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1 avage of a host protein-derived peptide (pro-neuropeptide Y).
2 OM), vasoactive intestinal peptide (VIP), or neuropeptide Y.
3 es such as stromal cell-derived factor-1 and neuropeptide Y.
4  decarboxylase-67 (GAD67), somatostatin, and neuropeptide Y.
5 ell type-specific distribution of endogenous neuropeptide Y.
6 e pool neurons that already express GABA and neuropeptide Y.
7 amage, and compensatory changes in EAAT3 and neuropeptide Y.
8  estrogen receptor-alpha colocalization with neuropeptide Y.
9 es and peptides such as leptin, ghrelin, and neuropeptide Y.
10  abundant levels of tyrosine hydroxylase and neuropeptide Y.
11 ferent orexigenic molecules: AgRP; GABA; and neuropeptide Y.
12 lar (3V) infusions of the orexigenic peptide neuropeptide Y 3-36 in awake, freely moving rats and det
13         dNPF is an ortholog of the mammalian neuropeptide Y, a highly conserved neuromodulator that s
14 rocessed for immunocytochemistry for SST and neuropeptide Y, a neuropeptide partially coexpressed in
15          In the current study, we found that neuropeptide Y, a protein produced by neurons, affected
16                         Neurons coexpressing neuropeptide Y, agouti-related peptide, and GABA (NAG) p
17                                 Hypothalamic neuropeptide Y, agouti-related peptide, and proopiomelan
18 ial role of central alpha-klotho to modulate neuropeptide Y/agouti-related peptide (NPY/AgRP)-express
19 icted neuronal subsets (proopiomelanocortin, neuropeptide Y/agouti-related peptide, and steroidogenic
20 ng hormone neurons but has limited effect on neuropeptide Y/agouti-related protein and proopiomelanoc
21 ons of the arcuate nucleus, specifically the neuropeptide Y/agouti-related protein neurons and the do
22 of tanycytes ex vivo depolarized orexigenic (neuropeptide Y/agouti-related protein; NPY/AgRP) and ano
23 ances the activity of agouti-related protein/neuropeptide Y (AgRP/NPY)-expressing neurons but induces
24 ls containing parvalbumin, somatostatin, and neuropeptide Y also show unique topographical distributi
25  approaches we show this plasticity requires neuropeptide Y, an adrenal cotransmitter and the activat
26 f sympathetic nerves and tissue abundance of neuropeptide-Y, an indicator of sympathetic nerve activi
27 (-/-) mice, and other transcripts, including neuropeptide Y and activating transcription factor-3, ar
28  reduced appetite-stimulating neuropeptides, neuropeptide Y and Agouti-related peptide.
29 d by arcuate nucleus neurons that co-express neuropeptide Y and agouti-related protein (NPY/AgRP neur
30 d food intake, body weight, and hypothalamic neuropeptide Y and Agouti-related protein mRNA expressio
31 nduced hepatic FGF21, which caused increased neuropeptide Y and agouti-related protein mRNAs in the h
32 he regulation of social behavior (especially neuropeptide Y and corticotropin-releasing factor) are m
33  disorder are enhanced hippocampal levels of neuropeptide Y and EAAT3 and increased calpain proteolys
34 at co-express agouti-related protein (AgRP), neuropeptide Y and gamma-aminobutyric acid (GABA) are kn
35 he receptor binding motifs of members of the neuropeptide Y and glucagon families modulate receptor a
36  control animals and had lower expression of neuropeptide Y and higher expression of proopiomelanocor
37                 In the arcuate nucleus, both Neuropeptide Y and proopiomelanocortin cells expressed t
38 f brainstem and hypothalamic neurons express neuropeptide Y and proopiomelanocortin in the arcuate nu
39  of GABAergic GAD67+ interneurons expressing neuropeptide Y and somatostatin are reduced in the hippo
40 2.2 is not required for the specification of neuropeptide Y and vasoactive intestinal polypeptide, in
41 coneogenic enzymes and elevated hypothalamic neuropeptide-Y and agouti-related protein mRNA levels.
42 calized with cholinergic starburst ACs, NPY (neuropeptide Y)- and EBF1 (early B-cell factor 1)-positi
43 n analogue of the C-terminal pentapeptide of neuropeptide Y, and a neuropeptide FF analogue) were sub
44 othalamic neuropeptides proopiomelanocortin, neuropeptide Y, and agouti-related peptide in T1D mice.
