusions in a transgenic mouse m
25 cts is directly relevant to the formation of neuronal intranuclear inclusions in Huntington's disease
26                   The discovery last year of neuronal intranuclear inclusions in Huntington's disease
27 cle pathology and by the late development of neuronal intranuclear inclusions in spinal neurons.
28 deficits, weight loss, cerebral atrophy, and neuronal intranuclear inclusions in the R6/2 transgenic
29                                              Neuronal intranuclear inclusion (NII) formation and cell
30             Patient autopsy material reveals neuronal intranuclear inclusions (NII) in affected regio
31 agment of mutant huntingtin was localized to neuronal intranuclear inclusions (NIIs) and dystrophic n
32  brain and hastened both the presentation of neuronal intranuclear inclusions (NIIs) and the onset of
33 (AF) as early as 9 weeks of age and striatal neuronal intranuclear inclusions (NIIs) by 20 weeks.
34                                              Neuronal intranuclear inclusions (NIIs) characteristical
35 The cellular localization and development of neuronal intranuclear inclusions (NIIs) in cortex and st
36   The transgenic mice described here develop neuronal intranuclear inclusions (NIIs), a hallmark of S
37 criptional dysregulation and accumulation of neuronal intranuclear inclusions (NIIs).
38 analysis demonstrates widespread ubiquinated neuronal intranuclear inclusions (NIIs).
39 ith development of both disease symptoms and neuronal intranuclear inclusions (NIIs).
40 t death, other than widespread ubiquitinated neuronal intranuclear inclusions (NIIs).
41  neurites, and in some familial cases UBQ-ir neuronal intranuclear inclusions of a lentiform appearan
42 ities, reactive gliosis and the formation of neuronal intranuclear inclusions predominating in the st
43 hin the hippocampus mirror the appearance of neuronal intranuclear inclusions, suggesting a relations
44 ic mouse models of Huntington's disease with neuronal intranuclear inclusions, the identification of