45     Ivy cells express nitric oxide synthase, neuropeptide Y, and high levels of GABA(A) receptor alph
46  against calbindin, parvalbumin, calretinin, neuropeptide Y, and somatostatin, and the number of inte
47 -regulating proteins agouti-related peptide, neuropeptide Y, and uncoupling protein 2 in the hypothal
48 hich express the orexigenic neuronal marker, Neuropeptide-Y, and respond to fasting and leptin-induce
49            Agouti-related peptide (AGRP) and Neuropeptide Y are potent orexigens and are coexpressed
50 ress characteristic combinations of GABA and neuropeptide Y as cotransmitters and Lim1,2 and Nurr1 tr
51 the insulin promotor (Ins2) were stained for neuropeptide Y before, during, and after virally induced
52 s that synthesize agouti-related peptide and neuropeptide Y but inhibited anorexigenic neurons that s
53 e BBB-impermeable neuropeptides dalargin and neuropeptide Y chemically conjugated with FC5-Fc fusion
54 .93]) after stressful training; lower plasma neuropeptide Y concentration after stressful training (d
55 ures were heart rate, breathing rate, plasma neuropeptide Y concentration, score on the Response to S
56                                              Neuropeptide Y-containing interneurons in the dentate hi
57 rvated by inhibitory interneurons expressing neuropeptide Y::Cre (NPY(+)) in the dorsal spinal cord.
58 eurons that are defined by the expression of neuropeptide Y::Cre (NPY::Cre) act to gate mechanical it
59                                              Neuropeptide Y detection was performed.
60 cholecystokinin, but inhibited by orexigenic neuropeptide Y, dynorphin and met-enkephalin, consistent
61 the mammalian target of rapamycin pathway in neuropeptide-Y-ergic neurons of the arcuate nucleus, and
62  A significant reduction of calbindin-, NPY (neuropeptide Y)-expressing, and cholinergic interneurons
63 ic inhibitory circuit involving a variety of neuropeptide Y-expressing interneurons.
64 endrites, whereas nitric oxide synthase- and neuropeptide Y-expressing ivy cells provided synaptic an
65                        In addition, we found neuropeptide Y expression almost exclusively in those GI
66                   Surprisingly, hypothalamic neuropeptide Y expression was completely suppressed, sug
67 nders to two model peptides, angiotensin and neuropeptide Y, following screening by solution phage pa
68 g mice showed increased levels of C-terminal neuropeptide Y fragments and increased neurogenesis.
69  single nucleotide polymorphism (SNP) in the neuropeptide Y gene has been associated with elevated se
70 bilaterian neuropeptide receptors, including neuropeptide Y, gonadotropin inhibitory hormone (GnIH),
71                    Both serotonin (5-HT) and neuropeptide Y have been shown to affect a variety of ma
72 essing Drosophila neuropeptide F (dNPF), the neuropeptide Y homolog, strongly correlates with food-od
73 leasing short neuropeptide F, the Drosophila neuropeptide Y homolog.
74 used immunohistochemistry for calretinin and neuropeptide Y in 24 age- and gender-matched patients wi
75 nthesize gamma-amino-butyric acid (GABA) and neuropeptide Y in adult mice leads to starvation within
76 d release of the orexigenic neurotransmitter neuropeptide Y in response to glucose.
77 ons expressing parvalbumin, somatostatin and neuropeptide Y in the dentate gyrus, reduced aberrant ne
78  pro-opiomelanocortin/agouti-related peptide/neuropeptide Y in the hypothalamus of the rats.
79 rvating (calretinin(+), somatostatin(+), and neuropeptide Y(+)) interneurons.
80                                   Numbers of neuropeptide Y-IR neurons were not affected.
81 osal plexus and was expressed exclusively by neuropeptide Y-IR neurons.
82                                              Neuropeptide Y is a key peptide affecting adiposity and
83  neuropeptides such as ghrelin, orexin A and neuropeptide Y is also discussed.
84          Multiple peptide systems, including neuropeptide Y, leptin, ghrelin, and others, are involve
85 P, corticotropin releasing factor (CRF), and neuropeptide Y levels in adult male Long-Evans rats defe
86 n and attraction is thought to depend on the neuropeptide Y-like receptor NPR-1, because solitary and
87 ting cells and identified neurons expressing neuropeptide Y-like short neuropeptide F (sNPF).
88 -expressing interneurons and initiation of a neuropeptide-y-like signaling axis that promotes elevate
89 cystokinin/CB(1) cannabinoid receptor(+) and neuropeptide Y(+) local-circuit interneurons upon SAVA m
90 f both Lifeact-GFP and the SG marker protein neuropeptide Y-mCherry reveals that SGs actively translo
91         Aptamer-modified microelectrodes for Neuropeptide Y measurement by electrochemical impedance
92  and inhibited by orexigenic neuromodulators neuropeptide Y, met-enkephalin, dynorphin and the catech
93 tissue weight, fecal output, arcuate nucleus neuropeptide Y mRNA expression, plasma corticosterone, a
94 ng hormone but surprisingly has no effect on neuropeptide Y mRNA, a neuropeptide previously shown to
95                                              Neuropeptide Y neurons have been shown to be particularl
96 s, stimulation of the agouti-related protein/neuropeptide Y neurons, and activation of the hypothalam
97  activated L-type Ca(2+) currents in arcuate neuropeptide Y neurons.
98 lly significant difference in the density of neuropeptide Y+ neurons between subjects with autism and
99 coordinated action of Caenorhabditis elegans neuropeptide Y/neuropeptide F and serotonin that could a
100 , we identified the evolutionary ortholog of neuropeptide Y/neuropeptide F in the nematode Caenorhabd
101 n, corticotropin releasing hormone, galanin, neuropeptide Y, neurotensin, preproenkephalin and tachyk
102 in, dynorphin) and brain antistress systems (neuropeptide Y, nociceptin [orphanin FQ]) in drug depend
103 r, metabotropic glutamate receptors-2 mGlu2, neuropeptide-Y NPY, and mineralocorticoid receptors MR).
104 l stages influences IGL neurons that express neuropeptide Y (NPY) (hereafter, IGL(NPY) neurons), guid
105 d identify the vasoconstrictive mechanism as Neuropeptide Y (NPY) acting on Y1 receptors.
106 e truncated cleaved form of neurotransmitter neuropeptide Y (NPY) actively promotes a breach of BM va
107                                              Neuropeptide Y (NPY) administration into the basolateral
108                 Bioconjugates containing the neuropeptide Y (NPY) analogue [F(7),P(34)]-NPY as target
109  In lean mice, the adipokine leptin inhibits neuropeptide Y (NPY) and agouti-related peptide (AgRP) n
110 bits neurotransmission in neurons expressing neuropeptide Y (NPY) and agouti-related peptide (AgRP) v
111                                              Neuropeptide Y (NPY) and Agouti-related protein (AgRP),
112                                   Endogenous neuropeptide Y (NPY) and corticotrophin-releasing factor
113 s that regulate stress and reward, including neuropeptide Y (NPY) and corticotropin-releasing factor
114 s, including somatostatin (SST), tachykinin, neuropeptide Y (NPY) and cortistatin, in a pattern remin
115 was developed for the sensitive detection of neuropeptide Y (NPY) and employed in the analysis of NPY
116 c), containing pro-opoiomelanocortin (POMC), neuropeptide Y (NPY) and growth hormone releasing hormon
117                                              Neuropeptide Y (NPY) and its receptors (especially Y1, Y
118 up-regulating specific target genes, such as neuropeptide Y (NPY) and its Y1 and Y5 receptors (Y5Rs).
119                                              Neuropeptide Y (NPY) and NPY receptors represent a highl
120 rgetic state, circulating steroids, and both neuropeptide Y (NPY) and orexin (OX) immunoreactivity.
121 and modulation of neurological signals, with Neuropeptide Y (NPY) and Orexin A (OXA) offering diagnos
122                                              Neuropeptide Y (NPY) and pancreatic polypeptide (PP) con
123  the lateral hypothalamus (LH) together with neuropeptide Y (NPY) and proopiomelanocortin (POMC) neur
124  the total number of interneurons expressing neuropeptide Y (NPY) and vasoactive intestinal polypepti
125                       Other peptides such as neuropeptide Y (NPY) are synthesized throughout the brai
126                           Orexin A (OXA) and neuropeptide Y (NPY) are two hypothalamic neuropeptides
127 sh, and here we report the identification of neuropeptide Y (NPY) as both necessary for normal daytim
128 in a structural model of the peptide hormone neuropeptide Y (NPY) bound to its human G-protein-couple
129 in the basolateral amygdaloid complex (BLA), neuropeptide Y (NPY) buffers against protracted anxiety
130 e ontogeny of proopiomelanocortin (POMC) and neuropeptide Y (NPY) cell populations, which exert oppos
131 stimulatory connections onto the neighboring neuropeptide Y (NPY) cells.
132                             The hypothalamic neuropeptide Y (NPY) circuitry is a key regulator of fee
133  strongly implicated hindbrain catecholamine/neuropeptide Y (NPY) coexpressing neurons as key mediato
134 ression of agouti-related protein (Agrp) and neuropeptide Y (Npy) coincide with the cyclic changes in
135 c (tg) mice affected primarily the levels of neuropeptide Y (NPY) compared with other neuropeptides.
136 subpopulation of CA1 interneurons expressing neuropeptide Y (NPY) contributes prominently to this dyn
137                                              Neuropeptide Y (NPY) counters stress and is involved in
138  we show that the claims of Zhou et al. that neuropeptide Y (NPY) diplotype-predicted expression is c
139          The DMH contains neurons expressing Neuropeptide Y (NPY) during specific physiological condi
140                                   Endogenous neuropeptide Y (NPY) exerts long-lasting spinal inhibito
141 s glucocorticoids have pronounced effects on neuropeptide Y (NPY) expression and as NPY neurons proje
142                                We found that neuropeptide Y (NPY) expression identifies a class of GA
143 pproach in mice of both sexes, we found that neuropeptide Y (NPY) expression identifies a major class
144 gered molecular plasticity including ectopic neuropeptide Y (NPY) expression in granule cells, but wi
145  element binding) binding protein (CBP), and neuropeptide Y (NPY) expression in the amygdaloid brain
146                                      Because neuropeptide Y (NPY) expression is strongly induced in D
147 pression of vGLUT1 but not vGAT, and reduced Neuropeptide Y (NPY) expression.
148 nes and their cognate receptors belonging to neuropeptide Y (NPY) family mediate diverse biological f
149 ucagon-like peptide-1 (GLP-1), glucagon, and neuropeptide Y (NPY) from circumvallate papillae of Tas1
150                                              Neuropeptide Y (NPY) gene expression in the hippocampal
151  anxiety and other psychiatric disorders and neuropeptide Y (NPY) has emerged as a key component of a
152                                              Neuropeptide Y (NPY) has robust anxiolytic properties an
153                      SIGNIFICANCE STATEMENT: Neuropeptide Y (NPY) has robust anxiolytic properties, a
154               While recent animal studies of neuropeptide Y (NPY) have suggested a facilitator role f
155         Previous studies have suggested that neuropeptide Y (NPY) in the dorsomedial hypothalamus (DM
156             The expression of the orexigenic neuropeptide Y (NPY) in the hypothalamus to the low lept
157 ted by changes in tonic inhibition of SNA by neuropeptide Y (NPY) in the paraventricular nucleus (PVN
158 ted with reduced tonic inhibition of LSNA by neuropeptide Y (NPY) in the PVN.
159                However, the specific role of neuropeptide Y (NPY) in this process has not been system
160                                              Neuropeptide Y (NPY) interacts with the Y(1) receptor, N
161 ic inputs to the two populations of striatal neuropeptide Y (NPY) interneurons, plateau low threshold
162                                              Neuropeptide Y (NPY) is a 36-amino acid peptide circulat
163                                              Neuropeptide Y (NPY) is a 36-amino-acid peptide transmit
164                                              Neuropeptide Y (NPY) is a hypothalamic neuropeptide that
165                                              Neuropeptide Y (NPY) is a peptide known for its anti-anx
166                                              Neuropeptide Y (NPY) is a putative endogenous anxiolytic
167                                              Neuropeptide Y (NPY) is a well-established orexigenic pe
168                                              Neuropeptide Y (NPY) is an important modulatory neuropep
169                                              Neuropeptide Y (NPY) is anxiolytic and its release is in
170 EMENT Within the basolateral amygdala (BLA), neuropeptide Y (NPY) is associated with buffering the ne
171                                              Neuropeptide Y (NPY) is induced in peripheral tissues su
172                                              Neuropeptide Y (NPY) is one of the major neuropeptides p
173                                              Neuropeptide Y (NPY) is one of the most abundant protein
174                                              Neuropeptide Y (NPY) is one of the most widespread neuro
175 cord following inflammation or nerve injury, neuropeptide Y (NPY) is poised to regulate the transmiss
176 ported that the sympathetic neurotransmitter neuropeptide Y (NPY) is potently angiogenic, primarily t
177                                              Neuropeptide Y (NPY) is produced in the intergeniculate
178                            Here we show that Neuropeptide Y (NPY) is uniquely required for the long-l
179 evious observations that fluorescent-labeled neuropeptide Y (NPY) is usually released within 200 ms a
180                                  Among them, neuropeptide Y (NPY) is well known to promote aversive m
181                                              Neuropeptide Y (NPY) is widely expressed in the central
182                            ICV TTR decreased neuropeptide Y (NPY) levels in the DMH and the paraventr
183  understand whether genetic variation at the Neuropeptide Y (NPY) locus governs secretion and stress
184                                              Neuropeptide Y (NPY) mediates stress-induced obesity in
185 cordings were made from GFP (Renilla)-tagged neuropeptide Y (NPY) neurons from the arcuate nucleus of
186                                              Neuropeptide Y (NPY) neurons in both the arcuate nucleus
187 nhibitory effect of Dyn-A on arcuate nucleus neuropeptide Y (NPY) neurons mediated by both 1 and (kap
188  In contrast, kisspeptin inhibits orexigenic neuropeptide Y (NPY) neurons through an indirect mechani
189                  The DMH contains orexigenic neuropeptide Y (NPY) neurons, but the role of these neur
190 he purpose of this study was to characterize neuropeptide Y (NPY) overflow and metabolism from isolat
191            Recent studies suggest that human neuropeptide Y (NPY) plays a prominent role in managemen
192                                              Neuropeptide Y (NPY) produces anxiolytic effects in rode
193 nt protein is expressed under control of the neuropeptide Y (NPY) promoter and striatal NPY-expressin
194  a green fluorescent protein (GFP) under the neuropeptide Y (NPY) promoter.
195                         Within the family of neuropeptide Y (NPY) receptors, the Y(4) receptor (Y(4)R
196 he autonomic nervous system and hypothalamic neuropeptide Y (NPY) release during fasting regulates he
197                                              Neuropeptide Y (NPY) signaling in the CNS was modulated
198                                              Neuropeptide Y (NPY) signaling via limbic NPY1 and 2 rec
199          In the current study, we found that neuropeptide Y (NPY) suppressed monocyte recruitment to
200 on requires simultaneous withdrawal of tonic neuropeptide Y (NPY) sympathoinhibition.
201           It has been speculated this alters neuropeptide Y (NPY) synthesis, trafficking, or secretio
202              Functional genetic variation of neuropeptide Y (NPY) was recently identified as a source
203                                We found that Neuropeptide Y (NPY) was significantly down-regulated in
204 We applied this BiFC system to study example neuropeptide Y (NPY) Y1 receptor dimers.
205 d the role of alpha-adrenergic receptor- and neuropeptide Y (NPY) Y1 receptor-mediated vasoconstricti
206                                Activation of neuropeptide Y (NPY) Y1 receptors (Y1r) in the rat basol
207 ations of GABAergic interneurones expressing neuropeptide Y (NPY)(+), parvalbumin (PV)(+) and tyrosin
208  neurons [gamma-aminobutyric acid (GABA)(+), neuropeptide Y (NPY)(+), proopiomelanocortin (POMC)(+),
209                                              Neuropeptide Y (NPY), a 36 aa peptide, regulates stress
210                                              Neuropeptide Y (NPY), a brain neuromodulator that has be
211 study, we identified increased expression of neuropeptide Y (NPY), a pleiotropic growth factor which
212 have studied the subcellular distribution of neuropeptide Y (NPY), a prototypical and broadly express
213                                              Neuropeptide Y (NPY), a stress modulatory transmitter, i
214         Our previous studies have identified neuropeptide Y (NPY), a sympathetic neurotransmitter exp
215 In birds, CART inhibits food intake, whereas neuropeptide Y (NPY), a well-known orexigenic peptide, s
216 ry indicate that cortical astrocytes contain neuropeptide Y (NPY), a widespread neuronal transmitter.
217  from chemotherapy-induced cell death, while neuropeptide Y (NPY), acting via its Y2 receptor (Y2R),
218 c neuron markers proopiomelanocortin (POMC), neuropeptide Y (NPY), agouti-related peptide (AGRP), som
219 ment, hypothalamic mRNA expression levels of neuropeptide Y (NPY), agouti-related protein (AGRP), pro
220                    We studied the actions of neuropeptide Y (NPY), an abundant neocortical neuropepti
221 se changes are regulated postsynaptically by neuropeptide Y (NPY), an adrenal cotransmitter.
222 creased mRNA levels of hypothalamic GHS-R1a, neuropeptide Y (NPY), and agouti-related protein (AgRP),
223  (CB), parvalbumin (PV), calretinin (CR) and neuropeptide Y (NPY), and somatostatin (SOM) and glial f
224        Over 80% of these neuron also express neuropeptide Y (NPY), and this coexpression is maintaine
225  augments the SCN clock-resetting effects of neuropeptide Y (NPY), another neurochemical correlate of
226 ctor 3 (ATF3), the neuropeptides galanin and neuropeptide Y (NPY), brain-derived neurotrophic factor
227 -melanocyte-stimulating hormone (alpha-MSH), neuropeptide Y (NPY), glutamate, and GABA from first-ord
228 ), galanin, gastrin-releasing peptide (GRP), neuropeptide Y (NPY), nitric oxide synthase (NOS), serot
229 ed by either gamma-aminobutyric acid (GABA), neuropeptide Y (NPY), or beta-endorphin receptor blockad
230 vels of genes that regulate appetite, namely neuropeptide Y (NPY), pro-opiomelanocortin (POMC) and th
231 he well-characterized neuropeptides, such as neuropeptide Y (NPY), proopiomelanocortin (POMC), orexin
232 ent with PEDF + DHA induced transcription of neuropeptide y (npy), small proline-rich protein 1a (spr
233 xp4 expression induces ectopic expression of neuropeptide Y (Npy), which has been reported to be pres
234  effects were due to disrupted signalling of neuropeptide Y (NPY), which is known to mediate non-phot
235 ar interneurons of the dentate gyrus express neuropeptide Y (NPY), which modulates granule cell activ
236                       One relevant target is neuropeptide Y (NPY), which regulates both stress and fo
237 l transduction pathway modulating release of neuropeptide Y (NPY), which stimulates OE stem cell acti
238 cell activation and promotes the activity of neuropeptide Y (NPY)- and agouti-related peptide (AgRP)-
239 nterneurons express col19a1 mRNA; subsets of neuropeptide Y (NPY)-, somatostatin (Som)-, and calbindi
240 rotein (Tg2576), Abeta causes dysfunction in neuropeptide Y (NPY)-expressing hypothalamic arcuate neu
241 e investigated the properties of neostriatal neuropeptide Y (NPY)-expressing interneurons in transgen
242 1 (GAD1) in either cholecystokinin (CCK)- or neuropeptide Y (NPY)-expressing interneurons.
243  (Cell 2019;176:687-701) determined that the neuropeptide Y (NPY)-like receptor 7 (NPYLR7) controls m
244 on, appeared to be precipitated by a loss of neuropeptide Y (NPY)-mediated local circuit inhibition a
245 ood attraction to aversion is regulated by a neuropeptide Y (NPY)-related brain signaling peptide.
246                   We investigated a role for neuropeptide Y (NPY)-related signaling in long-term beha
247 ropeptides agouti-related protein (AgRP) and neuropeptide Y (NPY).
248 e-like ethanol drinking to study the role of neuropeptide Y (NPY).
249 r producing the neuropeptides enkephalin and neuropeptide Y (NPY).
250 III are GABAergic, and some of these express neuropeptide Y (NPY).
251 rsors and express neuronal proteins, such as neuropeptide Y (NPY).
252 F co-receptors GFRalpha1 and RET, as well as neuropeptide Y (NPY).
253  release of the anticonvulsant neuropeptide, neuropeptide Y (NPY).
254 expressing agouti-related peptide (AgRP) and neuropeptide Y (Npy).
255 n enzyme that truncates the neurotransmitter neuropeptide Y (NPY).
256 asting discharge rates of fluorescent-tagged neuropeptide-Y (NPY) (within 200 ms) and tissue plasmino
257  that express pro-opiomelanocortin (POMC) or neuropeptide-Y (NPY) and agouti-related protein (AgRP).
258                      Orthologs of vertebrate neuropeptide-Y (NPY) known as neuropeptide-F (NPF) have
259 ridization to localize appetite-stimulating (neuropeptide Y, NPY; agouti-related protein, AGRP) and a
260 cal activity: the N-terminal region of human neuropeptide Y (NPY1-9, Tyr(1)-Pro(2)-Ser(3)-Lys(4)-Pro(
261 tostatin, vasoactive intestinal polypeptide, neuropeptide Y, or cholecystokinin (antigens commonly co
262              Furthermore, loss of calbindin, neuropeptide Y, parvalbumin, and GAD65-positive interneu
263                                          The neuropeptide Y pathway acts together with SRB-13 to anta
264             Moreover, an increased number of neuropeptide Y-positive interneurons in the DG correlate
265 h changes in parvalbumin-, somatostatin- and neuropeptide Y-positive interneurons.
266 e submucosal plexus, DOReGFP was detected in neuropeptide Y-positive secretomotor and vasodilator neu
267                                      Loss of neuropeptide Y prevents a fasting-induced increase in ep
268 pressing green fluorescent protein under the neuropeptide Y promoter, we find that, across all layers
269 tance P, calcitonin gene-related peptide, or neuropeptide Y protein expression in DRGs and spinal cor
270                         Lower mRNA levels of neuropeptide Y receptor 1 (NPY1R) and NPY5R but not NPY
271                       Infusion of a specific neuropeptide Y receptor 5 agonist into the PVN consequen
272 -related behavior, while pretreatment with a neuropeptide Y receptor 5 antagonist prevented the anxio
273 e proteins that is upregulated by OXT is the neuropeptide Y receptor 5.
274 boratory-derived, mutation controlled by the neuropeptide Y receptor homolog npr-1 can affect dauer l
275                                          The neuropeptide Y receptor homolog, NPR-1, and an inhibitor
276  multiple signaling molecules, including the neuropeptide Y receptor NPR-1 and the calcineurin subuni
277                    The neuropeptide receptor neuropeptide Y receptor type 1 (Npy1r) amalgamated multi
278     Here, the conformational dynamics of the neuropeptide Y receptor type 2 (Y2R) during activation w
279 on and this effect was blocked by a specific neuropeptide Y receptor Y1 (NPY1R) antagonist.
280 ion, although this was blocked by a specific neuropeptide Y receptor Y1 receptor antagonist, suggesti
281 ne, which encodes a homolog of the mammalian neuropeptide Y receptor.
282 coupled receptor (GPCR) related to mammalian neuropeptide Y receptors, functions to suppress innate i
283 uantification of neuronal glucocorticoid and neuropeptide Y receptors.
284  Y1 receptor (Y1R) and Y5 receptor (Y5R) for neuropeptide Y share similar actions in the regulation o
285 ive deficits, as well as endocannabinoid and neuropeptide Y system alterations and altered circadian
286                                          The neuropeptide Y system consists of several neuropeptides
287 ic, hypothalamic-pituitary-adrenal axis, and neuropeptide Y systems.
288 er was monitored by observing DCV-associated neuropeptide Y tagged with a fluorescent protein.
289 receptor systems (bombesin, neurotensin, and neuropeptide-Y) that offer high potential in the field o
290 d tumor necrosis factor, whereas addition of neuropeptide Y to wild-type macrophages attenuates the r
291 s-associated increase of immunoreactivity of neuropeptide Y, tyrosine hydroxylase, and somatostatin.
292         In contrast, interneurons containing neuropeptide Y, vasoactive intestinal peptide, or the 5-
293  hypothalamic expression of the orexic gene, neuropeptide Y, was lower and expression of the anorexic
294  (AgRP) neurons (which also release AgRP and neuropeptide Y), we generated mice with an AgRP neuron-s
295 ropeptide F (sNPF), an ortholog of mammalian neuropeptide Y, which we show here is a direct target of
296                                              Neuropeptide Y Y(1) receptors (Y(1)R) have been found to
297                     The results suggest that neuropeptide Y Y1R differentially expressed in the limbi
298 Some in the trafficking of G protein-coupled neuropeptide Y Y2 receptor (NPY(2)R) and serotonin 5-hyd
299                                  The role of neuropeptide Y Y2 receptor (Y2R) in human diseases such
300 ereomeric mixture) is a potent and selective neuropeptide Y Y4 receptor agonist.

 
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